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1. Biomedical articles (top 50; 2009 to 2014)
1. ||||||.... 62%  Seward SL Jr, Gahl WA: Hermansky-Pudlak syndrome: health care throughout life. Pediatrics; 2013 Jul;132(1):153-60
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Hermansky-Pudlak syndrome (HPS) is a rare autosomal recessive disease that displays genetic heterogeneity; there are 9 known subtypes.
  • HPS is characterized by oculocutaneous albinism, a platelet storage pool deficiency and resultant bleeding diathesis, and lysosomal accumulation of ceroid lipofuscin.
  • Patients with HPS, specifically those with the genotypes HPS-1, HPS-2, or HPS-4, are predisposed to interstitial lung disease.
  • [MeSH-minor] Albinism, Oculocutaneous / diagnosis. Albinism, Oculocutaneous / epidemiology. Albinism, Oculocutaneous / genetics. Albinism, Oculocutaneous / therapy. Child. Chromosome Aberrations. Cooperative Behavior. Cross-Cultural Comparison. Cross-Sectional Studies. DNA Mutational Analysis. Disability Evaluation. Early Diagnosis. Genes, Recessive. Genotype. Humans. Interdisciplinary Communication. Phenotype. Platelet Storage Pool Deficiency / diagnosis. Platelet Storage Pool Deficiency / epidemiology. Platelet Storage Pool Deficiency / genetics. Platelet Storage Pool Deficiency / therapy. Puerto Rico

  • Genetic Alliance. consumer health - Hermansky-Pudlak syndrome.
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  • (PMID = 23753089.001).
  • [ISSN] 1098-4275
  • [Journal-full-title] Pediatrics
  • [ISO-abbreviation] Pediatrics
  • [Language] eng
  • [Publication-type] Comparative Study; Journal Article; Review
  • [Publication-country] United States
  • [Keywords] NOTNLM ; Hermansky-Pudlak / albinism / platelet storage pool deficiency / standard of care
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2. |||....... 30%  Cooper IF, Siadaty MS: 'Cells' associated with 'Pearl': Top Publications. BioMedLib Review; Cell;Pearl:705814498. ISSN: 2331-5717. 2014/4/28
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  • Finding such articles is important for researchers, clinicians, and patients.
  • In group one there are publications with the strongest evidence of association. We focused finding the most relevant publications pertinent to our goal, rather than combining them into a conclusion section. Such textual synthesis will be the focus of our next project.
  • Zhen L et al: Abnormal expression and subcellular distribution of subunit proteins of the AP-3 adaptor complex lead to platelet storage pool deficiency in the pearl mouse.
  • Leknes IL: Eosinophilic granule cells and endocytic cells in intestinal wall of pearl gouramy (Anabantidae: Teleostei).
  • O'Brien EP et al: Molecular markers near two mouse chromosome 13 genes, muted and pearl, which cause platelet storage pool deficiency (SPD).

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
  • (UID = 705814498.001).
  • [ISSN] 2331-5717
  • [Journal-full-title] BioMedLib Review
  • [Language] eng
  • [Publication-type] Review
  • [Publication-country] UNITED STATES
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3. |||....... 26%  Cooper IF, Siadaty MS: 'Cells' associated with 'Prolonged Bleeding': Top Publications. BioMedLib Review; Cell;ProlongedBleeding:705824241. ISSN: 2331-5717. 2014/4/28
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  • Finding such articles is important for researchers, clinicians, and patients.
  • In group one there are publications with the strongest evidence of association. We focused finding the most relevant publications pertinent to our goal, rather than combining them into a conclusion section. Such textual synthesis will be the focus of our next project.
  • Stenberg PE et al: Prolonged bleeding time with defective platelet filopodia formation in the Wistar Furth rat.
  • Deckmyn H et al: Disturbed platelet aggregation to collagen associated with an antibody against an 85- to 90-Kd platelet glycoprotein in a patient with prolonged bleeding time.
  • Schleinitz N et al: Monoclonal antibody immunospecific platelet assay in patients with lupus anticoagulant and prolonged bleeding time: Comment on the article by Urbanus et al.
  • Cowles BE et al: Prolonged bleeding time of Chediak-Higashi cats corrected by platelet transfusion.
  • Swank RT et al: Inherited prolonged bleeding time and platelet storage pool deficiency in the subtle gray (sut) mouse.
  • Castaman G et al: Characterization of a novel bleeding disorder with isolated prolonged bleeding time and deficiency of platelet microvesicle generation.
  • Israels SJ et al: Platelet storage pool deficiency: diagnosis in patients with prolonged bleeding times and normal platelet aggregation.
  • Wisløff F et al: Prolonged bleeding time with adequate platelet count in hospital patients.
  • Stavem P et al: Hereditary thrombocytopenia with excessively prolonged bleeding time, corrected by infusions of platelet poor plasma.

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
  • (UID = 705824241.001).
  • [ISSN] 2331-5717
  • [Journal-full-title] BioMedLib Review
  • [Language] eng
  • [Publication-type] Review
  • [Publication-country] UNITED STATES
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4. ||........ 24%  Cooper IF, Siadaty MS: 'Amino Acids, Peptides, or Proteins' associated with 'Deficiency Platelet Storage Pool': Top Publications. BioMedLib Review; AminoAcidPeptide;DeficiencyPlateletStorage:705562357. ISSN: 2331-5717. 2014/4/24
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  • [Title] 'Amino Acids, Peptides, or Proteins' associated with 'Deficiency Platelet Storage Pool': Top Publications.
  • Background: There are articles published each month which present 'amino acid peptide or protein' for 'deficiency platelet storage pool'.
  • Finding such articles is important for researchers, clinicians, and patients.
  • In group one there are publications with the strongest evidence of association. We focused finding the most relevant publications pertinent to our goal, rather than combining them into a conclusion section. Such textual synthesis will be the focus of our next project.
  • Holmsen H et al: Content and thrombin-induced release of acid hydrolases in gel-filtered platelets from patients with storage pool disease.
  • Lages B et al: Decreased content and surface expression of alpha-granule membrane protein GMP-140 in one of two types of platelet alpha delta storage pool deficiency.
  • Zhen L et al: Abnormal expression and subcellular distribution of subunit proteins of the AP-3 adaptor complex lead to platelet storage pool deficiency in the pearl mouse.
  • Arderiu G et al: Phosphotyrosine proteins in platelets from patients with storage pool disease: direct relation between granule defects and defective signal transduction.
  • Lages B et al: Metal ion contents of gel-filtered platelets from patients with storage pool disease.
  • Weiss HJ et al: Platelet prothrombinase activity and intracellular calcium responses in patients with storage pool deficiency, glycoprotein IIb-IIIa deficiency, or impaired platelet coagulant activity--a comparison with Scott syndrome.
  • Weiss HJ et al: Heterogeneity in storage pool deficiency: studies on granule-bound substances in 18 patients including variants deficient in alpha-granules, platelet factor 4, beta-thromboglobulin, and platelet-derived growth factor.
  • Kosch A et al: [Thrombocytic alpha-delta-storage-pool-disease: shortening of bleeding time after infusion of 1-desamino-8-D-arginine vasopressin].
  • Barone I et al: Management of platelet storage pool disease during pregnancy with recombinant factor VIIa.
  • Jamieson GA et al: Platelet membrane glycoproteins in thrombasthenia, Bernard-Soulier syndrome, and storage pool disease.

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
  • (UID = 705562357.001).
  • [ISSN] 2331-5717
  • [Journal-full-title] BioMedLib Review
  • [Language] eng
  • [Publication-type] Review
  • [Publication-country] UNITED STATES
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5. ||........ 21%  Cooper IF, Siadaty MS: 'Cells' associated with 'Albinism Oculocutaneous': Top Publications. BioMedLib Review; Cell;AlbinismOculocutaneous:705725229. ISSN: 2331-5717. 2014/4/26
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  • Finding such articles is important for researchers, clinicians, and patients.
  • In group one there are publications with the strongest evidence of association. We focused finding the most relevant publications pertinent to our goal, rather than combining them into a conclusion section. Such textual synthesis will be the focus of our next project.
  • Johnston M et al: Observation of a unique case of metastatic basal cell carcinoma found by radiographic evaluation in a patient with oculocutaneous albinism.
  • Contopoulos-Ioannidis D et al: Recurrent rhabdomyolysis in a patient with oculocutaneous albinism type 1 and platelet storage-pool deficiency.
  • Hamel BC et al: Oculocutaneous albinism associated with motor neuron disease.
  • Bomalaski JS et al: Oculocutaneous albinism, platelet storage pool disease, and progressive lupus nephritis.
  • Reich S et al: Oculocutaneous albinism accompanied by minor morphologic stigmata and reduced number and function of NK cells. A new variant of NK cell defect?.
  • Witkop CJ Jr et al: Ophthalmologic, biochemical, platelet, and ultrastructural defects in the various types of oculocutaneous albinism.

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
  • (UID = 705725229.001).
  • [ISSN] 2331-5717
  • [Journal-full-title] BioMedLib Review
  • [Language] eng
  • [Publication-type] Review
  • [Publication-country] UNITED STATES
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6. ||........ 21%  Cooper IF, Siadaty MS: 'Steroids' associated with 'Ap 3 Adaptor Complex': Top Publications. BioMedLib Review; Steroid;Ap3Adaptor:705926085. ISSN: 2331-5717. 2014/5/5
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  • Finding such articles is important for researchers, clinicians, and patients.
  • In group one there are publications with the strongest evidence of association. We focused finding the most relevant publications pertinent to our goal, rather than combining them into a conclusion section. Such textual synthesis will be the focus of our next project.
  • Zhen L et al: Abnormal expression and subcellular distribution of subunit proteins of the AP-3 adaptor complex lead to platelet storage pool deficiency in the pearl mouse.

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
  • (UID = 705926085.001).
  • [ISSN] 2331-5717
  • [Journal-full-title] BioMedLib Review
  • [Language] eng
  • [Publication-type] Review
  • [Publication-country] UNITED STATES
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7. ||||||.... 62%  Mora AJ, Wolfsohn DM: The management of gastrointestinal disease in Hermansky-Pudlak syndrome. J Clin Gastroenterol; 2011 Sep;45(8):700-2
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] The management of gastrointestinal disease in Hermansky-Pudlak syndrome.
  • Clinically, HPS is characterized by oculocutaneous albinism, platelet storage pool deficiency, and ceroid tissue accumulation.
  • It is a rare disorder that has been described globally but has the highest frequency in a cluster population in Puerto Rico.

  • Genetic Alliance. consumer health - Hermansky-Pudlak syndrome.
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  • (PMID = 21085008.001).
  • [ISSN] 1539-2031
  • [Journal-full-title] Journal of clinical gastroenterology
  • [ISO-abbreviation] J. Clin. Gastroenterol.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Anti-Bacterial Agents; 0 / Anti-Inflammatory Agents, Non-Steroidal; 0 / Gastrointestinal Agents; 0 / Steroids
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8. ||||||.... 62%  Spencer J, Rosengren S: Hermansky-Pudlak syndrome in pregnancy. Am J Perinatol; 2009 Oct;26(9):617-9
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Hermansky-Pudlak syndrome (HPS) is a multisystem, autosomal-recessive disorder characterized by oculocutaneous albinism, platelet storage pool deficiency resulting in prolonged bleeding, and ceroid lipofuscin deposition.


9. ||||||.... 61%  Harris-Glocker M, Thornburg LL, Pressman EK: Hermansky-Pudlak syndrome in a pregnant patient: a case report. J Reprod Med; 2013 May-Jun;58(5-6):267-70
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • BACKGROUND: Hermansky-Pudlak syndrome (HPS), a rare autosomal-recessive disorder encompassing multiple organs, is characterized by oculocutaneous albinism, platelet storage pool deficiency resulting in bleeding diathesis, and ceroid lipofuscin deposition which can lead to pulmonary fibrosis, colitis, cardiomyopathy and renal failure.
  • CASE: We present the case of a patient with HPS also complicated by spontaneous triplet pregnancy.
  • [MeSH-minor] Adult. Albinism, Oculocutaneous. Cesarean Section. Deamino Arginine Vasopressin / administration & dosage. Female. Gestational Age. Hemostatics. Humans. Platelet Storage Pool Deficiency. Pregnancy. Pregnancy Outcome. Premature Birth


10. ||||||.... 56%  Vo KT, Grooms L, Klima J, Holland-Hall C, O'Brien SH: Menstrual bleeding patterns and prevalence of bleeding disorders in a multidisciplinary adolescent haematology clinic. Haemophilia; 2013 Jan;19(1):71-5
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Sixty-two per cent of patients were diagnosed with a bleeding disorder, including platelet storage pool deficiency (36%), von Willebrand's disease (9%), other platelet function defect (8%), Ehlers-Danlos syndrome (7%) and combined bleeding disorders (2%).
  • Comparison of the bleeding profiles for females with and without a bleeding disorder revealed only three factors that were significantly different, including the reported regularity of patients' periods (P = 0.02), description of period flow (P = 0.04) and number of days of each period that the bleeding was described as 'heavy' (P = 0.007).
  • Specifically, a relatively high proportion of adolescents were diagnosed with platelet storage pool deficiency.
  • In our small population, menstrual bleeding profiles, as examined by a standardized questionnaire, could not identify females with an underlying bleeding disorder, demonstrating the important role of haemostasis testing in the evaluation of adolescents with HMB.
  • [MeSH-minor] Adolescent. Child. Female. Humans. Ohio / epidemiology. Platelet Storage Pool Deficiency / complications. Prevalence. Retrospective Studies

  • MedlinePlus Health Information. consumer health - Menstruation.
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  • [Copyright] © 2012 Blackwell Publishing Ltd.
  • (PMID = 23005346.001).
  • [ISSN] 1365-2516
  • [Journal-full-title] Haemophilia : the official journal of the World Federation of Hemophilia
  • [ISO-abbreviation] Haemophilia
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
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11. ||||...... 38%  Bachmann C, Abele H, Wallwiener D, Kagan KO: Neonatal and maternal risk in Hermansky-Pudlak syndrome: peripartum management-brief report and review of literature. Arch Gynecol Obstet; 2014 Jun;289(6):1193-5
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • HPS is a rare disease and belongs to a heterogeneous group of autosomal recessive disorders characterized by the triad of partial oculocutaneous albinism, disorder of "ceroid" metabolism and platelet storage pool deficiency with bleeding disorder.

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  • (PMID = 24337786.001).
  • [ISSN] 1432-0711
  • [Journal-full-title] Archives of gynecology and obstetrics
  • [ISO-abbreviation] Arch. Gynecol. Obstet.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Germany
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12. ||||...... 37%  Carmona-Rivera C, Hess RA, O'Brien K, Golas G, Tsilou E, White JG, Gahl WA, Huizing M: Novel mutations in the HPS1 gene among Puerto Rican patients. Clin Genet; 2011 Jun;79(6):561-7
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Hermansky-Pudlak syndrome (HPS) is a disorder of oculocutaneous albinism (OCA) and platelet storage pool deficiency.
  • Eight different disease-causing genes have been identified, whose gene products are thought to be involved in the biogenesis of lysosome-related organelles.
  • This mutation resulted in activation of a cryptic intronic splice site causing an aberrantly spliced HPS1 mRNA that included 144-bp of intronic sequence, producing 11 novel amino acids followed by a stop codon.

  • Genetics Home Reference. consumer health - Hermansky-Pudlak syndrome.
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  • [Copyright] Published 2010. This article is a US Government work and is in the public domain in the USA.
  • (PMID = 20662851.001).
  • [ISSN] 1399-0004
  • [Journal-full-title] Clinical genetics
  • [ISO-abbreviation] Clin. Genet.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Research Support, N.I.H., Extramural; Research Support, N.I.H., Intramural
  • [Publication-country] Denmark
  • [Chemical-registry-number] 0 / HPS1 protein, human; 0 / Membrane Proteins
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13. ||........ 21%  Cooper IF, Siadaty MS: 'Hormones' associated with 'Ear Abnormalities': Top Publications. BioMedLib Review; Hormone;EarAbnormalities:705873923. ISSN: 2331-5717. 2014/5/1
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  • Finding such articles is important for researchers, clinicians, and patients.
  • In group one there are publications with the strongest evidence of association. We focused finding the most relevant publications pertinent to our goal, rather than combining them into a conclusion section. Such textual synthesis will be the focus of our next project.
  • Swank RT et al: Platelet storage pool deficiency associated with inherited abnormalities of the inner ear in the mouse pigment mutants muted and mocha.

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
  • (UID = 705873923.001).
  • [ISSN] 2331-5717
  • [Journal-full-title] BioMedLib Review
  • [Language] eng
  • [Publication-type] Review
  • [Publication-country] UNITED STATES
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14. ||........ 20%  Cooper IF, Siadaty MS: 'Cells' associated with 'Gene Pool': Top Publications. BioMedLib Review; Cell;GenePool:705698894. ISSN: 2331-5717. 2014/4/25
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  • [Title] 'Cells' associated with 'Gene Pool': Top Publications.
  • Background: There are articles published each month which present 'cell' for 'gene pool'.
  • Finding such articles is important for researchers, clinicians, and patients.
  • In group one there are publications with the strongest evidence of association. We focused finding the most relevant publications pertinent to our goal, rather than combining them into a conclusion section. Such textual synthesis will be the focus of our next project.
  • McCormack WT et al: Germ line maintenance of the pseudogene donor pool for somatic immunoglobulin gene conversion in chickens.
  • Kong FK et al: T cell receptor gene deletion circles identify recent thymic emigrants in the peripheral T cell pool.
  • Wataganara T et al: Plasma gamma-globin gene expression suggests that fetal hematopoietic cells contribute to the pool of circulating cell-free fetal nucleic acids during pregnancy.
  • Meuth M: Sensitivity of a mutator gene in Chinese hamster ovary cell to deoxynucleoside triphosphate pool alterations.
  • Paredes JA et al: Gene expression deregulation in postnatal skeletal muscle of TK2 deficient mice reveals a lower pool of proliferating myogenic progenitor cells.
  • Cohn M: On a key postulate of T-cell receptor restrictive function: the V-gene loci act as a single pool encoding recognition of the polymorphic alleles of the species major histocompatibility complex.
  • Wiesenfeld SL: Sickle-cell trait in human biological and cultural evolution. Development of agriculture causing increased malaria is bound to gene-pool changes causing malaria reduction.
  • Balanovskaia EV et al: [The Russian gene pool. Genogeography of erythrocyte genetic markers (ACP1, PGM1, ESD, GLO1, 6-PGD)].
  • Radzhabov MO et al: [The Dagestan gene pool: interethnic and intraethnic differentiation of eight aboriginal ethnic groups: analysis based on data on the AB0 and Rhesus erythrocyte antigen systems].
  • Gwynn B et al: The gene encoding protein 4.2 is distinct from the mouse platelet storage pool deficiency mutation pallid.

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
  • (UID = 705698894.001).
  • [ISSN] 2331-5717
  • [Journal-full-title] BioMedLib Review
  • [Language] eng
  • [Publication-type] Review
  • [Publication-country] UNITED STATES
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15. ||........ 18%  Cooper IF, Siadaty MS: 'Amino Acids, Peptides, or Proteins' associated with 'Ap 3 Adaptor Complex': Top Publications. BioMedLib Review; AminoAcidPeptide;Ap3Adaptor:705869207. ISSN: 2331-5717. 2014/5/1
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  • Finding such articles is important for researchers, clinicians, and patients.
  • In group one there are publications with the strongest evidence of association. We focused finding the most relevant publications pertinent to our goal, rather than combining them into a conclusion section. Such textual synthesis will be the focus of our next project.
  • Zhen L et al: Abnormal expression and subcellular distribution of subunit proteins of the AP-3 adaptor complex lead to platelet storage pool deficiency in the pearl mouse.

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
  • (UID = 705869207.001).
  • [ISSN] 2331-5717
  • [Journal-full-title] BioMedLib Review
  • [Language] eng
  • [Publication-type] Review
  • [Publication-country] UNITED STATES
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16. ||........ 18%  Cooper IF, Siadaty MS: 'Cells' associated with 'Adaptor Protein Complex 3': Top Publications. BioMedLib Review; Cell;AdaptorProteinComplex:705776304. ISSN: 2331-5717. 2014/4/27
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  • Finding such articles is important for researchers, clinicians, and patients.
  • In group one there are publications with the strongest evidence of association. We focused finding the most relevant publications pertinent to our goal, rather than combining them into a conclusion section. Such textual synthesis will be the focus of our next project.
  • Zhen L et al: Abnormal expression and subcellular distribution of subunit proteins of the AP-3 adaptor complex lead to platelet storage pool deficiency in the pearl mouse.
  • Huizing M et al: AP-3 mediates tyrosinase but not TRP-1 trafficking in human melanocytes.
  • Suzuki T et al: The mouse homolog of the bovine leukemia virus receptor is closely related to the delta subunit of adaptor-related protein complex AP-3, not associated with the cell surface.
  • Baguma-Nibasheka M et al: Altered retinal cell differentiation in the AP-3 delta mutant (Mocha) mouse.

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
  • (UID = 705776304.001).
  • [ISSN] 2331-5717
  • [Journal-full-title] BioMedLib Review
  • [Language] eng
  • [Publication-type] Review
  • [Publication-country] UNITED STATES
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17. ||........ 18%  Cooper IF, Siadaty MS: 'Cells' associated with 'Ashkenazi Jew': Top Publications. BioMedLib Review; Cell;AshkenaziJew:705747038. ISSN: 2331-5717. 2014/4/26
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  • Finding such articles is important for researchers, clinicians, and patients.
  • In group one there are publications with the strongest evidence of association. We focused finding the most relevant publications pertinent to our goal, rather than combining them into a conclusion section. Such textual synthesis will be the focus of our next project.
  • Huizing M et al: Hermansky-Pudlak syndrome type 3 in Ashkenazi Jews and other non-Puerto Rican patients with hypopigmentation and platelet storage-pool deficiency.

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
  • (UID = 705747038.001).
  • [ISSN] 2331-5717
  • [Journal-full-title] BioMedLib Review
  • [Language] eng
  • [Publication-type] Review
  • [Publication-country] UNITED STATES
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18. ||........ 18%  Cooper IF, Siadaty MS: 'Cells' associated with 'Rhabdomyolyses': Top Publications. BioMedLib Review; Cell;Rhabdomyolyses:705714558. ISSN: 2331-5717. 2014/4/26
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  • Finding such articles is important for researchers, clinicians, and patients.
  • In group one there are publications with the strongest evidence of association. We focused finding the most relevant publications pertinent to our goal, rather than combining them into a conclusion section. Such textual synthesis will be the focus of our next project.
  • Contopoulos-Ioannidis D et al: Recurrent rhabdomyolysis in a patient with oculocutaneous albinism type 1 and platelet storage-pool deficiency.
  • Messonnier L et al: Moderate endurance exercise is not a risk for rhabdomyolysis or renal failure in sickle cell trait carriers.

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
  • (UID = 705714558.001).
  • [ISSN] 2331-5717
  • [Journal-full-title] BioMedLib Review
  • [Language] eng
  • [Publication-type] Review
  • [Publication-country] UNITED STATES
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19. ||........ 18%  Cooper IF, Siadaty MS: 'Cell Components' associated with 'Ap 3 Adaptor Complex': Top Publications. BioMedLib Review; CellComponent;Ap3Adaptor:705648958. ISSN: 2331-5717. 2014/4/25
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  • Finding such articles is important for researchers, clinicians, and patients.
  • In group one there are publications with the strongest evidence of association. We focused finding the most relevant publications pertinent to our goal, rather than combining them into a conclusion section. Such textual synthesis will be the focus of our next project.
  • Zhen L et al: Abnormal expression and subcellular distribution of subunit proteins of the AP-3 adaptor complex lead to platelet storage pool deficiency in the pearl mouse.

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
  • (UID = 705648958.001).
  • [ISSN] 2331-5717
  • [Journal-full-title] BioMedLib Review
  • [Language] eng
  • [Publication-type] Review
  • [Publication-country] UNITED STATES
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20. ||........ 18%  Masliah-Planchon J, Darnige L, Bellucci S: Molecular determinants of platelet delta storage pool deficiencies: an update. Br J Haematol; 2013 Jan;160(1):5-11
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Molecular determinants of platelet delta storage pool deficiencies: an update.
  • Delta storage pool deficiency (δ-SPD) is a rare heterogeneous group of platelet disorders characterized by a reduction in the number or content of dense granules.
  • However, during the last few years, important insights into the pathophysiology of δ-SPD have been achieved using mouse models and dense granule deficiency-associated congenital diseases, such as Hermansky-Pudlak syndrome and Chediak-Higashi syndrome.
  • [MeSH-major] Platelet Storage Pool Deficiency / genetics

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  • [Copyright] © 2012 Blackwell Publishing Ltd.
  • (PMID = 23025459.001).
  • [ISSN] 1365-2141
  • [Journal-full-title] British journal of haematology
  • [ISO-abbreviation] Br. J. Haematol.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] England
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21. |......... 15%  De Leeuw M, Beuls E, Jorens P, Parizel P, Jacobs W: Delta-storage pool disease as a mimic of abusive head trauma in a 7-month-old baby: a case report. J Forensic Leg Med; 2013 Jul;20(5):520-1
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Delta-storage pool disease as a mimic of abusive head trauma in a 7-month-old baby: a case report.
  • Eventually, platelet aggregation tests and electron microscopy diagnosed a delta-storage pool disease; that is, a haemostatic disorder involving dense granules of the platelets.
  • Initial minor blunt trauma may have resulted in subdural bleeding, while subsequent retinal haemorrhage could have been facilitated by the underlying haemostatic disorder.
  • Delta-storage pool disease should be considered as a possible mimic of abusive head trauma similar to other rare conditions such as Menkes disease and type 1 glutaric aciduria.
  • [MeSH-major] Platelet Storage Pool Deficiency / diagnosis
  • [MeSH-minor] Child Abuse / diagnosis. Diagnosis, Differential. Forensic Medicine. Hematoma, Subdural / etiology. Humans. Infant. Male. Microscopy, Electron. Platelet Function Tests. Retinal Hemorrhage / etiology. Shaken Baby Syndrome / diagnosis

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  • [Copyright] Copyright © 2013 Elsevier Ltd and Faculty of Forensic and Legal Medicine. All rights reserved.
  • [CommentIn] J Forensic Leg Med. 2013 Nov;20(8):1149-50 [24237842.001]
  • (PMID = 23756525.001).
  • [ISSN] 1878-7487
  • [Journal-full-title] Journal of forensic and legal medicine
  • [ISO-abbreviation] J Forensic Leg Med
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] England
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22. |......... 9%  Brodsky MA, Machovec KA, Chambers BP, Nielsen VG: Platelet-mediated thrombolysis in patients with δ-storage pool deficiency: a thrombelastographic analysis. Blood Coagul Fibrinolysis; 2011 Oct;22(7):610-2
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Platelet-mediated thrombolysis in patients with δ-storage pool deficiency: a thrombelastographic analysis.
  • We present the first thrombelastographic descriptions of three patients with δ-storage pool deficiency, a platelet disorder that involves a deficiency of dense granules and moderate bleeding.
  • The patients demonstrated a 49-54% loss of platelet-mediated clot strength over a 1-2-h period after normal thrombus formation.
  • This pattern persisted, with some attenuation of loss of strength following administration of epsilon aminocaproic acid, desmopressin and platelets for tonsillectomy.
  • Assessment of platelet function in patients with platelet granule disorders can be accomplished with thrombelastographic methods in ambulatory and perioperative settings; however, the effects of therapy for this disorder cannot be monitored with thrombelastography without obtaining a blood sample prior to prophylactic hemostatic intervention.
  • [MeSH-major] Blood Platelets / drug effects. Hemostasis / drug effects. Hemostatics / administration & dosage. Platelet Storage Pool Deficiency / blood. Platelet Storage Pool Deficiency / drug therapy. Thrombelastography / methods
  • [MeSH-minor] Adolescent. Aminocaproic Acid / administration & dosage. Aminocaproic Acid / therapeutic use. Cytoplasmic Granules. Deamino Arginine Vasopressin / administration & dosage. Deamino Arginine Vasopressin / therapeutic use. Female. Fibrinolysis / drug effects. Humans. Male. Platelet Aggregation / drug effects. Platelet Count. Platelet Transfusion. Tonsillectomy

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  • (PMID = 21822127.001).
  • [ISSN] 1473-5733
  • [Journal-full-title] Blood coagulation & fibrinolysis : an international journal in haemostasis and thrombosis
  • [ISO-abbreviation] Blood Coagul. Fibrinolysis
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Hemostatics; ENR1LLB0FP / Deamino Arginine Vasopressin; U6F3787206 / Aminocaproic Acid
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23. |......... 8%  Rajpal G, Pomerantz JM, Ragni MV, Waters JH, Vallejo MC: The use of thromboelastography for the peripartum management of a patient with platelet storage pool disorder. Int J Obstet Anesth; 2011 Apr;20(2):173-7
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] The use of thromboelastography for the peripartum management of a patient with platelet storage pool disorder.
  • We describe the peripartum management of a 26-year-old primigravida with a platelet storage pool disorder who underwent spontaneous vaginal delivery of twins with epidural analgesia.
  • The use of thromboelastography in the assessment and management of bleeding risk in the setting of platelet storage pool disorder is described.
  • [MeSH-major] Platelet Storage Pool Deficiency / blood. Postpartum Hemorrhage / therapy. Pregnancy Complications / blood. Thrombelastography / methods

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  • [Copyright] Copyright © 2010 Elsevier Ltd. All rights reserved.
  • [CommentIn] Int J Obstet Anesth. 2011 Oct;20(4):368-9 [21907567.001]
  • [CommentIn] Int J Obstet Anesth. 2011 Oct;20(4):360; author reply 361 [21839631.001]
  • (PMID = 21168326.001).
  • [ISSN] 1532-3374
  • [Journal-full-title] International journal of obstetric anesthesia
  • [ISO-abbreviation] Int J Obstet Anesth
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Netherlands
  • [Chemical-registry-number] ENR1LLB0FP / Deamino Arginine Vasopressin
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24. |......... 8%  Ringeisen AL, Schimmenti LA, White JG, Schoonveld C, Summers CG: Hermansky-Pudlak syndrome (HPS5) in a nonagenarian. J AAPOS; 2013 Jun;17(3):334-6
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Hermansky-Pudlak syndrome (HPS) is an autosomal-recessive disorder clinically characterized by oculocutaneous albinism, bleeding diatheses, and lysosomal accumulation of ceroid lipofuscin, which in some cases may cause granulomatous colitis and pulmonary fibrosis.
  • This report highlights the importance of typing HPS to counsel patients regarding disease prognosis.
  • [MeSH-minor] Aged, 80 and over. Albinism, Oculocutaneous / diagnosis. Hemorrhage / diagnosis. Humans. Male. Platelet Storage Pool Deficiency / diagnosis. Pulmonary Fibrosis / diagnosis. Visual Acuity

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  • [Copyright] Copyright © 2013 American Association for Pediatric Ophthalmology and Strabismus. Published by Mosby, Inc. All rights reserved.
  • [CommentIn] J AAPOS. 2014 Apr;18(2):209-10 [24698632.001]
  • (PMID = 23607980.001).
  • [ISSN] 1528-3933
  • [Journal-full-title] Journal of AAPOS : the official publication of the American Association for Pediatric Ophthalmology and Strabismus / American Association for Pediatric Ophthalmology and Strabismus
  • [ISO-abbreviation] J AAPOS
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Carrier Proteins; 0 / HPS5 protein, human
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25. |......... 7%  Jedlitschky G, Cattaneo M, Lubenow LE, Rosskopf D, Lecchi A, Artoni A, Motta G, Niessen J, Kroemer HK, Greinacher A: Role of MRP4 (ABCC4) in platelet adenine nucleotide-storage: evidence from patients with delta-storage pool deficiencies. Am J Pathol; 2010 Mar;176(3):1097-103
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Role of MRP4 (ABCC4) in platelet adenine nucleotide-storage: evidence from patients with delta-storage pool deficiencies.
  • We previously showed that the MRP4 (ABCC4) transporter is expressed in human platelet delta-granules and may be involved in ADP transport.
  • We now demonstrate by immunoblotting and immunofluorescence microscopy that platelet MRP4 is absent in two patients with a platelet delta-storage pool deficiency (delta-SPD)-like phenotype with reduced platelet adenine nucleotide (AN) but normal serotonin levels, whereas their other membrane marker proteins of platelet granules were normally expressed and localized.
  • In these patients, MRP4 was present in lymphocytes, and the coding region of their MRP4/ABCC4 gene did not show any mutation that explained the lack of expression.
  • In platelets with "classic" delta-SPD (low AN and serotonin levels), MRP4 was quantitatively (immunoblot) normal, but, like other delta-granules membrane marker proteins (eg, LAMP2), was mostly displaced from delta-granules to patches at the plasma membrane, suggesting that platelets with classic delta-SPD have an abnormality that impairs the assembly of normal delta-granules.
  • Thus, defective expression of platelet MRP4 is associated with selective defect in AN storage.
  • The genetic basis of the new delta-SPD phenotype remains to be elucidated.
  • [MeSH-major] Adenine Nucleotides / metabolism. Blood Platelets / metabolism. Multidrug Resistance-Associated Proteins / metabolism. Platelet Storage Pool Deficiency / metabolism
  • [MeSH-minor] Adolescent. Adult. Female. Gene Expression Regulation. Humans. Leukocytes / metabolism. Male. Phenotype. Platelet Function Tests. Protein Transport. RNA, Messenger / genetics. RNA, Messenger / metabolism. Young Adult

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  • [Cites] Blood. 2004 Dec 1;104(12):3603-10 [15297306.001]
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  • (PMID = 20133816.001).
  • [ISSN] 1525-2191
  • [Journal-full-title] The American journal of pathology
  • [ISO-abbreviation] Am. J. Pathol.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / ABCC4 protein, human; 0 / Adenine Nucleotides; 0 / Multidrug Resistance-Associated Proteins; 0 / RNA, Messenger
  • [Other-IDs] NLM/ PMC2832132
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26. |......... 6%  White JG, Thomas A: Platelet structural pathology in a patient with the X-linked GATA-1, R216Q mutation. Platelets; 2009 Feb;20(1):41-9
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Platelet structural pathology in a patient with the X-linked GATA-1, R216Q mutation.
  • Some of the pathological features observed in macrothrombocytes from patients with the V205M and G208S variations including hypo- and agranular platelets, tubular inclusions and platelets within platelets, as well as platelets within platelets within platelets were identified.
  • However, tubular membrane sheets in megakaryocytes and platelets of the V205M and G208S types and large groups of platelets attached to platelets to form megathrombocytes were not observed.
  • The unique pathology of the megathrombocytes from this patient was the near absence of dense bodies in his giant cells.
  • Storage Pool Deficiency, together with large platelets, defective adhesion and aggregation of his macrocytes under shear stress to vWF and collagen and defective clot retraction may contribute to the pathogenesis of his bleeding disorder.
  • [MeSH-major] Blood Platelets / pathology. GATA1 Transcription Factor / genetics. Genes, X-Linked / genetics. Hemorrhagic Disorders / pathology. Mutation, Missense
  • [MeSH-minor] Cell Shape. Cell Size. Cytoplasmic Granules / ultrastructure. Cytoplasmic Structures / ultrastructure. Humans. Intracellular Space / ultrastructure. Male. Microbodies / ultrastructure. Microscopy, Electron, Transmission. Platelet Adhesiveness. Platelet Aggregation. Platelet Storage Pool Deficiency / complications. Platelet Storage Pool Deficiency / etiology. Platelet Storage Pool Deficiency / pathology

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  • (PMID = 19172521.001).
  • [ISSN] 1369-1635
  • [Journal-full-title] Platelets
  • [ISO-abbreviation] Platelets
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Chemical-registry-number] 0 / GATA1 Transcription Factor
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27. |......... 6%  Di Michele M, Thys C, Waelkens E, Overbergh L, D'Hertog W, Mathieu C, De Vos R, Peerlinck K, Van Geet C, Freson K: An integrated proteomics and genomics analysis to unravel a heterogeneous platelet secretion defect. J Proteomics; 2011 May 16;74(6):902-13
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] An integrated proteomics and genomics analysis to unravel a heterogeneous platelet secretion defect.
  • Eight patients with clinical bleeding problems have evidence for platelet storage pool disease as they present with impaired platelet aggregation and secretion with low concentrations of ADP and collagen and an absence of second phase aggregation with epinephrine.
  • Electron microscopy analysis further showed a reduced but not absent amount of platelet dense granules, and CD63 staining was decreased compared to healthy controls.
  • The presence of alpha granules and CD62P expression after platelet activation was normal.
  • This work aimed at identifying differentially expressed proteins in the platelet releasate and its remaining pellet after activation with A23187 and TRAP in patients and controls using DIGE-based proteomic technology.
  • In addition, 29 cytoskeleton-related genes showed an altered expression in platelet mRNA from patients using a real-time PCR array.
  • In conclusion, our study shows that the dense granule secretion defect in patients with platelet storage pool disease is highly heterogeneous with evidence of an underlying cytoskeleton defect.
  • [MeSH-major] Blood Platelets / metabolism. Cytoskeletal Proteins / genetics. Genomics. Platelet Storage Pool Deficiency / metabolism. Proteomics
  • [MeSH-minor] Adolescent. Adult. Calcimycin / pharmacology. Child. Cytoplasmic Granules / metabolism. Electrophoresis, Gel, Two-Dimensional. Female. Humans. Male. Middle Aged. Platelet Aggregation. RNA, Messenger / blood

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  • [Copyright] Copyright © 2011 Elsevier B.V. All rights reserved.
  • (PMID = 21406263.001).
  • [ISSN] 1876-7737
  • [Journal-full-title] Journal of proteomics
  • [ISO-abbreviation] J Proteomics
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / Cytoskeletal Proteins; 0 / RNA, Messenger; 37H9VM9WZL / Calcimycin
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28. |......... 6%  Jackson SP, Schoenwaelder SM: Procoagulant platelets: are they necrotic? Blood; 2010 Sep 23;116(12):2011-8
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  • [Title] Procoagulant platelets: are they necrotic?
  • Apoptosis and necrosis represent distinct cell death processes that regulate mammalian development, physiology and disease.
  • Although anucleate, platelets undergo programmed cell death, with apoptosis playing an important role in clearing effete platelets from the circulation.
  • While it has long been recognized that procoagulant platelets exhibit characteristic features of dying cells, recent studies have demonstrated that platelet procoagulant function can occur independent of apoptosis.
  • A growing body of evidence suggest that the biochemical, morphologic and functional changes underlying agonist-induced platelet procoagulant function are broadly consistent with cell necrosis, raising the possibility that distinct death pathways regulate platelet function and survival.
  • In this article, we will discuss the mechanisms underlying apoptotic and necrotic cell death pathways and examine the evidence linking these pathways to the platelet procoagulant response.
  • We will also discuss the potential contribution of these pathways to the platelet storage lesion and propose a simplified nomenclature to describe procoagulant platelets.
  • [MeSH-major] Blood Platelets / pathology. Necrosis / blood. Thrombophilia / blood
  • [MeSH-minor] Apoptosis. Blood Coagulation. Humans. Inflammation / blood. Platelet Storage Pool Deficiency / etiology

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  • (PMID = 20538794.001).
  • [ISSN] 1528-0020
  • [Journal-full-title] Blood
  • [ISO-abbreviation] Blood
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] United States
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29. |......... 6%  Wei AH, Schoenwaelder SM, Andrews RK, Jackson SP: New insights into the haemostatic function of platelets. Br J Haematol; 2009 Nov;147(4):415-30
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] New insights into the haemostatic function of platelets.
  • Considerable progress has been made over the last two decades in delineating the key molecular events regulating the haemostatic function of platelets.
  • Much of this new insight has been derived from the study of mouse models, in which the expression or structure of one or more platelet proteins has been genetically altered.
  • There is a dearth of literature available to help physicians integrate and apply the burgeoning knowledge on platelet biology to diagnosing patients with atypical or unexplained platelet dysfunction.
  • The purpose of this review is to summarise the major primary platelet disorders relevant to pathological bleeding in humans (excluding those primarily due to thrombocytopenia or acquired functional disorders), with a focus on lesions identified in mouse models that could represent candidate molecules for study in patients with impaired platelet function.
  • [MeSH-major] Blood Platelets / physiology. Hemostasis / physiology
  • [MeSH-minor] Animals. Blood Platelet Disorders / blood. Humans. Mice. Platelet Adhesiveness / physiology. Platelet Aggregation / physiology. Platelet Storage Pool Deficiency / blood

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  • (PMID = 19656155.001).
  • [ISSN] 1365-2141
  • [Journal-full-title] British journal of haematology
  • [ISO-abbreviation] Br. J. Haematol.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] England
  • [Number-of-references] 130
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30. |......... 6%  Bluteau D, Gilles L, Hilpert M, Antony-Debré I, James C, Debili N, Camara-Clayette V, Wagner-Ballon O, Cordette-Lagarde V, Robert T, Ripoche H, Gonin P, Swierczek S, Prchal J, Vainchenker W, Favier R, Raslova H: Down-regulation of the RUNX1-target gene NR4A3 contributes to hematopoiesis deregulation in familial platelet disorder/acute myelogenous leukemia. Blood; 2011 Dec 8;118(24):6310-20
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Down-regulation of the RUNX1-target gene NR4A3 contributes to hematopoiesis deregulation in familial platelet disorder/acute myelogenous leukemia.
  • Inactivating or dominant-negative mutations in the RUNX1 gene have been also identified in pedigrees of familial platelet disorders with a variable propensity to develop acute myeloid leukemia (FPD/AML).
  • We propose that the down-regulation of NR4A3 in RUNX1-mutated hematopoietic progenitors leads to an increase in the pool of cells susceptible to be hit by secondary leukemic genetic events.
  • [MeSH-major] Core Binding Factor Alpha 2 Subunit / genetics. DNA-Binding Proteins / metabolism. Down-Regulation. Hematopoiesis. Leukemia, Myeloid, Acute / genetics. Platelet Storage Pool Deficiency / genetics. Receptors, Steroid / metabolism. Receptors, Thyroid Hormone / metabolism

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  • (PMID = 21725049.001).
  • [ISSN] 1528-0020
  • [Journal-full-title] Blood
  • [ISO-abbreviation] Blood
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Core Binding Factor Alpha 2 Subunit; 0 / DNA-Binding Proteins; 0 / NR4A3 protein, human; 0 / RUNX1 protein, human; 0 / Receptors, Steroid; 0 / Receptors, Thyroid Hormone
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31. |......... 5%  Gunning W, Dole M, Brecher M, White JG: The Medich giant platelet syndrome: two new cases. Platelets; 2013;24(2):107-12
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] The Medich giant platelet syndrome: two new cases.
  • Hypogranular platelet disorders in human subjects are relatively rare.
  • They include the gray platelet syndrome, αδ storage pool deficiency, the Hermansky-Pudlak syndrome, and the white platelet syndrome.
  • Perhaps the rarest of them all is the Medich giant platelet disorder.
  • This study describes two children with thrombocytopenia and giant, hypogranular platelets found shortly after birth.
  • Electron microscopic study of their platelets revealed sheets of membrane wrapped into tubes resembling scrolls.
  • As a result, the disorder can now be referred to as the Medich giant platelet syndrome.
  • [MeSH-major] Bernard-Soulier Syndrome / diagnosis. Blood Platelet Disorders / congenital. Hermanski-Pudlak Syndrome / diagnosis. Platelet Storage Pool Deficiency / diagnosis
  • [MeSH-minor] Blood Platelets / ultrastructure. Female. Humans. Male. Thrombocytopenia / diagnosis

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  • (PMID = 22931192.001).
  • [ISSN] 1369-1635
  • [Journal-full-title] Platelets
  • [ISO-abbreviation] Platelets
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] England
  • [Chemical-registry-number] White platelet syndrome
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32. |......... 4%  Kirchmaier CM, Pillitteri D: [Congenital thrombocytopathies]. Internist (Berl); 2010 Sep;51(9):1109-14, 1116-7
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Inherited thrombocytopathies are much less frequent in comparison to acquired platelet function disorders.
  • However, congenital disorders can lead to severe bleeding tendency and are often not diagnosed.
  • They are induced by different platelet defects based on disorders of platelet adhesion, receptors, secretion and signal transduction.
  • In some cases they are associated with thrombocytopenia, giant platelets and various comorbidities.
  • [MeSH-major] Blood Platelet Disorders / genetics. Blood Platelets / physiology. Genotype. Hemorrhagic Disorders / genetics
  • [MeSH-minor] Bernard-Soulier Syndrome / blood. Bernard-Soulier Syndrome / diagnosis. Bernard-Soulier Syndrome / genetics. Bernard-Soulier Syndrome / therapy. Diagnosis, Differential. Humans. Mass Screening. Platelet Aggregation / genetics. Platelet Aggregation / physiology. Platelet Function Tests. Platelet Storage Pool Deficiency / blood. Platelet Storage Pool Deficiency / diagnosis. Platelet Storage Pool Deficiency / genetics. Platelet Storage Pool Deficiency / therapy. Prostaglandin-Endoperoxide Synthases / genetics. Receptors, Collagen / genetics. Receptors, Collagen / physiology. Thrombasthenia / blood. Thrombasthenia / diagnosis. Thrombasthenia / genetics. Thrombasthenia / therapy. Thromboxane-A Synthase / genetics

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  • (PMID = 20725709.001).
  • [ISSN] 1432-1289
  • [Journal-full-title] Der Internist
  • [ISO-abbreviation] Internist (Berl)
  • [Language] ger
  • [Publication-type] English Abstract; Journal Article; Review
  • [Publication-country] Germany
  • [Chemical-registry-number] 0 / Receptors, Collagen; EC 1.14.99.1 / Prostaglandin-Endoperoxide Synthases; EC 5.3.99.5 / Thromboxane-A Synthase
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33. |......... 4%  Hernández-Ruiz L, Sáez-Benito A, Pujol-Moix N, Rodríguez-Martorell J, Ruiz FA: Platelet inorganic polyphosphate decreases in patients with delta storage pool disease. J Thromb Haemost; 2009 Feb;7(2):361-3
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Platelet inorganic polyphosphate decreases in patients with delta storage pool disease.
  • [MeSH-major] Platelet Storage Pool Deficiency / metabolism. Polyphosphates / metabolism
  • [MeSH-minor] Blood Platelets / chemistry. Case-Control Studies. Humans

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  • (PMID = 19036063.001).
  • [ISSN] 1538-7836
  • [Journal-full-title] Journal of thrombosis and haemostasis : JTH
  • [ISO-abbreviation] J. Thromb. Haemost.
  • [Language] eng
  • [Publication-type] Letter; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Polyphosphates
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34. |......... 4%  Masliah-Planchon J, Bellucci S, Darnige L, Callebert J, Fischer AM, Tapon-Bretaudière J: Absence of mutation in the RAB27B gene in patients with platelet delta-storage pool deficiency. Eur J Haematol; 2011 Apr;86(4):350-1
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Absence of mutation in the RAB27B gene in patients with platelet delta-storage pool deficiency.
  • [MeSH-major] Platelet Storage Pool Deficiency / genetics. rab GTP-Binding Proteins / genetics
  • [MeSH-minor] Adult. Child. Child, Preschool. DNA Mutational Analysis. Female. Genetic Testing. Humans. Male. Mutation. Platelet Aggregation / genetics

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  • (PMID = 21198862.001).
  • [ISSN] 1600-0609
  • [Journal-full-title] European journal of haematology
  • [ISO-abbreviation] Eur. J. Haematol.
  • [Language] eng
  • [Publication-type] Letter
  • [Publication-country] England
  • [Chemical-registry-number] EC 3.6.1.- / rab GTP-Binding Proteins; EC 3.6.1.-. / Rab27B protein, human
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35. |......... 4%  Barone I, Meccariello G, Cazzato L, Capalbo SF, Matteo M, Greco P: Management of platelet storage pool disease during pregnancy with recombinant factor VIIa. Eur J Obstet Gynecol Reprod Biol; 2013 Oct;170(2):576-7
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Management of platelet storage pool disease during pregnancy with recombinant factor VIIa.
  • [MeSH-major] Factor VIIa / therapeutic use. Platelet Storage Pool Deficiency / drug therapy. Pregnancy Complications, Hematologic / drug therapy

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  • (PMID = 23850139.001).
  • [ISSN] 1872-7654
  • [Journal-full-title] European journal of obstetrics, gynecology, and reproductive biology
  • [ISO-abbreviation] Eur. J. Obstet. Gynecol. Reprod. Biol.
  • [Language] eng
  • [Publication-type] Case Reports; Letter
  • [Publication-country] Ireland
  • [Chemical-registry-number] 0 / Recombinant Proteins; 0 / recombinant FVIIa; EC 3.4.21.21 / Factor VIIa
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36. |......... 4%  Campbell J, Yentis S: The use of thromboelastography for the peripartum management of a patient with platelet storage pool disorder. Int J Obstet Anesth; 2011 Oct;20(4):360; author reply 361
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] The use of thromboelastography for the peripartum management of a patient with platelet storage pool disorder.
  • [MeSH-major] Platelet Storage Pool Deficiency / blood. Postpartum Hemorrhage / therapy. Pregnancy Complications / blood. Thrombelastography / methods

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  • [CommentOn] Int J Obstet Anesth. 2011 Apr;20(2):173-7 [21168326.001]
  • [ErratumIn] Int J Obstet Anesth. 2012 Jan;21(1):103
  • (PMID = 21839631.001).
  • [ISSN] 1532-3374
  • [Journal-full-title] International journal of obstetric anesthesia
  • [ISO-abbreviation] Int J Obstet Anesth
  • [Language] eng
  • [Publication-type] Comment; Letter
  • [Publication-country] Netherlands
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37. |......... 3%  Gauthama P, Maybury H, Brooks H: Management of a parturient with TAR syndrome during caesarean section and the use of thromboelastography. Int J Obstet Anesth; 2011 Oct;20(4):368-9
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Management of a parturient with TAR syndrome during caesarean section and the use of thromboelastography.
  • [MeSH-major] Platelet Storage Pool Deficiency / blood. Postpartum Hemorrhage / therapy. Pregnancy Complications / blood. Thrombelastography / methods

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  • [CommentOn] Int J Obstet Anesth. 2011 Apr;20(2):173-7 [21168326.001]
  • (PMID = 21907567.001).
  • [ISSN] 1532-3374
  • [Journal-full-title] International journal of obstetric anesthesia
  • [ISO-abbreviation] Int J Obstet Anesth
  • [Language] eng
  • [Publication-type] Comment; Letter
  • [Publication-country] Netherlands
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38. |......... 3%  Agarwal U, Zaid RZ, Kadir RA: Uterine artery embolization: an approach to achieve haemostasis in post hysterectomy haemorrhages. Haemophilia; 2009 Jan;15(1):357-8
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [MeSH-major] Hysterectomy / adverse effects. Platelet Storage Pool Deficiency / complications. Postoperative Hemorrhage / therapy. Uterine Artery Embolization / methods

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  • (PMID = 18721152.001).
  • [ISSN] 1365-2516
  • [Journal-full-title] Haemophilia : the official journal of the World Federation of Hemophilia
  • [ISO-abbreviation] Haemophilia
  • [Language] eng
  • [Publication-type] Case Reports; Letter
  • [Publication-country] England
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39. |......... 2%  Nisal M, Pavord S, Oppenheimer CA, Francis S, Khare M: Hermansky-Pudlak syndrome: management of a rare bleeding disorder in a twin pregnancy. J Obstet Gynaecol; 2012 Feb;32(2):185-6
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Hermansky-Pudlak syndrome: management of a rare bleeding disorder in a twin pregnancy.
  • [MeSH-major] Hermanski-Pudlak Syndrome / therapy. Platelet Storage Pool Deficiency / therapy. Pregnancy Complications, Hematologic / therapy. Pregnancy, Twin


40. |......... 1%  Seligsohn U: Treatment of inherited platelet disorders. Haemophilia; 2012 Jul;18 Suppl 4:161-5
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Treatment of inherited platelet disorders.
  • For patients affected by severe inherited platelet dysfunctions, e.g.
  • Glanzmann thrombasthenia (GT) or Bernard-Soulier syndrome (BSS), platelet transfusion is frequently needed for controlling spontaneous bleeding, and is always needed when trauma occurs or surgery is performed.
  • For the mild-to-moderate bleeding entities, e.g. storage pool disease, thrombaxane A2 receptor defect, platelet transfusion is usually unnecessary.
  • Transfusion of platelets should be used selectively and sparingly because of the substantial risk of alloimmunization against HLA antigens and/or platelet glycoproteins (GP) αIIb, β(3), or αIIbβ(3) in GT, and GPI-IX-V in BSS, which may lead to refractoriness to therapy.
  • To reduce the risk, HLA-matched single donors of platelets should be used.
  • Therapy other than platelet transfusion includes: (i) Prevention (vaccination against hepatitis B, avoidance of non-steroidal anti-inflammatory drugs, preservation of dental hygiene, correction of iron deficiency and prenatal diagnosis). (ii) Topical measures (compression with gauze soaked with tranexamic acid, fibrin sealants, splints for dental extractions and packing for nose bleeds). (iii) Antifibrinolytic agents that are useful for minor surgery and as adjuncts for other treatment modalities. (iv) Desmopressin that increases plasma levels of von Willebrand factor and factor VIII giving rise to increased platelet adhesiveness and aggregation associated with shortened bleeding time. (v) Recombinant factor VIIa (rFVIIa).
  • The mechanism by which rFVIIa arrests bleeding is probably related to increased thrombin generation by a tissue factor-independent process, enhanced platelet adhesion and restoration of platelet aggregation. (vi) Female hormones.
  • [MeSH-major] Blood Coagulation Disorders, Inherited / therapy. Blood Platelet Disorders / therapy
  • [MeSH-minor] Antifibrinolytic Agents / therapeutic use. Coagulants / therapeutic use. Factor VIIa / therapeutic use. Female. Hematopoietic Stem Cell Transplantation. Hormones / therapeutic use. Humans. Male. Platelet Transfusion. Pregnancy. Pregnancy Complications, Hematologic / therapy. Recombinant Proteins / therapeutic use

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  • [Copyright] © 2012 Blackwell Publishing Ltd.
  • (PMID = 22726101.001).
  • [ISSN] 1365-2516
  • [Journal-full-title] Haemophilia : the official journal of the World Federation of Hemophilia
  • [ISO-abbreviation] Haemophilia
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Antifibrinolytic Agents; 0 / Coagulants; 0 / Hormones; 0 / Recombinant Proteins; 0 / recombinant FVIIa; EC 3.4.21.21 / Factor VIIa
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41. |......... 1%  McGlasson DL, Fritsma GA: Whole blood platelet aggregometry and platelet function testing. Semin Thromb Hemost; 2009 Mar;35(2):168-80
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Whole blood platelet aggregometry and platelet function testing.
  • Platelet aggregometry has been the reference method employed to detect, diagnose, and monitor qualitative platelet disorders since the early 1960s.
  • Lumiaggregometry and impedance-based whole blood lumiaggregometry have advantages over light transmittance aggregometry in that they provide for enhanced specimen management and increase the test sensitivity to impairment of platelet granule secretion.
  • Whole blood lumiaggregometry detects and identifies congenital and acquired platelet plasma membrane receptor defects, metabolic pathway secretion disorders, and storage pool deficiency.
  • There is growing interest in using impedance-based whole blood lumiaggregometry for near-patient whole blood platelet analysis and antiplatelet therapy monitoring.
  • This article will also discuss other whole blood testing processes for assessing platelet function, particularly as applied to assessing the effect of antiplatelet medication.
  • [MeSH-major] Blood Platelet Disorders / diagnosis. Platelet Aggregation / physiology. Platelet Function Tests / methods
  • [MeSH-minor] Blood Platelets / physiology. Humans

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  • (PMID = 19408190.001).
  • [ISSN] 1098-9064
  • [Journal-full-title] Seminars in thrombosis and hemostasis
  • [ISO-abbreviation] Semin. Thromb. Hemost.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] United States
  • [Number-of-references] 93
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42. |......... 1%  Cooper IF, Siadaty MS: 'Cells' associated with 'Intracellular Storage': Top Publications. BioMedLib Review; Cell;IntracellularStorage:705813074. ISSN: 2331-5717. 2014/4/28
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  • [Title] 'Cells' associated with 'Intracellular Storage': Top Publications.
  • Background: There are articles published each month which present 'cell' for 'intracellular storage'.
  • Finding such articles is important for researchers, clinicians, and patients.
  • In group one there are publications with the strongest evidence of association. We focused finding the most relevant publications pertinent to our goal, rather than combining them into a conclusion section. Such textual synthesis will be the focus of our next project.
  • Jost C et al: Intracellular storage and regulated plasma membrane expression of human complement receptor type 1 in rat basophil leukemia cell transfectants.
  • Becich MJ et al: Intracellular transport and storage of secretory proteins in relation to cytodifferentiation in neoplastic pancreatic acinar cells.
  • Tamir H et al: Serotonin storage pools in basophil leukemia and mast cells: characterization of two types of serotonin binding protein and radioautographic analysis of the intracellular distribution of [3H]serotonin.
  • Aardema H et al: Bovine cumulus cells protect maturing oocytes from increased fatty acid levels by massive intracellular lipid storage.
  • Jy W et al: Intracellular calcium storage and release in the human platelet. Chlorotetracycline as a continuous monitor.
  • Fasolato C et al: Intracellular Ca2+ pools in PC12 cells. Three intracellular pools are distinguished by their turnover and mechanisms of Ca2+ accumulation, storage, and release.
  • Weiss HJ et al: Platelet prothrombinase activity and intracellular calcium responses in patients with storage pool deficiency, glycoprotein IIb-IIIa deficiency, or impaired platelet coagulant activity--a comparison with Scott syndrome.
  • Kobayashi S et al: K+-depolarization induces a direct release of Ca2+ from intracellular storage sites in cultured vascular smooth muscle cells from rat aorta.
  • Schröder AK et al: Crosslinking of CD66B on peripheral blood neutrophils mediates the release of interleukin-8 from intracellular storage.
  • Beyer E et al: Change of isoforms' spectra of alpha-L-fucosidase from human skin fibroblasts in intracellular storage of nonhydrolyzable substances.

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
  • (UID = 705813074.001).
  • [ISSN] 2331-5717
  • [Journal-full-title] BioMedLib Review
  • [Language] eng
  • [Publication-type] Review
  • [Publication-country] UNITED STATES
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43. |......... 1%  Weiss HJ: Impaired platelet procoagulant mechanisms in patients with bleeding disorders. Semin Thromb Hemost; 2009 Mar;35(2):233-41
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Impaired platelet procoagulant mechanisms in patients with bleeding disorders.
  • Activated platelets contribute to the arrest of bleeding by forming aggregates at sites of vascular injury and by providing a surface for assembling enzyme complexes involved in fibrin formation (platelet procoagulant activity; PCA).
  • Impairment in the latter property of platelets has been observed in some disorders of hemostasis.
  • In Scott syndrome, there is a defect in membrane vesiculation and in the surface expression of phosphatidylserine (PS), the phospholipid that is necessary for assembling the factor VIIIa/IXa (tenase) and factor Va/Xa (prothrombinase) complexes involved in thrombin formation.
  • A family with an isolated defect in vesiculation, but normal prothrombinase activity, has also been reported.
  • In the Quebec platelet disorder, overexpression of the fibrinolytic enzyme urokinase-type plasminogen activator results in the degradation of alpha-granule proteins, including factor V, and a specific abnormality in platelet factor V is the basis for the prothrombinase defect in platelet factor V-New York.
  • The impaired prothrombinase activity in patients with delta-storage pool deficiency may be due to a failure to provide sufficient amounts of secreted adenine nucleotides which, when bound to P2 purinergic receptors, are necessary to maintain the intracellular Ca (2+) levels that are required for the surface expression of PS.
  • Platelet prothrombinase activity and thrombin potential in patients with Glanzmann thrombasthenia (GPIIb-IIIa deficiency) may be decreased, normal, or increased, depending on the experimental conditions, for reasons that are not currently clear.
  • The most consistent platelet PCA abnormality in the Bernard-Soulier syndrome (GPIb-complex deficiency) is an abnormally short serum prothrombin time, associated with a defect in the process by which an interaction between fibrin, von Willebrand factor, and GPIb promotes PCA.
  • [MeSH-major] Blood Coagulation Disorders / blood. Blood Platelets / pathology

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  • [ErratumIn] Semin Thromb Hemost. 2009 Jun;35(4):447
  • (PMID = 19408196.001).
  • [ISSN] 1098-9064
  • [Journal-full-title] Seminars in thrombosis and hemostasis
  • [ISO-abbreviation] Semin. Thromb. Hemost.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
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44. |......... 1%  Cooper IF, Siadaty MS: 'Mammals' associated with 'Platelet Changes': Top Publications. BioMedLib Review; Mammal;PlateletChanges:705574797. ISSN: 2331-5717. 2014/4/24
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  • [Title] 'Mammals' associated with 'Platelet Changes': Top Publications.
  • Background: There are articles published each month which present 'mammal' for 'platelet changes'.
  • Finding such articles is important for researchers, clinicians, and patients.
  • In group one there are publications with the strongest evidence of association. We focused finding the most relevant publications pertinent to our goal, rather than combining them into a conclusion section. Such textual synthesis will be the focus of our next project.
  • Zhao J et al: Changes in Otx2 and parvalbumin immunoreactivity in the superior colliculus in the platelet-derived growth factor receptor-β knockout mice.
  • Fushuku S et al: Morphological changes of platelets following platelet aggregation induced by collagen in Japanese Black cattle with delta-storage pool deficiency.
  • Hasegawa-Sasaki H: Early changes in inositol lipids and their metabolites induced by platelet-derived growth factor in quiescent Swiss mouse 3T3 cells.
  • Fushuku S et al: Changes of platelet cytoskeletal proteins following platelet aggregation induced by collagen and platelet membrane glycoprotein in Japanese black cattle with delta-storage pool deficiency.
  • Dard B et al: [Changes in the inhibitory processes of platelet activation in the SHR rat].
  • Williams EF et al: Changes in renal, platelet and cardiac nitrobenzylthioinosine binding in spontaneously hypertensive rats.
  • Wilson DV et al: Histologic and ultrastructural changes after large-colon torsion, with and without use of a specific platelet-activating factor antagonist (WEB 2086), in ponies.
  • Moesgaard SG et al: Changes in platelet function in Dachshunds with early stages of myxomatous mitral valve disease.
  • Bo P et al: EEG changes and platelet aggregation in experimental cerebral focal ischemia in rabbits.
  • Ibe BO et al: Maturational changes in ovine pulmonary metabolism of platelet-activating factor: implications for postnatal adaptation.

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
  • (UID = 705574797.001).
  • [ISSN] 2331-5717
  • [Journal-full-title] BioMedLib Review
  • [Language] eng
  • [Publication-type] Review
  • [Publication-country] UNITED STATES
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45. |......... 1%  Cooper IF, Siadaty MS: 'Mammals' associated with 'Induced Platelet Aggregation': Top Publications. BioMedLib Review; Mammal;InducedPlateletAggregation:705654850. ISSN: 2331-5717. 2014/4/25
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  • [Title] 'Mammals' associated with 'Induced Platelet Aggregation': Top Publications.
  • Background: There are articles published each month which present 'mammal' for 'induced platelet aggregation'.
  • Finding such articles is important for researchers, clinicians, and patients.
  • In group one there are publications with the strongest evidence of association. We focused finding the most relevant publications pertinent to our goal, rather than combining them into a conclusion section. Such textual synthesis will be the focus of our next project.
  • Shan CW et al: Blockage of clonidine-induced platelet aggregation in rabbits by procainamide.
  • Bednarek-Tupikowska G et al: Influence of dehydroepiandrosterone on platelet aggregation, superoxide dismutase activity and serum lipid peroxide concentrations in rabbits with induced hypercholesterolemia.
  • Fushuku S et al: Morphological changes of platelets following platelet aggregation induced by collagen in Japanese Black cattle with delta-storage pool deficiency.
  • Larrucea S et al: Agonist-induced aggregation of Chinese hamster ovary cells coexpressing the human receptors for fibrinogen (integrin alphaIIbbeta3) and the platelet-activating factor: dissociation between adhesion and aggregation.
  • Fushuku S et al: Changes of platelet cytoskeletal proteins following platelet aggregation induced by collagen and platelet membrane glycoprotein in Japanese black cattle with delta-storage pool deficiency.
  • Miyamae T et al: Calcium-induced platelet aggregation in washed platelets from guinea pigs.
  • Weiss DJ et al: Effect of a competitive inhibitor of platelet aggregation on experimentally induced laminitis in ponies.
  • Li P et al: Losartan inhibits thromboxane A2-induced platelet aggregation and vascular constriction in spontaneously hypertensive rats.
  • Jarvis GE et al: Endotoxin-induced platelet aggregation in heparinised equine whole blood in vitro.
  • Rickards KJ et al: Differential effects of phosphodiesterase inhibitors on platelet activating factor (PAF)- and adenosine diphosphate (ADP)-induced equine platelet aggregation.

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
  • (UID = 705654850.001).
  • [ISSN] 2331-5717
  • [Journal-full-title] BioMedLib Review
  • [Language] eng
  • [Publication-type] Review
  • [Publication-country] UNITED STATES
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46. |......... 1%  Cooper IF, Siadaty MS: 'Cell Components' associated with 'Platelet Changes': Top Publications. BioMedLib Review; CellComponent;PlateletChanges:705571490. ISSN: 2331-5717. 2014/4/24
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  • [Title] 'Cell Components' associated with 'Platelet Changes': Top Publications.
  • Background: There are articles published each month which present 'cell component' for 'platelet changes'.
  • Finding such articles is important for researchers, clinicians, and patients.
  • In group one there are publications with the strongest evidence of association. We focused finding the most relevant publications pertinent to our goal, rather than combining them into a conclusion section. Such textual synthesis will be the focus of our next project.
  • Newman PJ et al: Activation-dependent changes in human platelet PECAM-1: phosphorylation, cytoskeletal association, and surface membrane redistribution.
  • Thompson P: Platelet and erythrocyte membrane fluidity changes in alcohol-dependent patients undergoing acute withdrawal.
  • Fushuku S et al: Changes of platelet cytoskeletal proteins following platelet aggregation induced by collagen and platelet membrane glycoprotein in Japanese black cattle with delta-storage pool deficiency.
  • DeLisser HM et al: Deletions in the cytoplasmic domain of platelet-endothelial cell adhesion molecule-1 (PECAM-1, CD31) result in changes in ligand binding properties.
  • Kuwahara M et al: Cytosolic calcium changes in a process of platelet adhesion and cohesion on a von Willebrand factor-coated surface under flow conditions.
  • Fagotto F et al: Changes in yolk platelet pH during Xenopus laevis development correlate with yolk utilization. A quantitative confocal microscopy study.
  • Annie-Jeyachristy S et al: Changes in the level of cytosolic calcium, nitric oxide and nitric oxide synthase activity during platelet aggregation: an in vitro study in platelets from normal subjects and those with cirrhosis.
  • Labrouche S et al: Changes in phospholipid composition of blood cell membranes (erythrocyte, platelet, and polymorphonuclear) in different types of diabetes--clinical and biological correlations.
  • Ishikawa Y et al: Changes of platelet functions by extracellular sodium ion.
  • Hajimohammadreza I et al: Platelet and erythrocyte membrane changes in Alzheimer's disease.

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
  • (UID = 705571490.001).
  • [ISSN] 2331-5717
  • [Journal-full-title] BioMedLib Review
  • [Language] eng
  • [Publication-type] Review
  • [Publication-country] UNITED STATES
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47. |......... 1%  Cooper IF, Siadaty MS: 'Amino Acids, Peptides, or Proteins' associated with 'Platelet Types': Top Publications. BioMedLib Review; AminoAcidPeptide;PlateletTypes:705691374. ISSN: 2331-5717. 2014/4/25
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  • [Title] 'Amino Acids, Peptides, or Proteins' associated with 'Platelet Types': Top Publications.
  • Background: There are articles published each month which present 'amino acid peptide or protein' for 'platelet types'.
  • Finding such articles is important for researchers, clinicians, and patients.
  • In group one there are publications with the strongest evidence of association. We focused finding the most relevant publications pertinent to our goal, rather than combining them into a conclusion section. Such textual synthesis will be the focus of our next project.
  • Lages B et al: Decreased content and surface expression of alpha-granule membrane protein GMP-140 in one of two types of platelet alpha delta storage pool deficiency.
  • Saelman EU et al: Platelet adhesion to collagen types I through VIII under conditions of stasis and flow is mediated by GPIa/IIa (alpha 2 beta 1-integrin).
  • Eriksson A et al: Demonstration of functionally different interactions between phospholipase C-gamma and the two types of platelet-derived growth factor receptors.
  • Vassbotn FS et al: Neomycin is a platelet-derived growth factor (PDGF) antagonist that allows discrimination of PDGF alpha- and beta-receptor signals in cells expressing both receptor types.
  • McGowan EB et al: Modified platelet responses to thrombin. Evidence for two types of receptors or coupling mechanisms.
  • Satoh T et al: Platelet-derived growth factor receptor mediates activation of ras through different signaling pathways in different cell types.
  • Yoshikawa T et al: Thymidine phosphorylase/platelet-derived endothelial cell growth factor is upregulated in advanced solid types of gastric cancer.
  • Salvarani C et al: HLA typing, platelet associated immunoglobulins and anticardiolipin antibody.
  • Lipitz S et al: Neonatal alloimmune thrombocytopenia due to anti-P1A1 (anti-HPA-1a): importance of paternal and fetal platelet typing for assessment of fetal risk.
  • Chiang TM et al: Peptides derived from platelet non-integrin collagen-receptors or types I and III collagen inhibit collagen-platelet interaction.

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
  • (UID = 705691374.001).
  • [ISSN] 2331-5717
  • [Journal-full-title] BioMedLib Review
  • [Language] eng
  • [Publication-type] Review
  • [Publication-country] UNITED STATES
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48. |......... 1%  Glembotsky AC, Bluteau D, Espasandin YR, Goette NP, Marta RF, Marin Oyarzun CP, Korin L, Lev PR, Laguens RP, Molinas FC, Raslova H, Heller PG: Mechanisms underlying platelet function defect in a pedigree with familial platelet disorder with a predisposition to acute myelogenous leukemia: potential role for candidate RUNX1 targets. J Thromb Haemost; 2014 May;12(5):761-72
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Mechanisms underlying platelet function defect in a pedigree with familial platelet disorder with a predisposition to acute myelogenous leukemia: potential role for candidate RUNX1 targets.
  • BACKGROUND: Familial platelet disorder with a predisposition to acute myelogenous leukemia (FPD/AML) is an inherited platelet disorder caused by a germline RUNX1 mutation and characterized by thrombocytopenia, a platelet function defect, and leukemia predisposition.
  • The mechanisms underlying FPD/AML platelet dysfunction remain incompletely clarified.
  • We aimed to determine the contribution of platelet structural abnormalities and defective activation pathways to the platelet phenotype.
  • METHODS: Lumiaggregometry, α-granule and dense granule content and release, platelet ultrastructure, αIIb β3 integrin activation and outside-in signaling were assessed in members of one FPD/AML pedigree.
  • RESULTS: A severe decrease in platelet aggregation, defective αIIb β3 integrin activation and combined αδ storage pool deficiency were found.
  • However, whereas the number of dense granules was markedly reduced, α-granule content was heterogeneous.
  • A trend towards decreased platelet spreading was found, and β3 integrin phosphorylation was impaired, reflecting altered outside-in signaling.
  • A decrease in the level of transcription factor p45 NF-E2 was shown in platelet RNA and lysates, and other deregulated genes included RAB27B and MYL9.
  • RUNX1 was shown to bind to the NF-E2 promoter in primary megakaryocytes, and wild-type RUNX1, but not FPD/AML mutants, was able to activate NF-E2 expression.
  • CONCLUSIONS: The FPD/AML platelet function defect represents a complex trait, and RUNX1 orchestrates platelet function by regulating diverse aspects of this process.
  • This study highlights the RUNX1 target NF-E2 as part of the molecular network by which RUNX1 regulates platelet biogenesis and function.

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  • [Copyright] © 2014 International Society on Thrombosis and Haemostasis.
  • (PMID = 24606315.001).
  • [ISSN] 1538-7836
  • [Journal-full-title] Journal of thrombosis and haemostasis : JTH
  • [ISO-abbreviation] J. Thromb. Haemost.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Keywords] NOTNLM ; FPD/AML / NF-E2 transcription factor / RUNX1 protein / Rab27B protein, human / blood platelet disorder / platelet function tests
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49. |......... 1%  Amesse LS, Pfaff-Amesse T, Gunning WT, Duffy N, French JA 2nd: Clinical and laboratory characteristics of adolescents with platelet function disorders and heavy menstrual bleeding. Exp Hematol Oncol; 2013;2(1):3
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Clinical and laboratory characteristics of adolescents with platelet function disorders and heavy menstrual bleeding.
  • BACKGROUND: Platelet function disorders (PFDs) have emerged as an important etiology of heavy menstrual bleeding (HMB) in adolescents.
  • The objective of this study was to evaluate these parameters in order to distinguish characteristics of the disorder that in turn will lead to earlier diagnosis and therapy initiation.
  • Interestingly, 20 girls were diagnosed with a PFD prior to menarche and of these, only 6 (30%) went on to develop HMB after pubertal transition, while the majority (14; 70%) did not.
  • The average age-at-PFD diagnosis was 14.5yrs, significantly differing from the 10.9yrs average age-at-PFD diagnosis in their counterparts that, after menarche, did not develop HMB (P<.01) Blood type O occurred significantly more frequently (76%) than national norms (P <.037).
  • Incidence of δ-Storage Pool deficiency (δ-SPD) was significantly higher (74%) than their non-HMB cohorts (45%) (P <.007).
  • In 25.6% for whom standard platelet studies were normal, electron microscopy detected reduced platelet δ-granules numbers (δ-SPD).
  • High false negative standard platelet function study results indicate additional diagnostic strategies, particularly for δ-SPD, should be considered.

  • ExactAntigen/Labome. author profiles.
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  • (PMID = 23347697.001).
  • [ISSN] 2162-3619
  • [Journal-full-title] Experimental hematology & oncology
  • [ISO-abbreviation] Exp Hematol Oncol
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Other-IDs] NLM/ PMC3584827
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50. |......... 1%  Cooper IF, Siadaty MS: 'Laboratory Procedures' associated with 'Induced Platelet Aggregation': Top Publications. BioMedLib Review; LaboratoryProcedure;InducedPlateletAggregation:705347938. ISSN: 2331-5717. 2014/4/28
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  • [Title] 'Laboratory Procedures' associated with 'Induced Platelet Aggregation': Top Publications.
  • Background: There are articles published each month which present 'laboratory procedure' for 'induced platelet aggregation'.
  • Finding such articles is important for researchers, clinicians, and patients.
  • In group one there are publications with the strongest evidence of association. We focused finding the most relevant publications pertinent to our goal, rather than combining them into a conclusion section. Such textual synthesis will be the focus of our next project.
  • Bednarek-Tupikowska G et al: Influence of dehydroepiandrosterone on platelet aggregation, superoxide dismutase activity and serum lipid peroxide concentrations in rabbits with induced hypercholesterolemia.
  • Fontana P et al: Adenosine diphosphate-induced platelet aggregation is associated with P2Y12 gene sequence variations in healthy subjects.
  • Fushuku S et al: Morphological changes of platelets following platelet aggregation induced by collagen in Japanese Black cattle with delta-storage pool deficiency.
  • Fushuku S et al: Changes of platelet cytoskeletal proteins following platelet aggregation induced by collagen and platelet membrane glycoprotein in Japanese black cattle with delta-storage pool deficiency.
  • Cook JJ et al: Nonpeptide glycoprotein IIb/IIIa inhibitors. 15. Antithrombotic efficacy of L-738,167, a long-acting GPIIb/IIIa antagonist, correlates with inhibition of adenosine diphosphate-induced platelet aggregation but not with bleeding time prolongation.
  • Lopez-Johnston JC et al: Inhibition of adrenaline and adenosine diphosphate induced platelet aggregation by Lansberg's hognose pit viper (Porthidium lansbergii hutmanni) venom.
  • Pérez de León AA et al: Apyrase activity and adenosine diphosphate induced platelet aggregation inhibition by the salivary gland proteins of Culicoides variipennis, the North American vector of bluetongue viruses.
  • García M et al: Adenosine diphosphate-induced platelet aggregation correlates with platelet activation identified with the use of flow cytometry.
  • Greinacher A et al: Laboratory diagnosis of heparin-associated thrombocytopenia and comparison of platelet aggregation test, heparin-induced platelet activation test, and platelet factor 4/heparin enzyme-linked immunosorbent assay.
  • Nubile G et al: [Collagen-induced platelet aggregation in K562 neoplastic cells culture media].

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
  • (UID = 705347938.001).
  • [ISSN] 2331-5717
  • [Journal-full-title] BioMedLib Review
  • [Language] eng
  • [Publication-type] Review
  • [Publication-country] UNITED STATES
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2. Definitions


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