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1. Biomedical articles (top 50; 2010 to 2015)
1. Donnenberg MS, Finlay BB: Combating enteropathogenic Escherichia coli (EPEC) infections: the way forward. Trends Microbiol; 2013 Jul;21(7):317-9
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Combating enteropathogenic Escherichia coli (EPEC) infections: the way forward.
  • Enteropathogenic Escherichia coli (EPEC) strains continue to cause severe and sometimes fatal infantile diarrhea, particularly in Africa.
  • Increased efforts at diagnosis, defining the clinical spectrum of disease, understanding pathogenic mechanisms, and delineating immune responses are desperately needed to develop new strategies to combat EPEC.
  • [MeSH-major] Diarrhea / epidemiology. Diarrhea / microbiology. Enteropathogenic Escherichia coli / immunology. Enteropathogenic Escherichia coli / pathogenicity. Escherichia coli Infections / epidemiology. Escherichia coli Infections / microbiology
  • [MeSH-minor] Africa / epidemiology. Escherichia coli Proteins / immunology. Immunization / methods. Virulence Factors / immunology

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  • [Copyright] Copyright © 2013 Elsevier Ltd. All rights reserved.
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  • (PMID = 23815982.001).
  • [ISSN] 1878-4380
  • [Journal-full-title] Trends in microbiology
  • [ISO-abbreviation] Trends Microbiol.
  • [Language] eng
  • [Grant] United States / NIAID NIH HHS / AI / U19 AI 090873; United States / NIAID NIH HHS / AI / U19 AI090873; Canada / Canadian Institutes of Health Research / /
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Escherichia coli Proteins; 0 / Virulence Factors
  • [Other-IDs] NLM/ NIHMS559478; NLM/ PMC4312613
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2. Swennes AG, Buckley EM, Madden CM, Byrd CP, Donocoff RS, Rodriguez L, Parry NM, Fox JG: Enteropathogenic Escherichia coli prevalence in laboratory rabbits. Vet Microbiol; 2013 May 3;163(3-4):395-8
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Enteropathogenic Escherichia coli prevalence in laboratory rabbits.
  • Rabbit-origin enteropathogenic Escherichia coli (EPEC) causes substantial diarrhea-associated morbidity and has zoonotic potential.
  • EPEC was isolated from 6/141 (4.3%) commercially-acquired laboratory rabbits.
  • Three of these did not have diarrhea or EPEC-typical intestinal lesions; they instead had background plasmacytic intestinal inflammation.
  • Asymptomatically infected rabbits may function as EPEC reservoirs.
  • [MeSH-major] Enteropathogenic Escherichia coli / physiology. Escherichia coli Infections / epidemiology
  • [MeSH-minor] Animals. Anti-Bacterial Agents / pharmacology. Disease Reservoirs. Escherichia coli Proteins / genetics. Female. Intestines / pathology. Prevalence. Rabbits. Serotyping. Virulence Factors / genetics

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  • [Copyright] Copyright © 2013 Elsevier B.V. All rights reserved.
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  • (PMID = 23391439.001).
  • [ISSN] 1873-2542
  • [Journal-full-title] Veterinary microbiology
  • [ISO-abbreviation] Vet. Microbiol.
  • [Language] eng
  • [Grant] United States / NIEHS NIH HHS / ES / P30 ES002109; United States / NIH HHS / OD / T32 OD010978; United States / NCRR NIH HHS / RR / T32 RR007036; United States / NCRR NIH HHS / RR / T32 RR007036
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / Anti-Bacterial Agents; 0 / Escherichia coli Proteins; 0 / Virulence Factors
  • [Other-IDs] NLM/ NIHMS438439; NLM/ PMC3594591
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3. Ruiz RC, Melo KC, Rossato SS, Barbosa CM, Corrêa LM, Elias WP, Piazza RM: Atypical enteropathogenic Escherichia coli secretes plasmid encoded toxin. Biomed Res Int; 2014;2014:896235
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  • [Title] Atypical enteropathogenic Escherichia coli secretes plasmid encoded toxin.
  • Plasmid encoded toxin (Pet) is a serine protease originally described in enteroaggregative Escherichia coli (EAEC) prototype strain 042 whose entire characterization was essentially obtained from studies performed with the purified toxin.
  • Atypical enteropathogenic Escherichia coli (aEPEC) strains, isolated from diarrhea cases, express Pet and its detection in supernatants of infected HEp-2 cells coincides with the appearance of cell damage, which, in turn, were similar to those described with purified Pet.

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  • (PMID = 24949475.001).
  • [ISSN] 2314-6141
  • [Journal-full-title] BioMed research international
  • [ISO-abbreviation] Biomed Res Int
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Other-IDs] NLM/ PMC4037613
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4. Thomassin JL, Brannon JR, Kaiser J, Gruenheid S, Le Moual H: Enterohemorrhagic and enteropathogenic Escherichia coli evolved different strategies to resist antimicrobial peptides. Gut Microbes; 2012 Nov-Dec;3(6):556-61
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Enterohemorrhagic and enteropathogenic Escherichia coli evolved different strategies to resist antimicrobial peptides.
  • Enterohemorrhagic and enteropathogenic Escherichia coli (EHEC and EPEC) are enteric human pathogens that colonize the large and small intestines, respectively.
  • To establish infection EHEC and EPEC must overcome innate host defenses, such as antimicrobial peptides (AMPs) produced by the intestinal epithelium.
  • We showed that the protease OmpT degrades the human AMP LL-37 more rapidly in EHEC than in EPEC.
  • Here, we propose that the different ompT expression in EHEC and EPEC reflects the varying levels of LL-37 throughout the human intestinal tract.
  • These data suggest that EHEC and EPEC adapted to their specific niches by developing distinct AMP-specific resistance mechanisms.
  • [MeSH-major] Antimicrobial Cationic Peptides / pharmacology. Drug Resistance, Microbial. Enterohemorrhagic Escherichia coli / drug effects. Enterohemorrhagic Escherichia coli / pathogenicity. Enteropathogenic Escherichia coli / drug effects. Enteropathogenic Escherichia coli / pathogenicity. Immune Evasion
  • [MeSH-minor] Bacterial Outer Membrane Proteins / metabolism. Escherichia coli Infections / immunology. Escherichia coli Infections / microbiology. Escherichia coli Proteins / metabolism. Gene Expression. Humans. Peptide Hydrolases / metabolism. Promoter Regions, Genetic. Proteolysis

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  • (PMID = 22895086.001).
  • [ISSN] 1949-0984
  • [Journal-full-title] Gut microbes
  • [ISO-abbreviation] Gut Microbes
  • [Language] eng
  • [Grant] Canada / Canadian Institutes of Health Research / / MOP-15551
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't; Review
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Antimicrobial Cationic Peptides; 0 / Bacterial Outer Membrane Proteins; 0 / Escherichia coli Proteins; 0 / ompT protein, E coli; 143108-26-3 / CAP18 lipopolysaccharide-binding protein; EC 3.4.- / Peptide Hydrolases
  • [Other-IDs] NLM/ PMC3495793
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5. Hazen TH, Humphrys MS, Ochieng JB, Parsons M, Bopp CA, O'Reilly CE, Mintz E, Rasko DA: Draft Genome Sequences of Nine Enteropathogenic Escherichia coli Strains from Kenya. Genome Announc; 2014;2(3)
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  • [Title] Draft Genome Sequences of Nine Enteropathogenic Escherichia coli Strains from Kenya.
  • We report here the draft genome sequences of nine enteropathogenic Escherichia coli (EPEC) strains isolated from children in Kenya who died during hospitalization with diarrhea.
  • Each of the isolates possess the EPEC adherence factor (EAF) plasmid encoding the bundle-forming pilus, which is characteristic of EPEC.
  • These isolates represent diverse serogroups and EPEC phylogenomic lineages.


6. Ruetz TJ, Vogl AW, Guttman JA: Detailed examination of cytoskeletal networks within enteropathogenic Escherichia coli pedestals. Anat Rec (Hoboken); 2012 Feb;295(2):201-7
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  • [Title] Detailed examination of cytoskeletal networks within enteropathogenic Escherichia coli pedestals.
  • Enteropathogenic Escherichia coli (EPEC) manipulate the cytoskeleton of host intestinal epithelial cells, producing membrane protrusions termed pedestals that the bacteria reside on throughout the course of their infections.
  • By definition pedestals are actin-based structures, however recent work has identified the spectrin cytoskeleton as a necessary component of EPEC pedestals.
  • Myosin S1 fragment decorated actin filaments examined by electron microscopy demonstrated that actin filaments orientate with their fast-growing barbed ends toward the lateral membranes of EPEC pedestals.
  • [MeSH-major] Cell Surface Extensions / ultrastructure. Cytoskeleton / ultrastructure. Enteropathogenic Escherichia coli / ultrastructure. HeLa Cells / ultrastructure

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  • [Copyright] Copyright © 2011 Wiley Periodicals, Inc.
  • (PMID = 22190417.001).
  • [ISSN] 1932-8494
  • [Journal-full-title] Anatomical record (Hoboken, N.J. : 2007)
  • [ISO-abbreviation] Anat Rec (Hoboken)
  • [Language] eng
  • [Grant] Canada / Canadian Institutes of Health Research / /
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Cytoskeletal Proteins; 0 / Myosin Subfragments; 12634-43-4 / Spectrin
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7. Spira B, Ferreira GM, de Almeida LG: relA enhances the adherence of enteropathogenic Escherichia coli. PLoS One; 2014;9(3):e91703
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  • [Title] relA enhances the adherence of enteropathogenic Escherichia coli.
  • Enteropathogenic Escherichia coli (EPEC) is a known causative agent of diarrhea in children.
  • In the process of colonization of the small intestine, EPEC synthesizes two types of adhesins, the bundle-forming pilus (BFP) and intimin.
  • The BFP pilus is an adhesin associated with the initial stages of adherence of EPEC to epithelial cells, while the outer membrane protein intimin carries out the intimate adherence that takes place at the third stage of infection.
  • BFP is encoded by the bfp operon located in plasmid EAF, present only in typical EPEC isolates, while eae, the gene that encodes intimin is situated in the LEE, a chromosomal pathogenicity island.
  • Here we show that deletion of relA, that encodes a guanosine penta and tetraphosphate synthetase impairs EPEC adherence to epithelial cells in vitro.
  • [MeSH-major] Adhesins, Bacterial / genetics. Enteropathogenic Escherichia coli / genetics. Escherichia coli Proteins / genetics. Fimbriae Proteins / genetics. Gene Expression Regulation, Bacterial. Ligases / genetics. Protein Subunits / genetics

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  • (PMID = 24643076.001).
  • [ISSN] 1932-6203
  • [Journal-full-title] PloS one
  • [ISO-abbreviation] PLoS ONE
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Adhesins, Bacterial; 0 / BfpA protein, E coli; 0 / Escherichia coli Proteins; 0 / Protein Subunits; 147094-99-3 / eaeA protein, E coli; 147680-16-8 / Fimbriae Proteins; EC 6.- / Ligases; EC 6.- / guanosine 3',5'-polyphosphate synthetases
  • [Other-IDs] NLM/ PMC3958371
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8. Cooper IF, Siadaty MS: 'Diseases or Syndromes' associated with 'Enteritis Escherichia Coli Enteropathogenic': Top Publications. BioMedLib Review; DiseaseOrSyndrome;EnteritisEscherichiaColi:707058446. ISSN: 2331-5717. 2014/3/4
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  • [Title] 'Diseases or Syndromes' associated with 'Enteritis Escherichia Coli Enteropathogenic': Top Publications.
  • [Transliterated title]
  • Background: There are articles published each month which present 'Disease or Syndrome' for 'enteritis escherichia coli enteropathogenic'.
  • Finding such articles is important for researchers, clinicians, and patients.
  • However these articles are spread across thousands of journals, and there are many types of 'Disease or Syndrome'.
  • This makes searching and locating the relevant publications a challenge.
  • We have used BioMedLib's semantic search technology to address the issue, and gathered all the pertinent publications in this review article.
  • Methods: We categorized the publications we found into two groups.
  • We used the strength of textual-association to separate the groups.
  • In group one there are publications with the strongest evidence of association. We focused finding the most relevant publications pertinent to our goal, rather than combining them into a conclusion section. Such textual synthesis will be the focus of our next project.
  • Results: Group one includes 26 publications, and group two 127 publications.
  • Here are the top 10.
  • Swennes AG et al: Enzootic enteropathogenic Escherichia coli infection in laboratory rabbits.
  • Ochoa TJ et al: Enteropathogenic escherichia coli infection in children.
  • Cantarelli VV et al: Cortactin is necessary for F-actin accumulation in pedestal structures induced by enteropathogenic Escherichia coli infection.
  • Crane JK et al: Effect of zinc in enteropathogenic Escherichia coli infection.
  • Wu Y et al: Enteropathogenic Escherichia coli infection triggers host phospholipid metabolism perturbations.
  • Savkovic SD et al: Mouse model of enteropathogenic Escherichia coli infection.
  • Clarke SC et al: Enteropathogenic Escherichia coli infection: history and clinical aspects.
  • Hemrajani C et al: NleH effectors interact with Bax inhibitor-1 to block apoptosis during enteropathogenic Escherichia coli infection.
  • Crane JK et al: Two pathways for ATP release from host cells in enteropathogenic Escherichia coli infection.
  • Michail S et al: Lactobacillus plantarum reduces the in vitro secretory response of intestinal epithelial cells to enteropathogenic Escherichia coli infection.

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
  • (UID = 707058446.001).
  • [ISSN] 2331-5717
  • [Journal-full-title] BioMedLib Review
  • [Language] eng
  • [Publication-type] Review
  • [Publication-country] UNITED STATES
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9. Cooper IF, Siadaty MS: 'Qualitative Concepts' associated with 'Enteritis Escherichia Coli Enteropathogenic': Top Publications. BioMedLib Review; QualitativeConcept;EnteritisEscherichiaColi:707087351. ISSN: 2331-5717. 2014/5/4
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  • [Title] 'Qualitative Concepts' associated with 'Enteritis Escherichia Coli Enteropathogenic': Top Publications.
  • [Transliterated title]
  • Background: There are articles published each month which present 'Qualitative Concept' for 'enteritis escherichia coli enteropathogenic'.
  • Finding such articles is important for researchers, clinicians, and patients.
  • However these articles are spread across thousands of journals, and there are many types of 'Qualitative Concept'.
  • This makes searching and locating the relevant publications a challenge.
  • We have used BioMedLib's semantic search technology to address the issue, and gathered all the pertinent publications in this review article.
  • Methods: We categorized the publications we found into two groups.
  • We used the strength of textual-association to separate the groups.
  • In group one there are publications with the strongest evidence of association. We focused finding the most relevant publications pertinent to our goal, rather than combining them into a conclusion section. Such textual synthesis will be the focus of our next project.
  • Results: Group one includes 26 publications, and group two 127 publications.
  • Here are the top 10.
  • Ochoa TJ et al: Enteropathogenic escherichia coli infection in children.
  • Swennes AG et al: Enzootic enteropathogenic Escherichia coli infection in laboratory rabbits.
  • Cantarelli VV et al: Cortactin is necessary for F-actin accumulation in pedestal structures induced by enteropathogenic Escherichia coli infection.
  • Crane JK et al: Effect of zinc in enteropathogenic Escherichia coli infection.
  • Wu Y et al: Enteropathogenic Escherichia coli infection triggers host phospholipid metabolism perturbations.
  • Savkovic SD et al: Mouse model of enteropathogenic Escherichia coli infection.
  • Clarke SC et al: Enteropathogenic Escherichia coli infection: history and clinical aspects.
  • Hemrajani C et al: NleH effectors interact with Bax inhibitor-1 to block apoptosis during enteropathogenic Escherichia coli infection.
  • Crane JK et al: Two pathways for ATP release from host cells in enteropathogenic Escherichia coli infection.
  • Michail S et al: Lactobacillus plantarum reduces the in vitro secretory response of intestinal epithelial cells to enteropathogenic Escherichia coli infection.

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
  • (UID = 707087351.001).
  • [ISSN] 2331-5717
  • [Journal-full-title] BioMedLib Review
  • [Language] eng
  • [Publication-type] Review
  • [Publication-country] UNITED STATES
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10. Staples M, Doyle CJ, Graham RM, Jennison AV: Molecular epidemiological typing of enteropathogenic Escherichia coli strains from Australian patients. Diagn Microbiol Infect Dis; 2013 Mar;75(3):320-4
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Molecular epidemiological typing of enteropathogenic Escherichia coli strains from Australian patients.
  • Enteropathogenic Escherichia coli (EPEC) are an important cause of diarrhoea worldwide, particularly in children.
  • Sixty-one EPEC strains isolated from stool specimens of symptomatic persons from 2008 to 2011 were characterised for the prevalence of diarrhoea-associated putative virulence genes.
  • The EPEC isolates were highly heterogeneous, representing all 4 phylogenetic groups and comprising 59 MLVA types, 49 MLST types, and 43 serotypes.
  • This diversity is indicative of the complexity of the human enteric EPEC population, which may be either commensal or pathogenic.
  • [MeSH-major] Bacterial Typing Techniques / methods. Enteropathogenic Escherichia coli / genetics. Multilocus Sequence Typing / methods
  • [MeSH-minor] Australia / epidemiology. Diarrhea / microbiology. Escherichia coli Proteins / genetics. Feces / microbiology. Genes, Bacterial. Genetic Variation. Humans. Molecular Epidemiology. Phylogeny. Prevalence. Virulence Factors / genetics

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  • [Copyright] Crown Copyright © 2013. Published by Elsevier Inc. All rights reserved.
  • (PMID = 23357294.001).
  • [ISSN] 1879-0070
  • [Journal-full-title] Diagnostic microbiology and infectious disease
  • [ISO-abbreviation] Diagn. Microbiol. Infect. Dis.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Escherichia coli Proteins; 0 / NleB protein, E coli; 0 / Virulence Factors
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11. de Almeida PM, Arais LR, Andrade JR, Prado EH, Irino K, Cerqueira Ade M: Characterization of atypical Enteropathogenic Escherichia coli (aEPEC) isolated from dogs. Vet Microbiol; 2012 Aug 17;158(3-4):420-4
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Characterization of atypical Enteropathogenic Escherichia coli (aEPEC) isolated from dogs.
  • Enteropathogenic Escherichia coli (EPEC), an important human pathogen has the ability to form attaching and effacing lesions on the intestinal epithelium and has been isolated from a wide range of species.
  • Two EPEC subgroups are recognized: typical (tEPEC) and atypical (aEPEC) strains, differing by the presence of EAF plasmid and bundle-forming pilus (BFP) in typical strains and their absence in atypical strains.
  • This study searched the presence of EPEC strains in 101 fecal samples of diarrheic (n=65) and non-diarrheic (n=36) dogs from two cities in Rio de Janeiro State, Brazil.
  • Most isolates was FAS-positive and showed a localized adherence-like (LAL) in a 6h HeLa cell-adherence assay.
  • [MeSH-major] Dog Diseases / microbiology. Enteropathogenic Escherichia coli / genetics. Escherichia coli Infections / veterinary
  • [MeSH-minor] Adhesins, Bacterial / genetics. Animals. Bacterial Adhesion. Base Sequence. Brazil. Dogs. Escherichia coli Proteins / genetics. Feces / microbiology. Genotype. HeLa Cells. Humans. Serotyping

  • MedlinePlus Health Information. consumer health - E. Coli Infections.
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  • [Copyright] Copyright © 2012. Published by Elsevier B.V.
  • (PMID = 22421111.001).
  • [ISSN] 1873-2542
  • [Journal-full-title] Veterinary microbiology
  • [ISO-abbreviation] Vet. Microbiol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / Adhesins, Bacterial; 0 / Escherichia coli Proteins
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12. Bustamante VH, Villalba MI, García-Angulo VA, Vázquez A, Martínez LC, Jiménez R, Puente JL: PerC and GrlA independently regulate Ler expression in enteropathogenic Escherichia coli. Mol Microbiol; 2011 Oct;82(2):398-415
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] PerC and GrlA independently regulate Ler expression in enteropathogenic Escherichia coli.
  • Typical enteropathogenic Escherichia coli (EPEC) strains contain the EAF plasmid, which carries the perABC locus encoding PerC.
  • The precise role of PerC in EPEC virulence gene regulation has remained unclear, mainly because EPEC strains lacking the pEAF still express the LEE genes and because PerC is not present in other A/E pathogens such as Citrobacter rodentium.
  • Our results substantiate the role of PerC and GrlA in EPEC virulence gene regulation and suggest that these convergent regulatory mechanisms may have represented an evolutionary adaptation in EPEC to co-ordinate the expression of plasmid- and chromosome-encoded virulence factors needed to successfully colonize its intestinal niche.
  • [MeSH-major] Enteropathogenic Escherichia coli / metabolism. Escherichia coli Proteins / metabolism. Gene Expression Regulation, Bacterial. Trans-Activators / metabolism

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  • [Copyright] © 2011 Blackwell Publishing Ltd.
  • (PMID = 21895790.001).
  • [ISSN] 1365-2958
  • [Journal-full-title] Molecular microbiology
  • [ISO-abbreviation] Mol. Microbiol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Escherichia coli Proteins; 0 / GrlA protein, E coli; 0 / Ler protein, E coli; 0 / PerC protein, E coli; 0 / Trans-Activators
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13. Hazen TH, Sahl JW, Fraser CM, Donnenberg MS, Scheutz F, Rasko DA: Draft Genome Sequences of Three O157 Enteropathogenic Escherichia coli Isolates. Genome Announc; 2013;1(4)
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Draft Genome Sequences of Three O157 Enteropathogenic Escherichia coli Isolates.
  • We report the draft genome sequences of three enteropathogenic Escherichia coli (EPEC) isolates that display the O157 serogroup but do not have the Shiga toxin genes (stx), which are characteristic of O157 enterohemorrhagic E. coli (EHEC). E. coli strain RN587/1 has the O157:H8 serotype and possesses the EAF plasmid characteristic of typical EPEC (J. B.


14. Hernandes RT, Velsko I, Sampaio SC, Elias WP, Robins-Browne RM, Gomes TA, Girón JA: Fimbrial adhesins produced by atypical enteropathogenic Escherichia coli strains. Appl Environ Microbiol; 2011 Dec;77(23):8391-9
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Fimbrial adhesins produced by atypical enteropathogenic Escherichia coli strains.
  • Atypical enteropathogenic Escherichia coli (aEPEC) has emerged as a significant cause of pediatric diarrhea worldwide; however, information regarding its adherence mechanisms to the human gut mucosa is lacking.
  • These genes are associated with adhesion and/or biofilm formation of pathogenic and commensal E. coli.
  • We were not able to detect the hemorrhagic coli pilus (HCP) or the E. coli laminin-binding fimbriae (ELF) in these strains by using immunofluorescence.
  • The E. coli common pilus (ECP) was evidenced in 36.6% of the strains on bacteria adhering to HeLa cells by immunofluorescence, suggesting that ECP could play an important role in cell adherence for some aEPEC strains.
  • [MeSH-major] Adhesins, Bacterial / biosynthesis. Adhesins, Escherichia coli / biosynthesis. Enteropathogenic Escherichia coli / metabolism
  • [MeSH-minor] Australia. Bacterial Adhesion. Brazil. DNA, Bacterial / genetics. Diarrhea / microbiology. Epithelial Cells / microbiology. Escherichia coli Infections / microbiology. Gene Expression Profiling. HeLa Cells. Humans. Polymerase Chain Reaction

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  • (PMID = 21926222.001).
  • [ISSN] 1098-5336
  • [Journal-full-title] Applied and environmental microbiology
  • [ISO-abbreviation] Appl. Environ. Microbiol.
  • [Language] eng
  • [Grant] United States / NIAID NIH HHS / AI / AI66012
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Adhesins, Bacterial; 0 / Adhesins, Escherichia coli; 0 / DNA, Bacterial
  • [Other-IDs] NLM/ PMC3233042
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15. Asadi Karam M, Bouzari S, Oloomi M, Aslani M, Jafari A: Phenotypic and Genotypic Characterization of Enteropathogenic Escherichia coli (EPEC) strains in Tehran, Iran. Iran J Microbiol; 2010 Mar;2(1):3-7
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Phenotypic and Genotypic Characterization of Enteropathogenic Escherichia coli (EPEC) strains in Tehran, Iran.
  • BACKGROUND AND OBJECTIVES: Enteropathogenic Escherichia coli (EPEC) strains can be detected by serogrouping and the presence of enterocyte attaching- effacing (eae) gene.
  • Most EPEC strains belong to a certain O antigenic group.
  • The aim of the present study was to genetically characterize EPEC strains isolated from children with diarrhea.
  • MATERIALS AND METHODS: Serogrouping was performed by EPEC antisera in 321 E. coli isolates.
  • RESULTS: Seventeen (5.3%) isolates belonging to 7 EPEC serogroups were identified among the whole material and all carried the eae gene.
  • None of the 321 isolates showed presence of stx gene indicating that all 17 isolates classified as EPEC by O serogrouping did not belong to the enterohaemorrhagic E. coli (EHEC) group.
  • CONCLUSION: Serogrouping and detection of the eae gene showed to be reliable for detection of EPEC strains.
  • No Shigatoxin- producing E. coli (STEC) strain was found among the isolates.
  • Detection of the insertion site of LEE showed that selC, pheU or PheV are insertion sites of LEE in the EPEC strains.

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  • (PMID = 22347543.001).
  • [ISSN] 2008-4447
  • [Journal-full-title] Iranian journal of microbiology
  • [ISO-abbreviation] Iran J Microbiol
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Iran
  • [Other-IDs] NLM/ PMC3279762
  • [Keywords] NOTNLM ; Attaching- effacing / Intimin / Iran / Pathogenicity island / Serogrouping
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16. Tilak GP, Mudaliar JL: Role of enteropathogenic Escherichia coli in paediatric diarrhoeas in South India. Mater Sociomed; 2012;24(3):178-81
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Role of enteropathogenic Escherichia coli in paediatric diarrhoeas in South India.
  • BACKGROUND: Enteropathogenic Escherichia coli(EPEC) is a major cause of diarrhoea in children below 5 years of age.
  • Serotyping is classical method for identification of EPEC strains.
  • But serotypic markers are rarely sufficient to reliably identify the strains as Escherichia coli.
  • Introduction of PCR methodology which depends on detection of virulence factors has provided a practical and rapid way of detecting diarrhoeagenic Esch.coli.
  • Multiantibiotic resistant EPEC strains are a common phenomenon with world wide extension.
  • OBJECTIVES: To study the role of EPEC in Paediatric diarrhoea by both Serogrouping and Molecular characterisation by PCR and to analyse the antibiotic susceptabililty patterns of EPEC strains in our area.
  • Escherichia.coli isolates were identified by Microscopy, Culture and Biochemical reactions.
  • Among the Escherichia coli isolates, EPEC isolates were identified by Serogrouping.
  • Escherichia coli isolates were also subjected to Molecular characterisation by Multiplex PCR assay and those isolates which showed pathogenic genes were futher serotyped.
  • Antibiotic susceptibility pattern of EPEC isolates was determined by CLSI guidelines.
  • RESULTS: Among the Escherichia coli isolates 36.8% in the diarrhoeal group and none of them from the nondiarrhoeal group were identified as EPEC by serogrouping.
  • 73.3% of the EPEC isolates were below 2 years of age and no much difference in the sex distribution was observed.
  • In the diarrhoeal group 38.8% of Escherichia coli were EAEC and no other diarrhoeagenic Escherichia coli group was found by molecular characterisation.
  • In the nondiarrhoeal Escherichia coli strains, 46.6% showed EAEC genes.
  • Among the Escherichia coli isolates which agglutinated with EPEC polyvalent antisera, 33.3% were positive for Enteroaggregative genes. (Table 1).
  • CONCLUSION: EPEC is still an important pathogen in paediatric diarrhoeas.
  • O serogrouping can still be relied upon for detection of EPEC.
  • Dehydration is one of the clinical features of EPEC diarrhoea.

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  • (PMID = 23922527.001).
  • [ISSN] 1512-7680
  • [Journal-full-title] Materia socio-medica
  • [ISO-abbreviation] Mater Sociomed
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Bosnia and Hercegovina
  • [Other-IDs] NLM/ PMC3732348
  • [Keywords] NOTNLM ; Andhra Pradesh / Antibiotic Susceptibility / Enteroaggregative Escherchia coli (EAEC). / Enteropathogenic Escherichia coli (EPEC) / Molecular characterisation / Serotyping
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17. Vieira MA, Salvador FA, Silva RM, Irino K, Vaz TM, Rockstroh AC, Guth BE, Gomes TA: Prevalence and characteristics of the O122 pathogenicity island in typical and atypical enteropathogenic Escherichia coli strains. J Clin Microbiol; 2010 Apr;48(4):1452-5
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  • [Title] Prevalence and characteristics of the O122 pathogenicity island in typical and atypical enteropathogenic Escherichia coli strains.
  • The presence of the pathogenicity island (PAI) O122 genes, efa1 (lifA), sen, pagC, nleB, and nleE, in typical and atypical enteropathogenic Escherichia coli (EPEC) strains was investigated.
  • The simultaneous occurrence of all genes was statistically associated with diarrhea due to atypical EPEC.
  • Detection of the complete PAI O122 could aid in the identification of potential pathogenic strains of atypical EPEC.
  • [MeSH-major] DNA, Bacterial / genetics. Enteropathogenic Escherichia coli / genetics. Escherichia coli Infections / microbiology. Genomic Islands
  • [MeSH-minor] Diarrhea / microbiology. Escherichia coli Proteins / genetics. Humans. Prevalence. Virulence Factors / genetics

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  • (PMID = 20181917.001).
  • [ISSN] 1098-660X
  • [Journal-full-title] Journal of clinical microbiology
  • [ISO-abbreviation] J. Clin. Microbiol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / DNA, Bacterial; 0 / Escherichia coli Proteins; 0 / Virulence Factors
  • [Other-IDs] NLM/ PMC2849565
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18. Culler HF, Mota CM, Abe CM, Elias WP, Sircili MP, Franzolin MR: Atypical enteropathogenic Escherichia coli strains form biofilm on abiotic surfaces regardless of their adherence pattern on cultured epithelial cells. Biomed Res Int; 2014;2014:845147
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  • [Title] Atypical enteropathogenic Escherichia coli strains form biofilm on abiotic surfaces regardless of their adherence pattern on cultured epithelial cells.
  • The aim of this study was to determine the capacity of biofilm formation of atypical enteropathogenic Escherichia coli (aEPEC) strains on abiotic and biotic surfaces.
  • [MeSH-major] Bacterial Adhesion. Biofilms / growth & development. Enteropathogenic Escherichia coli / growth & development. Escherichia coli Infections / microbiology

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  • (PMID = 24883330.001).
  • [ISSN] 2314-6141
  • [Journal-full-title] BioMed research international
  • [ISO-abbreviation] Biomed Res Int
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Receptors, Artificial
  • [Other-IDs] NLM/ PMC4032706
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19. Wilbur JS, Byrd W, Ramamurthy S, Ledvina HE, Khirfan K, Riggs MW, Boedeker EC, Vedantam G, Viswanathan VK: The secreted effector protein EspZ is essential for virulence of rabbit enteropathogenic Escherichia coli. Infect Immun; 2015 Mar;83(3):1139-49
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  • [Title] The secreted effector protein EspZ is essential for virulence of rabbit enteropathogenic Escherichia coli.
  • To explore the role of EspZ in A/E pathogen virulence, we generated an isogenic espZ deletion strain (ΔespZ) and corresponding cis-complemented derivatives of rabbit enteropathogenic Escherichia coli and compared their abilities to regulate the T3SS and influence host cell survival in vitro.
  • Also, the ΔespZ strain induced greater host cell death than did the parent and complemented strains.
  • In rabbit infections, fecal ΔespZ strain levels were 10-fold lower than those of the parent strain at 1 day postinfection, while the complemented strain was recovered at intermediate levels.
  • Terminal deoxynucleotidyltransferase-mediated dUTP-biotin nick end labeling (TUNEL) staining of intestinal sections revealed increased epithelial cell apoptosis on day 1 after infection with the ΔespZ strain compared to animals infected with the parent or complemented strains.
  • [MeSH-major] Enterocytes / microbiology. Enteropathogenic Escherichia coli / genetics. Enteropathogenic Escherichia coli / pathogenicity. Escherichia coli Infections / microbiology. Escherichia coli Proteins / genetics. Microvilli / microbiology

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  • [Copyright] Copyright © 2015, American Society for Microbiology. All Rights Reserved.
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  • (PMID = 25561713.001).
  • [ISSN] 1098-5522
  • [Journal-full-title] Infection and immunity
  • [ISO-abbreviation] Infect. Immun.
  • [Language] eng
  • [Grant] United States / PHS HHS / / NIAID1R01AI081742; United States / NIAID NIH HHS / AI / R01 AI081742
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Escherichia coli Proteins; 0 / EspZ protein, E coli
  • [Other-IDs] NLM/ PMC4333479 [Available on 09/01/15]
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20. Swennes AG, Buckley EM, Parry NM, Madden CM, García A, Morgan PB, Astrofsky KM, Fox JG: Enzootic enteropathogenic Escherichia coli infection in laboratory rabbits. J Clin Microbiol; 2012 Jul;50(7):2353-8
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  • [Title] Enzootic enteropathogenic Escherichia coli infection in laboratory rabbits.
  • Enteropathogenic Escherichia coli (EPEC) is the most important cause of persistent diarrhea in children, particularly in developing countries.
  • Animals serve as pathogenic E. coli reservoirs, and compelling evidence for cross-species EPEC transmission exists.
  • In this report, enzootic EPEC infection associated with up to 10.5% diarrhea-associated morbidity in a large laboratory Dutch Belted rabbit colony was investigated.
  • Fecal culture of 20 rabbits yielded 48 E. coli isolates, 83% of which were eae positive.
  • Repetitive sequence-based PCR (REP-PCR) and serologic analysis identified a single disease-associated EPEC O145:H2 strain.
  • In sampled rabbits, EPEC-positive culture and the presence of diarrhea were significantly associated.
  • This strain displayed a localized adherence-like HEp-2 cell adherence pattern, as seen in diarrheic human infant EPEC isolates.
  • This report emphasizes the zoonotic potential of animal EPEC strains and the need for virulence determinant-based screening of E. coli isolates from diarrheic animals.
  • [MeSH-major] Animals, Laboratory / microbiology. Diarrhea / veterinary. Disease Outbreaks. Enteropathogenic Escherichia coli / isolation & purification. Escherichia coli Infections / veterinary. Rabbits / microbiology

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  • (PMID = 22573597.001).
  • [ISSN] 1098-660X
  • [Journal-full-title] Journal of clinical microbiology
  • [ISO-abbreviation] J. Clin. Microbiol.
  • [Language] eng
  • [Databank-accession-numbers] GENBANK/ JQ700206
  • [Grant] United States / NIEHS NIH HHS / ES / P30 ES002109; United States / NIH HHS / OD / T32 OD010978; United States / NCRR NIH HHS / RR / T32 RR070036
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Anti-Bacterial Agents; 0 / DNA, Bacterial; 0 / Fluoroquinolones; 3DX3XEK1BN / enrofloxacin
  • [Other-IDs] NLM/ PMC3405579
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21. Wong Fok Lung T, Pearson JS, Schuelein R, Hartland EL: The cell death response to enteropathogenic Escherichia coli infection. Cell Microbiol; 2014 Dec;16(12):1736-45
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  • [Title] The cell death response to enteropathogenic Escherichia coli infection.
  • The causative agent of infant diarrhoea, enteropathogenic Escherichia coli (EPEC), is an extracellular, intestinal pathogen that blocks both inflammation and programmed cell death.
  • EPEC attaches to enterocytes, remains in the gut lumen and utilizes a type III secretion system (T3SS) to inject multiple virulence effector proteins directly into the infected cell, many of which subvert host antimicrobial processes through the disruption of signalling pathways.
  • Recently, T3SS effector proteins from EPEC have been identified that inhibit death receptor-induced apoptosis.
  • Here we review the mechanisms used by EPEC T3SS effectors to manipulate apoptosis and promote host cell survival and discuss the role of these activities during infection.

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  • [Copyright] © 2014 John Wiley & Sons Ltd.
  • (PMID = 25266336.001).
  • [ISSN] 1462-5822
  • [Journal-full-title] Cellular microbiology
  • [ISO-abbreviation] Cell. Microbiol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
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22. Pearson JS, Hartland EL: A surprising sweetener from enteropathogenic Escherichia coli. Gut Microbes; 2014;5(6):766-9
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  • [Title] A surprising sweetener from enteropathogenic Escherichia coli.
  • Infections with enteropathogenic Escherichia coli (EPEC) are remarkably devoid of gut inflammation and necrotic damage compared to infections caused by invasive pathogens such as Salmonella and Shigella.
  • Recently, we observed that EPEC blocks cell death using the type III secretion system (T3SS) effector NleB.
  • Hence, by blocking cell death, NleB promotes enterocyte survival and thereby prolongs EPEC attachment to the gut epithelium.

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  • (PMID = 25536377.001).
  • [ISSN] 1949-0984
  • [Journal-full-title] Gut microbes
  • [ISO-abbreviation] Gut Microbes
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Keywords] NOTNLM ; Fas ligand / T3SS / colitis / death domain / enteropathogenic E. coli / glycosyltransferase
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23. Alonso MZ, Padola NL, Parma AE, Lucchesi PM: Enteropathogenic Escherichia coli contamination at different stages of the chicken slaughtering process. Poult Sci; 2011 Nov;90(11):2638-41
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  • [Title] Enteropathogenic Escherichia coli contamination at different stages of the chicken slaughtering process.
  • Enteropathogenic Escherichia coli is a foodborne pathogen that produces potentially fatal infant diarrhea, noticeably in developing countries.
  • The aim of this study was to detect EPEC contamination by PCR at different stages of the chicken slaughtering process.
  • Enteropathogenic Escherichia coli was detected in 6 to 28% of cloacal samples, 39 and 56% of unwashed eviscerated carcasses, and 4 to 58% of washed carcasses.
  • None of the samples were positive for bfpA, suggesting contamination with atypical EPEC.
  • The detection of EPEC at different stages of the chicken slaughtering process showed that the proportion of contaminated samples remained or even increased during processing.
  • [MeSH-major] Abattoirs. Enteropathogenic Escherichia coli / isolation & purification. Food Microbiology

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  • (PMID = 22010252.001).
  • [ISSN] 0032-5791
  • [Journal-full-title] Poultry science
  • [ISO-abbreviation] Poult. Sci.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
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24. Gouveia EM, Silva IS, Nakazato G, Araujo FR, Chang MR: Experimental infection with enteropathogenic Escherichia coli identified by PCR using enteric-coated capsules in boxer pups. Acta Cir Bras; 2011 Apr;26(2):144-8
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  • [Title] Experimental infection with enteropathogenic Escherichia coli identified by PCR using enteric-coated capsules in boxer pups.
  • PURPOSE: To verify the possibility of an experimental infection with enteropathogenic Escherichia coli and to confirm by PCR that the symptoms manifested after infection were due to the virulence factors of the studied bacteria.
  • One animal from each litter was included in a control group and the remaining animals were divided into two groups: one inoculated with strain 4083, and another one inoculated with strain SPA14.
  • Gelatinous capsules coated with enteric-coating solution were used for the inoculation of strains. E. coli isolation from feces was performed for all tested animals, and the extracted DNA was subjected to Polymerase Chain Reaction (PCR).
  • CONCLUSION: The efficiency of PCR for the studied strains indicates that this technique can be recommended for the diagnosis of enteropathogenic Escherichia coli as a differential from other pathogens causing diarrhea.
  • [MeSH-major] Enteropathogenic Escherichia coli / genetics. Escherichia coli Infections / microbiology. Polymerase Chain Reaction

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  • (PMID = 21445479.001).
  • [ISSN] 1678-2674
  • [Journal-full-title] Acta cirúrgica brasileira / Sociedade Brasileira para Desenvolvimento Pesquisa em Cirurgia
  • [ISO-abbreviation] Acta Cir Bras
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Brazil
  • [Chemical-registry-number] 0 / DNA, Bacterial; 0 / Virulence Factors
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25. Flynn AN, Wang A, McKay DM, Buret AG: Apoptosis-inducing factor contributes to epithelial cell apoptosis induced by enteropathogenic Escherichia coli. Can J Physiol Pharmacol; 2011 Feb;89(2):143-8
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  • [Title] Apoptosis-inducing factor contributes to epithelial cell apoptosis induced by enteropathogenic Escherichia coli.
  • The mechanisms by which enteropathogenic Escherichia coli (EPEC) causes intestinal epithelial cell apoptosis remain unclear.
  • We tested the hypothesis that apoptosis-inducing factor (AIF) is involved in apoptosis induced by EPEC.
  • Infection of intestinal epithelial cells in vitro with EPEC led to the mitochondrial and cytosolic accumulation of AIF.
  • Knockdown of AIF with siRNA blocked cellular apoptosis in response to EPEC infection, as assessed by poly(ADP-ribose) polymerase cleavage and oligonucleosome formation.
  • Taken together, these data suggest that caspase-dependent mobilization of AIF contributes to EPEC-induced epithelial cell apoptosis.
  • [MeSH-major] Apoptosis / physiology. Apoptosis Inducing Factor / metabolism. Enteropathogenic Escherichia coli / physiology. Intestinal Mucosa / metabolism. Intestinal Mucosa / microbiology
  • [MeSH-minor] Caspases / metabolism. Cell Line. Cytosol / metabolism. Epithelial Cells / metabolism. Epithelial Cells / microbiology. Epithelial Cells / pathology. Escherichia coli Infections / metabolism. Escherichia coli Infections / microbiology. Escherichia coli Infections / pathology. Gene Knockdown Techniques. HeLa Cells. Humans. Mitochondria / metabolism. Poly(ADP-ribose) Polymerases / metabolism. RNA, Small Interfering / genetics

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  • (PMID = 21326346.001).
  • [ISSN] 1205-7541
  • [Journal-full-title] Canadian journal of physiology and pharmacology
  • [ISO-abbreviation] Can. J. Physiol. Pharmacol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Canada
  • [Chemical-registry-number] 0 / Apoptosis Inducing Factor; 0 / RNA, Small Interfering; EC 2.4.2.30 / Poly(ADP-ribose) Polymerases; EC 3.4.22.- / Caspases
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26. Roxas JL, Ryan K, Vedantam G, Viswanathan VK: Enteropathogenic Escherichia coli dynamically regulates EGFR signaling in intestinal epithelial cells. Am J Physiol Gastrointest Liver Physiol; 2014 Aug 1;307(3):G374-80
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  • [Title] Enteropathogenic Escherichia coli dynamically regulates EGFR signaling in intestinal epithelial cells.
  • The diarrheagenic pathogen enteropathogenic Escherichia coli (EPEC) dynamically modulates the survival of infected host intestinal epithelial cells.
  • To identify the specific EPEC effector molecule(s) that influenced EGFR stability, epithelial cells infected with isogenic mutant EPEC strains were examined.
  • An EPEC ΔespF strain failed to induce EGFR degradation, whereas EPEC ΔespZ accentuated receptor loss in infected cells.
  • The pan-caspase inhibitor Q-VD-OPh blocked EPEC-induced EGFR cleavage in a dose-dependent manner.
  • Taken together, our data suggest that EPEC EspF stimulates caspase-dependent EGFR cleavage and loss, whereas EspZ inhibits this process.
  • [MeSH-major] Enteropathogenic Escherichia coli / metabolism. Epithelial Cells / metabolism. Epithelial Cells / microbiology. Intestines / metabolism. Intestines / microbiology. Receptor, Epidermal Growth Factor / metabolism. Signal Transduction
  • [MeSH-minor] Caco-2 Cells. Carrier Proteins / genetics. Carrier Proteins / metabolism. Caspase Inhibitors / pharmacology. Cell Death. Dose-Response Relationship, Drug. Escherichia coli Proteins / genetics. Escherichia coli Proteins / metabolism. Host-Pathogen Interactions. Humans. Mutation. Phosphorylation. Protein Stability. Time Factors

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  • [Copyright] Copyright © 2014 the American Physiological Society.
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  • (PMID = 24904077.001).
  • [ISSN] 1522-1547
  • [Journal-full-title] American journal of physiology. Gastrointestinal and liver physiology
  • [ISO-abbreviation] Am. J. Physiol. Gastrointest. Liver Physiol.
  • [Language] eng
  • [Grant] United States / PHS HHS / / NIAID1R01AI081742
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Carrier Proteins; 0 / Caspase Inhibitors; 0 / Escherichia coli Proteins; 0 / EspFU protein, E coli; 0 / EspZ protein, E coli; EC 2.7.10.1 / EGFR protein, human; EC 2.7.10.1 / Receptor, Epidermal Growth Factor
  • [Other-IDs] NLM/ PMC4121633 [Available on 08/01/15]
  • [Keywords] NOTNLM ; EGFR degradation / EGFR phosphorylation / EPEC / EspF / EspZ / caspase
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27. Mohammadzadeh M, Oloomi M, Bouzari S: Genetic evaluation of Locus of enterocyte effacement pathogenicity island (LEE) in Enteropathogenic Escherichia coli isolates (EPEC). Iran J Microbiol; 2013 Dec;5(4):345-9
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  • [Title] Genetic evaluation of Locus of enterocyte effacement pathogenicity island (LEE) in Enteropathogenic Escherichia coli isolates (EPEC).
  • BACKGROUND AND OBJECTIVES: Enteropathogenic Escherichia coli (EPEC) divided into two groups typical and atypical (aspect).
  • MATERIALS AND METHODS: In this study 130 E. coli isolates confirmed by biochemical analysis from diarrheal patients, were evaluated for EPEC pathotype by PCR.
  • All EPEC strains tested for presence of some LEE encoded virulence genes and sites of LEE insertion by PCR method.
  • RESULTS: Among 50 strains of EPEC 28 (56%) and 22 (44%) were typical and atypical strains respectively.

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  • (PMID = 25848503.001).
  • [ISSN] 2008-3289
  • [Journal-full-title] Iranian journal of microbiology
  • [ISO-abbreviation] Iran J Microbiol
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Iran
  • [Other-IDs] NLM/ PMC4385159
  • [Keywords] NOTNLM ; EPEC / LEE / tRNA / virulence genes
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28. Horcajo P, Domínguez-Bernal G, Carrión J, De La Fuente R, Ruiz-Santa-Quiteria JA, Orden JA: Differences in virulence gene expression between atypical enteropathogenic Escherichia coli strains isolated from diarrheic and healthy ruminants. Can J Vet Res; 2013 Apr;77(2):158-60
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  • [Title] Differences in virulence gene expression between atypical enteropathogenic Escherichia coli strains isolated from diarrheic and healthy ruminants.
  • Differences in the pathogenicity of atypical enteropathogenic Escherichia coli (EPEC) strains may be due, at least partially, to different expression patterns of some virulence genes.
  • To investigate this hypothesis, the virulence gene expression patterns of 6 atypical EPEC strains isolated from healthy and diarrheic ruminants were compared using quantitative real-time reverse transcription polymerase chain reaction after growing the bacteria in culture medium alone or after binding it to HeLa epithelial cells.
  • Thus, the expression levels of some virulence genes may help determine which atypical EPEC strains cause diarrhea in ruminants.
  • [MeSH-major] Diarrhea / veterinary. Enteropathogenic Escherichia coli / metabolism. Gene Expression Regulation, Bacterial / physiology. Virulence Factors / metabolism

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  • (PMID = 24082409.001).
  • [ISSN] 0830-9000
  • [Journal-full-title] Canadian journal of veterinary research = Revue canadienne de recherche vétérinaire
  • [ISO-abbreviation] Can. J. Vet. Res.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Canada
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29. Scaletsky IC, Souza TB, Aranda KR, Okeke IN: Genetic elements associated with antimicrobial resistance in enteropathogenic Escherichia coli (EPEC) from Brazil. BMC Microbiol; 2010;10:25
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  • [Title] Genetic elements associated with antimicrobial resistance in enteropathogenic Escherichia coli (EPEC) from Brazil.
  • BACKGROUND: We recently observed an association of resistance with a certain enteropathogenic Escherichia coli (EPEC) serotypes and identified a conjugative plasmid, similar to plasmid pED208, that was conserved among archival O111:H2/NM and O119:H2 strains of diverse geographical origin.
  • In this study, we sought to determine the prevalence and distribution of this plasmid among a collection of EPEC isolates from Brazil, as well as to study the susceptibilities of these isolates to antimicrobial agents.
  • RESULTS: Resistance was more commonly seen in typical EPEC than atypical strains.
  • Markers for the EPEC conjugative multiresistance plasmid, were detected in 21 (30%) of typical but only 4 (5%) of atypical strains (p = 0.001, Chi-squared test).
  • CONCLUSION: Our data suggest a limited but expanding host range for the EPEC resistance plasmid.
  • [MeSH-major] Enteropathogenic Escherichia coli / drug effects. Enteropathogenic Escherichia coli / genetics. Escherichia coli Infections / microbiology

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  • (PMID = 20105329.001).
  • [ISSN] 1471-2180
  • [Journal-full-title] BMC microbiology
  • [ISO-abbreviation] BMC Microbiol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Anti-Bacterial Agents
  • [Other-IDs] NLM/ PMC2828443
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30. Srikhanta YN, Hocking DM, Praszkier J, Wakefield MJ, Robins-Browne RM, Yang J, Tauschek M: RegR virulence regulon of rabbit-specific enteropathogenic Escherichia coli strain E22. Infect Immun; 2013 Apr;81(4):1078-89
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  • [Title] RegR virulence regulon of rabbit-specific enteropathogenic Escherichia coli strain E22.
  • AraC-like regulators play a key role in the expression of virulence factors in enteric pathogens, such as enteropathogenic Escherichia coli (EPEC), enterotoxigenic E. coli, enteroaggregative E. coli, and Citrobacter rodentium.
  • Bioinformatic analysis of the genome of rabbit-specific EPEC (REPEC) strain E22 (O103:H2) revealed the presence of a gene encoding an AraC-like regulatory protein, RegR, which shares 71% identity to the global virulence regulator, RegA, of C. rodentium.
  • Taken together, our findings indicate that RegR controls the expression of a series of accessory adhesins that significantly enhance the virulence of REPEC strain E22.
  • [MeSH-major] Bacterial Proteins / metabolism. Enteropathogenic Escherichia coli / genetics. Enteropathogenic Escherichia coli / pathogenicity. Gene Expression Regulation, Bacterial. Regulon. Transcription Factors / metabolism. Virulence Factors / biosynthesis
  • [MeSH-minor] Animals. Computational Biology. DNA, Bacterial / metabolism. Disease Models, Animal. Electrophoretic Mobility Shift Assay. Escherichia coli Infections / microbiology. Escherichia coli Infections / pathology. Escherichia coli Proteins / analysis. Fimbriae Proteins / biosynthesis. Fimbriae Proteins / genetics. Gene Expression Profiling. Microarray Analysis. Promoter Regions, Genetic. Protein Binding. Proteome / analysis. Rabbits. Transcription, Genetic

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  • (PMID = 23340312.001).
  • [ISSN] 1098-5522
  • [Journal-full-title] Infection and immunity
  • [ISO-abbreviation] Infect. Immun.
  • [Language] eng
  • [Databank-accession-numbers] GEO/ GSE41093
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Bacterial Proteins; 0 / DNA, Bacterial; 0 / Escherichia coli Proteins; 0 / Proteome; 0 / RegR protein, bacteria; 0 / Transcription Factors; 0 / Virulence Factors; 147680-16-8 / Fimbriae Proteins
  • [Other-IDs] NLM/ PMC3639590
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31. Monaghan Á, Byrne B, Fanning S, Sweeney T, McDowell D, Bolton DJ: Serotypes and virulence profiles of atypical enteropathogenic Escherichia coli (EPEC) isolated from bovine farms and abattoirs. J Appl Microbiol; 2013 Feb;114(2):595-603
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Serotypes and virulence profiles of atypical enteropathogenic Escherichia coli (EPEC) isolated from bovine farms and abattoirs.
  • AIMS: The objective of this study was to examine the prevalence of enteropathogenic Escherichia coli (EPEC) on beef and dairy farms and in beef abattoirs and to characterize the isolates in terms of serogroup and virulence markers.
  • All 140 isolates obtained were atypical EPEC (aEPEC), while θ and β intimin types were common.
  • [MeSH-major] Cattle / microbiology. Enteropathogenic Escherichia coli / classification. Enteropathogenic Escherichia coli / pathogenicity

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  • [Copyright] © 2012 The Society for Applied Microbiology.
  • (PMID = 23163884.001).
  • [ISSN] 1365-2672
  • [Journal-full-title] Journal of applied microbiology
  • [ISO-abbreviation] J. Appl. Microbiol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Virulence Factors
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32. Ochoa TJ, Contreras CA: Enteropathogenic escherichia coli infection in children. Curr Opin Infect Dis; 2011 Oct;24(5):478-83
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Enteropathogenic escherichia coli infection in children.
  • PURPOSE OF REVIEW: Enteropathogenic Escherichia coli (EPEC) is an important diarrheal pathogen of young children.
  • As the diagnosis of EPEC is now based mainly on molecular criteria, there has been an important change in its prevalence.
  • The purpose of this study is to review the current epidemiology of EPEC infection and the new insights into its physiopathology.
  • RECENT FINDINGS: Recent epidemiological studies indicate that atypical EPEC (aEPEC) is more prevalent than typical EPEC (tEPEC) in both developed and developing countries, and that aEPEC is important in both pediatric endemic diarrhea and diarrhea outbreaks.
  • However, the exact mechanisms of diarrhea in EPEC infection are not completely understood.
  • SUMMARY: Remarkable progress has been made to identify virulence determinants required to mediate the pathogenesis of EPEC.
  • [MeSH-major] Enteropathogenic Escherichia coli. Escherichia coli Infections / epidemiology

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  • (PMID = 21857511.001).
  • [ISSN] 1473-6527
  • [Journal-full-title] Current opinion in infectious diseases
  • [ISO-abbreviation] Curr. Opin. Infect. Dis.
  • [Language] eng
  • [Grant] United States / FIC NIH HHS / TW / 1K01TW007405; United States / FIC NIH HHS / TW / K01 TW007405; United States / FIC NIH HHS / TW / K01 TW007405-04
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Review
  • [Publication-country] United States
  • [Other-IDs] NLM/ NIHMS348474; NLM/ PMC3277943
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33. Contreras CA, Ochoa TJ, Ruiz J, Lacher DW, Durand D, DebRoy C, Lanata CF, Cleary TG: Genetic diversity of locus of enterocyte effacement genes of enteropathogenic Escherichia coli isolated from Peruvian children. J Med Microbiol; 2012 Aug;61(Pt 8):1114-20
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  • [Title] Genetic diversity of locus of enterocyte effacement genes of enteropathogenic Escherichia coli isolated from Peruvian children.
  • The aim of this study was to determine the frequency and allele associations of locus of enterocyte effacement encoded esp and tir genes among 181 enteropathogenic Escherichia coli (EPEC) strains (90 diarrhoea-associated and 91 controls) isolated from Peruvian children under 18 months of age.
  • EPEC strains were isolated with higher frequency from healthy controls (91/424, 21.7%) than from diarrhoeal samples (90/936, 9.6%) (P<0.001); 28.9% of diarrhoeal and 17.6% of control samples were typical EPEC (tEPEC).
  • The distribution of espA alleles (alpha, beta, beta2 and gamma) and espD alleles (alpha, beta, gamma and a new variant, espD-N1) between tEPEC and atypical EPEC (aEPEC) was significantly different (P<0.05).
  • Our findings indicate that there is a high degree of heterogeneity among EPEC strains isolated from Peruvian children and that aEPEC and tEPEC variants cluster.
  • [MeSH-major] Enteropathogenic Escherichia coli / genetics. Enteropathogenic Escherichia coli / isolation & purification. Escherichia coli Infections / epidemiology. Escherichia coli Infections / microbiology. Escherichia coli Proteins / genetics. Genetic Variation. Phosphoproteins / genetics

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  • (PMID = 22493278.001).
  • [ISSN] 1473-5644
  • [Journal-full-title] Journal of medical microbiology
  • [ISO-abbreviation] J. Med. Microbiol.
  • [Language] eng
  • [Databank-accession-numbers] GENBANK/ JN571730/ JN571731/ JN571732/ JN571733
  • [Grant] United States / FIC NIH HHS / TW / 1K01TW007405; United States / NICHD NIH HHS / HD / R01-HD051716
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / DNA, Bacterial; 0 / Escherichia coli Proteins; 0 / LEE protein, E coli; 0 / Phosphoproteins
  • [Other-IDs] NLM/ PMC3542133
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34. Quintero-Villegas MI, Aam BB, Rupnow J, Sørlie M, Eijsink VG, Hutkins RW: Adherence inhibition of enteropathogenic Escherichia coli by chitooligosaccharides with specific degrees of acetylation and polymerization. J Agric Food Chem; 2013 Mar 20;61(11):2748-54
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Adherence inhibition of enteropathogenic Escherichia coli by chitooligosaccharides with specific degrees of acetylation and polymerization.
  • Adherence of enteropathogenic Escherichia coli (EPEC) to the surface of a human HEp-2 cell line was determined in the absence or presence of the various CHOS fractions.
  • The results showed that all CHOS fractions inhibited adherence of EPEC to HEp-2 cells.
  • [MeSH-major] Bacterial Adhesion / drug effects. Enteropathogenic Escherichia coli / drug effects. Oligosaccharides / chemistry. Oligosaccharides / pharmacology

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  • (PMID = 23428168.001).
  • [ISSN] 1520-5118
  • [Journal-full-title] Journal of agricultural and food chemistry
  • [ISO-abbreviation] J. Agric. Food Chem.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Oligosaccharides
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35. Zahavi EE, Lieberman JA, Donnenberg MS, Nitzan M, Baruch K, Rosenshine I, Turner JR, Melamed-Book N, Feinstein N, Zlotkin-Rivkin E, Aroeti B: Bundle-forming pilus retraction enhances enteropathogenic Escherichia coli infectivity. Mol Biol Cell; 2011 Jul 15;22(14):2436-47
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  • [Title] Bundle-forming pilus retraction enhances enteropathogenic Escherichia coli infectivity.
  • Enteropathogenic Escherichia coli (EPEC) is an important human pathogen that causes acute infantile diarrhea.
  • The type IV bundle-forming pili (BFP) of typical EPEC strains are dynamic fibrillar organelles that can extend out and retract into the bacterium.
  • How pili retraction contributes to EPEC pathogenesis at the cellular level remains largely obscure, however.
  • In this study, an effort has been made to address this question using engineered EPEC strains with induced BFP retraction capacity.
  • Our studies hence suggest novel insights into the involvement of pili retraction in EPEC pathogenesis.
  • [MeSH-major] Enteropathogenic Escherichia coli / pathogenicity. Escherichia coli Infections / microbiology. Fimbriae, Bacterial / physiology
  • [MeSH-minor] Animals. Arabinose / metabolism. Caco-2 Cells. Cell Line. Diarrhea, Infantile / microbiology. Dogs. Escherichia coli Proteins / genetics. Escherichia coli Proteins / metabolism. HeLa Cells. Humans. Infant, Newborn. Phosphorylation. Tyrosine / metabolism

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  • HSDB. structure - L-TYROSINE.
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  • (PMID = 21613538.001).
  • [ISSN] 1939-4586
  • [Journal-full-title] Molecular biology of the cell
  • [ISO-abbreviation] Mol. Biol. Cell
  • [Language] eng
  • [Grant] United States / NIAID NIH HHS / AI / R01 AI037606; United States / NIGMS NIH HHS / GM / T32 GM008181
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / BfpF protein, E coli; 0 / Escherichia coli Proteins; 42HK56048U / Tyrosine; B40ROO395Z / Arabinose
  • [Other-IDs] NLM/ PMC3135470
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36. Al Hilali SA, Almohana AM: Occurrence and molecular characterization of enteropathogenic Escherichia coli serotypes isolated from children with diarrhoea in Najaf, Iraq. Indian J Med Microbiol; 2011 Oct-Dec;29(4):383-8
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  • [Title] Occurrence and molecular characterization of enteropathogenic Escherichia coli serotypes isolated from children with diarrhoea in Najaf, Iraq.
  • PURPOSE: Enteropathogenic Escherichia coli (EPEC) are among the most important pathogens infecting children worldwide and are one of the main causes of diarrhoea.
  • The study was carried out to investigate the occurrence of EPEC as a cause of infectious diarrhoea in children younger than 2 years of age and characterize their virulence genes.
  • MATERIALS AND METHODS: During the study period, a total of 656 faecal specimens from children with diarrhoea and 54 from healthy children were analyzed. E. coli isolates were serotypically identified with EPEC polyvalent and monovalent antisera.
  • The isolated EPEC were examined for the presence of the attaching and effacing (eaeA), bundle-forming pilus (bfpA), Shiga like toxins (stx₁ and stx₂), enterohaemorrhagic E. coli enterohaemolysin (EHEC hlyA) and EPEC adherence factor (EAF) genes by the PCR assay.
  • RESULTS: The study has shown that 22 (3.4%) had diarrhoea due to EPEC, while no EPEC isolates were detected in asymptomatic children.
  • The highest number of the EPEC isolated belonging to polyvalent 2.
  • The primers encoding virulence genes were subjected to all the EPEC isolates.
  • Typical EPEC (eaeA⁺, bfpA⁺) was diagnosed in two isolates, while, atypical EPEC was manifested in four isolates.
  • CONCLUSIONS: According to the results, the frequency of EPEC isolates in Najaf was lower than what has been suspected and the investigation including the use of molecular technique and serotyping, are necessary to allow precise identification and epidemiological study of these pathogens.
  • [MeSH-major] Enteropathogenic Escherichia coli / classification. Enteropathogenic Escherichia coli / isolation & purification. Escherichia coli Infections / microbiology. Virulence Factors / genetics
  • [MeSH-minor] Escherichia coli Proteins / genetics. Feces / microbiology. Genotype. Humans. Infant. Iraq. Polymerase Chain Reaction. Serotyping

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  • (PMID = 22120799.001).
  • [ISSN] 1998-3646
  • [Journal-full-title] Indian journal of medical microbiology
  • [ISO-abbreviation] Indian J Med Microbiol
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] India
  • [Chemical-registry-number] 0 / Escherichia coli Proteins; 0 / Virulence Factors
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37. Magalhães CA, Rossato SS, Barbosa AS, Santos TO, Elias WP, Sircili MP, Piazza RM: The ability of haemolysins expressed by atypical enteropathogenic Escherichia coli to bind to extracellular matrix components. Mem Inst Oswaldo Cruz; 2011 Mar;106(2):146-52
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  • [Title] The ability of haemolysins expressed by atypical enteropathogenic Escherichia coli to bind to extracellular matrix components.
  • Typical and atypical enteropathogenic Escherichia coli (EPEC) are considered important bacterial causes of diarrhoea.
  • Considering the repertoire of virulence genes, atypical EPEC (aEPEC) is a heterogeneous group, harbouring genes that are found in other diarrheagenic E. coli pathotypes, such as those encoding haemolysins.
  • [MeSH-major] Enteropathogenic Escherichia coli / physiology. Escherichia coli Proteins / physiology. Extracellular Matrix / metabolism

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  • (PMID = 21537672.001).
  • [ISSN] 1678-8060
  • [Journal-full-title] Memórias do Instituto Oswaldo Cruz
  • [ISO-abbreviation] Mem. Inst. Oswaldo Cruz
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Brazil
  • [Chemical-registry-number] 0 / Escherichia coli Proteins; 0 / Hemolysin Proteins; 0 / Virulence Factors; 0 / enterohemolysin
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38. Liu J, Wang WD, Liu YJ, Liu S, Zhou B, Zhu LW, Ji X, Sun Y, Feng SZ: Mice vaccinated with enteropathogenic Escherichia coli ghosts show significant protection against lethal challenges. Lett Appl Microbiol; 2012 Mar;54(3):255-62
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  • [Title] Mice vaccinated with enteropathogenic Escherichia coli ghosts show significant protection against lethal challenges.
  • AIM: To prepare enteropathogenic Escherichia coli (EPEC) E2348/69 ghosts and investigate whether immunization with EPEC bacterial ghosts can elicit protective immune responses.
  • An EPEC reference strain E2348/69 (serotype O127:H6) was transformed with the lysis plasmids to produce EPEC ghosts.
  • Mice injected with bacterial ghosts EGE (EPEC ghosts produced using lysis protein E) or EGES (EPEC ghosts produced using a combination of lysis protein E and SNA) gained weight normally and showed no clinical signs of disease.
  • Vaccination trials showed that mice immunized with EGE or EGES were significantly protected against subsequent challenge with the wild-type virulent parent strain, EPEC E2348/69 (42/50 and 45/50 survival, respectively); in contrast, none of the 30 control mice survived.
  • CONCLUSIONS: Immunization with EPEC ghosts can elicit protective immune responses in BALB/c mice.
  • SIGNIFICANCE AND IMPACT OF THE STUDY: EPEC ghosts may represent a promising new approach for vaccination against EPEC infection.
  • [MeSH-major] Bacterial Vaccines / immunology. Enteropathogenic Escherichia coli / immunology. Escherichia coli Infections / prevention & control

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  • [Copyright] © 2011 The Authors. Letters in Applied Microbiology © 2011 The Society for Applied Microbiology.
  • (PMID = 22211792.001).
  • [ISSN] 1472-765X
  • [Journal-full-title] Letters in applied microbiology
  • [ISO-abbreviation] Lett. Appl. Microbiol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Antibodies, Bacterial; 0 / Bacterial Vaccines; 0 / Immunoglobulin G
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39. Bakhshi B, Fallahzad S, Pourshafie MR: The occurrence of atypical enteropathogenic Escherichia coli strains among children with diarrhea in Iran. J Infect Chemother; 2013 Aug;19(4):615-20
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  • [Title] The occurrence of atypical enteropathogenic Escherichia coli strains among children with diarrhea in Iran.
  • The aim of this study was to investigate the genetic diversity and class 2 integron content of typical and atypical enteropathogenic Escherichia coli (EPEC) strains isolated from children less than 5 years of age.
  • The identity of EPEC and their class 2 integron content was confirmed by PCR analysis and sequencing.
  • Subtyping of Escherichia coli spp. was performed through pulsed-field gel electrophoresis (PFGE) analysis.
  • All EPEC strains were resistant to 6 antimicrobial agents except for gentamycin.
  • The most prevalent serogroups among EPEC strains were found to be members of O86 and O127 serogroups (37.7 %) and O44, O125, and O128 (42.8 %).
  • The majority of our EPEC isolates (60.7 %) were identified as atypical.
  • No PFGE clustering was observed according to bundle-forming pilus (bfp) bacteria, suggesting that PFGE analysis could not discriminate between typical and atypical EPEC strains.
  • The high ratio of antibiotic-resistant strains and the large heterogeneity among EPEC isolates with low prevalence of class 2 integrons signify the need to examine for other mechanism(s) involved in conferring resistance in typical and atypical populations of EPEC.
  • [MeSH-major] Diarrhea / microbiology. Enteropathogenic Escherichia coli / genetics. Escherichia coli Infections / microbiology

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  • (PMID = 23203219.001).
  • [ISSN] 1437-7780
  • [Journal-full-title] Journal of infection and chemotherapy : official journal of the Japan Society of Chemotherapy
  • [ISO-abbreviation] J. Infect. Chemother.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Japan
  • [Chemical-registry-number] 0 / Anti-Bacterial Agents; 0 / DNA, Bacterial
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40. Orton DJ, Arsenault DJ, Thomas NA, Doucette AA: GELFrEE fractionation combined with mass spectrometry for proteome analysis of secreted toxins from Enteropathogenic Escherichia coli (EPEC). Mol Cell Probes; 2013 Oct-Dec;27(5-6):200-7
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  • [Title] GELFrEE fractionation combined with mass spectrometry for proteome analysis of secreted toxins from Enteropathogenic Escherichia coli (EPEC).
  • Enteropathogenic Escherichia coli, or EPEC, is a human pathogen associated with gastroenteritis and diarrheal disease whose pathogenicity is related to the secretion of effector proteins (exotoxins).
  • Here, we apply a solution-phase electrophoretic platform (GELFrEE) followed by MS to characterize the secreted proteome of a wild type and mutant strain of EPEC (ΔsepD), exhibiting enhanced secretion of effector proteins.
  • Semi-quantitative analysis of the MS data reveals the effector proteins EspB, EspC, and EspD appear in a relatively greater abundance from wild type EPEC, while two major virulence factors in EPEC, Tir and NleA appear in greater abundance from the secreted proteome of the mutant strain.
  • This study demonstrates the application of GELFrEE-MS to differentiate wild type and mutant strains of EPEC.
  • [MeSH-major] Bacterial Toxins / analysis. Electrophoresis, Polyacrylamide Gel. Enteropathogenic Escherichia coli / metabolism. Enteropathogenic Escherichia coli / pathogenicity. Escherichia coli Proteins / analysis. Mass Spectrometry. Proteome / analysis

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  • [Copyright] Copyright © 2013 Elsevier Ltd. All rights reserved.
  • [RepublishedIn] Mol Cell Probes. 2014 Apr-Jun;28(2-3):83-90 [24486296.001]
  • (PMID = 23831145.001).
  • [ISSN] 1096-1194
  • [Journal-full-title] Molecular and cellular probes
  • [ISO-abbreviation] Mol. Cell. Probes
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Bacterial Outer Membrane Proteins; 0 / Bacterial Toxins; 0 / EaeB protein, E coli; 0 / Escherichia coli Proteins; 0 / EspC protein, E coli; 0 / EspD protein, E coli; 0 / NleA protein, E coli; 0 / Proteome; 0 / Receptors, Cell Surface; 0 / Tir protein, E coli; 0 / Virulence Factors
  • [Keywords] NOTNLM ; Enteropathogenic Escherichia coli / GELFrEE / Mass spectrometry / Proteomics / Secreted proteins / Toxins
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41. Nakhjavani FA, Emaneini M, Hosseini H, Iman-Eini H, Aligholi M, Jabalameli F, Haghi-Ashtiani MT, Taherikalani M, Mirsalehian A: Molecular analysis of typical and atypical enteropathogenic Escherichia coli (EPEC) isolated from children with diarrhoea. J Med Microbiol; 2013 Feb;62(Pt 2):191-5
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  • [Title] Molecular analysis of typical and atypical enteropathogenic Escherichia coli (EPEC) isolated from children with diarrhoea.
  • To investigate the incidence, antimicrobial resistance and genetic relationships of enteropathogenic Escherichia coli (EPEC) in children with diarrhoea, a total of 612 stool specimens were collected in Tehran, Iran, and cultured to isolate strains of EPEC.
  • The genetic relationships between EPEC isolates were determined by pulsed-field gel electrophoresis (PFGE).
  • Out of the 412 strains of E. coli obtained from 612 diarrhoeal stool specimens, 23 (5.6 %) were identified as EPEC, of which seven (30.4 %) were classified as typical strains of EPEC and 16 (69.6 %) were classified as atypical.
  • Out of the 23 EPEC isolates, 69.5 % were resistant to ampicillin, 39.1 % were resistant to tetracycline and cotrimoxazole, 30.4 % were resistant to cefpodoxime, ceftazidime, ceftriaxone and aztreonam, and 26.1 % were resistant to imipenem.
  • The present study shows that typical and atypical EPEC isolates displayed considerable heterogeneity in PFGE profiles and EPEC infections were only sporadic in Tehran.
  • [MeSH-major] Diarrhea / microbiology. Drug Resistance, Multiple, Bacterial. Enteropathogenic Escherichia coli / drug effects. Escherichia coli Infections / microbiology. Feces / microbiology
  • [MeSH-minor] Adhesins, Bacterial / genetics. Child. Child, Preschool. DNA, Bacterial / genetics. Escherichia coli Proteins / genetics. Fimbriae Proteins / genetics. Humans. Infant. Microbial Sensitivity Tests. Shiga Toxin / genetics

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  • (PMID = 23065543.001).
  • [ISSN] 1473-5644
  • [Journal-full-title] Journal of medical microbiology
  • [ISO-abbreviation] J. Med. Microbiol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Adhesins, Bacterial; 0 / BfpA protein, E coli; 0 / DNA, Bacterial; 0 / Escherichia coli Proteins; 147094-99-3 / eaeA protein, E coli; 147680-16-8 / Fimbriae Proteins; 75757-64-1 / Shiga Toxin
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42. Nisa S, Hazen TH, Assatourian L, Nougayrède JP, Rasko DA, Donnenberg MS: In vitro evolution of an archetypal enteropathogenic Escherichia coli strain. J Bacteriol; 2013 Oct;195(19):4476-83
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  • [Title] In vitro evolution of an archetypal enteropathogenic Escherichia coli strain.
  • Enteropathogenic Escherichia coli (EPEC) is a leading cause of infantile diarrhea in developing countries.
  • EPEC strain E2348/69 is used worldwide as a prototype to study EPEC genetics and disease.
  • To identify the genomic and phenotypic changes in the prototype strain, we sequenced the genome of the nalidixic acid-resistant (Nal(r)) E2348/69 clone.
  • We also sequenced a recent nleF mutant derived by one-step PCR mutagenesis from the Nal(r) strain.
  • The sequencing results revealed no unintended changes between the mutant and the parent strain.
  • This report reconciles longstanding inconsistencies in phenotypic properties of an archetypal strain and provides both reassurance and cautions regarding intentional and unintentional evolution in vitro.
  • [MeSH-major] Enteropathogenic Escherichia coli / genetics. Enteropathogenic Escherichia coli / metabolism

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  • (PMID = 23913321.001).
  • [ISSN] 1098-5530
  • [Journal-full-title] Journal of bacteriology
  • [ISO-abbreviation] J. Bacteriol.
  • [Language] eng
  • [Databank-accession-numbers] GENBANK/ ASZR00000000/ ASZS00000000
  • [Grant] United States / NIAID NIH HHS / AI / U19 AI090873; United States / NIAID NIH HHS / AI / U19AI090873
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] United States
  • [Other-IDs] NLM/ PMC3807458
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43. Sakkejha H, Byrne L, Lawson AJ, Jenkins C: An update on the microbiology and epidemiology of enteropathogenic Escherichia coli in England 2010-2012. J Med Microbiol; 2013 Oct;62(Pt 10):1531-4
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  • [Title] An update on the microbiology and epidemiology of enteropathogenic Escherichia coli in England 2010-2012.
  • Historically, enteropathogenic Escherichia coli (EPEC) are a well-known cause of outbreaks of infantile diarrhoea associated with morbidity and mortality in England.
  • The aim of this study was to provide an update on the microbiology and epidemiology of strains of EPEC in England between 2010 and 2012.
  • A wide range of E. coli serogroups were identified, with the most common being E. coli O145, O49 and O157.
  • Few isolates (9%) had additional virulence factors (specifically bfp, vtx2f and espT genes) and the majority were classified as atypical EPEC.
  • No outbreaks were reported during this period; however, the data indicated that EPEC are still an important cause of sporadic cases of infantile diarrhoea in England.
  • [MeSH-major] Enteropathogenic Escherichia coli / isolation & purification. Escherichia coli Infections / epidemiology. Escherichia coli Infections / microbiology

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  • (PMID = 23893920.001).
  • [ISSN] 1473-5644
  • [Journal-full-title] Journal of medical microbiology
  • [ISO-abbreviation] J. Med. Microbiol.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Virulence Factors
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44. Reis RS, Horn F: Enteropathogenic Escherichia coli, Samonella, Shigella and Yersinia: cellular aspects of host-bacteria interactions in enteric diseases. Gut Pathog; 2010;2(1):8
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  • [Title] Enteropathogenic Escherichia coli, Samonella, Shigella and Yersinia: cellular aspects of host-bacteria interactions in enteric diseases.
  • A successful infection of the human intestine by enteropathogenic bacteria depends on the ability of bacteria to attach and colonize the intestinal epithelium and, in some cases, to invade the host cell, survive intracellularly and disseminate from cell to cell.
  • Here we overview the most important molecular strategies developed by enteropathogenic Escherichia coli, Salmonella enterica, Shigella flexneri, and Yersinia enterocolitica to cause enteric infections.

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  • (PMID = 20649986.001).
  • [ISSN] 1757-4749
  • [Journal-full-title] Gut pathogens
  • [ISO-abbreviation] Gut Pathog
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Other-IDs] NLM/ PMC2921366
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45. Liberatore AM, Moreira FC, Gomes TA, Menchaca-Diaz JL, Koh IH: Typical and atypical enteropathogenic Escherichia coli bacterial translocation associated with tissue hypoperfusion in rats. Braz J Med Biol Res; 2011 Oct;44(10):1018-24
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  • [Title] Typical and atypical enteropathogenic Escherichia coli bacterial translocation associated with tissue hypoperfusion in rats.
  • Although enteropathogenic Escherichia coli (EPEC) are well-recognized diarrheal agents, their ability to translocate and cause extraintestinal alterations is not known.
  • We investigated whether a typical EPEC (tEPEC) and an atypical EPEC (aEPEC) strain translocate and cause microcirculation injury under conditions of intestinal bacterial overgrowth.
  • Recovery of the positive control E. coli R-6 (N = 6) was 100% for all compartments.
  • Bacteria were not recovered from extraintestinal sites of controls inoculated with non-pathogenic E. coli strains HB101 (N = 6) and HS (N = 10), or saline.
  • Mesenteric microcirculation injuries were detected with both EPEC strains, but only aEPEC was similar to E. coli R-6 with regard to systemic tissue hypoperfusion.
  • [MeSH-major] Bacterial Translocation / physiology. Enteropathogenic Escherichia coli / physiology. Escherichia coli Infections / microbiology. Intestines / microbiology. Microcirculation

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  • (PMID = 21989977.001).
  • [ISSN] 1414-431X
  • [Journal-full-title] Brazilian journal of medical and biological research = Revista brasileira de pesquisas médicas e biológicas / Sociedade Brasileira de Biofísica ... [et al.]
  • [ISO-abbreviation] Braz. J. Med. Biol. Res.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Brazil
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46. Bouzari S, Aslani MM, Oloomi M, Jafari A, Dashti A: Comparison of multiplex PCR with serogrouping and PCR-RFLP of fliC gene for the detection of enteropathogenic Escherichia coli (EPEC). Braz J Infect Dis; 2011 Jul-Aug;15(4):365-9
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  • [Title] Comparison of multiplex PCR with serogrouping and PCR-RFLP of fliC gene for the detection of enteropathogenic Escherichia coli (EPEC).
  • Enteropathogenic Escherichia coli (EPEC) comprise one of the six categories of diarrhoeagenic E. coli (DEC).
  • EPEC is subgrouped into typical (tEPEC) and atypical (aEPEC).
  • The identification of DEC cannot be based only on cultural and biochemical criteria, since they are indistinguishable from the non-pathogenic E. coli commonly found in human feces.
  • In the present study five hundred E. coli isolates from children with diarrhea were subjected into multiplex PCR.
  • The results obtained revealed that overall 41 (8.2%) isolates could be detected as EPEC by this multiplex PCR assay.
  • Of these isolates; 27 (66%) were typical (escv+, bfp+) and 14 (34%) atypical EPEC (escv+, bfp-).
  • [MeSH-major] DNA, Bacterial / analysis. Enteropathogenic Escherichia coli / classification. Escherichia coli Proteins / genetics. Multiplex Polymerase Chain Reaction. O Antigens / analysis. Polymorphism, Restriction Fragment Length

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  • (PMID = 21861008.001).
  • [ISSN] 1678-4391
  • [Journal-full-title] The Brazilian journal of infectious diseases : an official publication of the Brazilian Society of Infectious Diseases
  • [ISO-abbreviation] Braz J Infect Dis
  • [Language] eng
  • [Publication-type] Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Brazil
  • [Chemical-registry-number] 0 / DNA, Bacterial; 0 / Escherichia coli Proteins; 0 / FliC protein, E coli; 0 / O Antigens; 0 / Shiga Toxin 1; 0 / Shiga Toxin 2
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47. Mainil JG, Bardiau M, Ooka T, Ogura Y, Murase K, Etoh Y, Ichihara S, Horikawa K, Buvens G, Piérard D, Itoh T, Hayashi T: Typing of O26 enterohaemorrhagic and enteropathogenic Escherichia coli isolated from humans and cattle with IS621 multiplex PCR-based fingerprinting. J Appl Microbiol; 2011 Sep;111(3):773-86
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  • [Title] Typing of O26 enterohaemorrhagic and enteropathogenic Escherichia coli isolated from humans and cattle with IS621 multiplex PCR-based fingerprinting.
  • AIMS: This study evaluated a typing method of O26:H11 enterohaemorrhagic and enteropathogenic Escherichia coli (EHEC and EPEC) based on the variation in genomic location and copy numbers of IS621.
  • Thirty-eight amplification profiles were observed amongst a collection of 69 human and bovine O26:H11 EHEC and EPEC.
  • Seventy-one per cent of the 45 EHEC and EPEC with identical IS621 fingerprints within groups of two, three or four isolates had >85% pulsed field gel electrophoresis (PFGE) profile similarity, including four groups of epidemiologically related EHEC or EPEC, while most of the groups had <85% similarity between each others.
  • CONCLUSIONS: The IS621 fingerprinting and the PFGE are complementary typing assays of EHEC and EPEC; though, the former is less discriminatory.
  • SIGNIFICANCE AND IMPACT OF THE STUDY: The IS621 printing method represents a rapid (24 h) first-line surveillance and typing assay, to compare and trace back O26:H11 EHEC and EPEC during surveys in farms, multiple human cases and outbreaks.
  • [MeSH-major] Bacterial Typing Techniques / methods. DNA Fingerprinting / methods. Enterohemorrhagic Escherichia coli / classification. Enteropathogenic Escherichia coli / classification. Multiplex Polymerase Chain Reaction / methods

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  • [Copyright] Journal of Applied Microbiology © 2011 The Society for Applied Microbiology. No claim to Belgian Government works.
  • (PMID = 21707882.001).
  • [ISSN] 1365-2672
  • [Journal-full-title] Journal of applied microbiology
  • [ISO-abbreviation] J. Appl. Microbiol.
  • [Language] eng
  • [Publication-type] Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / DNA, Bacterial; 0 / O Antigens
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48. Behiry IK, Abada EA, Ahmed EA, Labeeb RS: Enteropathogenic Escherichia coli associated with diarrhea in children in Cairo, Egypt. ScientificWorldJournal; 2011;11:2613-9
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  • [Title] Enteropathogenic Escherichia coli associated with diarrhea in children in Cairo, Egypt.
  • In this study we isolate and identify the Enteropathogenic Escherichia coli (EPEC) causing diarrhea in children less than five years in Cairo, Egypt, during different seasons.
  • The identified E. coli isolates were subjected to antimicrobial disc diffusion susceptibility test and further identified for EPEC serotype by slide agglutination test, using antiserum E. coli somatic trivalent I (O111, O55, O26) according to the instructions of the manufacturer.
  • Out of 134 patients 5.2% of them revealed EPEC in the fecal sample, while the 20 children control group showed no EPEC isolates in their samples.
  • Our EPEC frequency showed variations from the compared results of other studies.
  • Higher rate of EPEC (18.7%) was found in patients between 2 to 3 years, while EPEC rate was (7.5%) in patients less than 6 months old, with P < 0.05.
  • EPEC was identified from fecal specimens as a unique pathogen or associated with other pathogens in acute and chronic diarrhea in children.
  • EPEC were detected in all seasons except in winter, and was predominant in summer season.
  • Four (57%) EPEC isolates were resistant to ampicillin, ticarcillin, and cotrimoxazole, and (14.3%) to the third generation cephalosporins.
  • [MeSH-major] Diarrhea / microbiology. Enteropathogenic Escherichia coli / isolation & purification

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  • (PMID = 22262949.001).
  • [ISSN] 1537-744X
  • [Journal-full-title] TheScientificWorldJournal
  • [ISO-abbreviation] ScientificWorldJournal
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Anti-Bacterial Agents
  • [Other-IDs] NLM/ PMC3254012
  • [Keywords] NOTNLM ; children less than 5 years / enteropathogenic E. coli / slide agglutination test
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49. Smith K, Humphreys D, Hume PJ, Koronakis V: Enteropathogenic Escherichia coli recruits the cellular inositol phosphatase SHIP2 to regulate actin-pedestal formation. Cell Host Microbe; 2010 Jan 21;7(1):13-24
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Enteropathogenic Escherichia coli recruits the cellular inositol phosphatase SHIP2 to regulate actin-pedestal formation.
  • Adhesion of enteropathogenic Escherichia coli to epithelial cells triggers actin-rich pedestal formation beneath the bacteria.
  • [MeSH-major] Actins / metabolism. Enteropathogenic Escherichia coli / pathogenicity. Epithelial Cells / microbiology. Escherichia coli Proteins / physiology. Phosphoric Monoester Hydrolases / metabolism. Receptors, Cell Surface / physiology. Virulence Factors / physiology

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  • [Copyright] 2010 Elsevier Inc. All rights reserved.
  • [CommentIn] Cell Host Microbe. 2010 Jan 21;7(1):1-2 [20114020.001]
  • (PMID = 20114025.001).
  • [ISSN] 1934-6069
  • [Journal-full-title] Cell host & microbe
  • [ISO-abbreviation] Cell Host Microbe
  • [Language] eng
  • [Grant] United Kingdom / Medical Research Council / / G0500583; United Kingdom / Biotechnology and Biological Sciences Research Council / / ; United Kingdom / Medical Research Council / / ; United Kingdom / Wellcome Trust / /
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Actins; 0 / Carrier Proteins; 0 / Escherichia coli Proteins; 0 / Membrane Proteins; 0 / Phosphatidylinositols; 0 / RAPH1 protein, human; 0 / Receptors, Cell Surface; 0 / Shc Signaling Adaptor Proteins; 0 / Tir protein, E coli; 0 / Virulence Factors; EC 3.1.3.- / INPPL1 protein, human; EC 3.1.3.- / Phosphoric Monoester Hydrolases
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50. Iida M, Okamura N, Yamazaki M, Yatsuyanagi J, Kurazono T, Suzuki R, Hiruta N, Isobe J, Seto K, Kawano K, Narimatsu H, Ratchtrachenchai OA, Okabe N, Ito K: Classification of perA sequences and their correlation with autoaggregation in typical enteropathogenic Escherichia coli isolates collected in Japan and Thailand. Microbiol Immunol; 2010 Apr;54(4):184-95
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Classification of perA sequences and their correlation with autoaggregation in typical enteropathogenic Escherichia coli isolates collected in Japan and Thailand.
  • Enteropathogenic Escherichia coli (EPEC) strains produce a bundle-forming pilus (BFP) that mediates localized adherence (LA) to intestinal epithelial cells.
  • We analyzed perA gene polymorphism among typical (eae- and bfpA-positive) EPEC strains isolated from healthy and diarrheal persons in Japan (n=27) and Thailand (n=26) during the period 1995 to 2007 and compared this with virulence and phenotypic characteristics.
  • Our results showed that functional deficiency due to frameshift mutation and subsequent nonsense mutation in perA reduced BFP expression in typical EPEC strains isolated in Japan.
  • [MeSH-major] Enteropathogenic Escherichia coli / classification. Escherichia coli Proteins. Repressor Proteins
  • [MeSH-minor] Amino Acid Sequence. Bacterial Adhesion. Cell Line. Diarrhea / microbiology. Epithelial Cells / microbiology. Escherichia coli Infections / microbiology. Fimbriae Proteins / chemistry. Fimbriae Proteins / genetics. Fimbriae Proteins / metabolism. Gene Expression Regulation, Bacterial. Hemolysis. Heteroduplex Analysis. Humans. Japan. Molecular Sequence Data. Mutation. Phylogeny. Polymorphism, Genetic. Sequence Analysis, DNA. Serotyping. Thailand. Virulence

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  • (PMID = 20377747.001).
  • [ISSN] 0385-5600
  • [Journal-full-title] Microbiology and immunology
  • [ISO-abbreviation] Microbiol. Immunol.
  • [Language] eng
  • [Databank-accession-numbers] GENBANK/ AB364243/ AB364244/ AB523678/ AB523679/ AB523680/ AB523681/ AB523682/ AB523683/ AB523684/ AB523685/ AB523686/ AB523687/ AB523688/ AB523689/ AB523690/ AB523691/ AB523692/ AB523693/ AB523694/ AB523695/ AB523696/ AB523697/ AB523698/ AB523699/ AB523700/ AB523701/ AB523702
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Japan
  • [Chemical-registry-number] 0 / BfpA protein, E coli; 0 / Escherichia coli Proteins; 0 / Repressor Proteins; 0 / bfpT protein, E coli; 147680-16-8 / Fimbriae Proteins
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