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1. Biomedical articles (top 50; 2009 to 2014)
1. |||||||||. 100%  Scaletsky IC, Aranda KR, Souza TB, Silva NP, Morais MB: Evidence of pathogenic subgroups among atypical enteropathogenic Escherichia coli strains. J Clin Microbiol; 2009 Nov;47(11):3756-9
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Evidence of pathogenic subgroups among atypical enteropathogenic Escherichia coli strains.
  • We describe the characterization of 126 atypical enteropathogenic Escherichia coli (aEPEC) isolates from 1,749 Brazilian children.
  • [MeSH-major] Diarrhea / microbiology. Enteropathogenic Escherichia coli / classification. Enteropathogenic Escherichia coli / pathogenicity. Escherichia coli Infections / microbiology
  • [MeSH-minor] Brazil. DNA Primers / genetics. DNA, Bacterial / genetics. Escherichia coli Proteins / genetics. Genotype. Hemolysin Proteins / genetics. Humans. Infant. Serotyping. Virulence Factors / genetics

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  • (PMID = 19759223.001).
  • [ISSN] 1098-660X
  • [Journal-full-title] Journal of clinical microbiology
  • [ISO-abbreviation] J. Clin. Microbiol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, U.S. Gov't, Non-P.H.S.
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / DNA Primers; 0 / DNA, Bacterial; 0 / Escherichia coli Proteins; 0 / Hemolysin Proteins; 0 / Hlya protein, E coli; 0 / Paa protein, E coli; 0 / Virulence Factors
  • [Other-IDs] NLM/ PMC2772600
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2. |||||||||. 96%  Ochoa TJ, Contreras CA: Enteropathogenic escherichia coli infection in children. Curr Opin Infect Dis; 2011 Oct;24(5):478-83
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Enteropathogenic escherichia coli infection in children.
  • PURPOSE OF REVIEW: Enteropathogenic Escherichia coli (EPEC) is an important diarrheal pathogen of young children.
  • As the diagnosis of EPEC is now based mainly on molecular criteria, there has been an important change in its prevalence.
  • The purpose of this study is to review the current epidemiology of EPEC infection and the new insights into its physiopathology.
  • RECENT FINDINGS: Recent epidemiological studies indicate that atypical EPEC (aEPEC) is more prevalent than typical EPEC (tEPEC) in both developed and developing countries, and that aEPEC is important in both pediatric endemic diarrhea and diarrhea outbreaks.
  • However, the exact mechanisms of diarrhea in EPEC infection are not completely understood.
  • SUMMARY: Remarkable progress has been made to identify virulence determinants required to mediate the pathogenesis of EPEC.
  • [MeSH-major] Enteropathogenic Escherichia coli. Escherichia coli Infections / epidemiology

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  • (PMID = 21857511.001).
  • [ISSN] 1473-6527
  • [Journal-full-title] Current opinion in infectious diseases
  • [ISO-abbreviation] Curr. Opin. Infect. Dis.
  • [Language] eng
  • [Grant] United States / FIC NIH HHS / TW / 1K01TW007405; United States / FIC NIH HHS / TW / K01 TW007405-04
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Review
  • [Publication-country] United States
  • [Other-IDs] NLM/ NIHMS348474; NLM/ PMC3277943
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3. |||||||||. 95%  Hazen TH, Sahl JW, Fraser CM, Donnenberg MS, Scheutz F, Rasko DA: Draft Genome Sequences of Three O157 Enteropathogenic Escherichia coli Isolates. Genome Announc; 2013;1(4)
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  • [Title] Draft Genome Sequences of Three O157 Enteropathogenic Escherichia coli Isolates.
  • We report the draft genome sequences of three enteropathogenic Escherichia coli (EPEC) isolates that display the O157 serogroup but do not have the Shiga toxin genes (stx), which are characteristic of O157 enterohemorrhagic E. coli (EHEC). E. coli strain RN587/1 has the O157:H8 serotype and possesses the EAF plasmid characteristic of typical EPEC (J. B.

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  • (PMID = 23868135.001).
  • [ISSN] 2169-8287
  • [Journal-full-title] Genome announcements
  • [ISO-abbreviation] Genome Announc
  • [Language] eng
  • [Grant] United States / NIAID NIH HHS / AI / U19 AI090873
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Other-IDs] NLM/ PMC3715677
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4. |||||||||. 95%  Thomassin JL, Brannon JR, Kaiser J, Gruenheid S, Le Moual H: Enterohemorrhagic and enteropathogenic Escherichia coli evolved different strategies to resist antimicrobial peptides. Gut Microbes; 2012 Nov-Dec;3(6):556-61
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  • [Title] Enterohemorrhagic and enteropathogenic Escherichia coli evolved different strategies to resist antimicrobial peptides.
  • Enterohemorrhagic and enteropathogenic Escherichia coli (EHEC and EPEC) are enteric human pathogens that colonize the large and small intestines, respectively.
  • To establish infection EHEC and EPEC must overcome innate host defenses, such as antimicrobial peptides (AMPs) produced by the intestinal epithelium.
  • We showed that the protease OmpT degrades the human AMP LL-37 more rapidly in EHEC than in EPEC.
  • Here, we propose that the different ompT expression in EHEC and EPEC reflects the varying levels of LL-37 throughout the human intestinal tract.
  • These data suggest that EHEC and EPEC adapted to their specific niches by developing distinct AMP-specific resistance mechanisms.
  • [MeSH-major] Antimicrobial Cationic Peptides / pharmacology. Drug Resistance, Microbial. Enterohemorrhagic Escherichia coli / drug effects. Enterohemorrhagic Escherichia coli / pathogenicity. Enteropathogenic Escherichia coli / drug effects. Enteropathogenic Escherichia coli / pathogenicity. Immune Evasion
  • [MeSH-minor] Bacterial Outer Membrane Proteins / metabolism. Escherichia coli Infections / immunology. Escherichia coli Infections / microbiology. Escherichia coli Proteins / metabolism. Gene Expression. Humans. Peptide Hydrolases / metabolism. Promoter Regions, Genetic. Proteolysis

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  • (PMID = 22895086.001).
  • [ISSN] 1949-0984
  • [Journal-full-title] Gut microbes
  • [ISO-abbreviation] Gut Microbes
  • [Language] eng
  • [Grant] Canada / Canadian Institutes of Health Research / / MOP-15551
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't; Review
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Antimicrobial Cationic Peptides; 0 / Bacterial Outer Membrane Proteins; 0 / Escherichia coli Proteins; 0 / ompT protein, E coli; 143108-26-3 / CAP18 lipopolysaccharide-binding protein; EC 3.4.- / Peptide Hydrolases
  • [Other-IDs] NLM/ PMC3495793
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5. |||||||||. 95%  Ruetz TJ, Vogl AW, Guttman JA: Detailed examination of cytoskeletal networks within enteropathogenic Escherichia coli pedestals. Anat Rec (Hoboken); 2012 Feb;295(2):201-7
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  • [Title] Detailed examination of cytoskeletal networks within enteropathogenic Escherichia coli pedestals.
  • Enteropathogenic Escherichia coli (EPEC) manipulate the cytoskeleton of host intestinal epithelial cells, producing membrane protrusions termed pedestals that the bacteria reside on throughout the course of their infections.
  • By definition pedestals are actin-based structures, however recent work has identified the spectrin cytoskeleton as a necessary component of EPEC pedestals.
  • Myosin S1 fragment decorated actin filaments examined by electron microscopy demonstrated that actin filaments orientate with their fast-growing barbed ends toward the lateral membranes of EPEC pedestals.
  • [MeSH-major] Cell Surface Extensions / ultrastructure. Cytoskeleton / ultrastructure. Enteropathogenic Escherichia coli / ultrastructure. HeLa Cells / ultrastructure

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  • [Copyright] Copyright © 2011 Wiley Periodicals, Inc.
  • (PMID = 22190417.001).
  • [ISSN] 1932-8494
  • [Journal-full-title] Anatomical record (Hoboken, N.J. : 2007)
  • [ISO-abbreviation] Anat Rec (Hoboken)
  • [Language] eng
  • [Grant] Canada / Canadian Institutes of Health Research / /
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Cytoskeletal Proteins; 0 / Myosin Subfragments; 12634-43-4 / Spectrin
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6. |||||||||. 95%  Behiry IK, Abada EA, Ahmed EA, Labeeb RS: Enteropathogenic Escherichia coli associated with diarrhea in children in Cairo, Egypt. ScientificWorldJournal; 2011;11:2613-9
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  • [Title] Enteropathogenic Escherichia coli associated with diarrhea in children in Cairo, Egypt.
  • In this study we isolate and identify the Enteropathogenic Escherichia coli (EPEC) causing diarrhea in children less than five years in Cairo, Egypt, during different seasons.
  • The identified E. coli isolates were subjected to antimicrobial disc diffusion susceptibility test and further identified for EPEC serotype by slide agglutination test, using antiserum E. coli somatic trivalent I (O111, O55, O26) according to the instructions of the manufacturer.
  • Out of 134 patients 5.2% of them revealed EPEC in the fecal sample, while the 20 children control group showed no EPEC isolates in their samples.
  • Our EPEC frequency showed variations from the compared results of other studies.
  • Higher rate of EPEC (18.7%) was found in patients between 2 to 3 years, while EPEC rate was (7.5%) in patients less than 6 months old, with P < 0.05.
  • EPEC was identified from fecal specimens as a unique pathogen or associated with other pathogens in acute and chronic diarrhea in children.
  • EPEC were detected in all seasons except in winter, and was predominant in summer season.
  • Four (57%) EPEC isolates were resistant to ampicillin, ticarcillin, and cotrimoxazole, and (14.3%) to the third generation cephalosporins.
  • [MeSH-major] Diarrhea / microbiology. Enteropathogenic Escherichia coli / isolation & purification

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  • (PMID = 22262949.001).
  • [ISSN] 1537-744X
  • [Journal-full-title] TheScientificWorldJournal
  • [ISO-abbreviation] ScientificWorldJournal
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Anti-Bacterial Agents
  • [Other-IDs] NLM/ PMC3254012
  • [Keywords] NOTNLM ; children less than 5 years / enteropathogenic E. coli / slide agglutination test
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7. |||||||||. 93%  Hernandes RT, Velsko I, Sampaio SC, Elias WP, Robins-Browne RM, Gomes TA, Girón JA: Fimbrial adhesins produced by atypical enteropathogenic Escherichia coli strains. Appl Environ Microbiol; 2011 Dec;77(23):8391-9
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Fimbrial adhesins produced by atypical enteropathogenic Escherichia coli strains.
  • Atypical enteropathogenic Escherichia coli (aEPEC) has emerged as a significant cause of pediatric diarrhea worldwide; however, information regarding its adherence mechanisms to the human gut mucosa is lacking.
  • These genes are associated with adhesion and/or biofilm formation of pathogenic and commensal E. coli.
  • We were not able to detect the hemorrhagic coli pilus (HCP) or the E. coli laminin-binding fimbriae (ELF) in these strains by using immunofluorescence.
  • The E. coli common pilus (ECP) was evidenced in 36.6% of the strains on bacteria adhering to HeLa cells by immunofluorescence, suggesting that ECP could play an important role in cell adherence for some aEPEC strains.
  • [MeSH-major] Adhesins, Bacterial / biosynthesis. Adhesins, Escherichia coli / biosynthesis. Enteropathogenic Escherichia coli / metabolism
  • [MeSH-minor] Australia. Bacterial Adhesion. Brazil. DNA, Bacterial / genetics. Diarrhea / microbiology. Epithelial Cells / microbiology. Escherichia coli Infections / microbiology. Gene Expression Profiling. HeLa Cells. Humans. Polymerase Chain Reaction

  • NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .
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  • (PMID = 21926222.001).
  • [ISSN] 1098-5336
  • [Journal-full-title] Applied and environmental microbiology
  • [ISO-abbreviation] Appl. Environ. Microbiol.
  • [Language] eng
  • [Grant] United States / NIAID NIH HHS / AI / AI66012
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Adhesins, Bacterial; 0 / Adhesins, Escherichia coli; 0 / DNA, Bacterial
  • [Other-IDs] NLM/ PMC3233042
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8. |||||||||. 123%  Donnenberg MS, Finlay BB: Combating enteropathogenic Escherichia coli (EPEC) infections: the way forward. Trends Microbiol; 2013 Jul;21(7):317-9
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Combating enteropathogenic Escherichia coli (EPEC) infections: the way forward.
  • Enteropathogenic Escherichia coli (EPEC) strains continue to cause severe and sometimes fatal infantile diarrhea, particularly in Africa.
  • Increased efforts at diagnosis, defining the clinical spectrum of disease, understanding pathogenic mechanisms, and delineating immune responses are desperately needed to develop new strategies to combat EPEC.
  • [MeSH-major] Diarrhea / epidemiology. Diarrhea / microbiology. Enteropathogenic Escherichia coli / immunology. Enteropathogenic Escherichia coli / pathogenicity. Escherichia coli Infections / epidemiology. Escherichia coli Infections / microbiology
  • [MeSH-minor] Africa / epidemiology. Escherichia coli Proteins / immunology. Immunization / methods. Virulence Factors / immunology

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  • [Copyright] Copyright © 2013 Elsevier Ltd. All rights reserved.
  • (PMID = 23815982.001).
  • [ISSN] 1878-4380
  • [Journal-full-title] Trends in microbiology
  • [ISO-abbreviation] Trends Microbiol.
  • [Language] eng
  • [Grant] United States / NIAID NIH HHS / AI / U19 AI 090873; United States / NIAID NIH HHS / AI / U19 AI090873; Canada / Canadian Institutes of Health Research / /
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Escherichia coli Proteins; 0 / Virulence Factors
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9. |||||||||. 116%  Hernandes RT, Elias WP, Vieira MA, Gomes TA: An overview of atypical enteropathogenic Escherichia coli. FEMS Microbiol Lett; 2009 Aug;297(2):137-49
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  • [Title] An overview of atypical enteropathogenic Escherichia coli.
  • The enteropathogenic Escherichia coli (EPEC) pathotype is currently divided into two groups, typical EPEC (tEPEC) and atypical EPEC (aEPEC).
  • The property that distinguishes these two groups is the presence of the EPEC adherence factor plasmid, which is only found in tEPEC. aEPEC strains are emerging enteropathogens that have been detected worldwide.
  • Herein, we review the serotypes, virulence properties, genetic relationships, epidemiology, reservoir and diagnosis of aEPEC, including those strains not belonging to the classical EPEC serogroups (nonclassical EPEC serogroups).
  • The large variety of serotypes and genetic virulence properties of aEPEC strains from nonclassical EPEC serogroups makes it difficult to determine which strains are truly pathogenic.
  • [MeSH-major] Enteropathogenic Escherichia coli. Escherichia coli Infections / microbiology
  • [MeSH-minor] Animals. Escherichia coli Proteins / genetics. Escherichia coli Proteins / metabolism. Humans. Virulence. World Health

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  • (PMID = 19527295.001).
  • [ISSN] 1574-6968
  • [Journal-full-title] FEMS microbiology letters
  • [ISO-abbreviation] FEMS Microbiol. Lett.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't; Review
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Escherichia coli Proteins
  • [Number-of-references] 138
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10. |||||||||. 104%  Swennes AG, Buckley EM, Madden CM, Byrd CP, Donocoff RS, Rodriguez L, Parry NM, Fox JG: Enteropathogenic Escherichia coli prevalence in laboratory rabbits. Vet Microbiol; 2013 May 3;163(3-4):395-8
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  • [Title] Enteropathogenic Escherichia coli prevalence in laboratory rabbits.
  • Rabbit-origin enteropathogenic Escherichia coli (EPEC) causes substantial diarrhea-associated morbidity and has zoonotic potential.
  • EPEC was isolated from 6/141 (4.3%) commercially-acquired laboratory rabbits.
  • Three of these did not have diarrhea or EPEC-typical intestinal lesions; they instead had background plasmacytic intestinal inflammation.
  • Asymptomatically infected rabbits may function as EPEC reservoirs.
  • [MeSH-major] Enteropathogenic Escherichia coli / physiology. Escherichia coli Infections / epidemiology
  • [MeSH-minor] Animals. Anti-Bacterial Agents / pharmacology. Disease Reservoirs. Escherichia coli Proteins / genetics. Female. Intestines / pathology. Prevalence. Rabbits. Serotyping. Virulence Factors / genetics

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  • [Copyright] Copyright © 2013 Elsevier B.V. All rights reserved.
  • (PMID = 23391439.001).
  • [ISSN] 1873-2542
  • [Journal-full-title] Veterinary microbiology
  • [ISO-abbreviation] Vet. Microbiol.
  • [Language] eng
  • [Grant] United States / NIH HHS / OD / T32 OD010978; United States / NCRR NIH HHS / RR / T32 RR007036; United States / NCRR NIH HHS / RR / T32 RR007036
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / Anti-Bacterial Agents; 0 / Escherichia coli Proteins; 0 / Virulence Factors
  • [Other-IDs] NLM/ NIHMS438439 [Available on 05/03/14]; NLM/ PMC3594591 [Available on 05/03/14]
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11. |||||||||. 92%  Asadi Karam M, Bouzari S, Oloomi M, Aslani M, Jafari A: Phenotypic and Genotypic Characterization of Enteropathogenic Escherichia coli (EPEC) strains in Tehran, Iran. Iran J Microbiol; 2010 Mar;2(1):3-7
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  • [Title] Phenotypic and Genotypic Characterization of Enteropathogenic Escherichia coli (EPEC) strains in Tehran, Iran.
  • BACKGROUND AND OBJECTIVES: Enteropathogenic Escherichia coli (EPEC) strains can be detected by serogrouping and the presence of enterocyte attaching- effacing (eae) gene.
  • Most EPEC strains belong to a certain O antigenic group.
  • The aim of the present study was to genetically characterize EPEC strains isolated from children with diarrhea.
  • MATERIALS AND METHODS: Serogrouping was performed by EPEC antisera in 321 E. coli isolates.
  • RESULTS: Seventeen (5.3%) isolates belonging to 7 EPEC serogroups were identified among the whole material and all carried the eae gene.
  • None of the 321 isolates showed presence of stx gene indicating that all 17 isolates classified as EPEC by O serogrouping did not belong to the enterohaemorrhagic E. coli (EHEC) group.
  • CONCLUSION: Serogrouping and detection of the eae gene showed to be reliable for detection of EPEC strains.
  • No Shigatoxin- producing E. coli (STEC) strain was found among the isolates.
  • Detection of the insertion site of LEE showed that selC, pheU or PheV are insertion sites of LEE in the EPEC strains.

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  • (PMID = 22347543.001).
  • [ISSN] 2008-4447
  • [Journal-full-title] Iranian journal of microbiology
  • [ISO-abbreviation] Iran J Microbiol
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Iran
  • [Other-IDs] NLM/ PMC3279762
  • [Keywords] NOTNLM ; Attaching- effacing / Intimin / Iran / Pathogenicity island / Serogrouping
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12. |||||||||. 90%  Tilak GP, Mudaliar JL: Role of enteropathogenic Escherichia coli in paediatric diarrhoeas in South India. Mater Sociomed; 2012;24(3):178-81
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  • [Title] Role of enteropathogenic Escherichia coli in paediatric diarrhoeas in South India.
  • BACKGROUND: Enteropathogenic Escherichia coli(EPEC) is a major cause of diarrhoea in children below 5 years of age.
  • Serotyping is classical method for identification of EPEC strains.
  • But serotypic markers are rarely sufficient to reliably identify the strains as Escherichia coli.
  • Introduction of PCR methodology which depends on detection of virulence factors has provided a practical and rapid way of detecting diarrhoeagenic Esch.coli.
  • Multiantibiotic resistant EPEC strains are a common phenomenon with world wide extension.
  • OBJECTIVES: To study the role of EPEC in Paediatric diarrhoea by both Serogrouping and Molecular characterisation by PCR and to analyse the antibiotic susceptabililty patterns of EPEC strains in our area.
  • Escherichia.coli isolates were identified by Microscopy, Culture and Biochemical reactions.
  • Among the Escherichia coli isolates, EPEC isolates were identified by Serogrouping.
  • Escherichia coli isolates were also subjected to Molecular characterisation by Multiplex PCR assay and those isolates which showed pathogenic genes were futher serotyped.
  • Antibiotic susceptibility pattern of EPEC isolates was determined by CLSI guidelines.
  • RESULTS: Among the Escherichia coli isolates 36.8% in the diarrhoeal group and none of them from the nondiarrhoeal group were identified as EPEC by serogrouping.
  • 73.3% of the EPEC isolates were below 2 years of age and no much difference in the sex distribution was observed.
  • In the diarrhoeal group 38.8% of Escherichia coli were EAEC and no other diarrhoeagenic Escherichia coli group was found by molecular characterisation.
  • In the nondiarrhoeal Escherichia coli strains, 46.6% showed EAEC genes.
  • Among the Escherichia coli isolates which agglutinated with EPEC polyvalent antisera, 33.3% were positive for Enteroaggregative genes. (Table 1).
  • CONCLUSION: EPEC is still an important pathogen in paediatric diarrhoeas.
  • O serogrouping can still be relied upon for detection of EPEC.
  • Dehydration is one of the clinical features of EPEC diarrhoea.

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  • (PMID = 23922527.001).
  • [ISSN] 1512-7680
  • [Journal-full-title] Materia socio-medica
  • [ISO-abbreviation] Mater Sociomed
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Bosnia and Hercegovina
  • [Other-IDs] NLM/ PMC3732348
  • [Keywords] NOTNLM ; Andhra Pradesh / Antibiotic Susceptibility / Enteroaggregative Escherchia coli (EAEC). / Enteropathogenic Escherichia coli (EPEC) / Molecular characterisation / Serotyping
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13. ||||||||.. 82%  Munera D, Crepin VF, Marches O, Frankel G: N-terminal type III secretion signal of enteropathogenic Escherichia coli translocator proteins. J Bacteriol; 2010 Jul;192(13):3534-9
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  • [Title] N-terminal type III secretion signal of enteropathogenic Escherichia coli translocator proteins.
  • We report that the N terminus of the type III secretion system translocator proteins EspB, EspD, and EspA mediate protein secretion and translocation from wild-type enteropathogenic Escherichia coli and hypersecretion from sepL and sepD mutants.
  • [MeSH-major] Enteropathogenic Escherichia coli / metabolism. Escherichia coli Proteins / metabolism

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  • (PMID = 20400543.001).
  • [ISSN] 1098-5530
  • [Journal-full-title] Journal of bacteriology
  • [ISO-abbreviation] J. Bacteriol.
  • [Language] eng
  • [Grant] United Kingdom / Wellcome Trust / /
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Bacterial Outer Membrane Proteins; 0 / EaeB protein, E coli; 0 / Escherichia coli Proteins; 0 / EspA protein, E coli; 0 / EspD protein, E coli; 0 / Map protein, E coli; 0 / Receptors, Cell Surface; 0 / SepD protein ,E coli; 0 / SepL protein, E coli; 0 / Tir protein, E coli
  • [Other-IDs] NLM/ PMC2897685
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14. ||||||||.. 82%  Vieira MA, Salvador FA, Silva RM, Irino K, Vaz TM, Rockstroh AC, Guth BE, Gomes TA: Prevalence and characteristics of the O122 pathogenicity island in typical and atypical enteropathogenic Escherichia coli strains. J Clin Microbiol; 2010 Apr;48(4):1452-5
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  • [Title] Prevalence and characteristics of the O122 pathogenicity island in typical and atypical enteropathogenic Escherichia coli strains.
  • The presence of the pathogenicity island (PAI) O122 genes, efa1 (lifA), sen, pagC, nleB, and nleE, in typical and atypical enteropathogenic Escherichia coli (EPEC) strains was investigated.
  • The simultaneous occurrence of all genes was statistically associated with diarrhea due to atypical EPEC.
  • Detection of the complete PAI O122 could aid in the identification of potential pathogenic strains of atypical EPEC.
  • [MeSH-major] DNA, Bacterial / genetics. Enteropathogenic Escherichia coli / genetics. Escherichia coli Infections / microbiology. Genomic Islands
  • [MeSH-minor] Diarrhea / microbiology. Escherichia coli Proteins / genetics. Humans. Prevalence. Virulence Factors / genetics

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  • (PMID = 20181917.001).
  • [ISSN] 1098-660X
  • [Journal-full-title] Journal of clinical microbiology
  • [ISO-abbreviation] J. Clin. Microbiol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / DNA, Bacterial; 0 / Escherichia coli Proteins; 0 / Virulence Factors
  • [Other-IDs] NLM/ PMC2849565
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15. ||||||||.. 80%  Zahavi EE, Lieberman JA, Donnenberg MS, Nitzan M, Baruch K, Rosenshine I, Turner JR, Melamed-Book N, Feinstein N, Zlotkin-Rivkin E, Aroeti B: Bundle-forming pilus retraction enhances enteropathogenic Escherichia coli infectivity. Mol Biol Cell; 2011 Jul 15;22(14):2436-47
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  • [Title] Bundle-forming pilus retraction enhances enteropathogenic Escherichia coli infectivity.
  • Enteropathogenic Escherichia coli (EPEC) is an important human pathogen that causes acute infantile diarrhea.
  • The type IV bundle-forming pili (BFP) of typical EPEC strains are dynamic fibrillar organelles that can extend out and retract into the bacterium.
  • How pili retraction contributes to EPEC pathogenesis at the cellular level remains largely obscure, however.
  • In this study, an effort has been made to address this question using engineered EPEC strains with induced BFP retraction capacity.
  • Our studies hence suggest novel insights into the involvement of pili retraction in EPEC pathogenesis.
  • [MeSH-major] Enteropathogenic Escherichia coli / pathogenicity. Escherichia coli Infections / microbiology. Fimbriae, Bacterial / physiology
  • [MeSH-minor] Animals. Arabinose / metabolism. Caco-2 Cells. Cell Line. Diarrhea, Infantile / microbiology. Dogs. Escherichia coli Proteins / genetics. Escherichia coli Proteins / metabolism. HeLa Cells. Humans. Infant, Newborn. Phosphorylation. Tyrosine / metabolism

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  • (PMID = 21613538.001).
  • [ISSN] 1939-4586
  • [Journal-full-title] Molecular biology of the cell
  • [ISO-abbreviation] Mol. Biol. Cell
  • [Language] eng
  • [Grant] United States / NIAID NIH HHS / AI / R01 AI037606; United States / NIAID NIH HHS / AI / T32 AI007540; United States / NIGMS NIH HHS / GM / T32 GM008181
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / BfpF protein, E coli; 0 / Escherichia coli Proteins; 42HK56048U / Tyrosine; B40ROO395Z / Arabinose
  • [Other-IDs] NLM/ PMC3135470
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16. |||||||||. 98%  Staples M, Doyle CJ, Graham RM, Jennison AV: Molecular epidemiological typing of enteropathogenic Escherichia coli strains from Australian patients. Diagn Microbiol Infect Dis; 2013 Mar;75(3):320-4
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Molecular epidemiological typing of enteropathogenic Escherichia coli strains from Australian patients.
  • Enteropathogenic Escherichia coli (EPEC) are an important cause of diarrhoea worldwide, particularly in children.
  • Sixty-one EPEC strains isolated from stool specimens of symptomatic persons from 2008 to 2011 were characterised for the prevalence of diarrhoea-associated putative virulence genes.
  • The EPEC isolates were highly heterogeneous, representing all 4 phylogenetic groups and comprising 59 MLVA types, 49 MLST types, and 43 serotypes.
  • This diversity is indicative of the complexity of the human enteric EPEC population, which may be either commensal or pathogenic.
  • [MeSH-major] Bacterial Typing Techniques / methods. Enteropathogenic Escherichia coli / genetics. Multilocus Sequence Typing / methods
  • [MeSH-minor] Australia / epidemiology. Diarrhea / microbiology. Escherichia coli Proteins / genetics. Feces / microbiology. Genes, Bacterial. Genetic Variation. Humans. Molecular Epidemiology. Phylogeny. Prevalence. Virulence Factors / genetics

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  • [Copyright] Crown Copyright © 2013. Published by Elsevier Inc. All rights reserved.
  • (PMID = 23357294.001).
  • [ISSN] 1879-0070
  • [Journal-full-title] Diagnostic microbiology and infectious disease
  • [ISO-abbreviation] Diagn. Microbiol. Infect. Dis.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Escherichia coli Proteins; 0 / NleB protein, E coli; 0 / Virulence Factors
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17. |||||||||. 95%  de Almeida PM, Arais LR, Andrade JR, Prado EH, Irino K, Cerqueira Ade M: Characterization of atypical Enteropathogenic Escherichia coli (aEPEC) isolated from dogs. Vet Microbiol; 2012 Aug 17;158(3-4):420-4
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Characterization of atypical Enteropathogenic Escherichia coli (aEPEC) isolated from dogs.
  • Enteropathogenic Escherichia coli (EPEC), an important human pathogen has the ability to form attaching and effacing lesions on the intestinal epithelium and has been isolated from a wide range of species.
  • Two EPEC subgroups are recognized: typical (tEPEC) and atypical (aEPEC) strains, differing by the presence of EAF plasmid and bundle-forming pilus (BFP) in typical strains and their absence in atypical strains.
  • This study searched the presence of EPEC strains in 101 fecal samples of diarrheic (n=65) and non-diarrheic (n=36) dogs from two cities in Rio de Janeiro State, Brazil.
  • Most isolates was FAS-positive and showed a localized adherence-like (LAL) in a 6h HeLa cell-adherence assay.
  • [MeSH-major] Dog Diseases / microbiology. Enteropathogenic Escherichia coli / genetics. Escherichia coli Infections / veterinary
  • [MeSH-minor] Adhesins, Bacterial / genetics. Animals. Bacterial Adhesion. Base Sequence. Brazil. Dogs. Escherichia coli Proteins / genetics. Feces / microbiology. Genotype. HeLa Cells. Humans. Serotyping

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  • [Copyright] Copyright © 2012. Published by Elsevier B.V.
  • (PMID = 22421111.001).
  • [ISSN] 1873-2542
  • [Journal-full-title] Veterinary microbiology
  • [ISO-abbreviation] Vet. Microbiol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / Adhesins, Bacterial; 0 / Escherichia coli Proteins
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18. |||||||||. 94%  Bustamante VH, Villalba MI, García-Angulo VA, Vázquez A, Martínez LC, Jiménez R, Puente JL: PerC and GrlA independently regulate Ler expression in enteropathogenic Escherichia coli. Mol Microbiol; 2011 Oct;82(2):398-415
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  • [Title] PerC and GrlA independently regulate Ler expression in enteropathogenic Escherichia coli.
  • Typical enteropathogenic Escherichia coli (EPEC) strains contain the EAF plasmid, which carries the perABC locus encoding PerC.
  • The precise role of PerC in EPEC virulence gene regulation has remained unclear, mainly because EPEC strains lacking the pEAF still express the LEE genes and because PerC is not present in other A/E pathogens such as Citrobacter rodentium.
  • Our results substantiate the role of PerC and GrlA in EPEC virulence gene regulation and suggest that these convergent regulatory mechanisms may have represented an evolutionary adaptation in EPEC to co-ordinate the expression of plasmid- and chromosome-encoded virulence factors needed to successfully colonize its intestinal niche.
  • [MeSH-major] Enteropathogenic Escherichia coli / metabolism. Escherichia coli Proteins / metabolism. Gene Expression Regulation, Bacterial. Trans-Activators / metabolism

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  • [Copyright] © 2011 Blackwell Publishing Ltd.
  • (PMID = 21895790.001).
  • [ISSN] 1365-2958
  • [Journal-full-title] Molecular microbiology
  • [ISO-abbreviation] Mol. Microbiol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Escherichia coli Proteins; 0 / GrlA protein, E coli; 0 / Ler protein, E coli; 0 / PerC protein, E coli; 0 / Trans-Activators
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19. ||||||||.. 82%  Nisa S, Hazen TH, Assatourian L, Nougayrède JP, Rasko DA, Donnenberg MS: In vitro evolution of an archetypal enteropathogenic Escherichia coli strain. J Bacteriol; 2013 Oct;195(19):4476-83
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] In vitro evolution of an archetypal enteropathogenic Escherichia coli strain.
  • Enteropathogenic Escherichia coli (EPEC) is a leading cause of infantile diarrhea in developing countries.
  • EPEC strain E2348/69 is used worldwide as a prototype to study EPEC genetics and disease.
  • To identify the genomic and phenotypic changes in the prototype strain, we sequenced the genome of the nalidixic acid-resistant (Nal(r)) E2348/69 clone.
  • We also sequenced a recent nleF mutant derived by one-step PCR mutagenesis from the Nal(r) strain.
  • The sequencing results revealed no unintended changes between the mutant and the parent strain.
  • This report reconciles longstanding inconsistencies in phenotypic properties of an archetypal strain and provides both reassurance and cautions regarding intentional and unintentional evolution in vitro.
  • [MeSH-major] Enteropathogenic Escherichia coli / genetics. Enteropathogenic Escherichia coli / metabolism

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  • (PMID = 23913321.001).
  • [ISSN] 1098-5530
  • [Journal-full-title] Journal of bacteriology
  • [ISO-abbreviation] J. Bacteriol.
  • [Language] eng
  • [Databank-accession-numbers] GENBANK/ ASZR00000000/ ASZS00000000
  • [Grant] United States / NIAID NIH HHS / AI / U19 AI090873; United States / NIAID NIH HHS / AI / U19AI090873
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] United States
  • [Other-IDs] NLM/ PMC3807458 [Available on 04/01/14]
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20. ||||||||.. 81%  Monaghan Á, Byrne B, Fanning S, Sweeney T, McDowell D, Bolton DJ: Serotypes and virulence profiles of atypical enteropathogenic Escherichia coli (EPEC) isolated from bovine farms and abattoirs. J Appl Microbiol; 2013 Feb;114(2):595-603
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  • [Title] Serotypes and virulence profiles of atypical enteropathogenic Escherichia coli (EPEC) isolated from bovine farms and abattoirs.
  • AIMS: The objective of this study was to examine the prevalence of enteropathogenic Escherichia coli (EPEC) on beef and dairy farms and in beef abattoirs and to characterize the isolates in terms of serogroup and virulence markers.
  • All 140 isolates obtained were atypical EPEC (aEPEC), while θ and β intimin types were common.
  • [MeSH-major] Cattle / microbiology. Enteropathogenic Escherichia coli / classification. Enteropathogenic Escherichia coli / pathogenicity

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  • [Copyright] © 2012 The Society for Applied Microbiology.
  • (PMID = 23163884.001).
  • [ISSN] 1365-2672
  • [Journal-full-title] Journal of applied microbiology
  • [ISO-abbreviation] J. Appl. Microbiol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Virulence Factors
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21. ||||||||.. 81%  Yan D, Quan H, Wang L, Liu F, Liu H, Chen J, Cao X, Ge B: Enteropathogenic Escherichia coli Tir recruits cellular SHP-2 through ITIM motifs to suppress host immune response. Cell Signal; 2013 Sep;25(9):1887-94
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  • [Title] Enteropathogenic Escherichia coli Tir recruits cellular SHP-2 through ITIM motifs to suppress host immune response.
  • The important diarrheal pathogen enteropathogenic Escherichia coli (EPEC) require delivery and insertion of the bacterial translocated intimin receptor (Tir) into the host plasma membrane for pedestal formation.
  • Here, we show that EPEC Tir suppresses the production of inflammatory cytokines by recruitment of SHP-2 and subsequent deubiquitination of TRAF6 in an ITIM dependent manner.
  • Our findings revealed a novel mechanism by which the EPEC utilize its ITIM motifs to suppress and evade the host innate immune response, which could lead to the development of novel therapeutics to prevent bacterial infection.
  • [MeSH-major] Enteropathogenic Escherichia coli / physiology. Escherichia coli Infections / immunology. Escherichia coli Proteins / immunology. Host-Parasite Interactions. Immunoreceptor Tyrosine-Based Activation Motif. Protein Tyrosine Phosphatase, Non-Receptor Type 11 / immunology. Receptors, Cell Surface / immunology

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  • [Copyright] Copyright © 2013 The Authors. Published by Elsevier Inc. All rights reserved.
  • (PMID = 23707390.001).
  • [ISSN] 1873-3913
  • [Journal-full-title] Cellular signalling
  • [ISO-abbreviation] Cell. Signal.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Cytokines; 0 / Escherichia coli Proteins; 0 / Receptors, Cell Surface; 0 / TNF Receptor-Associated Factor 6; 0 / Tir protein, E coli; EC 3.1.3.48 / Protein Tyrosine Phosphatase, Non-Receptor Type 11
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22. ||||||||.. 80%  Salinger N, Kokona B, Fairman R, Okeke IN: The plasmid-encoded regulator activates factors conferring lysozyme resistance on enteropathogenic Escherichia coli strains. Appl Environ Microbiol; 2009 Jan;75(1):275-80
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  • [Title] The plasmid-encoded regulator activates factors conferring lysozyme resistance on enteropathogenic Escherichia coli strains.
  • We demonstrate that enhanced lysozyme resistance of enteropathogenic Escherichia coli requires the plasmid-encoded regulator, Per, and is mediated by factors outside the locus for enterocyte effacement.
  • EspC, a Per-activated serine protease autotransporter protein, conferred enhanced resistance on nonpathogenic E. coli, and a second Per-regulated, espC-independent lysozyme resistance mechanism was identified.
  • [MeSH-major] Anti-Bacterial Agents / antagonists & inhibitors. Drug Resistance, Bacterial. Enteropathogenic Escherichia coli / drug effects. Enteropathogenic Escherichia coli / physiology. Muramidase / antagonists & inhibitors. Plasmids. Transcription Factors / metabolism
  • [MeSH-minor] Amino Acid Sequence. Escherichia coli Proteins / metabolism. Humans. Microbial Sensitivity Tests. Microbial Viability. Molecular Sequence Data

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  • (PMID = 18997020.001).
  • [ISSN] 1098-5336
  • [Journal-full-title] Applied and environmental microbiology
  • [ISO-abbreviation] Appl. Environ. Microbiol.
  • [Language] eng
  • [Grant] United States / Howard Hughes Medical Institute / /
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Anti-Bacterial Agents; 0 / Escherichia coli Proteins; 0 / EspC protein, E coli; 0 / Transcription Factors; EC 3.2.1.17 / Muramidase
  • [Other-IDs] NLM/ PMC2612221
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23. ||||||||.. 80%  Berger CN, Crepin VF, Jepson MA, Arbeloa A, Frankel G: The mechanisms used by enteropathogenic Escherichia coli to control filopodia dynamics. Cell Microbiol; 2009 Feb;11(2):309-22
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  • [Title] The mechanisms used by enteropathogenic Escherichia coli to control filopodia dynamics.
  • Enteropathogenic Escherichia coli (EPEC) subverts actin dynamics in eukaryotic cells by injecting effector proteins via a type III secretion system.
  • Then, following recovery from the filopodial signals, EPEC triggers robust actin polymerization via a signalling complex comprising Tir and the adaptor proteins Nck.
  • Recovery from the filopodial signals requires phosphorylation of a Tir tyrosine (Y474) residue and actin polymerization pathway as both infection of cells with EPEC expressing TirY474S or infection of Nck knockout cells with wild-type EPEC resulted in persistence of filopodia.
  • These results show that EPEC effectors modulate actin dynamics by temporal subverting the Rho GTPases and other actin polymerization pathways for the benefit of the adherent pathogen.
  • [MeSH-major] Enteropathogenic Escherichia coli / pathogenicity. Escherichia coli Proteins / metabolism. Pseudopodia / physiology. Virulence Factors / metabolism

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  • (PMID = 19046338.001).
  • [ISSN] 1462-5822
  • [Journal-full-title] Cellular microbiology
  • [ISO-abbreviation] Cell. Microbiol.
  • [Language] eng
  • [Grant] United Kingdom / Medical Research Council / / G0401551; United Kingdom / Medical Research Council / / ; United Kingdom / Wellcome Trust / /
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Actins; 0 / Adaptor Proteins, Signal Transducing; 0 / Cytoskeletal Proteins; 0 / Escherichia coli Proteins; 0 / Map protein, E coli; 0 / Nck protein; 0 / Oncogene Proteins; 0 / Phosphoproteins; 0 / Platelet Glycoprotein GPIb-IX Complex; 0 / Receptors, Cell Surface; 0 / Sodium-Hydrogen Antiporter; 0 / Tir protein, E coli; 0 / Virulence Factors; 0 / ezrin; 0 / sodium-hydrogen exchanger regulatory factor; EC 2.7.11.1 / rho-Associated Kinases; EC 3.6.5.2 / rhoA GTP-Binding Protein
  • [Other-IDs] NLM/ PMC2688667
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24. ||||||||.. 80%  Girard F, Dziva F, Stevens MP, Frankel G: Interactions of typical and atypical enteropathogenic Escherichia coli strains with the calf intestinal mucosa ex vivo. Appl Environ Microbiol; 2009 Sep;75(18):5991-5
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  • [Title] Interactions of typical and atypical enteropathogenic Escherichia coli strains with the calf intestinal mucosa ex vivo.
  • Enteropathogenic Escherichia coli (EPEC) can be found in healthy and diarrheic cattle; however, little is known about the role of attaching and effacing (A/E) lesion formation in colonization of bovine intestinal mucosa by such strains.
  • We show that typical and atypical EPEC induce A/E lesions on calf intestinal explants independently of Tir tyrosine phosphorylation and TccP.
  • Our data support the existence of conserved Tir- and TccP-independent mechanisms of A/E lesion formation in a range of hosts and reinforce the zoonotic potential of EPEC in cattle.
  • [MeSH-major] Bacterial Adhesion. Enteropathogenic Escherichia coli / pathogenicity. Intestinal Mucosa / microbiology
  • [MeSH-minor] Animals. Cattle. Escherichia coli Proteins / metabolism. Phosphorylation

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  • (PMID = 19633123.001).
  • [ISSN] 1098-5336
  • [Journal-full-title] Applied and environmental microbiology
  • [ISO-abbreviation] Appl. Environ. Microbiol.
  • [Language] eng
  • [Grant] United Kingdom / Biotechnology and Biological Sciences Research Council / /
  • [Publication-type] In Vitro; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Escherichia coli Proteins; 0 / tccP protein, E coli
  • [Other-IDs] NLM/ PMC2747864
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25. ||||||||.. 79%  Luo W, Donnenberg MS: Interactions and predicted host membrane topology of the enteropathogenic Escherichia coli translocator protein EspB. J Bacteriol; 2011 Jun;193(12):2972-80
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  • [Title] Interactions and predicted host membrane topology of the enteropathogenic Escherichia coli translocator protein EspB.
  • Translocator proteins include two hydrophobic proteins, represented in enteropathogenic Escherichia coli (EPEC) by EspB and EspD, which are thought to interact and form a pore in the host membrane.
  • Here we adapted a sequence motif recognized by a host kinase to demonstrate that residues on the carboxyl-terminal side of the EspB transmembrane domain are localized to the host cell cytoplasm.
  • [MeSH-major] Bacterial Outer Membrane Proteins / metabolism. Enteropathogenic Escherichia coli / metabolism. Escherichia coli Proteins / metabolism. Gene Expression Regulation, Bacterial / physiology

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  • (PMID = 21498649.001).
  • [ISSN] 1098-5530
  • [Journal-full-title] Journal of bacteriology
  • [ISO-abbreviation] J. Bacteriol.
  • [Language] eng
  • [Grant] United States / NIAID NIH HHS / AI / AI32074; United States / NIAID NIH HHS / AI / R01 AI032074
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Bacterial Outer Membrane Proteins; 0 / EaeB protein, E coli; 0 / Escherichia coli Proteins; 0 / EspA protein, E coli; 0 / EspD protein, E coli; EC 2.7.- / Phosphotransferases
  • [Other-IDs] NLM/ PMC3133209
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26. ||||||||.. 79%  Gouveia EM, Silva IS, Nakazato G, Araujo FR, Chang MR: Experimental infection with enteropathogenic Escherichia coli identified by PCR using enteric-coated capsules in boxer pups. Acta Cir Bras; 2011 Apr;26(2):144-8
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  • [Title] Experimental infection with enteropathogenic Escherichia coli identified by PCR using enteric-coated capsules in boxer pups.
  • PURPOSE: To verify the possibility of an experimental infection with enteropathogenic Escherichia coli and to confirm by PCR that the symptoms manifested after infection were due to the virulence factors of the studied bacteria.
  • One animal from each litter was included in a control group and the remaining animals were divided into two groups: one inoculated with strain 4083, and another one inoculated with strain SPA14.
  • Gelatinous capsules coated with enteric-coating solution were used for the inoculation of strains. E. coli isolation from feces was performed for all tested animals, and the extracted DNA was subjected to Polymerase Chain Reaction (PCR).
  • CONCLUSION: The efficiency of PCR for the studied strains indicates that this technique can be recommended for the diagnosis of enteropathogenic Escherichia coli as a differential from other pathogens causing diarrhea.
  • [MeSH-major] Enteropathogenic Escherichia coli / genetics. Escherichia coli Infections / microbiology. Polymerase Chain Reaction

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  • (PMID = 21445479.001).
  • [ISSN] 1678-2674
  • [Journal-full-title] Acta cirúrgica brasileira / Sociedade Brasileira para Desenvolvimento Pesquisa em Cirurgia
  • [ISO-abbreviation] Acta Cir Bras
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Brazil
  • [Chemical-registry-number] 0 / DNA, Bacterial; 0 / Virulence Factors
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27. ||||||||.. 79%  Smith K, Humphreys D, Hume PJ, Koronakis V: Enteropathogenic Escherichia coli recruits the cellular inositol phosphatase SHIP2 to regulate actin-pedestal formation. Cell Host Microbe; 2010 Jan 21;7(1):13-24
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  • [Title] Enteropathogenic Escherichia coli recruits the cellular inositol phosphatase SHIP2 to regulate actin-pedestal formation.
  • Adhesion of enteropathogenic Escherichia coli to epithelial cells triggers actin-rich pedestal formation beneath the bacteria.
  • [MeSH-major] Actins / metabolism. Enteropathogenic Escherichia coli / pathogenicity. Epithelial Cells / microbiology. Escherichia coli Proteins / physiology. Phosphoric Monoester Hydrolases / metabolism. Receptors, Cell Surface / physiology. Virulence Factors / physiology

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  • [Copyright] 2010 Elsevier Inc. All rights reserved.
  • [CommentIn] Cell Host Microbe. 2010 Jan 21;7(1):1-2 [20114020.001]
  • (PMID = 20114025.001).
  • [ISSN] 1934-6069
  • [Journal-full-title] Cell host & microbe
  • [ISO-abbreviation] Cell Host Microbe
  • [Language] eng
  • [Grant] United Kingdom / Medical Research Council / / G0500583; United Kingdom / Biotechnology and Biological Sciences Research Council / / ; United Kingdom / Medical Research Council / / ; United Kingdom / Wellcome Trust / /
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Actins; 0 / Carrier Proteins; 0 / Escherichia coli Proteins; 0 / Membrane Proteins; 0 / Phosphatidylinositols; 0 / RAPH1 protein, human; 0 / Receptors, Cell Surface; 0 / Shc Signaling Adaptor Proteins; 0 / Tir protein, E coli; 0 / Virulence Factors; EC 3.1.3.- / INPPL1 protein, human; EC 3.1.3.- / Phosphoric Monoester Hydrolases
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28. ||||||||.. 79%  Alonso MZ, Padola NL, Parma AE, Lucchesi PM: Enteropathogenic Escherichia coli contamination at different stages of the chicken slaughtering process. Poult Sci; 2011 Nov;90(11):2638-41
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  • [Title] Enteropathogenic Escherichia coli contamination at different stages of the chicken slaughtering process.
  • Enteropathogenic Escherichia coli is a foodborne pathogen that produces potentially fatal infant diarrhea, noticeably in developing countries.
  • The aim of this study was to detect EPEC contamination by PCR at different stages of the chicken slaughtering process.
  • Enteropathogenic Escherichia coli was detected in 6 to 28% of cloacal samples, 39 and 56% of unwashed eviscerated carcasses, and 4 to 58% of washed carcasses.
  • None of the samples were positive for bfpA, suggesting contamination with atypical EPEC.
  • The detection of EPEC at different stages of the chicken slaughtering process showed that the proportion of contaminated samples remained or even increased during processing.
  • [MeSH-major] Abattoirs. Enteropathogenic Escherichia coli / isolation & purification. Food Microbiology

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  • (PMID = 22010252.001).
  • [ISSN] 0032-5791
  • [Journal-full-title] Poultry science
  • [ISO-abbreviation] Poult. Sci.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
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29. ||||||||.. 78%  Moura RA, Sircili MP, Leomil L, Matté MH, Trabulsi LR, Elias WP, Irino K, Pestana de Castro AF: Clonal relationship among atypical enteropathogenic Escherichia coli strains isolated from different animal species and humans. Appl Environ Microbiol; 2009 Dec;75(23):7399-408
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  • [Title] Clonal relationship among atypical enteropathogenic Escherichia coli strains isolated from different animal species and humans.
  • Forty-nine typical and atypical enteropathogenic Escherichia coli (EPEC) strains belonging to different serotypes and isolated from humans, pets (cats and dogs), farm animals (bovines, sheep, and rabbits), and wild animals (monkeys) were investigated for virulence markers and clonal similarity by pulsed-field gel electrophoresis (PFGE) and multilocus sequence typing (MLST).
  • The virulence markers analyzed revealed that atypical EPEC strains isolated from animals have the potential to cause diarrhea in humans.
  • These results indicate that these animals act as atypical EPEC reservoirs and may represent sources of infection for humans.
  • Since humans also act as a reservoir of atypical EPEC strains, the cycle of mutual infection of atypical EPEC between animals and humans, mainly pets and their owners, cannot be ruled out since the transmission dynamics between the reservoirs are not yet clearly understood.
  • [MeSH-major] Animals, Domestic / microbiology. Animals, Wild / microbiology. Bacterial Typing Techniques. DNA Fingerprinting. DNA, Bacterial / genetics. Enteropathogenic Escherichia coli / classification. Enteropathogenic Escherichia coli / genetics
  • [MeSH-minor] Animals. Brazil. Cluster Analysis. Electrophoresis, Gel, Pulsed-Field. Escherichia coli Proteins / genetics. Genotype. Humans. Molecular Epidemiology. Molecular Sequence Data. Sequence Analysis, DNA. Serotyping. Virulence Factors / genetics

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  • (PMID = 19801470.001).
  • [ISSN] 1098-5336
  • [Journal-full-title] Applied and environmental microbiology
  • [ISO-abbreviation] Appl. Environ. Microbiol.
  • [Language] eng
  • [Databank-accession-numbers] GENBANK/ FJ561750/ FJ561751/ FJ561752/ FJ561753/ FJ561754/ FJ561755/ FJ561756/ FJ561757/ FJ561758/ FJ561759/ FJ561760/ FJ561761/ FJ561762/ FJ561763/ FJ561764/ FJ561765/ FJ561766/ FJ561767/ FJ561768/ FJ561769/ FJ561770/ FJ561771/ FJ561772/ FJ561773/ FJ561774/ FJ561775/ FJ561776/ FJ561777/ FJ561778/ FJ561779/ FJ561780/ FJ561781/ FJ561782/ FJ561783/ FJ561784/ FJ561785/ FJ561786/ FJ561787/ FJ561788/ FJ561789/ FJ561790/ FJ561791/ FJ561792/ FJ561793/ FJ561794/ FJ561795/ FJ561796/ FJ561797/ FJ561798/ FJ561799/ FJ561800/ FJ561801/ FJ561802/ FJ561803/ FJ561804/ FJ561805/ FJ561806/ FJ561807/ FJ561808/ FJ561809/ FJ561810/ FJ561811/ FJ561812/ FJ561813/ FJ561814/ FJ561815/ FJ561816/ FJ561817/ FJ561818/ FJ561819/ FJ561820/ FJ561821/ FJ561822/ FJ561823/ FJ561824/ FJ561825/ FJ561826/ FJ561827/ FJ561828/ FJ561829/ FJ561830/ FJ561831/ FJ561832/ FJ561833/ FJ561834/ FJ561835/ FJ561836/ FJ561837/ FJ561838/ FJ561839/ FJ561840/ FJ561841/ FJ561842/ FJ561843/ FJ561844/ FJ561845/ FJ561846/ FJ561847/ FJ561848/ FJ561849/ FJ561850/ FJ561851/ FJ561852/ FJ561853/ FJ561854/ FJ561855/ FJ561856/ FJ561857/ FJ561858/ FJ561859/ FJ561860/ FJ561861/ FJ561862/ FJ561863/ FJ561864/ FJ561865/ FJ561866/ FJ561867/ FJ561868/ FJ561869/ FJ561870/ FJ561871/ FJ561872/ FJ561873/ FJ561874/ FJ561875/ FJ561876/ FJ561877/ FJ561878/ FJ561879/ FJ561880/ FJ561881/ FJ561882/ FJ561883/ FJ561884/ FJ561885/ FJ561886/ FJ561887/ FJ561888/ FJ561889/ FJ561890/ FJ561891/ FJ561892/ FJ561893/ FJ561894/ FJ561895/ FJ561896/ FJ561897/ FJ561898/ FJ561899/ FJ561900/ FJ561901/ FJ561902/ FJ561903/ FJ561904/ FJ561905/ FJ561906/ FJ561907/ FJ561908/ FJ561909/ FJ561910/ FJ561911/ FJ561912/ FJ561913/ FJ561914/ FJ561915/ FJ561916/ FJ561917/ FJ561918/ FJ561919/ FJ561920/ FJ561921/ FJ561922/ FJ561923/ FJ561924/ FJ561925/ FJ561926/ FJ561927/ FJ561928/ FJ561929/ FJ561930/ FJ561931/ FJ561932/ FJ561933/ FJ561934/ FJ561935/ FJ561936/ FJ561937/ FJ561938/ FJ561939/ FJ561940/ FJ561941/ FJ561942/ FJ561943/ FJ561944/ FJ561945/ FJ561946/ FJ561947/ FJ561948/ FJ561949/ FJ561950/ FJ561951/ FJ561952/ FJ561953/ FJ561954/ FJ561955/ FJ561956/ FJ561957/ FJ561958/ FJ561959/ FJ561960/ FJ561961/ FJ561962/ FJ561963/ FJ561964/ FJ561965/ FJ561966/ FJ561967/ FJ561968/ FJ561969/ FJ561970/ FJ561971/ FJ561972/ FJ561973/ FJ561974/ FJ561975/ FJ561976/ FJ561977/ FJ561978/ FJ561979/ FJ561980/ FJ561981/ FJ561982/ FJ561983/ FJ561984/ FJ561985/ FJ561986/ FJ561987/ FJ561988/ FJ561989/ FJ561990/ FJ561991/ FJ561992/ FJ561993/ FJ561994/ FJ561995/ FJ561996/ FJ561997/ FJ561998/ FJ561999/ FJ562000/ FJ562001/ FJ562002/ FJ562003/ FJ562004/ FJ562005/ FJ562006/ FJ562007/ FJ562008/ FJ562009/ FJ562010/ FJ562011/ FJ562012/ FJ562013/ FJ562014/ FJ562015/ FJ562016/ FJ562017/ FJ562018/ FJ562019/ FJ562020/ FJ562021/ FJ562022/ FJ562023/ FJ562024/ FJ562025/ FJ562026/ FJ562027/ FJ562028/ FJ562029/ FJ562030/ FJ562031/ FJ562032/ FJ562033/ FJ562034/ FJ562035/ FJ562036/ FJ562037/ FJ562038/ FJ562039/ FJ562040/ FJ562041/ FJ562042/ FJ562043/ FJ562044/ FJ562045/ FJ562046/ FJ562047/ FJ562048/ FJ562049/ FJ562050/ FJ562051/ FJ562052/ FJ562053/ FJ562054/ FJ562055/ FJ562056/ FJ562057/ FJ562058/ FJ562059/ FJ562060/ FJ562061/ FJ562062/ FJ562063/ FJ562064/ FJ562065/ FJ562066/ FJ562067/ FJ562068/ FJ562069/ FJ562070/ FJ562071/ FJ562072/ FJ562073/ FJ562074/ FJ562075/ FJ562076/ FJ562077/ FJ562078/ FJ562079/ FJ562080/ FJ562081/ FJ562082/ FJ562083/ FJ562084/ FJ562085/ FJ562086/ FJ562087/ FJ562088/ FJ562089/ FJ562090/ FJ562091/ FJ562092
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / DNA, Bacterial; 0 / Escherichia coli Proteins; 0 / Virulence Factors
  • [Other-IDs] NLM/ PMC2786407
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30. ||||||||.. 78%  Flynn AN, Wang A, McKay DM, Buret AG: Apoptosis-inducing factor contributes to epithelial cell apoptosis induced by enteropathogenic Escherichia coli. Can J Physiol Pharmacol; 2011 Feb;89(2):143-8
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  • [Title] Apoptosis-inducing factor contributes to epithelial cell apoptosis induced by enteropathogenic Escherichia coli.
  • The mechanisms by which enteropathogenic Escherichia coli (EPEC) causes intestinal epithelial cell apoptosis remain unclear.
  • We tested the hypothesis that apoptosis-inducing factor (AIF) is involved in apoptosis induced by EPEC.
  • Infection of intestinal epithelial cells in vitro with EPEC led to the mitochondrial and cytosolic accumulation of AIF.
  • Knockdown of AIF with siRNA blocked cellular apoptosis in response to EPEC infection, as assessed by poly(ADP-ribose) polymerase cleavage and oligonucleosome formation.
  • Taken together, these data suggest that caspase-dependent mobilization of AIF contributes to EPEC-induced epithelial cell apoptosis.
  • [MeSH-major] Apoptosis / physiology. Apoptosis Inducing Factor / metabolism. Enteropathogenic Escherichia coli / physiology. Intestinal Mucosa / metabolism. Intestinal Mucosa / microbiology
  • [MeSH-minor] Caspases / metabolism. Cell Line. Cytosol / metabolism. Epithelial Cells / metabolism. Epithelial Cells / microbiology. Epithelial Cells / pathology. Escherichia coli Infections / metabolism. Escherichia coli Infections / microbiology. Escherichia coli Infections / pathology. Gene Knockdown Techniques. HeLa Cells. Humans. Mitochondria / metabolism. Poly(ADP-ribose) Polymerases / metabolism. RNA, Small Interfering / genetics

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  • (PMID = 21326346.001).
  • [ISSN] 1205-7541
  • [Journal-full-title] Canadian journal of physiology and pharmacology
  • [ISO-abbreviation] Can. J. Physiol. Pharmacol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Canada
  • [Chemical-registry-number] 0 / Apoptosis Inducing Factor; 0 / RNA, Small Interfering; EC 2.4.2.30 / Poly(ADP-ribose) Polymerases; EC 3.4.22.- / Caspases
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31. ||||||||.. 78%  Liu J, Wang WD, Liu YJ, Liu S, Zhou B, Zhu LW, Ji X, Sun Y, Feng SZ: Mice vaccinated with enteropathogenic Escherichia coli ghosts show significant protection against lethal challenges. Lett Appl Microbiol; 2012 Mar;54(3):255-62
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  • [Title] Mice vaccinated with enteropathogenic Escherichia coli ghosts show significant protection against lethal challenges.
  • AIM: To prepare enteropathogenic Escherichia coli (EPEC) E2348/69 ghosts and investigate whether immunization with EPEC bacterial ghosts can elicit protective immune responses.
  • An EPEC reference strain E2348/69 (serotype O127:H6) was transformed with the lysis plasmids to produce EPEC ghosts.
  • Mice injected with bacterial ghosts EGE (EPEC ghosts produced using lysis protein E) or EGES (EPEC ghosts produced using a combination of lysis protein E and SNA) gained weight normally and showed no clinical signs of disease.
  • Vaccination trials showed that mice immunized with EGE or EGES were significantly protected against subsequent challenge with the wild-type virulent parent strain, EPEC E2348/69 (42/50 and 45/50 survival, respectively); in contrast, none of the 30 control mice survived.
  • CONCLUSIONS: Immunization with EPEC ghosts can elicit protective immune responses in BALB/c mice.
  • SIGNIFICANCE AND IMPACT OF THE STUDY: EPEC ghosts may represent a promising new approach for vaccination against EPEC infection.
  • [MeSH-major] Bacterial Vaccines / immunology. Enteropathogenic Escherichia coli / immunology. Escherichia coli Infections / prevention & control

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  • [Copyright] © 2011 The Authors. Letters in Applied Microbiology © 2011 The Society for Applied Microbiology.
  • (PMID = 22211792.001).
  • [ISSN] 1472-765X
  • [Journal-full-title] Letters in applied microbiology
  • [ISO-abbreviation] Lett. Appl. Microbiol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Antibodies, Bacterial; 0 / Bacterial Vaccines; 0 / Immunoglobulin G
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32. ||||||||.. 78%  Quintero-Villegas MI, Aam BB, Rupnow J, Sørlie M, Eijsink VG, Hutkins RW: Adherence inhibition of enteropathogenic Escherichia coli by chitooligosaccharides with specific degrees of acetylation and polymerization. J Agric Food Chem; 2013 Mar 20;61(11):2748-54
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  • [Title] Adherence inhibition of enteropathogenic Escherichia coli by chitooligosaccharides with specific degrees of acetylation and polymerization.
  • Adherence of enteropathogenic Escherichia coli (EPEC) to the surface of a human HEp-2 cell line was determined in the absence or presence of the various CHOS fractions.
  • The results showed that all CHOS fractions inhibited adherence of EPEC to HEp-2 cells.

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  • (PMID = 23428168.001).
  • [ISSN] 1520-5118
  • [Journal-full-title] Journal of agricultural and food chemistry
  • [ISO-abbreviation] J. Agric. Food Chem.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
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33. ||||||||.. 78%  Horcajo P, Domínguez-Bernal G, Carrión J, De La Fuente R, Ruiz-Santa-Quiteria JA, Orden JA: Differences in virulence gene expression between atypical enteropathogenic Escherichia coli strains isolated from diarrheic and healthy ruminants. Can J Vet Res; 2013 Apr;77(2):158-60
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  • [Title] Differences in virulence gene expression between atypical enteropathogenic Escherichia coli strains isolated from diarrheic and healthy ruminants.
  • Differences in the pathogenicity of atypical enteropathogenic Escherichia coli (EPEC) strains may be due, at least partially, to different expression patterns of some virulence genes.
  • To investigate this hypothesis, the virulence gene expression patterns of 6 atypical EPEC strains isolated from healthy and diarrheic ruminants were compared using quantitative real-time reverse transcription polymerase chain reaction after growing the bacteria in culture medium alone or after binding it to HeLa epithelial cells.
  • Thus, the expression levels of some virulence genes may help determine which atypical EPEC strains cause diarrhea in ruminants.

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  • (PMID = 24082409.001).
  • [ISSN] 0830-9000
  • [Journal-full-title] Canadian journal of veterinary research = Revue canadienne de recherche vétérinaire
  • [ISO-abbreviation] Can. J. Vet. Res.
  • [Language] eng
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34. ||||||||.. 78%  Scaletsky IC, Souza TB, Aranda KR, Okeke IN: Genetic elements associated with antimicrobial resistance in enteropathogenic Escherichia coli (EPEC) from Brazil. BMC Microbiol; 2010;10:25
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  • [Title] Genetic elements associated with antimicrobial resistance in enteropathogenic Escherichia coli (EPEC) from Brazil.
  • BACKGROUND: We recently observed an association of resistance with a certain enteropathogenic Escherichia coli (EPEC) serotypes and identified a conjugative plasmid, similar to plasmid pED208, that was conserved among archival O111:H2/NM and O119:H2 strains of diverse geographical origin.
  • In this study, we sought to determine the prevalence and distribution of this plasmid among a collection of EPEC isolates from Brazil, as well as to study the susceptibilities of these isolates to antimicrobial agents.
  • RESULTS: Resistance was more commonly seen in typical EPEC than atypical strains.
  • Markers for the EPEC conjugative multiresistance plasmid, were detected in 21 (30%) of typical but only 4 (5%) of atypical strains (p = 0.001, Chi-squared test).
  • CONCLUSION: Our data suggest a limited but expanding host range for the EPEC resistance plasmid.
  • [MeSH-major] Enteropathogenic Escherichia coli / drug effects. Enteropathogenic Escherichia coli / genetics. Escherichia coli Infections / microbiology

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  • (PMID = 20105329.001).
  • [ISSN] 1471-2180
  • [Journal-full-title] BMC microbiology
  • [ISO-abbreviation] BMC Microbiol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Anti-Bacterial Agents
  • [Other-IDs] NLM/ PMC2828443
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35. ||||||||.. 77%  Magalhães CA, Rossato SS, Barbosa AS, Santos TO, Elias WP, Sircili MP, Piazza RM: The ability of haemolysins expressed by atypical enteropathogenic Escherichia coli to bind to extracellular matrix components. Mem Inst Oswaldo Cruz; 2011 Mar;106(2):146-52
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  • [Title] The ability of haemolysins expressed by atypical enteropathogenic Escherichia coli to bind to extracellular matrix components.
  • Typical and atypical enteropathogenic Escherichia coli (EPEC) are considered important bacterial causes of diarrhoea.
  • Considering the repertoire of virulence genes, atypical EPEC (aEPEC) is a heterogeneous group, harbouring genes that are found in other diarrheagenic E. coli pathotypes, such as those encoding haemolysins.
  • [MeSH-major] Enteropathogenic Escherichia coli / physiology. Escherichia coli Proteins / physiology. Extracellular Matrix / metabolism

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  • (PMID = 21537672.001).
  • [ISSN] 1678-8060
  • [Journal-full-title] Memórias do Instituto Oswaldo Cruz
  • [ISO-abbreviation] Mem. Inst. Oswaldo Cruz
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Brazil
  • [Chemical-registry-number] 0 / Escherichia coli Proteins; 0 / Hemolysin Proteins; 0 / Virulence Factors; 0 / enterohemolysin
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36. ||||||||.. 77%  Roxas JL, Wilbur JS, Zhang X, Martinez G, Vedantam G, Viswanathan VK: The enteropathogenic Escherichia coli-secreted protein EspZ inhibits host cell apoptosis. Infect Immun; 2012 Nov;80(11):3850-7
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  • [Title] The enteropathogenic Escherichia coli-secreted protein EspZ inhibits host cell apoptosis.
  • The diarrheagenic pathogen enteropathogenic Escherichia coli (EPEC) limits the death of infected enterocytes early in infection.
  • A number of bacterial molecules and host signaling pathways contribute to the enhanced survival of EPEC-infected host cells.
  • EspZ, a type III secreted effector protein that is unique to EPEC and related "attaching and effacing" (A/E) pathogens, plays a role in limiting host cell death, but the precise host signaling pathways responsible for this phenotype are not known.
  • Consistent with previous studies, scanning electron microscopy analysis of intestinal epithelial cells infected with an EPEC espZ mutant (ΔespZ) showed increased levels of apoptotic and necrotic cells compared to cells infected with the isogenic parent strain.
  • Correspondingly, higher levels of cytosolic cytochrome c and increased activation of caspases 9, 7, and 3 were observed for ΔespZ strain-infected cells compared to wild-type (WT) EPEC-infected cells.
  • [MeSH-major] Apoptosis / physiology. Enteropathogenic Escherichia coli / metabolism. Epithelial Cells / metabolism. Escherichia coli Proteins / metabolism. Intestines / metabolism

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  • [Cites] Infect Immun. 2001 Jul;69(7):4580-9 [11402002.001]
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  • (PMID = 22907816.001).
  • [ISSN] 1098-5522
  • [Journal-full-title] Infection and immunity
  • [ISO-abbreviation] Infect. Immun.
  • [Language] eng
  • [Grant] United States / NIAID NIH HHS / AI / 1R01AI081742; United States / NIAID NIH HHS / AI / R01 AI081742
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Escherichia coli Proteins; 0 / EspZ protein, E coli
  • [Other-IDs] NLM/ PMC3486051
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37. ||||||||.. 77%  Al Hilali SA, Almohana AM: Occurrence and molecular characterization of enteropathogenic Escherichia coli serotypes isolated from children with diarrhoea in Najaf, Iraq. Indian J Med Microbiol; 2011 Oct-Dec;29(4):383-8
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  • [Title] Occurrence and molecular characterization of enteropathogenic Escherichia coli serotypes isolated from children with diarrhoea in Najaf, Iraq.
  • PURPOSE: Enteropathogenic Escherichia coli (EPEC) are among the most important pathogens infecting children worldwide and are one of the main causes of diarrhoea.
  • The study was carried out to investigate the occurrence of EPEC as a cause of infectious diarrhoea in children younger than 2 years of age and characterize their virulence genes.
  • MATERIALS AND METHODS: During the study period, a total of 656 faecal specimens from children with diarrhoea and 54 from healthy children were analyzed. E. coli isolates were serotypically identified with EPEC polyvalent and monovalent antisera.
  • The isolated EPEC were examined for the presence of the attaching and effacing (eaeA), bundle-forming pilus (bfpA), Shiga like toxins (stx₁ and stx₂), enterohaemorrhagic E. coli enterohaemolysin (EHEC hlyA) and EPEC adherence factor (EAF) genes by the PCR assay.
  • RESULTS: The study has shown that 22 (3.4%) had diarrhoea due to EPEC, while no EPEC isolates were detected in asymptomatic children.
  • The highest number of the EPEC isolated belonging to polyvalent 2.
  • The primers encoding virulence genes were subjected to all the EPEC isolates.
  • Typical EPEC (eaeA⁺, bfpA⁺) was diagnosed in two isolates, while, atypical EPEC was manifested in four isolates.
  • CONCLUSIONS: According to the results, the frequency of EPEC isolates in Najaf was lower than what has been suspected and the investigation including the use of molecular technique and serotyping, are necessary to allow precise identification and epidemiological study of these pathogens.
  • [MeSH-major] Enteropathogenic Escherichia coli / classification. Enteropathogenic Escherichia coli / isolation & purification. Escherichia coli Infections / microbiology. Virulence Factors / genetics
  • [MeSH-minor] Escherichia coli Proteins / genetics. Feces / microbiology. Genotype. Humans. Infant. Iraq. Polymerase Chain Reaction. Serotyping

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  • (PMID = 22120799.001).
  • [ISSN] 1998-3646
  • [Journal-full-title] Indian journal of medical microbiology
  • [ISO-abbreviation] Indian J Med Microbiol
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] India
  • [Chemical-registry-number] 0 / Escherichia coli Proteins; 0 / Virulence Factors
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38. ||||||||.. 77%  Srikhanta YN, Hocking DM, Praszkier J, Wakefield MJ, Robins-Browne RM, Yang J, Tauschek M: RegR virulence regulon of rabbit-specific enteropathogenic Escherichia coli strain E22. Infect Immun; 2013 Apr;81(4):1078-89
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  • [Title] RegR virulence regulon of rabbit-specific enteropathogenic Escherichia coli strain E22.
  • AraC-like regulators play a key role in the expression of virulence factors in enteric pathogens, such as enteropathogenic Escherichia coli (EPEC), enterotoxigenic E. coli, enteroaggregative E. coli, and Citrobacter rodentium.
  • Bioinformatic analysis of the genome of rabbit-specific EPEC (REPEC) strain E22 (O103:H2) revealed the presence of a gene encoding an AraC-like regulatory protein, RegR, which shares 71% identity to the global virulence regulator, RegA, of C. rodentium.
  • Taken together, our findings indicate that RegR controls the expression of a series of accessory adhesins that significantly enhance the virulence of REPEC strain E22.
  • [MeSH-major] Bacterial Proteins / metabolism. Enteropathogenic Escherichia coli / genetics. Enteropathogenic Escherichia coli / pathogenicity. Gene Expression Regulation, Bacterial. Regulon. Transcription Factors / metabolism. Virulence Factors / biosynthesis
  • [MeSH-minor] Animals. Computational Biology. DNA, Bacterial / metabolism. Disease Models, Animal. Electrophoretic Mobility Shift Assay. Escherichia coli Infections / microbiology. Escherichia coli Infections / pathology. Escherichia coli Proteins / analysis. Fimbriae Proteins / biosynthesis. Fimbriae Proteins / genetics. Gene Expression Profiling. Microarray Analysis. Promoter Regions, Genetic. Protein Binding. Proteome / analysis. Rabbits. Transcription, Genetic

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  • (PMID = 23340312.001).
  • [ISSN] 1098-5522
  • [Journal-full-title] Infection and immunity
  • [ISO-abbreviation] Infect. Immun.
  • [Language] eng
  • [Databank-accession-numbers] GEO/ GSE41093
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Bacterial Proteins; 0 / DNA, Bacterial; 0 / Escherichia coli Proteins; 0 / Proteome; 0 / RegR protein, bacteria; 0 / Transcription Factors; 0 / Virulence Factors; 147680-16-8 / Fimbriae Proteins
  • [Other-IDs] NLM/ PMC3639590
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39. ||||||||.. 77%  Contreras CA, Ochoa TJ, Ruiz J, Lacher DW, Durand D, DebRoy C, Lanata CF, Cleary TG: Genetic diversity of locus of enterocyte effacement genes of enteropathogenic Escherichia coli isolated from Peruvian children. J Med Microbiol; 2012 Aug;61(Pt 8):1114-20
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  • [Title] Genetic diversity of locus of enterocyte effacement genes of enteropathogenic Escherichia coli isolated from Peruvian children.
  • The aim of this study was to determine the frequency and allele associations of locus of enterocyte effacement encoded esp and tir genes among 181 enteropathogenic Escherichia coli (EPEC) strains (90 diarrhoea-associated and 91 controls) isolated from Peruvian children under 18 months of age.
  • EPEC strains were isolated with higher frequency from healthy controls (91/424, 21.7%) than from diarrhoeal samples (90/936, 9.6%) (P<0.001); 28.9% of diarrhoeal and 17.6% of control samples were typical EPEC (tEPEC).
  • The distribution of espA alleles (alpha, beta, beta2 and gamma) and espD alleles (alpha, beta, gamma and a new variant, espD-N1) between tEPEC and atypical EPEC (aEPEC) was significantly different (P<0.05).
  • Our findings indicate that there is a high degree of heterogeneity among EPEC strains isolated from Peruvian children and that aEPEC and tEPEC variants cluster.
  • [MeSH-major] Enteropathogenic Escherichia coli / genetics. Enteropathogenic Escherichia coli / isolation & purification. Escherichia coli Infections / epidemiology. Escherichia coli Infections / microbiology. Escherichia coli Proteins / genetics. Genetic Variation. Phosphoproteins / genetics

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  • (PMID = 22493278.001).
  • [ISSN] 1473-5644
  • [Journal-full-title] Journal of medical microbiology
  • [ISO-abbreviation] J. Med. Microbiol.
  • [Language] eng
  • [Databank-accession-numbers] GENBANK/ JN571730/ JN571731/ JN571732/ JN571733
  • [Grant] United States / FIC NIH HHS / TW / 1K01TW007405; United States / NICHD NIH HHS / HD / R01-HD051716
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / DNA, Bacterial; 0 / Escherichia coli Proteins; 0 / LEE protein, E coli; 0 / Phosphoproteins
  • [Other-IDs] NLM/ PMC3542133
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40. ||||||||.. 77%  Pitondo-Silva A, Minarini LA, Camargo IL, Darini AL: Clonal relationships determined by multilocus sequence typing among enteropathogenic Escherichia coli isolated in Brazil. Can J Microbiol; 2009 Jun;55(6):672-9
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Clonal relationships determined by multilocus sequence typing among enteropathogenic Escherichia coli isolated in Brazil.
  • Enteropathogenic Escherichia coli (EPEC) infections are a leading cause of infantile diarrhea in developing nations.
  • Little is known about strains of EPEC analyzed by MLST from Brazil.
  • In this study, a diverse collection of 29 EPEC strains isolated from patients with diarrhea, admitted to the University Hospital of Ribeirao Preto, was characterized by MLST.
  • Strain analysis demonstrated 22 different sequence types (STs), of which almost half (48%) were new, indicating a high genotype diversity.
  • It was not possible to correlate typical and atypical EPEC with other strains in the MLST database.
  • This is the first study that analyzed EPEC strains from South America that are included in the E. coli MLST database.
  • Nine (31%) out of 29 strains are part of the CC10 clonal complex, the major clonal complex in the database, which comprises 174 strains and 86 different STs, suggesting that these strains might be the most important intestinal pathogenic E. coli worldwide.
  • Genetic relationships between typical and atypical EPEC, enterohemorrhagic E. coli, and enteroaggregative E. coli strains were not established by MLST.
  • [MeSH-major] Diarrhea / microbiology. Enteropathogenic Escherichia coli / isolation & purification. Escherichia coli Infections / microbiology

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  • (PMID = 19767837.001).
  • [ISSN] 1480-3275
  • [Journal-full-title] Canadian journal of microbiology
  • [ISO-abbreviation] Can. J. Microbiol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Canada
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41. ||||||||.. 77%  Iida M, Okamura N, Yamazaki M, Yatsuyanagi J, Kurazono T, Suzuki R, Hiruta N, Isobe J, Seto K, Kawano K, Narimatsu H, Ratchtrachenchai OA, Okabe N, Ito K: Classification of perA sequences and their correlation with autoaggregation in typical enteropathogenic Escherichia coli isolates collected in Japan and Thailand. Microbiol Immunol; 2010 Apr;54(4):184-95
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Classification of perA sequences and their correlation with autoaggregation in typical enteropathogenic Escherichia coli isolates collected in Japan and Thailand.
  • Enteropathogenic Escherichia coli (EPEC) strains produce a bundle-forming pilus (BFP) that mediates localized adherence (LA) to intestinal epithelial cells.
  • We analyzed perA gene polymorphism among typical (eae- and bfpA-positive) EPEC strains isolated from healthy and diarrheal persons in Japan (n=27) and Thailand (n=26) during the period 1995 to 2007 and compared this with virulence and phenotypic characteristics.
  • Our results showed that functional deficiency due to frameshift mutation and subsequent nonsense mutation in perA reduced BFP expression in typical EPEC strains isolated in Japan.
  • [MeSH-major] Enteropathogenic Escherichia coli / classification. Escherichia coli Proteins. Repressor Proteins
  • [MeSH-minor] Amino Acid Sequence. Bacterial Adhesion. Cell Line. Diarrhea / microbiology. Epithelial Cells / microbiology. Escherichia coli Infections / microbiology. Fimbriae Proteins / chemistry. Fimbriae Proteins / genetics. Fimbriae Proteins / metabolism. Gene Expression Regulation, Bacterial. Hemolysis. Heteroduplex Analysis. Humans. Japan. Molecular Sequence Data. Mutation. Phylogeny. Polymorphism, Genetic. Sequence Analysis, DNA. Serotyping. Thailand. Virulence

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  • (PMID = 20377747.001).
  • [ISSN] 0385-5600
  • [Journal-full-title] Microbiology and immunology
  • [ISO-abbreviation] Microbiol. Immunol.
  • [Language] eng
  • [Databank-accession-numbers] GENBANK/ AB364243/ AB364244/ AB523678/ AB523679/ AB523680/ AB523681/ AB523682/ AB523683/ AB523684/ AB523685/ AB523686/ AB523687/ AB523688/ AB523689/ AB523690/ AB523691/ AB523692/ AB523693/ AB523694/ AB523695/ AB523696/ AB523697/ AB523698/ AB523699/ AB523700/ AB523701/ AB523702
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Japan
  • [Chemical-registry-number] 0 / BfpA protein, E coli; 0 / Escherichia coli Proteins; 0 / Repressor Proteins; 0 / bfpT protein, E coli; 147680-16-8 / Fimbriae Proteins
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42. ||||||||.. 77%  Sakkejha H, Byrne L, Lawson AJ, Jenkins C: An update on the microbiology and epidemiology of enteropathogenic Escherichia coli in England 2010-2012. J Med Microbiol; 2013 Oct;62(Pt 10):1531-4
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  • [Title] An update on the microbiology and epidemiology of enteropathogenic Escherichia coli in England 2010-2012.
  • Historically, enteropathogenic Escherichia coli (EPEC) are a well-known cause of outbreaks of infantile diarrhoea associated with morbidity and mortality in England.
  • The aim of this study was to provide an update on the microbiology and epidemiology of strains of EPEC in England between 2010 and 2012.
  • A wide range of E. coli serogroups were identified, with the most common being E. coli O145, O49 and O157.
  • Few isolates (9%) had additional virulence factors (specifically bfp, vtx2f and espT genes) and the majority were classified as atypical EPEC.
  • No outbreaks were reported during this period; however, the data indicated that EPEC are still an important cause of sporadic cases of infantile diarrhoea in England.
  • [MeSH-major] Enteropathogenic Escherichia coli / isolation & purification. Escherichia coli Infections / epidemiology. Escherichia coli Infections / microbiology

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  • (PMID = 23893920.001).
  • [ISSN] 1473-5644
  • [Journal-full-title] Journal of medical microbiology
  • [ISO-abbreviation] J. Med. Microbiol.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Virulence Factors
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43. ||||||||.. 77%  Bouzari S, Aslani MM, Oloomi M, Jafari A, Dashti A: Comparison of multiplex PCR with serogrouping and PCR-RFLP of fliC gene for the detection of enteropathogenic Escherichia coli (EPEC). Braz J Infect Dis; 2011 Jul-Aug;15(4):365-9
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  • [Title] Comparison of multiplex PCR with serogrouping and PCR-RFLP of fliC gene for the detection of enteropathogenic Escherichia coli (EPEC).
  • Enteropathogenic Escherichia coli (EPEC) comprise one of the six categories of diarrhoeagenic E. coli (DEC).
  • EPEC is subgrouped into typical (tEPEC) and atypical (aEPEC).
  • The identification of DEC cannot be based only on cultural and biochemical criteria, since they are indistinguishable from the non-pathogenic E. coli commonly found in human feces.
  • In the present study five hundred E. coli isolates from children with diarrhea were subjected into multiplex PCR.
  • The results obtained revealed that overall 41 (8.2%) isolates could be detected as EPEC by this multiplex PCR assay.
  • Of these isolates; 27 (66%) were typical (escv+, bfp+) and 14 (34%) atypical EPEC (escv+, bfp-).
  • [MeSH-major] DNA, Bacterial / analysis. Enteropathogenic Escherichia coli / classification. Escherichia coli Proteins / genetics. Multiplex Polymerase Chain Reaction. O Antigens / analysis. Polymorphism, Restriction Fragment Length

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  • (PMID = 21861008.001).
  • [ISSN] 1678-4391
  • [Journal-full-title] The Brazilian journal of infectious diseases : an official publication of the Brazilian Society of Infectious Diseases
  • [ISO-abbreviation] Braz J Infect Dis
  • [Language] eng
  • [Publication-type] Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Brazil
  • [Chemical-registry-number] 0 / DNA, Bacterial; 0 / Escherichia coli Proteins; 0 / FliC protein, E coli; 0 / O Antigens; 0 / Shiga Toxin 1; 0 / Shiga Toxin 2
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44. ||||||||.. 77%  Nakhjavani FA, Emaneini M, Hosseini H, Iman-Eini H, Aligholi M, Jabalameli F, Haghi-Ashtiani MT, Taherikalani M, Mirsalehian A: Molecular analysis of typical and atypical enteropathogenic Escherichia coli (EPEC) isolated from children with diarrhoea. J Med Microbiol; 2013 Feb;62(Pt 2):191-5
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  • [Title] Molecular analysis of typical and atypical enteropathogenic Escherichia coli (EPEC) isolated from children with diarrhoea.
  • To investigate the incidence, antimicrobial resistance and genetic relationships of enteropathogenic Escherichia coli (EPEC) in children with diarrhoea, a total of 612 stool specimens were collected in Tehran, Iran, and cultured to isolate strains of EPEC.
  • The genetic relationships between EPEC isolates were determined by pulsed-field gel electrophoresis (PFGE).
  • Out of the 412 strains of E. coli obtained from 612 diarrhoeal stool specimens, 23 (5.6 %) were identified as EPEC, of which seven (30.4 %) were classified as typical strains of EPEC and 16 (69.6 %) were classified as atypical.
  • Out of the 23 EPEC isolates, 69.5 % were resistant to ampicillin, 39.1 % were resistant to tetracycline and cotrimoxazole, 30.4 % were resistant to cefpodoxime, ceftazidime, ceftriaxone and aztreonam, and 26.1 % were resistant to imipenem.
  • The present study shows that typical and atypical EPEC isolates displayed considerable heterogeneity in PFGE profiles and EPEC infections were only sporadic in Tehran.
  • [MeSH-major] Diarrhea / microbiology. Drug Resistance, Multiple, Bacterial. Enteropathogenic Escherichia coli / drug effects. Escherichia coli Infections / microbiology. Feces / microbiology
  • [MeSH-minor] Adhesins, Bacterial / genetics. Child. Child, Preschool. DNA, Bacterial / genetics. Escherichia coli Proteins / genetics. Fimbriae Proteins / genetics. Humans. Infant. Microbial Sensitivity Tests. Shiga Toxin / genetics

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  • (PMID = 23065543.001).
  • [ISSN] 1473-5644
  • [Journal-full-title] Journal of medical microbiology
  • [ISO-abbreviation] J. Med. Microbiol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Adhesins, Bacterial; 0 / BfpA protein, E coli; 0 / DNA, Bacterial; 0 / Escherichia coli Proteins; 147094-99-3 / eaeA protein, E coli; 147680-16-8 / Fimbriae Proteins; 75757-64-1 / Shiga Toxin
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45. ||||||||.. 77%  Reis RS, Horn F: Enteropathogenic Escherichia coli, Samonella, Shigella and Yersinia: cellular aspects of host-bacteria interactions in enteric diseases. Gut Pathog; 2010;2(1):8
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  • [Title] Enteropathogenic Escherichia coli, Samonella, Shigella and Yersinia: cellular aspects of host-bacteria interactions in enteric diseases.
  • A successful infection of the human intestine by enteropathogenic bacteria depends on the ability of bacteria to attach and colonize the intestinal epithelium and, in some cases, to invade the host cell, survive intracellularly and disseminate from cell to cell.
  • Here we overview the most important molecular strategies developed by enteropathogenic Escherichia coli, Salmonella enterica, Shigella flexneri, and Yersinia enterocolitica to cause enteric infections.

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  • (PMID = 20649986.001).
  • [ISSN] 1757-4749
  • [Journal-full-title] Gut pathogens
  • [ISO-abbreviation] Gut Pathog
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Other-IDs] NLM/ PMC2921366
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46. ||||||||.. 77%  Malik A, Tóth I, Nagy B: Colonisation of conventional weaned pigs by enteropathogenic Escherichia coli (EPEC) and its hazard potential for human health. Acta Vet Hung; 2012 Sep;60(3):297-307
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  • [Title] Colonisation of conventional weaned pigs by enteropathogenic Escherichia coli (EPEC) and its hazard potential for human health.
  • Enteropathogenic Escherichia coli (EPEC) bacteria frequently cause severe enteric diseases primarily in children and in young rabbits.
  • Their pathogenicity for pigs has been tested by oral infection of colostrum-deprived newborn, and of severely immunosuppressed weaned pigs, but colonisation of conventional weaned pigs by porcine EPEC has not been experimentally studied.
  • EPEC show similarities to enterohaemorrhagic E. coli (EHEC) additionally carrying shiga toxin genes integrated into the chromosome by lambdoid phages.
  • We have demonstrated earlier that the porcine EPEC prototype strain P86-1390 (O45) could be transduced in vivo (in ligated loops of weaned pigs), by Stx2 phage derived from a human EHEC.
  • Thus, the ability of this porcine EPEC strain to colonise conventional weaned pigs under farming conditions became a question of relevance to human health.
  • The EPEC P86-1390 and other well-characterised porcine EPEC strains were applied to 54 pigs, leaving 41 weaned pigs as negative controls.
  • In three experiments moderate predispositions were applied: coinfections with enterotoxigenic E. coli (ETEC) or with low-virulence TGE coronavirus, application of fumonisin B1 with a normal therapeutic dose of dexamethasone, and the increase of soybean protein concentration in the feed.
  • A total of 41 weaned pigs served as negative controls inoculated with a commensal porcine E. coli.
  • As an overall result, ileal segments of 18.5% of infected pigs were shown to be colonised by EPEC, while no EPEC were detected in the ilea of controls.
  • Among predisposing factors occurring on farms, feed protein content increased by 20% (26.3% crude protein, provided by 48% soybean meal) seemed to enhance EPEC colonisation and resulted in the mobilisation of spontaneous latent EPEC/ETEC infection.
  • The results indicate that under normal farm conditions porcine EPEC may colonise conventional weaned pigs by inducing ileal attaching effacing (AE) lesions with reasonable frequency, without clinical signs.
  • The results also suggest that conventional weaned pigs may represent undetected reservoirs of porcine EPEC, potentially giving rise to the emergence of new types of EHEC due to natural transduction by Stx phages.

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  • (PMID = 22903075.001).
  • [ISSN] 0236-6290
  • [Journal-full-title] Acta veterinaria Hungarica
  • [ISO-abbreviation] Acta Vet. Hung.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Hungary
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47. ||||||||.. 77%  Burgos YK, Pries K, Pestana de Castro AF, Beutin L: Characterization of the alpha-haemolysin determinant from the human enteropathogenic Escherichia coli O26 plasmid pEO5. FEMS Microbiol Lett; 2009 Mar;292(2):194-202
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  • [Title] Characterization of the alpha-haemolysin determinant from the human enteropathogenic Escherichia coli O26 plasmid pEO5.
  • The 157-kb conjugative plasmid pEO5 encoding alpha-haemolysin in strains of human enteropathogenic Escherichia coli (EPEC) O26 was investigated for its relationship with EHEC-haemolysin-encoding plasmids of enterohaemorrhagic E. coli (EHEC) O26 and O157 strains.
  • Plasmid pEO5 was found to be compatible with EHEC-virulence plasmids and did not hybridize in Southern blots with plasmid pO157 from the EHEC O157:H7 strain EDL933, indicating that both plasmids were unrelated.
  • The alpha-hly determinant of pEO5 (7252 bp) and its upstream region was most similar to corresponding sequences of the murine E. coli alpha-hly plasmid pHly152, in particular, the structural alpha-hlyCABD genes (99.2% identity) and the regulatory hlyR regions (98.8% identity).
  • pEO5 and alpha-hly plasmids of EPEC O26 strains from humans and cattle were very similar for the regions encompassing the structural alpha-hlyCABD genes.
  • [MeSH-major] Enteropathogenic Escherichia coli / genetics. Escherichia coli Proteins / genetics. Hemolysin Proteins / genetics. Plasmids

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  • (PMID = 19175679.001).
  • [ISSN] 1574-6968
  • [Journal-full-title] FEMS microbiology letters
  • [ISO-abbreviation] FEMS Microbiol. Lett.
  • [Language] eng
  • [Databank-accession-numbers] GENBANK/ FM180012
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / DNA Transposable Elements; 0 / Escherichia coli Proteins; 0 / Hemolysin Proteins
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48. ||||||||.. 77%  Liberatore AM, Moreira FC, Gomes TA, Menchaca-Diaz JL, Koh IH: Typical and atypical enteropathogenic Escherichia coli bacterial translocation associated with tissue hypoperfusion in rats. Braz J Med Biol Res; 2011 Oct;44(10):1018-24
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  • [Title] Typical and atypical enteropathogenic Escherichia coli bacterial translocation associated with tissue hypoperfusion in rats.
  • Although enteropathogenic Escherichia coli (EPEC) are well-recognized diarrheal agents, their ability to translocate and cause extraintestinal alterations is not known.
  • We investigated whether a typical EPEC (tEPEC) and an atypical EPEC (aEPEC) strain translocate and cause microcirculation injury under conditions of intestinal bacterial overgrowth.
  • Recovery of the positive control E. coli R-6 (N = 6) was 100% for all compartments.
  • Bacteria were not recovered from extraintestinal sites of controls inoculated with non-pathogenic E. coli strains HB101 (N = 6) and HS (N = 10), or saline.
  • Mesenteric microcirculation injuries were detected with both EPEC strains, but only aEPEC was similar to E. coli R-6 with regard to systemic tissue hypoperfusion.
  • [MeSH-major] Bacterial Translocation / physiology. Enteropathogenic Escherichia coli / physiology. Escherichia coli Infections / microbiology. Intestines / microbiology. Microcirculation

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  • (PMID = 21989977.001).
  • [ISSN] 1414-431X
  • [Journal-full-title] Brazilian journal of medical and biological research = Revista brasileira de pesquisas médicas e biológicas / Sociedade Brasileira de Biofísica ... [et al.]
  • [ISO-abbreviation] Braz. J. Med. Biol. Res.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Brazil
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49. ||||||||.. 77%  Bandyopadhyay S, Lodh C, Rahaman H, Bhattacharya D, Bera AK, Ahmed FA, Mahanti A, Samanta I, Mondal DK, Bandyopadhyay S, Sarkar S, Dutta TK, Maity S, Paul V, Ghosh MK, Sarkar M, Baruah KK: Characterization of shiga toxin producing (STEC) and enteropathogenic Escherichia coli (EPEC) in raw yak (Poephagus grunniens) milk and milk products. Res Vet Sci; 2012 Oct;93(2):604-10
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  • [Title] Characterization of shiga toxin producing (STEC) and enteropathogenic Escherichia coli (EPEC) in raw yak (Poephagus grunniens) milk and milk products.
  • Thirty-one shiga toxin-producing (STEC) and 6 enteropathogenic Escherichia coli (EPEC) were isolated from 87 raw yak milk and 63 'churpi' samples.
  • Dendogram constructed on the basis of RAPD and ERIC PCR profile distributed the STEC and EPEC isolates in separate clusters irrespective of their sources and serotypes.
  • The STEC and EPEC isolates exhibited resistance against erythromycin, amikacin, azithromycin, amoxicillin, ampicillin+cloxacillin, cephalothin, furazolidone, gentamicin, kanamycin, streptomycin and tetracycline.
  • This is the first ever report on occurrence and characterization of STEC and EPEC isolated from yak milk and milk products.
  • [MeSH-major] Cheese / microbiology. Enteropathogenic Escherichia coli / isolation & purification. Food Microbiology. Milk / microbiology. Shiga-Toxigenic Escherichia coli / isolation & purification
  • [MeSH-minor] Animals. Cattle. Escherichia coli Proteins / genetics. Escherichia coli Proteins / metabolism. Female. Odds Ratio

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  • [Copyright] Copyright © 2012. Published by Elsevier India Pvt Ltd.
  • (PMID = 22226073.001).
  • [ISSN] 1532-2661
  • [Journal-full-title] Research in veterinary science
  • [ISO-abbreviation] Res. Vet. Sci.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Escherichia coli Proteins
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50. ||||||||.. 77%  Hu Y, Song J, Zhao B: [Construction and immunization of an attenuated vaccine candidate enteropathogenic Escherichia coli O45 ]. Sheng Wu Gong Cheng Xue Bao; 2009 Feb;25(2):181-8
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [Construction and immunization of an attenuated vaccine candidate enteropathogenic Escherichia coli O45 ].
  • In order to obtain an attenuated vaccine candidate for enteropathogenic Escherichia coli (EPEC) O45, a ler deletion mutant of pig enteropathogenic E. coli (PEPEC) O45 was constructed by using the suicide vector pCVD442, termed as PEPEC O45(deltaler).
  • Test group and control group of mice were orogstrically inoculated with the PEPEC O45(deltaler) deletion mutant and the virulent strain O45 respectively.
  • In contrast, control group of mice inoculated with virulent strain O45 exhibited weight loss and all died in four days.
  • Oral immunization can induce specific immune responses in mice and pig, and this mutant strain could be used as an attenuated vaccine candidate against PEPEC O45.
  • [MeSH-major] Enteropathogenic Escherichia coli / immunology. Escherichia coli Infections / prevention & control. Escherichia coli Vaccines / biosynthesis. Escherichia coli Vaccines / immunology. Swine Diseases / prevention & control
  • [MeSH-minor] Animals. Escherichia coli Proteins / genetics. Gene Deletion. Mice. Mutagenesis, Site-Directed. Swine / microbiology. Trans-Activators / genetics. Vaccines, Attenuated / biosynthesis. Vaccines, Attenuated / genetics. Vaccines, Attenuated / immunology

  • MedlinePlus Health Information. consumer health - E. Coli Infections.
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  • (PMID = 19459321.001).
  • [ISSN] 1000-3061
  • [Journal-full-title] Sheng wu gong cheng xue bao = Chinese journal of biotechnology
  • [ISO-abbreviation] Sheng Wu Gong Cheng Xue Bao
  • [Language] chi
  • [Publication-type] English Abstract; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] China
  • [Chemical-registry-number] 0 / Escherichia coli Proteins; 0 / Escherichia coli Vaccines; 0 / Ler protein, E coli; 0 / Trans-Activators; 0 / Vaccines, Attenuated
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