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1. Biomedical articles (top 50; 2009 to 2014)
1. |||||||||. 100%  Swennes AG, Buckley EM, Madden CM, Byrd CP, Donocoff RS, Rodriguez L, Parry NM, Fox JG: Enteropathogenic Escherichia coli prevalence in laboratory rabbits. Vet Microbiol; 2013 May 3;163(3-4):395-8
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Enteropathogenic Escherichia coli prevalence in laboratory rabbits.
  • Rabbit-origin enteropathogenic Escherichia coli (EPEC) causes substantial diarrhea-associated morbidity and has zoonotic potential.
  • EPEC was isolated from 6/141 (4.3%) commercially-acquired laboratory rabbits.
  • Three of these did not have diarrhea or EPEC-typical intestinal lesions; they instead had background plasmacytic intestinal inflammation.
  • Asymptomatically infected rabbits may function as EPEC reservoirs.
  • [MeSH-major] Enteropathogenic Escherichia coli / physiology. Escherichia coli Infections / epidemiology
  • [MeSH-minor] Animals. Anti-Bacterial Agents / pharmacology. Disease Reservoirs. Escherichia coli Proteins / genetics. Female. Intestines / pathology. Prevalence. Rabbits. Serotyping. Virulence Factors / genetics

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  • [Copyright] Copyright © 2013 Elsevier B.V. All rights reserved.
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  • (PMID = 23391439.001).
  • [ISSN] 1873-2542
  • [Journal-full-title] Veterinary microbiology
  • [ISO-abbreviation] Vet. Microbiol.
  • [Language] eng
  • [Grant] United States / NIH HHS / OD / T32 OD010978; United States / NCRR NIH HHS / RR / T32 RR007036; United States / NCRR NIH HHS / RR / T32 RR007036
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / Anti-Bacterial Agents; 0 / Escherichia coli Proteins; 0 / Virulence Factors
  • [Other-IDs] NLM/ NIHMS438439; NLM/ PMC3594591
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2. |||||||||. 98%  Hazen TH, Humphrys MS, Ochieng JB, Parsons M, Bopp CA, O'Reilly CE, Mintz E, Rasko DA: Draft Genome Sequences of Nine Enteropathogenic Escherichia coli Strains from Kenya. Genome Announc; 2014;2(3)
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  • [Title] Draft Genome Sequences of Nine Enteropathogenic Escherichia coli Strains from Kenya.
  • We report here the draft genome sequences of nine enteropathogenic Escherichia coli (EPEC) strains isolated from children in Kenya who died during hospitalization with diarrhea.
  • Each of the isolates possess the EPEC adherence factor (EAF) plasmid encoding the bundle-forming pilus, which is characteristic of EPEC.
  • These isolates represent diverse serogroups and EPEC phylogenomic lineages.

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  • [Copyright] Copyright © 2014 Hazen et al.
  • (PMID = 24926061.001).
  • [ISSN] 2169-8287
  • [Journal-full-title] Genome announcements
  • [ISO-abbreviation] Genome Announc
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Other-IDs] NLM/ PMC4056304
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3. |||||||||. 97%  Thomassin JL, Brannon JR, Kaiser J, Gruenheid S, Le Moual H: Enterohemorrhagic and enteropathogenic Escherichia coli evolved different strategies to resist antimicrobial peptides. Gut Microbes; 2012 Nov-Dec;3(6):556-61
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  • [Title] Enterohemorrhagic and enteropathogenic Escherichia coli evolved different strategies to resist antimicrobial peptides.
  • Enterohemorrhagic and enteropathogenic Escherichia coli (EHEC and EPEC) are enteric human pathogens that colonize the large and small intestines, respectively.
  • To establish infection EHEC and EPEC must overcome innate host defenses, such as antimicrobial peptides (AMPs) produced by the intestinal epithelium.
  • We showed that the protease OmpT degrades the human AMP LL-37 more rapidly in EHEC than in EPEC.
  • Here, we propose that the different ompT expression in EHEC and EPEC reflects the varying levels of LL-37 throughout the human intestinal tract.
  • These data suggest that EHEC and EPEC adapted to their specific niches by developing distinct AMP-specific resistance mechanisms.
  • [MeSH-major] Antimicrobial Cationic Peptides / pharmacology. Drug Resistance, Microbial. Enterohemorrhagic Escherichia coli / drug effects. Enterohemorrhagic Escherichia coli / pathogenicity. Enteropathogenic Escherichia coli / drug effects. Enteropathogenic Escherichia coli / pathogenicity. Immune Evasion
  • [MeSH-minor] Bacterial Outer Membrane Proteins / metabolism. Escherichia coli Infections / immunology. Escherichia coli Infections / microbiology. Escherichia coli Proteins / metabolism. Gene Expression. Humans. Peptide Hydrolases / metabolism. Promoter Regions, Genetic. Proteolysis

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  • (PMID = 22895086.001).
  • [ISSN] 1949-0984
  • [Journal-full-title] Gut microbes
  • [ISO-abbreviation] Gut Microbes
  • [Language] eng
  • [Grant] Canada / Canadian Institutes of Health Research / / MOP-15551
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't; Review
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Antimicrobial Cationic Peptides; 0 / Bacterial Outer Membrane Proteins; 0 / Escherichia coli Proteins; 0 / ompT protein, E coli; 143108-26-3 / CAP18 lipopolysaccharide-binding protein; EC 3.4.- / Peptide Hydrolases
  • [Other-IDs] NLM/ PMC3495793
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4. |||||||||. 97%  Ochoa TJ, Contreras CA: Enteropathogenic escherichia coli infection in children. Curr Opin Infect Dis; 2011 Oct;24(5):478-83
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Enteropathogenic escherichia coli infection in children.
  • PURPOSE OF REVIEW: Enteropathogenic Escherichia coli (EPEC) is an important diarrheal pathogen of young children.
  • As the diagnosis of EPEC is now based mainly on molecular criteria, there has been an important change in its prevalence.
  • The purpose of this study is to review the current epidemiology of EPEC infection and the new insights into its physiopathology.
  • RECENT FINDINGS: Recent epidemiological studies indicate that atypical EPEC (aEPEC) is more prevalent than typical EPEC (tEPEC) in both developed and developing countries, and that aEPEC is important in both pediatric endemic diarrhea and diarrhea outbreaks.
  • However, the exact mechanisms of diarrhea in EPEC infection are not completely understood.
  • SUMMARY: Remarkable progress has been made to identify virulence determinants required to mediate the pathogenesis of EPEC.
  • [MeSH-major] Enteropathogenic Escherichia coli. Escherichia coli Infections / epidemiology

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  • (PMID = 21857511.001).
  • [ISSN] 1473-6527
  • [Journal-full-title] Current opinion in infectious diseases
  • [ISO-abbreviation] Curr. Opin. Infect. Dis.
  • [Language] eng
  • [Grant] United States / FIC NIH HHS / TW / 1K01TW007405; United States / FIC NIH HHS / TW / K01 TW007405; United States / FIC NIH HHS / TW / K01 TW007405-04
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Review
  • [Publication-country] United States
  • [Other-IDs] NLM/ NIHMS348474; NLM/ PMC3277943
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5. |||||||||. 96%  Scaletsky IC, Aranda KR, Souza TB, Silva NP, Morais MB: Evidence of pathogenic subgroups among atypical enteropathogenic Escherichia coli strains. J Clin Microbiol; 2009 Nov;47(11):3756-9
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  • [Title] Evidence of pathogenic subgroups among atypical enteropathogenic Escherichia coli strains.
  • We describe the characterization of 126 atypical enteropathogenic Escherichia coli (aEPEC) isolates from 1,749 Brazilian children.
  • [MeSH-major] Diarrhea / microbiology. Enteropathogenic Escherichia coli / classification. Enteropathogenic Escherichia coli / pathogenicity. Escherichia coli Infections / microbiology
  • [MeSH-minor] Brazil. DNA Primers / genetics. DNA, Bacterial / genetics. Escherichia coli Proteins / genetics. Genotype. Hemolysin Proteins / genetics. Humans. Infant. Serotyping. Virulence Factors / genetics

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  • (PMID = 19759223.001).
  • [ISSN] 1098-660X
  • [Journal-full-title] Journal of clinical microbiology
  • [ISO-abbreviation] J. Clin. Microbiol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, U.S. Gov't, Non-P.H.S.
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / DNA Primers; 0 / DNA, Bacterial; 0 / Escherichia coli Proteins; 0 / Hemolysin Proteins; 0 / Hlya protein, E coli; 0 / Paa protein, E coli; 0 / Virulence Factors
  • [Other-IDs] NLM/ PMC2772600
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6. |||||||||. 94%  Hazen TH, Sahl JW, Fraser CM, Donnenberg MS, Scheutz F, Rasko DA: Draft Genome Sequences of Three O157 Enteropathogenic Escherichia coli Isolates. Genome Announc; 2013;1(4)
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  • [Title] Draft Genome Sequences of Three O157 Enteropathogenic Escherichia coli Isolates.
  • We report the draft genome sequences of three enteropathogenic Escherichia coli (EPEC) isolates that display the O157 serogroup but do not have the Shiga toxin genes (stx), which are characteristic of O157 enterohemorrhagic E. coli (EHEC). E. coli strain RN587/1 has the O157:H8 serotype and possesses the EAF plasmid characteristic of typical EPEC (J. B.

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  • (PMID = 23868135.001).
  • [ISSN] 2169-8287
  • [Journal-full-title] Genome announcements
  • [ISO-abbreviation] Genome Announc
  • [Language] eng
  • [Grant] United States / NIAID NIH HHS / AI / U19 AI090873
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Other-IDs] NLM/ PMC3715677
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7. |......... 6%  Cooper IF, Siadaty MS: 'Signs or Symptoms' associated with 'E Coli Enteropathogenic': Top Publications. BioMedLib Review; SignOrSymptom;EColiEnteropathogenic:705206342. ISSN: 2331-5717. 2014/5/30
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  • [Title] 'Signs or Symptoms' associated with 'E Coli Enteropathogenic': Top Publications.
  • [Transliterated title]
  • Background: There are articles published each month which present 'Sign or Symptom' for 'e coli enteropathogenic'.
  • Finding such articles is important for researchers, clinicians, and patients.
  • However these articles are spread across thousands of journals, and there are many types of 'Sign or Symptom'.
  • This makes searching and locating the relevant publications a challenge.
  • We have used BioMedLib's semantic search technology to address the issue, and gathered all the pertinent publications in this review article.
  • Methods: We categorized the publications we found into two groups.
  • We used the strength of textual-association to separate the groups.
  • In group one there are publications with the strongest evidence of association. We focused finding the most relevant publications pertinent to our goal, rather than combining them into a conclusion section. Such textual synthesis will be the focus of our next project.
  • Results: Group one includes 21 publications, and group two 1500 publications.
  • Here are the top 10.
  • Vidotto MC et al: Unidentified serogroups of enteropathogenic Escherichia coli (EPEC) associated with diarrhoea in infants in Londrina, Parana, Brazil.
  • Dutta TK et al: Detection & characterization of Shiga toxin producing Escherichia coli (STEC) & enteropathogenic Escherichia coli (EPEC) in poultry birds with diarrhoea.
  • Ansaruzzaman M et al: Clonal groups of enteropathogenic Escherichia coli isolated in case-control studies of diarrhoea in Bangladesh.
  • Al Hilali SA et al: Occurrence and molecular characterization of enteropathogenic Escherichia coli serotypes isolated from children with diarrhoea in Najaf, Iraq.
  • Afset JE et al: High prevalence of atypical enteropathogenic Escherichia coli (EPEC) in Norwegian children with diarrhoea.
  • Alikhani MY et al: Detection of typical and atypical enteropathogenic Escherichia coli (EPEC) in Iranian children with and without diarrhoea.
  • Mani R et al: Study of enteropathogenic Escherichia coli(EPEC) diarrhoea in children.
  • Vaishnavi C et al: Enteropathogenic Escherichia coli isolates in paediatric diarrhoea.
  • Wani SA et al: Isolation and characterization of Shiga toxin-producing Escherichia coli (STEC) and enteropathogenic Escherichia coli (EPEC) from calves and lambs with diarrhoea in India.
  • Nakhjavani FA et al: Molecular analysis of typical and atypical enteropathogenic Escherichia coli (EPEC) isolated from children with diarrhoea.

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
  • (UID = 705206342.001).
  • [ISSN] 2331-5717
  • [Journal-full-title] BioMedLib Review
  • [Language] eng
  • [Publication-type] Review
  • [Publication-country] UNITED STATES
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8. |||||||||. 121%  Donnenberg MS, Finlay BB: Combating enteropathogenic Escherichia coli (EPEC) infections: the way forward. Trends Microbiol; 2013 Jul;21(7):317-9
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Combating enteropathogenic Escherichia coli (EPEC) infections: the way forward.
  • Enteropathogenic Escherichia coli (EPEC) strains continue to cause severe and sometimes fatal infantile diarrhea, particularly in Africa.
  • Increased efforts at diagnosis, defining the clinical spectrum of disease, understanding pathogenic mechanisms, and delineating immune responses are desperately needed to develop new strategies to combat EPEC.
  • [MeSH-major] Diarrhea / epidemiology. Diarrhea / microbiology. Enteropathogenic Escherichia coli / immunology. Enteropathogenic Escherichia coli / pathogenicity. Escherichia coli Infections / epidemiology. Escherichia coli Infections / microbiology
  • [MeSH-minor] Africa / epidemiology. Escherichia coli Proteins / immunology. Immunization / methods. Virulence Factors / immunology

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  • MedlinePlus Health Information. consumer health - E. Coli Infections.
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  • [Copyright] Copyright © 2013 Elsevier Ltd. All rights reserved.
  • (PMID = 23815982.001).
  • [ISSN] 1878-4380
  • [Journal-full-title] Trends in microbiology
  • [ISO-abbreviation] Trends Microbiol.
  • [Language] eng
  • [Grant] United States / NIAID NIH HHS / AI / U19 AI 090873; United States / NIAID NIH HHS / AI / U19 AI090873; Canada / Canadian Institutes of Health Research / /
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Escherichia coli Proteins; 0 / Virulence Factors
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9. |||||||||. 112%  Hernandes RT, Elias WP, Vieira MA, Gomes TA: An overview of atypical enteropathogenic Escherichia coli. FEMS Microbiol Lett; 2009 Aug;297(2):137-49
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  • [Title] An overview of atypical enteropathogenic Escherichia coli.
  • The enteropathogenic Escherichia coli (EPEC) pathotype is currently divided into two groups, typical EPEC (tEPEC) and atypical EPEC (aEPEC).
  • The property that distinguishes these two groups is the presence of the EPEC adherence factor plasmid, which is only found in tEPEC. aEPEC strains are emerging enteropathogens that have been detected worldwide.
  • Herein, we review the serotypes, virulence properties, genetic relationships, epidemiology, reservoir and diagnosis of aEPEC, including those strains not belonging to the classical EPEC serogroups (nonclassical EPEC serogroups).
  • The large variety of serotypes and genetic virulence properties of aEPEC strains from nonclassical EPEC serogroups makes it difficult to determine which strains are truly pathogenic.
  • [MeSH-major] Enteropathogenic Escherichia coli. Escherichia coli Infections / microbiology
  • [MeSH-minor] Animals. Escherichia coli Proteins / genetics. Escherichia coli Proteins / metabolism. Humans. Virulence. World Health

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  • (PMID = 19527295.001).
  • [ISSN] 1574-6968
  • [Journal-full-title] FEMS microbiology letters
  • [ISO-abbreviation] FEMS Microbiol. Lett.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't; Review
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Escherichia coli Proteins
  • [Number-of-references] 138
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10. |||||||||. 94%  Staples M, Doyle CJ, Graham RM, Jennison AV: Molecular epidemiological typing of enteropathogenic Escherichia coli strains from Australian patients. Diagn Microbiol Infect Dis; 2013 Mar;75(3):320-4
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  • [Title] Molecular epidemiological typing of enteropathogenic Escherichia coli strains from Australian patients.
  • Enteropathogenic Escherichia coli (EPEC) are an important cause of diarrhoea worldwide, particularly in children.
  • Sixty-one EPEC strains isolated from stool specimens of symptomatic persons from 2008 to 2011 were characterised for the prevalence of diarrhoea-associated putative virulence genes.
  • The EPEC isolates were highly heterogeneous, representing all 4 phylogenetic groups and comprising 59 MLVA types, 49 MLST types, and 43 serotypes.
  • This diversity is indicative of the complexity of the human enteric EPEC population, which may be either commensal or pathogenic.
  • [MeSH-major] Bacterial Typing Techniques / methods. Enteropathogenic Escherichia coli / genetics. Multilocus Sequence Typing / methods
  • [MeSH-minor] Australia / epidemiology. Diarrhea / microbiology. Escherichia coli Proteins / genetics. Feces / microbiology. Genes, Bacterial. Genetic Variation. Humans. Molecular Epidemiology. Phylogeny. Prevalence. Virulence Factors / genetics

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  • [Copyright] Crown Copyright © 2013. Published by Elsevier Inc. All rights reserved.
  • (PMID = 23357294.001).
  • [ISSN] 1879-0070
  • [Journal-full-title] Diagnostic microbiology and infectious disease
  • [ISO-abbreviation] Diagn. Microbiol. Infect. Dis.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Escherichia coli Proteins; 0 / NleB protein, E coli; 0 / Virulence Factors
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11. |||||||||. 92%  de Almeida PM, Arais LR, Andrade JR, Prado EH, Irino K, Cerqueira Ade M: Characterization of atypical Enteropathogenic Escherichia coli (aEPEC) isolated from dogs. Vet Microbiol; 2012 Aug 17;158(3-4):420-4
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  • [Title] Characterization of atypical Enteropathogenic Escherichia coli (aEPEC) isolated from dogs.
  • Enteropathogenic Escherichia coli (EPEC), an important human pathogen has the ability to form attaching and effacing lesions on the intestinal epithelium and has been isolated from a wide range of species.
  • Two EPEC subgroups are recognized: typical (tEPEC) and atypical (aEPEC) strains, differing by the presence of EAF plasmid and bundle-forming pilus (BFP) in typical strains and their absence in atypical strains.
  • This study searched the presence of EPEC strains in 101 fecal samples of diarrheic (n=65) and non-diarrheic (n=36) dogs from two cities in Rio de Janeiro State, Brazil.
  • Most isolates was FAS-positive and showed a localized adherence-like (LAL) in a 6h HeLa cell-adherence assay.
  • [MeSH-major] Dog Diseases / microbiology. Enteropathogenic Escherichia coli / genetics. Escherichia coli Infections / veterinary
  • [MeSH-minor] Adhesins, Bacterial / genetics. Animals. Bacterial Adhesion. Base Sequence. Brazil. Dogs. Escherichia coli Proteins / genetics. Feces / microbiology. Genotype. HeLa Cells. Humans. Serotyping

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  • [Copyright] Copyright © 2012. Published by Elsevier B.V.
  • (PMID = 22421111.001).
  • [ISSN] 1873-2542
  • [Journal-full-title] Veterinary microbiology
  • [ISO-abbreviation] Vet. Microbiol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / Adhesins, Bacterial; 0 / Escherichia coli Proteins
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12. |||||||||. 93%  Spira B, Ferreira GM, de Almeida LG: relA enhances the adherence of enteropathogenic Escherichia coli. PLoS One; 2014;9(3):e91703
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  • [Title] relA enhances the adherence of enteropathogenic Escherichia coli.
  • Enteropathogenic Escherichia coli (EPEC) is a known causative agent of diarrhea in children.
  • In the process of colonization of the small intestine, EPEC synthesizes two types of adhesins, the bundle-forming pilus (BFP) and intimin.
  • The BFP pilus is an adhesin associated with the initial stages of adherence of EPEC to epithelial cells, while the outer membrane protein intimin carries out the intimate adherence that takes place at the third stage of infection.
  • BFP is encoded by the bfp operon located in plasmid EAF, present only in typical EPEC isolates, while eae, the gene that encodes intimin is situated in the LEE, a chromosomal pathogenicity island.
  • Here we show that deletion of relA, that encodes a guanosine penta and tetraphosphate synthetase impairs EPEC adherence to epithelial cells in vitro.

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  • (PMID = 24643076.001).
  • [ISSN] 1932-6203
  • [Journal-full-title] PloS one
  • [ISO-abbreviation] PLoS ONE
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
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13. |||||||||. 93%  Ruiz RC, Melo KC, Rossato SS, Barbosa CM, Corrêa LM, Elias WP, Piazza RM: Atypical enteropathogenic Escherichia coli secretes plasmid encoded toxin. Biomed Res Int; 2014;2014:896235
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  • [Title] Atypical enteropathogenic Escherichia coli secretes plasmid encoded toxin.
  • Plasmid encoded toxin (Pet) is a serine protease originally described in enteroaggregative Escherichia coli (EAEC) prototype strain 042 whose entire characterization was essentially obtained from studies performed with the purified toxin.
  • Atypical enteropathogenic Escherichia coli (aEPEC) strains, isolated from diarrhea cases, express Pet and its detection in supernatants of infected HEp-2 cells coincides with the appearance of cell damage, which, in turn, were similar to those described with purified Pet.

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  • (PMID = 24949475.001).
  • [ISSN] 2314-6141
  • [Journal-full-title] BioMed research international
  • [ISO-abbreviation] Biomed Res Int
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Other-IDs] NLM/ PMC4037613
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14. |||||||||. 91%  Ruetz TJ, Vogl AW, Guttman JA: Detailed examination of cytoskeletal networks within enteropathogenic Escherichia coli pedestals. Anat Rec (Hoboken); 2012 Feb;295(2):201-7
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  • [Title] Detailed examination of cytoskeletal networks within enteropathogenic Escherichia coli pedestals.
  • Enteropathogenic Escherichia coli (EPEC) manipulate the cytoskeleton of host intestinal epithelial cells, producing membrane protrusions termed pedestals that the bacteria reside on throughout the course of their infections.
  • By definition pedestals are actin-based structures, however recent work has identified the spectrin cytoskeleton as a necessary component of EPEC pedestals.
  • Myosin S1 fragment decorated actin filaments examined by electron microscopy demonstrated that actin filaments orientate with their fast-growing barbed ends toward the lateral membranes of EPEC pedestals.
  • [MeSH-major] Cell Surface Extensions / ultrastructure. Cytoskeleton / ultrastructure. Enteropathogenic Escherichia coli / ultrastructure. HeLa Cells / ultrastructure

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  • [Copyright] Copyright © 2011 Wiley Periodicals, Inc.
  • (PMID = 22190417.001).
  • [ISSN] 1932-8494
  • [Journal-full-title] Anatomical record (Hoboken, N.J. : 2007)
  • [ISO-abbreviation] Anat Rec (Hoboken)
  • [Language] eng
  • [Grant] Canada / Canadian Institutes of Health Research / /
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Cytoskeletal Proteins; 0 / Myosin Subfragments; 12634-43-4 / Spectrin
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15. |||||||||. 91%  Behiry IK, Abada EA, Ahmed EA, Labeeb RS: Enteropathogenic Escherichia coli associated with diarrhea in children in Cairo, Egypt. ScientificWorldJournal; 2011;11:2613-9
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  • [Title] Enteropathogenic Escherichia coli associated with diarrhea in children in Cairo, Egypt.
  • In this study we isolate and identify the Enteropathogenic Escherichia coli (EPEC) causing diarrhea in children less than five years in Cairo, Egypt, during different seasons.
  • The identified E. coli isolates were subjected to antimicrobial disc diffusion susceptibility test and further identified for EPEC serotype by slide agglutination test, using antiserum E. coli somatic trivalent I (O111, O55, O26) according to the instructions of the manufacturer.
  • Out of 134 patients 5.2% of them revealed EPEC in the fecal sample, while the 20 children control group showed no EPEC isolates in their samples.
  • Our EPEC frequency showed variations from the compared results of other studies.
  • Higher rate of EPEC (18.7%) was found in patients between 2 to 3 years, while EPEC rate was (7.5%) in patients less than 6 months old, with P < 0.05.
  • EPEC was identified from fecal specimens as a unique pathogen or associated with other pathogens in acute and chronic diarrhea in children.
  • EPEC were detected in all seasons except in winter, and was predominant in summer season.
  • Four (57%) EPEC isolates were resistant to ampicillin, ticarcillin, and cotrimoxazole, and (14.3%) to the third generation cephalosporins.
  • [MeSH-major] Diarrhea / microbiology. Enteropathogenic Escherichia coli / isolation & purification

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  • (PMID = 22262949.001).
  • [ISSN] 1537-744X
  • [Journal-full-title] TheScientificWorldJournal
  • [ISO-abbreviation] ScientificWorldJournal
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Anti-Bacterial Agents
  • [Other-IDs] NLM/ PMC3254012
  • [Keywords] NOTNLM ; children less than 5 years / enteropathogenic E. coli / slide agglutination test
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16. |||||||||. 91%  Bustamante VH, Villalba MI, García-Angulo VA, Vázquez A, Martínez LC, Jiménez R, Puente JL: PerC and GrlA independently regulate Ler expression in enteropathogenic Escherichia coli. Mol Microbiol; 2011 Oct;82(2):398-415
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] PerC and GrlA independently regulate Ler expression in enteropathogenic Escherichia coli.
  • Typical enteropathogenic Escherichia coli (EPEC) strains contain the EAF plasmid, which carries the perABC locus encoding PerC.
  • The precise role of PerC in EPEC virulence gene regulation has remained unclear, mainly because EPEC strains lacking the pEAF still express the LEE genes and because PerC is not present in other A/E pathogens such as Citrobacter rodentium.
  • Our results substantiate the role of PerC and GrlA in EPEC virulence gene regulation and suggest that these convergent regulatory mechanisms may have represented an evolutionary adaptation in EPEC to co-ordinate the expression of plasmid- and chromosome-encoded virulence factors needed to successfully colonize its intestinal niche.
  • [MeSH-major] Enteropathogenic Escherichia coli / metabolism. Escherichia coli Proteins / metabolism. Gene Expression Regulation, Bacterial. Trans-Activators / metabolism

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  • [Copyright] © 2011 Blackwell Publishing Ltd.
  • (PMID = 21895790.001).
  • [ISSN] 1365-2958
  • [Journal-full-title] Molecular microbiology
  • [ISO-abbreviation] Mol. Microbiol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Escherichia coli Proteins; 0 / GrlA protein, E coli; 0 / Ler protein, E coli; 0 / PerC protein, E coli; 0 / Trans-Activators
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17. |||||||||. 90%  Hernandes RT, Velsko I, Sampaio SC, Elias WP, Robins-Browne RM, Gomes TA, Girón JA: Fimbrial adhesins produced by atypical enteropathogenic Escherichia coli strains. Appl Environ Microbiol; 2011 Dec;77(23):8391-9
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  • [Title] Fimbrial adhesins produced by atypical enteropathogenic Escherichia coli strains.
  • Atypical enteropathogenic Escherichia coli (aEPEC) has emerged as a significant cause of pediatric diarrhea worldwide; however, information regarding its adherence mechanisms to the human gut mucosa is lacking.
  • These genes are associated with adhesion and/or biofilm formation of pathogenic and commensal E. coli.
  • We were not able to detect the hemorrhagic coli pilus (HCP) or the E. coli laminin-binding fimbriae (ELF) in these strains by using immunofluorescence.
  • The E. coli common pilus (ECP) was evidenced in 36.6% of the strains on bacteria adhering to HeLa cells by immunofluorescence, suggesting that ECP could play an important role in cell adherence for some aEPEC strains.
  • [MeSH-major] Adhesins, Bacterial / biosynthesis. Adhesins, Escherichia coli / biosynthesis. Enteropathogenic Escherichia coli / metabolism
  • [MeSH-minor] Australia. Bacterial Adhesion. Brazil. DNA, Bacterial / genetics. Diarrhea / microbiology. Epithelial Cells / microbiology. Escherichia coli Infections / microbiology. Gene Expression Profiling. HeLa Cells. Humans. Polymerase Chain Reaction

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  • (PMID = 21926222.001).
  • [ISSN] 1098-5336
  • [Journal-full-title] Applied and environmental microbiology
  • [ISO-abbreviation] Appl. Environ. Microbiol.
  • [Language] eng
  • [Grant] United States / NIAID NIH HHS / AI / AI66012
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Adhesins, Bacterial; 0 / Adhesins, Escherichia coli; 0 / DNA, Bacterial
  • [Other-IDs] NLM/ PMC3233042
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18. |||||||||. 88%  Asadi Karam M, Bouzari S, Oloomi M, Aslani M, Jafari A: Phenotypic and Genotypic Characterization of Enteropathogenic Escherichia coli (EPEC) strains in Tehran, Iran. Iran J Microbiol; 2010 Mar;2(1):3-7
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  • [Title] Phenotypic and Genotypic Characterization of Enteropathogenic Escherichia coli (EPEC) strains in Tehran, Iran.
  • BACKGROUND AND OBJECTIVES: Enteropathogenic Escherichia coli (EPEC) strains can be detected by serogrouping and the presence of enterocyte attaching- effacing (eae) gene.
  • Most EPEC strains belong to a certain O antigenic group.
  • The aim of the present study was to genetically characterize EPEC strains isolated from children with diarrhea.
  • MATERIALS AND METHODS: Serogrouping was performed by EPEC antisera in 321 E. coli isolates.
  • RESULTS: Seventeen (5.3%) isolates belonging to 7 EPEC serogroups were identified among the whole material and all carried the eae gene.
  • None of the 321 isolates showed presence of stx gene indicating that all 17 isolates classified as EPEC by O serogrouping did not belong to the enterohaemorrhagic E. coli (EHEC) group.
  • CONCLUSION: Serogrouping and detection of the eae gene showed to be reliable for detection of EPEC strains.
  • No Shigatoxin- producing E. coli (STEC) strain was found among the isolates.
  • Detection of the insertion site of LEE showed that selC, pheU or PheV are insertion sites of LEE in the EPEC strains.

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  • (PMID = 22347543.001).
  • [ISSN] 2008-4447
  • [Journal-full-title] Iranian journal of microbiology
  • [ISO-abbreviation] Iran J Microbiol
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Iran
  • [Other-IDs] NLM/ PMC3279762
  • [Keywords] NOTNLM ; Attaching- effacing / Intimin / Iran / Pathogenicity island / Serogrouping
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19. |||||||||. 86%  Tilak GP, Mudaliar JL: Role of enteropathogenic Escherichia coli in paediatric diarrhoeas in South India. Mater Sociomed; 2012;24(3):178-81
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  • [Title] Role of enteropathogenic Escherichia coli in paediatric diarrhoeas in South India.
  • BACKGROUND: Enteropathogenic Escherichia coli(EPEC) is a major cause of diarrhoea in children below 5 years of age.
  • Serotyping is classical method for identification of EPEC strains.
  • But serotypic markers are rarely sufficient to reliably identify the strains as Escherichia coli.
  • Introduction of PCR methodology which depends on detection of virulence factors has provided a practical and rapid way of detecting diarrhoeagenic Esch.coli.
  • Multiantibiotic resistant EPEC strains are a common phenomenon with world wide extension.
  • OBJECTIVES: To study the role of EPEC in Paediatric diarrhoea by both Serogrouping and Molecular characterisation by PCR and to analyse the antibiotic susceptabililty patterns of EPEC strains in our area.
  • Escherichia.coli isolates were identified by Microscopy, Culture and Biochemical reactions.
  • Among the Escherichia coli isolates, EPEC isolates were identified by Serogrouping.
  • Escherichia coli isolates were also subjected to Molecular characterisation by Multiplex PCR assay and those isolates which showed pathogenic genes were futher serotyped.
  • Antibiotic susceptibility pattern of EPEC isolates was determined by CLSI guidelines.
  • RESULTS: Among the Escherichia coli isolates 36.8% in the diarrhoeal group and none of them from the nondiarrhoeal group were identified as EPEC by serogrouping.
  • 73.3% of the EPEC isolates were below 2 years of age and no much difference in the sex distribution was observed.
  • In the diarrhoeal group 38.8% of Escherichia coli were EAEC and no other diarrhoeagenic Escherichia coli group was found by molecular characterisation.
  • In the nondiarrhoeal Escherichia coli strains, 46.6% showed EAEC genes.
  • Among the Escherichia coli isolates which agglutinated with EPEC polyvalent antisera, 33.3% were positive for Enteroaggregative genes. (Table 1).
  • CONCLUSION: EPEC is still an important pathogen in paediatric diarrhoeas.
  • O serogrouping can still be relied upon for detection of EPEC.
  • Dehydration is one of the clinical features of EPEC diarrhoea.

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  • (PMID = 23922527.001).
  • [ISSN] 1512-7680
  • [Journal-full-title] Materia socio-medica
  • [ISO-abbreviation] Mater Sociomed
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Bosnia and Hercegovina
  • [Other-IDs] NLM/ PMC3732348
  • [Keywords] NOTNLM ; Andhra Pradesh / Antibiotic Susceptibility / Enteroaggregative Escherchia coli (EAEC). / Enteropathogenic Escherichia coli (EPEC) / Molecular characterisation / Serotyping
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20. ||||||||.. 79%  Horcajo P, Domínguez-Bernal G, Carrión J, De La Fuente R, Ruiz-Santa-Quiteria JA, Orden JA: Differences in virulence gene expression between atypical enteropathogenic Escherichia coli strains isolated from diarrheic and healthy ruminants. Can J Vet Res; 2013 Apr;77(2):158-60
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  • [Title] Differences in virulence gene expression between atypical enteropathogenic Escherichia coli strains isolated from diarrheic and healthy ruminants.
  • Differences in the pathogenicity of atypical enteropathogenic Escherichia coli (EPEC) strains may be due, at least partially, to different expression patterns of some virulence genes.
  • To investigate this hypothesis, the virulence gene expression patterns of 6 atypical EPEC strains isolated from healthy and diarrheic ruminants were compared using quantitative real-time reverse transcription polymerase chain reaction after growing the bacteria in culture medium alone or after binding it to HeLa epithelial cells.
  • Thus, the expression levels of some virulence genes may help determine which atypical EPEC strains cause diarrhea in ruminants.
  • [MeSH-major] Diarrhea / veterinary. Enteropathogenic Escherichia coli / metabolism. Gene Expression Regulation, Bacterial / physiology. Virulence Factors / metabolism

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  • (PMID = 24082409.001).
  • [ISSN] 0830-9000
  • [Journal-full-title] Canadian journal of veterinary research = Revue canadienne de recherche vétérinaire
  • [ISO-abbreviation] Can. J. Vet. Res.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Canada
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21. ||||||||.. 79%  Munera D, Crepin VF, Marches O, Frankel G: N-terminal type III secretion signal of enteropathogenic Escherichia coli translocator proteins. J Bacteriol; 2010 Jul;192(13):3534-9
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  • [Title] N-terminal type III secretion signal of enteropathogenic Escherichia coli translocator proteins.
  • We report that the N terminus of the type III secretion system translocator proteins EspB, EspD, and EspA mediate protein secretion and translocation from wild-type enteropathogenic Escherichia coli and hypersecretion from sepL and sepD mutants.
  • [MeSH-major] Enteropathogenic Escherichia coli / metabolism. Escherichia coli Proteins / metabolism

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  • (PMID = 20400543.001).
  • [ISSN] 1098-5530
  • [Journal-full-title] Journal of bacteriology
  • [ISO-abbreviation] J. Bacteriol.
  • [Language] eng
  • [Grant] United Kingdom / Wellcome Trust / /
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Bacterial Outer Membrane Proteins; 0 / EaeB protein, E coli; 0 / Escherichia coli Proteins; 0 / EspA protein, E coli; 0 / EspD protein, E coli; 0 / Map protein, E coli; 0 / Receptors, Cell Surface; 0 / SepD protein ,E coli; 0 / SepL protein, E coli; 0 / Tir protein, E coli
  • [Other-IDs] NLM/ PMC2897685
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22. ||||||||.. 79%  Nisa S, Hazen TH, Assatourian L, Nougayrède JP, Rasko DA, Donnenberg MS: In vitro evolution of an archetypal enteropathogenic Escherichia coli strain. J Bacteriol; 2013 Oct;195(19):4476-83
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  • [Title] In vitro evolution of an archetypal enteropathogenic Escherichia coli strain.
  • Enteropathogenic Escherichia coli (EPEC) is a leading cause of infantile diarrhea in developing countries.
  • EPEC strain E2348/69 is used worldwide as a prototype to study EPEC genetics and disease.
  • To identify the genomic and phenotypic changes in the prototype strain, we sequenced the genome of the nalidixic acid-resistant (Nal(r)) E2348/69 clone.
  • We also sequenced a recent nleF mutant derived by one-step PCR mutagenesis from the Nal(r) strain.
  • The sequencing results revealed no unintended changes between the mutant and the parent strain.
  • This report reconciles longstanding inconsistencies in phenotypic properties of an archetypal strain and provides both reassurance and cautions regarding intentional and unintentional evolution in vitro.
  • [MeSH-major] Enteropathogenic Escherichia coli / genetics. Enteropathogenic Escherichia coli / metabolism

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  • (PMID = 23913321.001).
  • [ISSN] 1098-5530
  • [Journal-full-title] Journal of bacteriology
  • [ISO-abbreviation] J. Bacteriol.
  • [Language] eng
  • [Databank-accession-numbers] GENBANK/ ASZR00000000/ ASZS00000000
  • [Grant] United States / NIAID NIH HHS / AI / U19 AI090873; United States / NIAID NIH HHS / AI / U19AI090873
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] United States
  • [Other-IDs] NLM/ PMC3807458
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23. ||||||||.. 79%  Vieira MA, Salvador FA, Silva RM, Irino K, Vaz TM, Rockstroh AC, Guth BE, Gomes TA: Prevalence and characteristics of the O122 pathogenicity island in typical and atypical enteropathogenic Escherichia coli strains. J Clin Microbiol; 2010 Apr;48(4):1452-5
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  • [Title] Prevalence and characteristics of the O122 pathogenicity island in typical and atypical enteropathogenic Escherichia coli strains.
  • The presence of the pathogenicity island (PAI) O122 genes, efa1 (lifA), sen, pagC, nleB, and nleE, in typical and atypical enteropathogenic Escherichia coli (EPEC) strains was investigated.
  • The simultaneous occurrence of all genes was statistically associated with diarrhea due to atypical EPEC.
  • Detection of the complete PAI O122 could aid in the identification of potential pathogenic strains of atypical EPEC.
  • [MeSH-major] DNA, Bacterial / genetics. Enteropathogenic Escherichia coli / genetics. Escherichia coli Infections / microbiology. Genomic Islands
  • [MeSH-minor] Diarrhea / microbiology. Escherichia coli Proteins / genetics. Humans. Prevalence. Virulence Factors / genetics

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  • (PMID = 20181917.001).
  • [ISSN] 1098-660X
  • [Journal-full-title] Journal of clinical microbiology
  • [ISO-abbreviation] J. Clin. Microbiol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / DNA, Bacterial; 0 / Escherichia coli Proteins; 0 / Virulence Factors
  • [Other-IDs] NLM/ PMC2849565
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24. ||||||||.. 79%  Monaghan Á, Byrne B, Fanning S, Sweeney T, McDowell D, Bolton DJ: Serotypes and virulence profiles of atypical enteropathogenic Escherichia coli (EPEC) isolated from bovine farms and abattoirs. J Appl Microbiol; 2013 Feb;114(2):595-603
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  • [Title] Serotypes and virulence profiles of atypical enteropathogenic Escherichia coli (EPEC) isolated from bovine farms and abattoirs.
  • AIMS: The objective of this study was to examine the prevalence of enteropathogenic Escherichia coli (EPEC) on beef and dairy farms and in beef abattoirs and to characterize the isolates in terms of serogroup and virulence markers.
  • All 140 isolates obtained were atypical EPEC (aEPEC), while θ and β intimin types were common.
  • [MeSH-major] Cattle / microbiology. Enteropathogenic Escherichia coli / classification. Enteropathogenic Escherichia coli / pathogenicity

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  • [Copyright] © 2012 The Society for Applied Microbiology.
  • (PMID = 23163884.001).
  • [ISSN] 1365-2672
  • [Journal-full-title] Journal of applied microbiology
  • [ISO-abbreviation] J. Appl. Microbiol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Virulence Factors
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25. ||||||||.. 78%  Bakhshi B, Fallahzad S, Pourshafie MR: The occurrence of atypical enteropathogenic Escherichia coli strains among children with diarrhea in Iran. J Infect Chemother; 2013 Aug;19(4):615-20
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  • [Title] The occurrence of atypical enteropathogenic Escherichia coli strains among children with diarrhea in Iran.
  • The aim of this study was to investigate the genetic diversity and class 2 integron content of typical and atypical enteropathogenic Escherichia coli (EPEC) strains isolated from children less than 5 years of age.
  • The identity of EPEC and their class 2 integron content was confirmed by PCR analysis and sequencing.
  • Subtyping of Escherichia coli spp. was performed through pulsed-field gel electrophoresis (PFGE) analysis.
  • All EPEC strains were resistant to 6 antimicrobial agents except for gentamycin.
  • The most prevalent serogroups among EPEC strains were found to be members of O86 and O127 serogroups (37.7 %) and O44, O125, and O128 (42.8 %).
  • The majority of our EPEC isolates (60.7 %) were identified as atypical.
  • No PFGE clustering was observed according to bundle-forming pilus (bfp) bacteria, suggesting that PFGE analysis could not discriminate between typical and atypical EPEC strains.
  • The high ratio of antibiotic-resistant strains and the large heterogeneity among EPEC isolates with low prevalence of class 2 integrons signify the need to examine for other mechanism(s) involved in conferring resistance in typical and atypical populations of EPEC.
  • [MeSH-major] Diarrhea / microbiology. Enteropathogenic Escherichia coli / genetics. Escherichia coli Infections / microbiology


26. ||||||||.. 78%  Quintero-Villegas MI, Aam BB, Rupnow J, Sørlie M, Eijsink VG, Hutkins RW: Adherence inhibition of enteropathogenic Escherichia coli by chitooligosaccharides with specific degrees of acetylation and polymerization. J Agric Food Chem; 2013 Mar 20;61(11):2748-54
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  • [Title] Adherence inhibition of enteropathogenic Escherichia coli by chitooligosaccharides with specific degrees of acetylation and polymerization.
  • Adherence of enteropathogenic Escherichia coli (EPEC) to the surface of a human HEp-2 cell line was determined in the absence or presence of the various CHOS fractions.
  • The results showed that all CHOS fractions inhibited adherence of EPEC to HEp-2 cells.
  • [MeSH-major] Bacterial Adhesion / drug effects. Enteropathogenic Escherichia coli / drug effects. Oligosaccharides / chemistry. Oligosaccharides / pharmacology

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  • (PMID = 23428168.001).
  • [ISSN] 1520-5118
  • [Journal-full-title] Journal of agricultural and food chemistry
  • [ISO-abbreviation] J. Agric. Food Chem.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Oligosaccharides
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27. ||||||||.. 78%  Orton DJ, Arsenault DJ, Thomas NA, Doucette AA: GELFrEE fractionation combined with mass spectrometry for proteome analysis of secreted toxins from Enteropathogenic Escherichia coli (EPEC). Mol Cell Probes; 2013 Oct-Dec;27(5-6):200-7
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  • [Title] GELFrEE fractionation combined with mass spectrometry for proteome analysis of secreted toxins from Enteropathogenic Escherichia coli (EPEC).
  • Enteropathogenic Escherichia coli, or EPEC, is a human pathogen associated with gastroenteritis and diarrheal disease whose pathogenicity is related to the secretion of effector proteins (exotoxins).
  • Here, we apply a solution-phase electrophoretic platform (GELFrEE) followed by MS to characterize the secreted proteome of a wild type and mutant strain of EPEC (ΔsepD), exhibiting enhanced secretion of effector proteins.
  • Semi-quantitative analysis of the MS data reveals the effector proteins EspB, EspC, and EspD appear in a relatively greater abundance from wild type EPEC, while two major virulence factors in EPEC, Tir and NleA appear in greater abundance from the secreted proteome of the mutant strain.
  • This study demonstrates the application of GELFrEE-MS to differentiate wild type and mutant strains of EPEC.
  • [MeSH-major] Bacterial Toxins / analysis. Electrophoresis, Polyacrylamide Gel. Enteropathogenic Escherichia coli / metabolism. Enteropathogenic Escherichia coli / pathogenicity. Escherichia coli Proteins / analysis. Mass Spectrometry. Proteome / analysis

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  • [Copyright] Copyright © 2013 Elsevier Ltd. All rights reserved.
  • [RepublishedIn] Mol Cell Probes. 2014 Apr-Jun;28(2-3):83-90 [24486296.001]
  • (PMID = 23831145.001).
  • [ISSN] 1096-1194
  • [Journal-full-title] Molecular and cellular probes
  • [ISO-abbreviation] Mol. Cell. Probes
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Bacterial Outer Membrane Proteins; 0 / Bacterial Toxins; 0 / EaeB protein, E coli; 0 / Escherichia coli Proteins; 0 / EspC protein, E coli; 0 / EspD protein, E coli; 0 / NleA protein, E coli; 0 / Proteome; 0 / Receptors, Cell Surface; 0 / Tir protein, E coli; 0 / Virulence Factors
  • [Keywords] NOTNLM ; Enteropathogenic Escherichia coli / GELFrEE / Mass spectrometry / Proteomics / Secreted proteins / Toxins
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28. ||||||||.. 78%  Berger CN, Crepin VF, Jepson MA, Arbeloa A, Frankel G: The mechanisms used by enteropathogenic Escherichia coli to control filopodia dynamics. Cell Microbiol; 2009 Feb;11(2):309-22
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  • [Title] The mechanisms used by enteropathogenic Escherichia coli to control filopodia dynamics.
  • Enteropathogenic Escherichia coli (EPEC) subverts actin dynamics in eukaryotic cells by injecting effector proteins via a type III secretion system.
  • Then, following recovery from the filopodial signals, EPEC triggers robust actin polymerization via a signalling complex comprising Tir and the adaptor proteins Nck.
  • Recovery from the filopodial signals requires phosphorylation of a Tir tyrosine (Y474) residue and actin polymerization pathway as both infection of cells with EPEC expressing TirY474S or infection of Nck knockout cells with wild-type EPEC resulted in persistence of filopodia.
  • These results show that EPEC effectors modulate actin dynamics by temporal subverting the Rho GTPases and other actin polymerization pathways for the benefit of the adherent pathogen.
  • [MeSH-major] Enteropathogenic Escherichia coli / pathogenicity. Escherichia coli Proteins / metabolism. Pseudopodia / physiology. Virulence Factors / metabolism

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  • (PMID = 19046338.001).
  • [ISSN] 1462-5822
  • [Journal-full-title] Cellular microbiology
  • [ISO-abbreviation] Cell. Microbiol.
  • [Language] eng
  • [Grant] United Kingdom / Medical Research Council / / G0401551; United Kingdom / Medical Research Council / / ; United Kingdom / Wellcome Trust / /
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Actins; 0 / Adaptor Proteins, Signal Transducing; 0 / Cytoskeletal Proteins; 0 / Escherichia coli Proteins; 0 / Map protein, E coli; 0 / Nck protein; 0 / Oncogene Proteins; 0 / Phosphoproteins; 0 / Platelet Glycoprotein GPIb-IX Complex; 0 / Receptors, Cell Surface; 0 / Sodium-Hydrogen Antiporter; 0 / Tir protein, E coli; 0 / Virulence Factors; 0 / ezrin; 0 / sodium-hydrogen exchanger regulatory factor; EC 2.7.11.1 / rho-Associated Kinases; EC 3.6.5.2 / rhoA GTP-Binding Protein
  • [Other-IDs] NLM/ PMC2688667
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29. ||||||||.. 78%  Zahavi EE, Lieberman JA, Donnenberg MS, Nitzan M, Baruch K, Rosenshine I, Turner JR, Melamed-Book N, Feinstein N, Zlotkin-Rivkin E, Aroeti B: Bundle-forming pilus retraction enhances enteropathogenic Escherichia coli infectivity. Mol Biol Cell; 2011 Jul 15;22(14):2436-47
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  • [Title] Bundle-forming pilus retraction enhances enteropathogenic Escherichia coli infectivity.
  • Enteropathogenic Escherichia coli (EPEC) is an important human pathogen that causes acute infantile diarrhea.
  • The type IV bundle-forming pili (BFP) of typical EPEC strains are dynamic fibrillar organelles that can extend out and retract into the bacterium.
  • How pili retraction contributes to EPEC pathogenesis at the cellular level remains largely obscure, however.
  • In this study, an effort has been made to address this question using engineered EPEC strains with induced BFP retraction capacity.
  • Our studies hence suggest novel insights into the involvement of pili retraction in EPEC pathogenesis.
  • [MeSH-major] Enteropathogenic Escherichia coli / pathogenicity. Escherichia coli Infections / microbiology. Fimbriae, Bacterial / physiology
  • [MeSH-minor] Animals. Arabinose / metabolism. Caco-2 Cells. Cell Line. Diarrhea, Infantile / microbiology. Dogs. Escherichia coli Proteins / genetics. Escherichia coli Proteins / metabolism. HeLa Cells. Humans. Infant, Newborn. Phosphorylation. Tyrosine / metabolism

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  • (PMID = 21613538.001).
  • [ISSN] 1939-4586
  • [Journal-full-title] Molecular biology of the cell
  • [ISO-abbreviation] Mol. Biol. Cell
  • [Language] eng
  • [Grant] United States / NIAID NIH HHS / AI / R01 AI037606; United States / NIGMS NIH HHS / GM / T32 GM008181
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / BfpF protein, E coli; 0 / Escherichia coli Proteins; 42HK56048U / Tyrosine; B40ROO395Z / Arabinose
  • [Other-IDs] NLM/ PMC3135470
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30. ||||||||.. 78%  Yan D, Quan H, Wang L, Liu F, Liu H, Chen J, Cao X, Ge B: Enteropathogenic Escherichia coli Tir recruits cellular SHP-2 through ITIM motifs to suppress host immune response. Cell Signal; 2013 Sep;25(9):1887-94
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  • [Title] Enteropathogenic Escherichia coli Tir recruits cellular SHP-2 through ITIM motifs to suppress host immune response.
  • The important diarrheal pathogen enteropathogenic Escherichia coli (EPEC) require delivery and insertion of the bacterial translocated intimin receptor (Tir) into the host plasma membrane for pedestal formation.
  • Here, we show that EPEC Tir suppresses the production of inflammatory cytokines by recruitment of SHP-2 and subsequent deubiquitination of TRAF6 in an ITIM dependent manner.
  • Our findings revealed a novel mechanism by which the EPEC utilize its ITIM motifs to suppress and evade the host innate immune response, which could lead to the development of novel therapeutics to prevent bacterial infection.
  • [MeSH-major] Enteropathogenic Escherichia coli / physiology. Escherichia coli Infections / immunology. Escherichia coli Proteins / immunology. Host-Parasite Interactions. Immunoreceptor Tyrosine-Based Activation Motif. Protein Tyrosine Phosphatase, Non-Receptor Type 11 / immunology. Receptors, Cell Surface / immunology

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  • [Copyright] Copyright © 2013 The Authors. Published by Elsevier Inc. All rights reserved.
  • (PMID = 23707390.001).
  • [ISSN] 1873-3913
  • [Journal-full-title] Cellular signalling
  • [ISO-abbreviation] Cell. Signal.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Cytokines; 0 / Escherichia coli Proteins; 0 / Receptors, Cell Surface; 0 / TNF Receptor-Associated Factor 6; 0 / Tir protein, E coli; EC 3.1.3.48 / Protein Tyrosine Phosphatase, Non-Receptor Type 11
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31. ||||||||.. 78%  Luo W, Donnenberg MS: Interactions and predicted host membrane topology of the enteropathogenic Escherichia coli translocator protein EspB. J Bacteriol; 2011 Jun;193(12):2972-80
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  • [Title] Interactions and predicted host membrane topology of the enteropathogenic Escherichia coli translocator protein EspB.
  • Translocator proteins include two hydrophobic proteins, represented in enteropathogenic Escherichia coli (EPEC) by EspB and EspD, which are thought to interact and form a pore in the host membrane.
  • Here we adapted a sequence motif recognized by a host kinase to demonstrate that residues on the carboxyl-terminal side of the EspB transmembrane domain are localized to the host cell cytoplasm.
  • [MeSH-major] Bacterial Outer Membrane Proteins / metabolism. Enteropathogenic Escherichia coli / metabolism. Escherichia coli Proteins / metabolism. Gene Expression Regulation, Bacterial / physiology

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  • (PMID = 21498649.001).
  • [ISSN] 1098-5530
  • [Journal-full-title] Journal of bacteriology
  • [ISO-abbreviation] J. Bacteriol.
  • [Language] eng
  • [Grant] United States / NIAID NIH HHS / AI / AI32074; United States / NIAID NIH HHS / AI / R01 AI032074
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Bacterial Outer Membrane Proteins; 0 / EaeB protein, E coli; 0 / Escherichia coli Proteins; 0 / EspA protein, E coli; 0 / EspD protein, E coli; EC 2.7.- / Phosphotransferases
  • [Other-IDs] NLM/ PMC3133209
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32. ||||||||.. 77%  Smith K, Humphreys D, Hume PJ, Koronakis V: Enteropathogenic Escherichia coli recruits the cellular inositol phosphatase SHIP2 to regulate actin-pedestal formation. Cell Host Microbe; 2010 Jan 21;7(1):13-24
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  • [Title] Enteropathogenic Escherichia coli recruits the cellular inositol phosphatase SHIP2 to regulate actin-pedestal formation.
  • Adhesion of enteropathogenic Escherichia coli to epithelial cells triggers actin-rich pedestal formation beneath the bacteria.
  • [MeSH-major] Actins / metabolism. Enteropathogenic Escherichia coli / pathogenicity. Epithelial Cells / microbiology. Escherichia coli Proteins / physiology. Phosphoric Monoester Hydrolases / metabolism. Receptors, Cell Surface / physiology. Virulence Factors / physiology

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  • [Copyright] 2010 Elsevier Inc. All rights reserved.
  • [CommentIn] Cell Host Microbe. 2010 Jan 21;7(1):1-2 [20114020.001]
  • (PMID = 20114025.001).
  • [ISSN] 1934-6069
  • [Journal-full-title] Cell host & microbe
  • [ISO-abbreviation] Cell Host Microbe
  • [Language] eng
  • [Grant] United Kingdom / Medical Research Council / / G0500583; United Kingdom / Biotechnology and Biological Sciences Research Council / / ; United Kingdom / Medical Research Council / / ; United Kingdom / Wellcome Trust / /
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Actins; 0 / Carrier Proteins; 0 / Escherichia coli Proteins; 0 / Membrane Proteins; 0 / Phosphatidylinositols; 0 / RAPH1 protein, human; 0 / Receptors, Cell Surface; 0 / Shc Signaling Adaptor Proteins; 0 / Tir protein, E coli; 0 / Virulence Factors; EC 3.1.3.- / INPPL1 protein, human; EC 3.1.3.- / Phosphoric Monoester Hydrolases
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33. ||||||||.. 77%  Nascimento HH, Silva LE, Souza RT, Silva NP, Scaletsky IC: Phenotypic and genotypic characteristics associated with biofilm formation in clinical isolates of atypical enteropathogenic Escherichia coli (aEPEC) strains. BMC Microbiol; 2014;14:184
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  • [Title] Phenotypic and genotypic characteristics associated with biofilm formation in clinical isolates of atypical enteropathogenic Escherichia coli (aEPEC) strains.
  • BACKGROUND: Biofilm formation by enteropathogenic Escherichia coli (EPEC) have been recently described in the prototype typical EPEC E2348/69 strain and in an atypical EPEC O55:H7 strain.
  • In this study, we sought to evaluate biofilm formation in a collection of 126 atypical EPEC strains isolated from 92 diarrheic and 34 nondiarrheic children, belonging to different serotypes.
  • Unlike the previously described aEPEC O55:H7, one aEPEC O119:HND strain (A111) formed a strong biofilm and pellicle at the air-liquid interface, but did not express curli.
  • [MeSH-major] Biofilms / growth & development. Enteropathogenic Escherichia coli / genetics. Enteropathogenic Escherichia coli / physiology. Escherichia coli Proteins / genetics. Fimbriae, Bacterial / genetics. Phosphorus-Oxygen Lyases / genetics
  • [MeSH-minor] DNA, Bacterial / chemistry. DNA, Bacterial / genetics. Escherichia coli Infections / microbiology. Humans. Infant. Molecular Sequence Data. Sequence Analysis, DNA. Serotyping. Temperature

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  • (PMID = 25012525.001).
  • [ISSN] 1471-2180
  • [Journal-full-title] BMC microbiology
  • [ISO-abbreviation] BMC Microbiol.
  • [Language] eng
  • [Databank-accession-numbers] GENBANK/ KM044265/ KM044266/ KM044267/ KM044268/ KM044269/ KM044270
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / DNA, Bacterial; 0 / Escherichia coli Proteins; EC 4.6.- / Phosphorus-Oxygen Lyases; EC 4.6.1.- / yddV protein, E coli
  • [Other-IDs] NLM/ PMC4100040
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34. ||||||||.. 77%  Salinger N, Kokona B, Fairman R, Okeke IN: The plasmid-encoded regulator activates factors conferring lysozyme resistance on enteropathogenic Escherichia coli strains. Appl Environ Microbiol; 2009 Jan;75(1):275-80
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  • [Title] The plasmid-encoded regulator activates factors conferring lysozyme resistance on enteropathogenic Escherichia coli strains.
  • We demonstrate that enhanced lysozyme resistance of enteropathogenic Escherichia coli requires the plasmid-encoded regulator, Per, and is mediated by factors outside the locus for enterocyte effacement.
  • EspC, a Per-activated serine protease autotransporter protein, conferred enhanced resistance on nonpathogenic E. coli, and a second Per-regulated, espC-independent lysozyme resistance mechanism was identified.
  • [MeSH-major] Anti-Bacterial Agents / antagonists & inhibitors. Drug Resistance, Bacterial. Enteropathogenic Escherichia coli / drug effects. Enteropathogenic Escherichia coli / physiology. Muramidase / antagonists & inhibitors. Plasmids. Transcription Factors / metabolism
  • [MeSH-minor] Amino Acid Sequence. Escherichia coli Proteins / metabolism. Humans. Microbial Sensitivity Tests. Microbial Viability. Molecular Sequence Data

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  • (PMID = 18997020.001).
  • [ISSN] 1098-5336
  • [Journal-full-title] Applied and environmental microbiology
  • [ISO-abbreviation] Appl. Environ. Microbiol.
  • [Language] eng
  • [Grant] United States / Howard Hughes Medical Institute / /
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Anti-Bacterial Agents; 0 / Escherichia coli Proteins; 0 / EspC protein, E coli; 0 / Transcription Factors; EC 3.2.1.17 / Muramidase
  • [Other-IDs] NLM/ PMC2612221
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35. ||||||||.. 77%  Chang AH, Haggerty TD, de Martel C, Leung CW, Parsonnet J: Effect of Helicobacter pylori infection on symptoms of gastroenteritis due to enteropathogenic Escherichia coli in adults. Dig Dis Sci; 2011 Feb;56(2):457-64
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  • [Title] Effect of Helicobacter pylori infection on symptoms of gastroenteritis due to enteropathogenic Escherichia coli in adults.
  • AIMS: We tested the hypothesis that chronic H. pylori infection increases the risk of diarrheal illness due to an acid-sensitive organism: enteropathogenic Escherichia coli (EPEC).
  • METHODS: After testing healthy adult volunteers for H. pylori, 19 infected and 26 uninfected subjects had gastric pH probes placed and were given 5-10 × 10(9) EPEC organisms; six had previously received a proton pump inhibitor.
  • After adjusting for hypochlorhydria and EPEC dose, H. pylori was not associated with diarrhea (OR 0.89, CI: 0.17-4.58).
  • [MeSH-major] Enteropathogenic Escherichia coli. Gastroenteritis / microbiology. Helicobacter Infections / complications. Helicobacter pylori

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  • (PMID = 20635147.001).
  • [ISSN] 1573-2568
  • [Journal-full-title] Digestive diseases and sciences
  • [ISO-abbreviation] Dig. Dis. Sci.
  • [Language] eng
  • [Grant] United States / NCRR NIH HHS / RR / 1UL1 RR025744; United States / NIDDK NIH HHS / DK / R01 DK053689; United States / NIDDK NIH HHS / DK / R01 DK53689
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / 2-Pyridinylmethylsulfinylbenzimidazoles; 0 / Anti-Ulcer Agents; D8TST4O562 / pantoprazole
  • [Other-IDs] NLM/ NIHMS503745; NLM/ PMC4005911
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36. ||||||||.. 77%  Bhat A, Shin M, Jeong JH, Kim HJ, Lim HJ, Rhee JH, Paik SY, Takeyasu K, Tobe T, Yen H, Lee G, Choy HE: DNA looping-dependent autorepression of LEE1 P1 promoters by Ler in enteropathogenic Escherichia coli (EPEC). Proc Natl Acad Sci U S A; 2014 Jun 24;111(25):E2586-95
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  • [Title] DNA looping-dependent autorepression of LEE1 P1 promoters by Ler in enteropathogenic Escherichia coli (EPEC).
  • Ler, a homolog of H-NS in enteropathogenic Escherichia coli (EPEC), plays a critical role in the expression of virulence genes encoded by the pathogenic island, locus of enterocyte effacement (LEE).
  • [MeSH-major] DNA, Bacterial / metabolism. Enteropathogenic Escherichia coli / metabolism. Escherichia coli Proteins / metabolism. Genetic Loci / physiology. Response Elements / physiology. Trans-Activators / metabolism. Transcription Initiation, Genetic / physiology

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  • (PMID = 24920590.001).
  • [ISSN] 1091-6490
  • [Journal-full-title] Proceedings of the National Academy of Sciences of the United States of America
  • [ISO-abbreviation] Proc. Natl. Acad. Sci. U.S.A.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / DNA, Bacterial; 0 / Escherichia coli Proteins; 0 / Ler protein, E coli; 0 / Trans-Activators
  • [Other-IDs] NLM/ PMC4078829
  • [Keywords] NOTNLM ; DNA-bending / first phosphodiester bond formation / road block transcript
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37. ||||||||.. 77%  Roxas JL, Ryan K, Vedantam G, Viswanathan VK: Enteropathogenic Escherichia coli dynamically regulates EGFR signaling in intestinal epithelial cells. Am J Physiol Gastrointest Liver Physiol; 2014 Aug 1;307(3):G374-80
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Enteropathogenic Escherichia coli dynamically regulates EGFR signaling in intestinal epithelial cells.
  • The diarrheagenic pathogen enteropathogenic Escherichia coli (EPEC) dynamically modulates the survival of infected host intestinal epithelial cells.
  • To identify the specific EPEC effector molecule(s) that influenced EGFR stability, epithelial cells infected with isogenic mutant EPEC strains were examined.
  • An EPEC ΔespF strain failed to induce EGFR degradation, whereas EPEC ΔespZ accentuated receptor loss in infected cells.
  • The pan-caspase inhibitor Q-VD-OPh blocked EPEC-induced EGFR cleavage in a dose-dependent manner.
  • Taken together, our data suggest that EPEC EspF stimulates caspase-dependent EGFR cleavage and loss, whereas EspZ inhibits this process.
  • [MeSH-major] Enteropathogenic Escherichia coli / metabolism. Epithelial Cells / metabolism. Epithelial Cells / microbiology. Intestines / metabolism. Intestines / microbiology. Receptor, Epidermal Growth Factor / metabolism. Signal Transduction
  • [MeSH-minor] Caco-2 Cells. Carrier Proteins / genetics. Carrier Proteins / metabolism. Caspase Inhibitors / pharmacology. Cell Death. Dose-Response Relationship, Drug. Escherichia coli Proteins / genetics. Escherichia coli Proteins / metabolism. Host-Pathogen Interactions. Humans. Mutation. Phosphorylation. Protein Stability. Time Factors

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  • [Copyright] Copyright © 2014 the American Physiological Society.
  • (PMID = 24904077.001).
  • [ISSN] 1522-1547
  • [Journal-full-title] American journal of physiology. Gastrointestinal and liver physiology
  • [ISO-abbreviation] Am. J. Physiol. Gastrointest. Liver Physiol.
  • [Language] eng
  • [Grant] United States / PHS HHS / / NIAID1R01AI081742
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Carrier Proteins; 0 / Caspase Inhibitors; 0 / Escherichia coli Proteins; 0 / EspFU protein, E coli; 0 / EspZ protein, E coli; EC 2.7.10.1 / EGFR protein, human; EC 2.7.10.1 / Receptor, Epidermal Growth Factor
  • [Other-IDs] NLM/ PMC4121633 [Available on 08/01/15]
  • [Keywords] NOTNLM ; EGFR degradation / EGFR phosphorylation / EPEC / EspF / EspZ / caspase
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38. ||||||||.. 77%  Girard F, Dziva F, Stevens MP, Frankel G: Interactions of typical and atypical enteropathogenic Escherichia coli strains with the calf intestinal mucosa ex vivo. Appl Environ Microbiol; 2009 Sep;75(18):5991-5
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  • [Title] Interactions of typical and atypical enteropathogenic Escherichia coli strains with the calf intestinal mucosa ex vivo.
  • Enteropathogenic Escherichia coli (EPEC) can be found in healthy and diarrheic cattle; however, little is known about the role of attaching and effacing (A/E) lesion formation in colonization of bovine intestinal mucosa by such strains.
  • We show that typical and atypical EPEC induce A/E lesions on calf intestinal explants independently of Tir tyrosine phosphorylation and TccP.
  • Our data support the existence of conserved Tir- and TccP-independent mechanisms of A/E lesion formation in a range of hosts and reinforce the zoonotic potential of EPEC in cattle.
  • [MeSH-major] Bacterial Adhesion. Enteropathogenic Escherichia coli / pathogenicity. Intestinal Mucosa / microbiology
  • [MeSH-minor] Animals. Cattle. Escherichia coli Proteins / metabolism. Phosphorylation

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  • (PMID = 19633123.001).
  • [ISSN] 1098-5336
  • [Journal-full-title] Applied and environmental microbiology
  • [ISO-abbreviation] Appl. Environ. Microbiol.
  • [Language] eng
  • [Grant] United Kingdom / Biotechnology and Biological Sciences Research Council / /
  • [Publication-type] In Vitro; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Escherichia coli Proteins; 0 / tccP protein, E coli
  • [Other-IDs] NLM/ PMC2747864
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39. ||||||||.. 76%  Gouveia EM, Silva IS, Nakazato G, Araujo FR, Chang MR: Experimental infection with enteropathogenic Escherichia coli identified by PCR using enteric-coated capsules in boxer pups. Acta Cir Bras; 2011 Apr;26(2):144-8
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  • [Title] Experimental infection with enteropathogenic Escherichia coli identified by PCR using enteric-coated capsules in boxer pups.
  • PURPOSE: To verify the possibility of an experimental infection with enteropathogenic Escherichia coli and to confirm by PCR that the symptoms manifested after infection were due to the virulence factors of the studied bacteria.
  • One animal from each litter was included in a control group and the remaining animals were divided into two groups: one inoculated with strain 4083, and another one inoculated with strain SPA14.
  • Gelatinous capsules coated with enteric-coating solution were used for the inoculation of strains. E. coli isolation from feces was performed for all tested animals, and the extracted DNA was subjected to Polymerase Chain Reaction (PCR).
  • CONCLUSION: The efficiency of PCR for the studied strains indicates that this technique can be recommended for the diagnosis of enteropathogenic Escherichia coli as a differential from other pathogens causing diarrhea.
  • [MeSH-major] Enteropathogenic Escherichia coli / genetics. Escherichia coli Infections / microbiology. Polymerase Chain Reaction

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  • (PMID = 21445479.001).
  • [ISSN] 1678-2674
  • [Journal-full-title] Acta cirúrgica brasileira / Sociedade Brasileira para Desenvolvimento Pesquisa em Cirurgia
  • [ISO-abbreviation] Acta Cir Bras
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Brazil
  • [Chemical-registry-number] 0 / DNA, Bacterial; 0 / Virulence Factors
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40. ||||||||.. 76%  Nieto-Pelegrin E, Meiler E, Martín-Villa JM, Benito-León M, Martinez-Quiles N: Crk adaptors negatively regulate actin polymerization in pedestals formed by enteropathogenic Escherichia coli (EPEC) by binding to Tir effector. PLoS Pathog; 2014 Mar;10(3):e1004022
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  • [Title] Crk adaptors negatively regulate actin polymerization in pedestals formed by enteropathogenic Escherichia coli (EPEC) by binding to Tir effector.
  • Infections by enteropathogenic Escherichia coli (EPEC) cause diarrhea linked to high infant mortality in developing countries.
  • EPEC adheres to epithelial cells and induces the formation of actin pedestals.
  • Here we investigate the function of two alternatively spliced isoforms of Crk adaptors (CrkI/II) and the paralog protein CrkL during pedestal formation by EPEC.
  • EPEC infection induces phosphorylation of the major regulatory tyrosine in CrkII and CrkL, possibly preventing the SH2 domain of these proteins from interacting with Tir.
  • Phosphorylated CrkII and CrkL proteins localize specifically to the plasma membrane in contact with EPEC.
  • [MeSH-major] Actins / metabolism. Escherichia coli Infections / metabolism. Escherichia coli Infections / pathology. Escherichia coli Proteins / metabolism. Oncogene Protein v-crk / metabolism. Receptors, Cell Surface / metabolism
  • [MeSH-minor] Adaptor Proteins, Signal Transducing / metabolism. Electroporation. Enteropathogenic Escherichia coli / metabolism. HeLa Cells. Humans. Microscopy, Fluorescence. Oncogene Proteins / metabolism. Protein Isoforms / metabolism. RNA, Small Interfering. Transfection

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  • (PMID = 24675776.001).
  • [ISSN] 1553-7374
  • [Journal-full-title] PLoS pathogens
  • [ISO-abbreviation] PLoS Pathog.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Actins; 0 / Adaptor Proteins, Signal Transducing; 0 / Escherichia coli Proteins; 0 / Nck protein; 0 / Oncogene Protein v-crk; 0 / Oncogene Proteins; 0 / Protein Isoforms; 0 / RNA, Small Interfering; 0 / Receptors, Cell Surface; 0 / Tir protein, E coli
  • [Other-IDs] NLM/ PMC3968158
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41. ||||||||.. 76%  Esmaili A, Nazir SF, Borthakur A, Yu D, Turner JR, Saksena S, Singla A, Hecht GA, Alrefai WA, Gill RK: Enteropathogenic Escherichia coli infection inhibits intestinal serotonin transporter function and expression. Gastroenterology; 2009 Dec;137(6):2074-83
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  • [Title] Enteropathogenic Escherichia coli infection inhibits intestinal serotonin transporter function and expression.
  • Whether alteration in SERT activity contributes to the pathophysiology of diarrhea induced by the food-borne pathogen enteropathogenic Escherichia coli (EPEC) is not known.
  • The present studies examined the effects of EPEC infection on SERT activity and expression in intestinal epithelial cells and elucidated the underlying mechanisms.
  • METHODS: Caco-2 cells as a model of human intestinal epithelia and EPEC-infected C57BL/6J mouse model of infection were utilized.
  • RESULTS: Infection of Caco-2 cells with EPEC for 30-120 minutes decreased apical SERT activity (P < .001) in a type 3 secretion system dependent manner and via involvement of protein tyrosine phosphatases.
  • EPEC infection decreased V(max) of the transporter; whereas cell surface biotinylation studies revealed no alteration in the cellular or plasma membrane content of SERT in Caco-2 cells.
  • EPEC infection of mice (24 hours) reduced SERT immunostaining with a corresponding decrease in SERT messenger RNA levels, 5-HT uptake, and mucosal 5-HT content in the small intestine.
  • CONCLUSIONS: Our results demonstrate inhibition of SERT by EPEC and define the mechanisms underlying these effects.
  • [MeSH-major] Enteropathogenic Escherichia coli / pathogenicity. Escherichia coli Infections / metabolism. Intestinal Mucosa / metabolism. Intestine, Small / metabolism. Serotonin / metabolism. Serotonin Plasma Membrane Transport Proteins / metabolism

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  • (PMID = 19747920.001).
  • [ISSN] 1528-0012
  • [Journal-full-title] Gastroenterology
  • [ISO-abbreviation] Gastroenterology
  • [Language] eng
  • [Grant] United States / NIDDK NIH HHS / DK / DK 061931; United States / NIDDK NIH HHS / DK / DK-074459; United States / NIDDK NIH HHS / DK / DK-09930; United States / NIDDK NIH HHS / DK / DK068271; United States / NIDDK NIH HHS / DK / K01 DK074458; United States / NIDDK NIH HHS / DK / P01 DK-067887; United States / NIDDK NIH HHS / DK / P01 DK067887; United States / NIDDK NIH HHS / DK / P01 DK067887-03; United States / NIDDK NIH HHS / DK / R01 DK050694; United States / NIDDK NIH HHS / DK / R01 DK050694-04; United States / NIDDK NIH HHS / DK / R01 DK061931; United States / NIDDK NIH HHS / DK / R01 DK061931-07; United States / NIDDK NIH HHS / DK / R01 DK071596
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / RNA, Messenger; 0 / SLC6A4 protein, human; 0 / Serotonin Plasma Membrane Transport Proteins; 0 / Slc6a4 protein, mouse; 333DO1RDJY / Serotonin; EC 3.1.3.48 / Protein Tyrosine Phosphatases
  • [Other-IDs] NLM/ NIHMS145181; NLM/ PMC3727418
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42. ||||||||.. 76%  Alonso MZ, Padola NL, Parma AE, Lucchesi PM: Enteropathogenic Escherichia coli contamination at different stages of the chicken slaughtering process. Poult Sci; 2011 Nov;90(11):2638-41
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Enteropathogenic Escherichia coli contamination at different stages of the chicken slaughtering process.
  • Enteropathogenic Escherichia coli is a foodborne pathogen that produces potentially fatal infant diarrhea, noticeably in developing countries.
  • The aim of this study was to detect EPEC contamination by PCR at different stages of the chicken slaughtering process.
  • Enteropathogenic Escherichia coli was detected in 6 to 28% of cloacal samples, 39 and 56% of unwashed eviscerated carcasses, and 4 to 58% of washed carcasses.
  • None of the samples were positive for bfpA, suggesting contamination with atypical EPEC.
  • The detection of EPEC at different stages of the chicken slaughtering process showed that the proportion of contaminated samples remained or even increased during processing.
  • [MeSH-major] Abattoirs. Enteropathogenic Escherichia coli / isolation & purification. Food Microbiology

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  • (PMID = 22010252.001).
  • [ISSN] 0032-5791
  • [Journal-full-title] Poultry science
  • [ISO-abbreviation] Poult. Sci.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
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43. ||||||||.. 76%  Moura RA, Sircili MP, Leomil L, Matté MH, Trabulsi LR, Elias WP, Irino K, Pestana de Castro AF: Clonal relationship among atypical enteropathogenic Escherichia coli strains isolated from different animal species and humans. Appl Environ Microbiol; 2009 Dec;75(23):7399-408
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  • [Title] Clonal relationship among atypical enteropathogenic Escherichia coli strains isolated from different animal species and humans.
  • Forty-nine typical and atypical enteropathogenic Escherichia coli (EPEC) strains belonging to different serotypes and isolated from humans, pets (cats and dogs), farm animals (bovines, sheep, and rabbits), and wild animals (monkeys) were investigated for virulence markers and clonal similarity by pulsed-field gel electrophoresis (PFGE) and multilocus sequence typing (MLST).
  • The virulence markers analyzed revealed that atypical EPEC strains isolated from animals have the potential to cause diarrhea in humans.
  • These results indicate that these animals act as atypical EPEC reservoirs and may represent sources of infection for humans.
  • Since humans also act as a reservoir of atypical EPEC strains, the cycle of mutual infection of atypical EPEC between animals and humans, mainly pets and their owners, cannot be ruled out since the transmission dynamics between the reservoirs are not yet clearly understood.
  • [MeSH-major] Animals, Domestic / microbiology. Animals, Wild / microbiology. Bacterial Typing Techniques. DNA Fingerprinting. DNA, Bacterial / genetics. Enteropathogenic Escherichia coli / classification. Enteropathogenic Escherichia coli / genetics
  • [MeSH-minor] Animals. Brazil. Cluster Analysis. Electrophoresis, Gel, Pulsed-Field. Escherichia coli Proteins / genetics. Genotype. Humans. Molecular Epidemiology. Molecular Sequence Data. Sequence Analysis, DNA. Serotyping. Virulence Factors / genetics

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  • [ISSN] 1098-5336
  • [Journal-full-title] Applied and environmental microbiology
  • [ISO-abbreviation] Appl. Environ. Microbiol.
  • [Language] eng
  • [Databank-accession-numbers] GENBANK/ FJ561750/ FJ561751/ FJ561752/ FJ561753/ FJ561754/ FJ561755/ FJ561756/ FJ561757/ FJ561758/ FJ561759/ FJ561760/ FJ561761/ FJ561762/ FJ561763/ FJ561764/ FJ561765/ FJ561766/ FJ561767/ FJ561768/ FJ561769/ FJ561770/ FJ561771/ FJ561772/ FJ561773/ FJ561774/ FJ561775/ FJ561776/ FJ561777/ FJ561778/ FJ561779/ FJ561780/ FJ561781/ FJ561782/ FJ561783/ FJ561784/ FJ561785/ FJ561786/ FJ561787/ FJ561788/ FJ561789/ FJ561790/ FJ561791/ FJ561792/ FJ561793/ FJ561794/ FJ561795/ FJ561796/ FJ561797/ FJ561798/ FJ561799/ FJ561800/ FJ561801/ FJ561802/ FJ561803/ FJ561804/ FJ561805/ FJ561806/ FJ561807/ FJ561808/ FJ561809/ FJ561810/ FJ561811/ FJ561812/ FJ561813/ FJ561814/ FJ561815/ FJ561816/ FJ561817/ FJ561818/ FJ561819/ FJ561820/ FJ561821/ FJ561822/ FJ561823/ FJ561824/ FJ561825/ FJ561826/ FJ561827/ FJ561828/ FJ561829/ FJ561830/ FJ561831/ FJ561832/ FJ561833/ FJ561834/ FJ561835/ FJ561836/ FJ561837/ FJ561838/ FJ561839/ FJ561840/ FJ561841/ FJ561842/ FJ561843/ FJ561844/ FJ561845/ FJ561846/ FJ561847/ FJ561848/ FJ561849/ FJ561850/ FJ561851/ FJ561852/ FJ561853/ FJ561854/ FJ561855/ FJ561856/ FJ561857/ FJ561858/ FJ561859/ FJ561860/ FJ561861/ FJ561862/ FJ561863/ FJ561864/ FJ561865/ FJ561866/ FJ561867/ FJ561868/ FJ561869/ FJ561870/ FJ561871/ FJ561872/ FJ561873/ FJ561874/ FJ561875/ FJ561876/ FJ561877/ FJ561878/ FJ561879/ FJ561880/ FJ561881/ FJ561882/ FJ561883/ FJ561884/ FJ561885/ FJ561886/ FJ561887/ FJ561888/ FJ561889/ FJ561890/ FJ561891/ FJ561892/ FJ561893/ FJ561894/ FJ561895/ FJ561896/ FJ561897/ FJ561898/ FJ561899/ FJ561900/ FJ561901/ FJ561902/ FJ561903/ FJ561904/ FJ561905/ FJ561906/ FJ561907/ FJ561908/ FJ561909/ FJ561910/ FJ561911/ FJ561912/ FJ561913/ FJ561914/ FJ561915/ FJ561916/ FJ561917/ FJ561918/ FJ561919/ FJ561920/ FJ561921/ FJ561922/ FJ561923/ FJ561924/ FJ561925/ FJ561926/ FJ561927/ FJ561928/ FJ561929/ FJ561930/ FJ561931/ FJ561932/ FJ561933/ FJ561934/ FJ561935/ FJ561936/ FJ561937/ FJ561938/ FJ561939/ FJ561940/ FJ561941/ FJ561942/ FJ561943/ FJ561944/ FJ561945/ FJ561946/ FJ561947/ FJ561948/ FJ561949/ FJ561950/ FJ561951/ FJ561952/ FJ561953/ FJ561954/ FJ561955/ FJ561956/ FJ561957/ FJ561958/ FJ561959/ FJ561960/ FJ561961/ FJ561962/ FJ561963/ FJ561964/ FJ561965/ FJ561966/ FJ561967/ FJ561968/ FJ561969/ FJ561970/ FJ561971/ FJ561972/ FJ561973/ FJ561974/ FJ561975/ FJ561976/ FJ561977/ FJ561978/ FJ561979/ FJ561980/ FJ561981/ FJ561982/ FJ561983/ FJ561984/ FJ561985/ FJ561986/ FJ561987/ FJ561988/ FJ561989/ FJ561990/ FJ561991/ FJ561992/ FJ561993/ FJ561994/ FJ561995/ FJ561996/ FJ561997/ FJ561998/ FJ561999/ FJ562000/ FJ562001/ FJ562002/ FJ562003/ FJ562004/ FJ562005/ FJ562006/ FJ562007/ FJ562008/ FJ562009/ FJ562010/ FJ562011/ FJ562012/ FJ562013/ FJ562014/ FJ562015/ FJ562016/ FJ562017/ FJ562018/ FJ562019/ FJ562020/ FJ562021/ FJ562022/ FJ562023/ FJ562024/ FJ562025/ FJ562026/ FJ562027/ FJ562028/ FJ562029/ FJ562030/ FJ562031/ FJ562032/ FJ562033/ FJ562034/ FJ562035/ FJ562036/ FJ562037/ FJ562038/ FJ562039/ FJ562040/ FJ562041/ FJ562042/ FJ562043/ FJ562044/ FJ562045/ FJ562046/ FJ562047/ FJ562048/ FJ562049/ FJ562050/ FJ562051/ FJ562052/ FJ562053/ FJ562054/ FJ562055/ FJ562056/ FJ562057/ FJ562058/ FJ562059/ FJ562060/ FJ562061/ FJ562062/ FJ562063/ FJ562064/ FJ562065/ FJ562066/ FJ562067/ FJ562068/ FJ562069/ FJ562070/ FJ562071/ FJ562072/ FJ562073/ FJ562074/ FJ562075/ FJ562076/ FJ562077/ FJ562078/ FJ562079/ FJ562080/ FJ562081/ FJ562082/ FJ562083/ FJ562084/ FJ562085/ FJ562086/ FJ562087/ FJ562088/ FJ562089/ FJ562090/ FJ562091/ FJ562092
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / DNA, Bacterial; 0 / Escherichia coli Proteins; 0 / Virulence Factors
  • [Other-IDs] NLM/ PMC2786407
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44. ||||||||.. 75%  Culler HF, Mota CM, Abe CM, Elias WP, Sircili MP, Franzolin MR: Atypical enteropathogenic Escherichia coli strains form biofilm on abiotic surfaces regardless of their adherence pattern on cultured epithelial cells. Biomed Res Int; 2014;2014:845147
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  • [Title] Atypical enteropathogenic Escherichia coli strains form biofilm on abiotic surfaces regardless of their adherence pattern on cultured epithelial cells.
  • The aim of this study was to determine the capacity of biofilm formation of atypical enteropathogenic Escherichia coli (aEPEC) strains on abiotic and biotic surfaces.

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  • (PMID = 24883330.001).
  • [ISSN] 2314-6141
  • [Journal-full-title] BioMed research international
  • [ISO-abbreviation] Biomed Res Int
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Other-IDs] NLM/ PMC4032706
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45. ||||||||.. 75%  Flynn AN, Wang A, McKay DM, Buret AG: Apoptosis-inducing factor contributes to epithelial cell apoptosis induced by enteropathogenic Escherichia coli. Can J Physiol Pharmacol; 2011 Feb;89(2):143-8
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  • [Title] Apoptosis-inducing factor contributes to epithelial cell apoptosis induced by enteropathogenic Escherichia coli.
  • The mechanisms by which enteropathogenic Escherichia coli (EPEC) causes intestinal epithelial cell apoptosis remain unclear.
  • We tested the hypothesis that apoptosis-inducing factor (AIF) is involved in apoptosis induced by EPEC.
  • Infection of intestinal epithelial cells in vitro with EPEC led to the mitochondrial and cytosolic accumulation of AIF.
  • Knockdown of AIF with siRNA blocked cellular apoptosis in response to EPEC infection, as assessed by poly(ADP-ribose) polymerase cleavage and oligonucleosome formation.
  • Taken together, these data suggest that caspase-dependent mobilization of AIF contributes to EPEC-induced epithelial cell apoptosis.
  • [MeSH-major] Apoptosis / physiology. Apoptosis Inducing Factor / metabolism. Enteropathogenic Escherichia coli / physiology. Intestinal Mucosa / metabolism. Intestinal Mucosa / microbiology
  • [MeSH-minor] Caspases / metabolism. Cell Line. Cytosol / metabolism. Epithelial Cells / metabolism. Epithelial Cells / microbiology. Epithelial Cells / pathology. Escherichia coli Infections / metabolism. Escherichia coli Infections / microbiology. Escherichia coli Infections / pathology. Gene Knockdown Techniques. HeLa Cells. Humans. Mitochondria / metabolism. Poly(ADP-ribose) Polymerases / metabolism. RNA, Small Interfering / genetics

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  • (PMID = 21326346.001).
  • [ISSN] 1205-7541
  • [Journal-full-title] Canadian journal of physiology and pharmacology
  • [ISO-abbreviation] Can. J. Physiol. Pharmacol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Canada
  • [Chemical-registry-number] 0 / Apoptosis Inducing Factor; 0 / RNA, Small Interfering; EC 2.4.2.30 / Poly(ADP-ribose) Polymerases; EC 3.4.22.- / Caspases
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46. ||||||||.. 75%  Liu J, Wang WD, Liu YJ, Liu S, Zhou B, Zhu LW, Ji X, Sun Y, Feng SZ: Mice vaccinated with enteropathogenic Escherichia coli ghosts show significant protection against lethal challenges. Lett Appl Microbiol; 2012 Mar;54(3):255-62
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  • [Title] Mice vaccinated with enteropathogenic Escherichia coli ghosts show significant protection against lethal challenges.
  • AIM: To prepare enteropathogenic Escherichia coli (EPEC) E2348/69 ghosts and investigate whether immunization with EPEC bacterial ghosts can elicit protective immune responses.
  • An EPEC reference strain E2348/69 (serotype O127:H6) was transformed with the lysis plasmids to produce EPEC ghosts.
  • Mice injected with bacterial ghosts EGE (EPEC ghosts produced using lysis protein E) or EGES (EPEC ghosts produced using a combination of lysis protein E and SNA) gained weight normally and showed no clinical signs of disease.
  • Vaccination trials showed that mice immunized with EGE or EGES were significantly protected against subsequent challenge with the wild-type virulent parent strain, EPEC E2348/69 (42/50 and 45/50 survival, respectively); in contrast, none of the 30 control mice survived.
  • CONCLUSIONS: Immunization with EPEC ghosts can elicit protective immune responses in BALB/c mice.
  • SIGNIFICANCE AND IMPACT OF THE STUDY: EPEC ghosts may represent a promising new approach for vaccination against EPEC infection.
  • [MeSH-major] Bacterial Vaccines / immunology. Enteropathogenic Escherichia coli / immunology. Escherichia coli Infections / prevention & control

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  • [Copyright] © 2011 The Authors. Letters in Applied Microbiology © 2011 The Society for Applied Microbiology.
  • (PMID = 22211792.001).
  • [ISSN] 1472-765X
  • [Journal-full-title] Letters in applied microbiology
  • [ISO-abbreviation] Lett. Appl. Microbiol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Antibodies, Bacterial; 0 / Bacterial Vaccines; 0 / Immunoglobulin G
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47. |||||||... 75%  Aslani MM, Alikhani MY: Molecular and phenotypic characterization of atypical enteropathogenic Escherichia coli serotypes isolated from children with and without diarrhea. J Microbiol Immunol Infect; 2011 Feb;44(1):27-32
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  • [Title] Molecular and phenotypic characterization of atypical enteropathogenic Escherichia coli serotypes isolated from children with and without diarrhea.
  • BACKGROUND: We characterized 36 atypical enteropathogenic Escherichia coli (EPEC) serotypes isolated from children with and without diarrhea in Iran.
  • Because the identification of atypical EPEC based on biochemical features is rather difficult and time consuming, we used a combination of three approaches, including a polymerase chain reaction-based method, culture adherence assay, and the restriction analysis of fliC gene (fliC-restriction fragment length polymorphism), to identify E coli serotypes.
  • METHODS: To distinguish typical and atypical EPEC strains, the presence of EPEC attaching effacing A gene (eaeA) gene and EPEC-attaching factor (EAF) plasmid were analyzed.
  • All E coli strains were identified based on the detection of the eaeA(+), bundle-forming pili A gene (bfpA(-)), EAF(-) or eaeA(-), bfpA(+), EAF(-) profiles and the absence of stx (encoded for shiga toxin) gene as atypical EPEC.
  • RESULTS: All strains studied belonged to 5 atypical EPEC serogroups and 15 serotypes based on the virulence profiles.
  • Of 36 atypical EPEC serotypes, 22 (61.2%) and 14 (38.8%) strains isolated from diarrheal and healthy cases, respectively.
  • CONCLUSIONS: The characteristics of the atypical EPEC serotypes from children with diarrhea were significantly different from those without diarrhea.
  • The compilation of data on atypical EPEC strains presented here indicates the importance of a combined approach of conventional and molecular tests to study the virulence and epidemiology of EPEC serotypes in human subjects.

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  • [Copyright] Copyright © 2011. Published by Elsevier B.V.
  • (PMID = 21531349.001).
  • [ISSN] 1684-1182
  • [Journal-full-title] Journal of microbiology, immunology, and infection = Wei mian yu gan ran za zhi
  • [ISO-abbreviation] J Microbiol Immunol Infect
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
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48. |||||||... 75%  Scaletsky IC, Souza TB, Aranda KR, Okeke IN: Genetic elements associated with antimicrobial resistance in enteropathogenic Escherichia coli (EPEC) from Brazil. BMC Microbiol; 2010;10:25
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  • [Title] Genetic elements associated with antimicrobial resistance in enteropathogenic Escherichia coli (EPEC) from Brazil.
  • BACKGROUND: We recently observed an association of resistance with a certain enteropathogenic Escherichia coli (EPEC) serotypes and identified a conjugative plasmid, similar to plasmid pED208, that was conserved among archival O111:H2/NM and O119:H2 strains of diverse geographical origin.
  • In this study, we sought to determine the prevalence and distribution of this plasmid among a collection of EPEC isolates from Brazil, as well as to study the susceptibilities of these isolates to antimicrobial agents.
  • RESULTS: Resistance was more commonly seen in typical EPEC than atypical strains.
  • Markers for the EPEC conjugative multiresistance plasmid, were detected in 21 (30%) of typical but only 4 (5%) of atypical strains (p = 0.001, Chi-squared test).
  • CONCLUSION: Our data suggest a limited but expanding host range for the EPEC resistance plasmid.
  • [MeSH-major] Enteropathogenic Escherichia coli / drug effects. Enteropathogenic Escherichia coli / genetics. Escherichia coli Infections / microbiology

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  • (PMID = 20105329.001).
  • [ISSN] 1471-2180
  • [Journal-full-title] BMC microbiology
  • [ISO-abbreviation] BMC Microbiol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Anti-Bacterial Agents
  • [Other-IDs] NLM/ PMC2828443
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49. |||||||... 75%  Magalhães CA, Rossato SS, Barbosa AS, Santos TO, Elias WP, Sircili MP, Piazza RM: The ability of haemolysins expressed by atypical enteropathogenic Escherichia coli to bind to extracellular matrix components. Mem Inst Oswaldo Cruz; 2011 Mar;106(2):146-52
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  • [Title] The ability of haemolysins expressed by atypical enteropathogenic Escherichia coli to bind to extracellular matrix components.
  • Typical and atypical enteropathogenic Escherichia coli (EPEC) are considered important bacterial causes of diarrhoea.
  • Considering the repertoire of virulence genes, atypical EPEC (aEPEC) is a heterogeneous group, harbouring genes that are found in other diarrheagenic E. coli pathotypes, such as those encoding haemolysins.
  • [MeSH-major] Enteropathogenic Escherichia coli / physiology. Escherichia coli Proteins / physiology. Extracellular Matrix / metabolism

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  • (PMID = 21537672.001).
  • [ISSN] 1678-8060
  • [Journal-full-title] Memórias do Instituto Oswaldo Cruz
  • [ISO-abbreviation] Mem. Inst. Oswaldo Cruz
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Brazil
  • [Chemical-registry-number] 0 / Escherichia coli Proteins; 0 / Hemolysin Proteins; 0 / Virulence Factors; 0 / enterohemolysin
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50. |||||||... 74%  Roxas JL, Wilbur JS, Zhang X, Martinez G, Vedantam G, Viswanathan VK: The enteropathogenic Escherichia coli-secreted protein EspZ inhibits host cell apoptosis. Infect Immun; 2012 Nov;80(11):3850-7
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] The enteropathogenic Escherichia coli-secreted protein EspZ inhibits host cell apoptosis.
  • The diarrheagenic pathogen enteropathogenic Escherichia coli (EPEC) limits the death of infected enterocytes early in infection.
  • A number of bacterial molecules and host signaling pathways contribute to the enhanced survival of EPEC-infected host cells.
  • EspZ, a type III secreted effector protein that is unique to EPEC and related "attaching and effacing" (A/E) pathogens, plays a role in limiting host cell death, but the precise host signaling pathways responsible for this phenotype are not known.
  • Consistent with previous studies, scanning electron microscopy analysis of intestinal epithelial cells infected with an EPEC espZ mutant (ΔespZ) showed increased levels of apoptotic and necrotic cells compared to cells infected with the isogenic parent strain.
  • Correspondingly, higher levels of cytosolic cytochrome c and increased activation of caspases 9, 7, and 3 were observed for ΔespZ strain-infected cells compared to wild-type (WT) EPEC-infected cells.
  • [MeSH-major] Apoptosis / physiology. Enteropathogenic Escherichia coli / metabolism. Epithelial Cells / metabolism. Escherichia coli Proteins / metabolism. Intestines / metabolism

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  • (PMID = 22907816.001).
  • [ISSN] 1098-5522
  • [Journal-full-title] Infection and immunity
  • [ISO-abbreviation] Infect. Immun.
  • [Language] eng
  • [Grant] United States / NIAID NIH HHS / AI / 1R01AI081742; United States / NIAID NIH HHS / AI / R01 AI081742
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Escherichia coli Proteins; 0 / EspZ protein, E coli
  • [Other-IDs] NLM/ PMC3486051
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