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1. |||||..... 50%  Wenning GK, Geser F, Krismer F, Seppi K, Duerr S, Boesch S, Köllensperger M, Goebel G, Pfeiffer KP, Barone P, Pellecchia MT, Quinn NP, Koukouni V, Fowler CJ, Schrag A, Mathias CJ, Giladi N, Gurevich T, Dupont E, Ostergaard K, Nilsson CF, Widner H, Oertel W, Eggert KM, Albanese A, del Sorbo F, Tolosa E, Cardozo A, Deuschl G, Hellriegel H, Klockgether T, Dodel R, Sampaio C, Coelho M, Djaldetti R, Melamed E, Gasser T, Kamm C, Meco G, Colosimo C, Rascol O, Meissner WG, Tison F, Poewe W, European Multiple System Atrophy Study Group: The natural history of multiple system atrophy: a prospective European cohort study. Lancet Neurol; 2013 Mar;12(3):264-74
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] The natural history of multiple system atrophy: a prospective European cohort study.
  • BACKGROUND: Multiple system atrophy (MSA) is a fatal and still poorly understood degenerative movement disorder that is characterised by autonomic failure, cerebellar ataxia, and parkinsonism in various combinations.
  • METHODS: Patients with a clinical diagnosis of MSA were recruited and followed up clinically for 2 years.
  • The parkinsonian variant of MSA (hazard ratio [HR] 2·08, 95% CI 1·09-3·97; p=0·026) and incomplete bladder emptying (HR 2·10, 1·02-4·30; p=0·044) predicted shorter survival.
  • [MeSH-major] Disease Progression. Multiple System Atrophy
  • [MeSH-minor] Aged. Autonomic Nervous System Diseases / diagnosis. Autonomic Nervous System Diseases / mortality. Autonomic Nervous System Diseases / physiopathology. Cerebellar Ataxia / diagnosis. Cerebellar Ataxia / mortality. Cerebellar Ataxia / physiopathology. Cohort Studies. Europe. Humans. Male. Middle Aged. Parkinson Disease / diagnosis. Parkinson Disease / mortality. Parkinson Disease / physiopathology. Phenotype. Prospective Studies. Severity of Illness Index


2. |||||..... 50%  Sakakibara R, Kishi M, Ogawa E, Tateno F, Uchiyama T, Yamamoto T, Yamanishi T: Bladder, bowel, and sexual dysfunction in Parkinson's disease. Parkinsons Dis; 2011;2011:924605
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  • [Title] Bladder, bowel, and sexual dysfunction in Parkinson's disease.
  • Bladder dysfunction (urinary urgency/frequency), bowel dysfunction (constipation), and sexual dysfunction (erectile dysfunction) (also called "pelvic organ" dysfunctions) are common nonmotor disorders in Parkinson's disease (PD).
  • The brain pathology causing the bladder dysfunction (appearance of overactivity) involves an altered dopamine-basal ganglia circuit, which normally suppresses the micturition reflex.
  • The pathophysiology of the pelvic organ dysfunction in PD differs from that in multiple system atrophy; therefore, it might aid in differential diagnosis.
  • Anticholinergic agents are used to treat bladder dysfunction in PD, although these drugs should be used with caution particularly in elderly patients who have cognitive decline.

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  • [Cites] Drugs Aging. 2004;21(14):911-30 [15554750.001]
  • [Cites] Am J Gastroenterol. 2005 Jan;100(1):124-9 [15654791.001]
  • [Cites] Mov Disord. 2005 Nov;20(11):1425-30 [16007638.001]
  • [Cites] Gut. 2005 Aug;54(8):1107-13 [16009682.001]
  • [Cites] Physiol Behav. 2005 Oct 15;86(3):356-68 [16135375.001]
  • [Cites] Neurogastroenterol Motil. 2005 Oct;17(5):637-42 [16185301.001]
  • [Cites] Neurology. 2005 Dec 27;65(12):1863-72 [16237129.001]
  • [Cites] J Comp Neurol. 2005 Dec 5;493(1):21-6 [16255005.001]
  • [Cites] J Comp Neurol. 2005 Dec 5;493(1):27-32 [16255006.001]
  • [Cites] Neurogastroenterol Motil. 2006 Feb;18(2):91-103 [16420287.001]
  • [Cites] J Neurosci. 2006 Mar 8;26(10):2798-807 [16525059.001]
  • [Cites] Neurology. 2006 Jul 11;67(1):125-30 [16832090.001]
  • [Cites] Brain. 2008 Jan;131(Pt 1):132-45 [17977862.001]
  • [Cites] J Urol. 2008 Oct;180(4):1241-8 [18707740.001]
  • [Cites] Neurobiol Dis. 2009 Oct;36(1):96-102 [19595768.001]
  • [Cites] CNS Neurosci Ther. 2010 Jun;16(3):e92-123 [20557568.001]
  • [Cites] Adv Neurol. 1987;45:19-34 [2881444.001]
  • [Cites] Am J Psychiatry. 1978 Dec;135(12):1552-5 [717578.001]
  • [Cites] Am J Physiol Gastrointest Liver Physiol. 2002 Nov;283(5):G1020-6 [12381514.001]
  • [Cites] Am J Physiol Gastrointest Liver Physiol. 2002 Dec;283(6):G1398-411 [12388212.001]
  • [Cites] Mov Disord. 2002 Nov;17(6):1371-4 [12465087.001]
  • [Cites] Jpn J Pharmacol. 2002 Dec;90(4):313-20 [12501007.001]
  • [Cites] Am J Gastroenterol. 2003 Feb;98(2):259-63 [12591038.001]
  • [Cites] Physiol Res. 2003;52(1):1-30 [12625803.001]
  • [Cites] J Urol. 2003 Apr;169(4):1388-91 [12629368.001]
  • [Cites] J Urol. 2003 May;169(5):1878-84 [12686866.001]
  • [Cites] Mov Disord. 2003 May;18(5):573-8 [12722172.001]
  • [Cites] J Neurol. 2003 Feb;250 Suppl 1:I28-30 [12761632.001]
  • [Cites] Physiol Res. 2003;52(3):275-84 [12790758.001]
  • [Cites] J Pharmacol Sci. 2003 Jun;92(2):143-8 [12832842.001]
  • [Cites] J Urol. 2003 Aug;170(2 Pt 2):S40-4; discussion S44-5 [12853772.001]
  • [Cites] Neuroscience. 2003;120(3):871-5 [12895527.001]
  • [Cites] Br J Pharmacol. 2003 Aug;139(8):1425-32 [12922929.001]
  • [Cites] Brain Res Bull. 1992 Aug;29(2):135-40 [1356067.001]
  • [Cites] Radiology. 1965 Jul;85:73-9 [14303073.001]
  • [Cites] Br J Urol. 1961 Dec;33:403-9 [14477379.001]
  • [Cites] Ann Neurol. 2004 Jan;55(1):118-20 [14705120.001]
  • [Cites] J Sex Marital Ther. 2004 Mar-Apr;30(2):95-105 [14742099.001]
  • [Cites] Parkinsonism Relat Disord. 2004 Mar;10(3):181-7 [15036175.001]
  • [Cites] Brain Res. 1992 May 15;580(1-2):92-9 [1504821.001]
  • [Cites] Neurourol Urodyn. 2004;23(3):265-72 [15098225.001]
  • [Cites] Drugs Aging. 2004;21(11):687-709 [15323576.001]
  • [Cites] Neurourol Urodyn. 2004;23(7):689-96 [15382192.001]
  • [Cites] Mov Disord. 2005 Jun;20(6):680-6 [15719424.001]
  • [Cites] Clin Ther. 2005 Feb;27(2):144-53 [15811477.001]
  • [Cites] Clin Auton Res. 2005 Apr;15(2):76-82 [15834763.001]
  • [Cites] World J Gastroenterol. 2005 May 14;11(18):2691-6 [15884105.001]
  • [Cites] Eur Neurol. 1992;32(3):134-40 [1592069.001]
  • [Cites] Sleep Med Rev. 2005 Aug;9(4):311-29 [15994100.001]
  • [Cites] Parkinsonism Relat Disord. 2005 Dec;11(8):509-12 [15994112.001]
  • [Cites] Prog Neurobiol. 2005 May;76(1):1-21 [16043278.001]
  • [Cites] Parkinsonism Relat Disord. 2005 Sep;11(6):381-6 [16109498.001]
  • [Cites] Neuroscience. 2005;135(1):299-303 [16111828.001]
  • [Cites] Mov Disord. 2006 Jan;21(1):115-6 [16142776.001]
  • [Cites] Prog Brain Res. 2006;152:335-43 [16198711.001]
  • [Cites] Prog Brain Res. 2006;152:359-72 [16198713.001]
  • [Cites] Eur J Neurol. 2005 Nov;12(11):842-50 [16241972.001]
  • [Cites] Parkinsonism Relat Disord. 2005 Dec;11(8):499-502 [16263322.001]
  • [Cites] Aliment Pharmacol Ther. 2005 Nov 15;22(10):997-1003 [16268975.001]
  • [Cites] BJU Int. 2005 Dec;96(9):1373-7 [16287461.001]
  • [Cites] Jpn J Psychiatry Neurol. 1992 Mar;46(1):181-6 [1635308.001]
  • [Cites] Br J Urol. 1992 Jul;70(1):100-2 [1638364.001]
  • [Cites] J Am Geriatr Soc. 2005 Dec;53(12):2195-201 [16398909.001]
  • [Cites] Pharmacol Res. 1999 Jan;39(1):11-9 [10051373.001]
  • [Cites] BJU Int. 1999 Mar;83 Suppl 2:1-6 [10210596.001]
  • [Cites] Neurourol Urodyn. 2000;19(1):101-9 [10602251.001]
  • [Cites] Urology. 2000 May;55(5A Suppl):51-7; discussion 59 [10767453.001]
  • [Cites] Intensive Care Med. 2000 Jul;26(7):901-7 [10990104.001]
  • [Cites] Trends Neurosci. 2000 Oct;23(10 Suppl):S2-7 [11052214.001]
  • [Cites] J Neurol Sci. 2001 Jun 15;187(1-2):55-9 [11440745.001]
  • [Cites] Horm Behav. 2002 Mar;41(2):170-7 [11855901.001]
  • [Cites] J Clin Gastroenterol. 2002 Mar;34(3):225-8 [11873100.001]
  • [Cites] J Gastrointest Surg. 2001 Jul-Aug;5(4):418-33 [11985985.001]
  • [Cites] Dis Colon Rectum. 2002 Aug;45(8):1016-22 [12195184.001]
  • [Cites] Asian J Androl. 2002 Dec;4(4):287-90 [12508130.001]
  • [Cites] J Comp Neurol. 2003 Apr 21;459(1):90-111 [12629668.001]
  • [Cites] Arq Neuropsiquiatr. 2003 Jun;61(2B):359-63 [12894267.001]
  • [Cites] Clin Neuropharmacol. 1992 Oct;15(5):375-80 [1330307.001]
  • [Cites] Arch Gen Psychiatry. 1965 Jan;12:29-45 [14221689.001]
  • [Cites] Gastroenterology. 1951 Mar;17(3):367-74 [14823189.001]
  • [Cites] Neurogastroenterol Motil. 2004 Apr;16 Suppl 1:143-7 [15066021.001]
  • [Cites] Neuroscience. 2004;126(4):1075-82 [15207340.001]
  • [Cites] World J Gastroenterol. 2006 Aug 7;12(29):4609-13 [16937428.001]
  • [Cites] Rev Urol. 2002;4 Suppl 4:S7-S18 [16986023.001]
  • [Cites] Neuroscience. 2006 Nov 3;142(4):1273-80 [16996219.001]
  • [Cites] Brain. 2006 Dec;129(Pt 12):3366-75 [17077105.001]
  • [Cites] World J Gastroenterol. 2006 Nov 28;12(44):7069-74 [17131466.001]
  • [Cites] Mov Disord. 2007 Jan 15;22(2):220-5 [17133504.001]
  • [Cites] Mov Disord. 2007 Aug 15;22(11):1672-3 [17149723.001]
  • [Cites] Urology. 1976 Jan;7(1):24-8 [174263.001]
  • [Cites] Curr Top Med Chem. 2007;7(11):1137-44 [17584134.001]
  • [Cites] Klin Wochenschr. 1991 Dec 11;69(20):906-9 [1795497.001]
  • [Cites] Int J Impot Res. 2008 Jan-Feb;20(1):105-10 [18059502.001]
  • [Cites] J Urol. 2008 Mar;179(3):1066-71 [18206919.001]
  • [Cites] J Neural Transm. 2008;115(3):443-60 [18327532.001]
  • [Cites] J Neurol. 1991 Feb;238(1):19-22 [1851513.001]
  • [Cites] Gut. 1991 Aug;32(8):941-4 [1885077.001]
  • [Cites] Urol Int. 2008;81(3):335-9 [18931554.001]
  • [Cites] Dig Dis Sci. 1990 Mar;35(3):327-32 [1968372.001]
  • [Cites] Acta Neuropathol. 1990;79(6):581-3 [1972853.001]
  • [Cites] Behav Brain Res. 2010 Jan 20;206(2):286-92 [19782104.001]
  • [Cites] J Sex Med. 2010 Feb;7(2 Pt 2):858-72 [19929916.001]
  • [Cites] J Sex Med. 2010 Jun;7(6):2056-67 [20345736.001]
  • [Cites] Mov Disord. 1991;6(2):151-6 [2057006.001]
  • [Cites] J Epidemiol Community Health. 1990 Sep;44(3):246-8 [2148771.001]
  • [Cites] Neurology. 1990 Sep;40(9):1347-9 [2392215.001]
  • [Cites] Arch Int Physiol Biochim. 1985 Jun;93(2):93-100 [2412517.001]
  • [Cites] Arch Neurol. 1989 Oct;46(10):1061-4 [2803065.001]
  • [Cites] Neurology. 1987 Jul;37(7):1253-5 [3037441.001]
  • [Cites] Neurosci Lett. 1986 Mar 28;65(1):79-83 [3486388.001]
  • [Cites] Ann Neurol. 1978 Feb;3(2):129-33 [350130.001]
  • [Cites] Clin Gastroenterol. 1986 Oct;15(4):937-65 [3536215.001]
  • [Cites] J Am Geriatr Soc. 1987 Jul;35(7):683-4 [3584772.001]
  • [Cites] J Pharmacol Exp Ther. 1987 Aug;242(2):548-51 [3612550.001]
  • [Cites] J Urol. 1987 Oct;138(4):836-8 [3656541.001]
  • [Cites] Clin Neuropharmacol. 1986;9(5):440-7 [3768866.001]
  • [Cites] Dig Dis Sci. 1985 May;30(5):419-25 [3987475.001]
  • [Cites] Int Urol Nephrol. 1985;17(1):35-41 [3997405.001]
  • [Cites] Gastroenterology. 1985 Dec;89(6):1411-4 [4054533.001]
  • [Cites] Eur J Clin Pharmacol. 1985;29(3):307-10 [4076326.001]
  • [Cites] Acta Neurol Scand. 1985 Nov;72(5):506-11 [4082917.001]
  • [Cites] Br J Urol. 1985 Dec;57(6):652-6 [4084724.001]
  • [Cites] Gastroenterology. 1984 Oct;87(4):848-56 [6088351.001]
  • [Cites] Br J Clin Pharmacol. 1984 Dec;18(6):959-62 [6529536.001]
  • [Cites] JAMA. 1983 Mar 4;249(9):1148 [6823069.001]
  • [Cites] J Urol. 1983 Jan;129(1):80-3 [6827689.001]
  • [Cites] Gastroenterology. 1983 Jul;85(1):105-13 [6852445.001]
  • [Cites] J Comp Neurol. 2000 Feb 21;417(4):399-414 [10701863.001]
  • [Cites] Brain. 2000 Apr;123 ( Pt 4):781-9 [10734009.001]
  • [Cites] Dis Colon Rectum. 2000 Jul;43(7):940-3 [10910239.001]
  • [Cites] Am J Physiol Gastrointest Liver Physiol. 2000 Aug;279(2):G311-8 [10915639.001]
  • [Cites] Urology. 2000 Aug 1;56(2):250-4 [10925088.001]
  • [Cites] Am J Physiol Regul Integr Comp Physiol. 2000 Aug;279(2):R599-609 [10938251.001]
  • [Cites] J Urol. 2000 Nov;164(5):1640-3 [11025724.001]
  • [Cites] Spinal Cord. 2000 Oct;38(10):615-21 [11093323.001]
  • [Cites] Neurourol Urodyn. 2001;20(1):105-13 [11135387.001]
  • [Cites] J Clin Pharmacol. 2001 Jun;41(6):636-44 [11402632.001]
  • [Cites] Neurology. 2001 Aug 14;57(3):456-62 [11502913.001]
  • [Cites] Mov Disord. 2001 Nov;16(6):1176-7 [11748757.001]
  • [Cites] Exp Neurol. 2002 Jan;173(1):129-35 [11771945.001]
  • [Cites] Trends Neurosci. 2002 Jan;25(1):45-50 [11801338.001]
  • [Cites] Mov Disord. 2002 Jan;17(1):223-5 [11835473.001]
  • [Cites] Neurourol Urodyn. 2002;21(2):167-78 [11857671.001]
  • [Cites] Mov Disord. 2002;17 Suppl 3:S63-8 [11948756.001]
  • [Cites] Parkinsonism Relat Disord. 2002 Mar;8(4):277-84 [12039423.001]
  • [Cites] Eur Urol. 2002 Apr;41(4):382-6 [12074807.001]
  • [Cites] Mov Disord. 1995 May;10(3):337-40 [7651453.001]
  • [Cites] Am J Gastroenterol. 1994 Jan;89(1):15-25 [8273792.001]
  • [Cites] Brain. 1995 Dec;118 ( Pt 6):1447-59 [8595476.001]
  • [Cites] Clin Neuropharmacol. 1995 Feb;18(1):76-8 [8665538.001]
  • [Cites] Neurourol Urodyn. 1997;16(2):111-5 [9042673.001]
  • [Cites] Eur Neurol. 1997;38 Suppl 2:2-7 [9387796.001]
  • [Cites] Mov Disord. 1997 Nov;12(6):923-7 [9399216.001]
  • [Cites] Mov Disord. 1997 Nov;12(6):946-51 [9399219.001]
  • [Cites] Mov Disord. 1997 Nov;12(6):952-7 [9399220.001]
  • [Cites] Brain Res Brain Res Rev. 1998 May;26(2-3):148-53 [9651511.001]
  • [Cites] Prog Neurobiol. 1998 Nov;56(4):433-506 [9775401.001]
  • [Cites] J Auton Nerv Syst. 1998 Dec 11;74(2-3):189-92 [9915636.001]
  • [Cites] Sheng Li Xue Bao. 1995 Jun;47(3):245-52 [7570109.001]
  • [Cites] Acta Neurol Scand. 1995 Jun;91(6):453-5 [7572039.001]
  • [Cites] Gut. 1976 Apr;17(4):306-12 [773793.001]
  • [Cites] Neurosci Lett. 1995 Jan 2;183(1-2):58-61 [7746488.001]
  • [Cites] Br J Surg. 1995 Feb;82(2):173-6 [7749679.001]
  • [Cites] World J Urol. 1995;13(1):30-3 [7773315.001]
  • [Cites] Mov Disord. 1995 Jan;10(1):66-70 [7885357.001]
  • [Cites] Mov Disord. 1995 Jan;10(1):81-4 [7885359.001]
  • [Cites] Dig Dis Sci. 1994 Nov;39(11):2364-7 [7956604.001]
  • [Cites] Mov Disord. 1993;8(1):83-6 [8093549.001]
  • [Cites] Acta Neuropathol. 1993;86(4):397-8 [8256591.001]
  • [Cites] Neurourol Urodyn. 1993;12(3):203-9 [8330043.001]
  • [Cites] Br J Surg. 1993 Aug;80(8):1070-1 [8402072.001]
  • [Cites] Mov Disord. 1993 Apr;8(2):151-7 [8474481.001]
  • [Cites] Ann Neurol. 1993 May;33(5):490-3 [8498826.001]
  • [Cites] Mov Disord. 1995 Sep;10(5):650-7 [8552119.001]
  • [Cites] Age Ageing. 1995 Nov;24(6):499-504 [8588540.001]
  • [Cites] Postgrad Med J. 1996 Mar;72(845):164-7 [8731708.001]
  • [Cites] Brain Res Brain Res Rev. 1995 Sep;21(2):162-84 [8866673.001]
  • [Cites] J Neurol Sci. 1996 Sep 15;141(1-2):105-10 [8880701.001]
  • [Cites] Pharmacol Res. 1996 Apr-May;33(4-5):217-26 [8938012.001]
  • [Cites] Mov Disord. 1997 May;12(3):423-5 [9159740.001]
  • [Cites] Clin Auton Res. 1997 Jun;7(3):163-5 [9232362.001]
  • [Cites] Br J Urol. 1997 Jul;80(1):100-4 [9240187.001]
  • [Cites] Neurosci Lett. 1997 Sep 19;233(2-3):109-12 [9350844.001]
  • [Cites] Adv Exp Med Biol. 1997;427:211-9 [9361846.001]
  • [Cites] Mov Disord. 1997 Sep;12(5):764-6 [9380063.001]
  • [Cites] Br J Urol. 1997 Dec;80(6):940-5 [9439415.001]
  • [Cites] J Pharmacol Exp Ther. 1998 Jul;286(1):228-33 [9655864.001]
  • [Cites] Am J Respir Crit Care Med. 1998 Aug;158(2):458-64 [9700121.001]
  • [Cites] Wien Klin Wochenschr. 1998 Aug 21;110(15):535-7 [9782572.001]
  • [Cites] Am J Physiol. 1998 Dec;275(6 Pt 2):R2049-55 [9843896.001]
  • [Cites] Neuroscience. 1998 Sep;86(2):353-87 [9881853.001]
  • [Cites] J Urol. 2006 Jan;175(1):202-6; discussion 206-7 [16406911.001]
  • [Cites] Int J Urol. 2006 Jan;13(1):29-35 [16448429.001]
  • [Cites] J Urol. 2006 Mar;175(3 Pt 2):S5-10 [16458739.001]
  • [Cites] Br J Pharmacol. 2006 Feb;147 Suppl 2:S14-24 [16465178.001]
  • [Cites] Br J Pharmacol. 2006 Feb;147 Suppl 2:S25-40 [16465182.001]
  • [Cites] Intern Med. 2006;45(1):35-6 [16467605.001]
  • [Cites] J Urol. 2006 Mar;175(3 Pt 1):994-8 [16469600.001]
  • [Cites] Urology. 2006 Mar;67(3):447-51 [16504269.001]
  • [Cites] Neurourol Urodyn. 2006;25(4):356-60 [16532465.001]
  • [Cites] Mol Psychiatry. 2006 Aug;11(8):782-6 [16619053.001]
  • [Cites] Int J Impot Res. 2006 Nov-Dec;18(6):550-8 [16625231.001]
  • [Cites] Br J Pharmacol. 2006 Jul;148(5):565-78 [16751797.001]
  • [Cites] Age Ageing. 2006 Sep;35(5):540-1 [16799178.001]
  • [Cites] Clin Auton Res. 2006 Aug;16(4):289-92 [16810464.001]
  • [Cites] J Urol. 2006 Aug;176(2):456-62 [16813864.001]
  • [Cites] J Urol. 2006 Aug;176(2):679-83 [16813919.001]
  • [Cites] BJU Int. 2006 Jul;98(1):125-31 [16831156.001]
  • [Cites] J Physiol Pharmacol. 2006 Jun;57(2):291-300 [16845232.001]
  • [Cites] Brain Res. 1990 Feb 26;510(1):104-7 [1691042.001]
  • (PMID = 21918729.001).
  • [ISSN] 2042-0080
  • [Journal-full-title] Parkinson's disease
  • [ISO-abbreviation] Parkinsons Dis
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Other-IDs] NLM/ PMC3171780
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3. |||||..... 49%  Rambau PF, Chandika A, Chalya PL, Jackson K: Scrotal Swelling and Testicular Atrophy due to Schistosomiasis in a 9-Year-Old Boy: A Case Report. Case Rep Infect Dis; 2011;2011:787961
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  • [Title] Scrotal Swelling and Testicular Atrophy due to Schistosomiasis in a 9-Year-Old Boy: A Case Report.
  • Schistosomiasis is a communicable disease which commonly involves urinary bladder causing hematuria, or large bowel causing bloody stool.

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  • (PMID = 22567478.001).
  • [ISSN] 2090-6633
  • [Journal-full-title] Case reports in infectious diseases
  • [ISO-abbreviation] Case Rep Infect Dis
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Egypt
  • [Other-IDs] NLM/ PMC3336235
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4. |||||..... 47%  Karthik TS, Prasad NR, Rani PR, Maheshwari R, Reddy PA, Chakradhar BV, Menon B: A rare case of short stature: Say Meyer syndrome. Indian J Endocrinol Metab; 2013 Oct;17(Suppl 1):S130-1
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  • He had global developmental delay and there was a lack of bowel and bladder control.
  • Magnetic resonance imaging (MRI) brain revealed mild diffuse frontotemporal atrophy with multiple irregular gliotic areas in bilateral frontal lobes.
  • Characteristic MRI brain findings include diffuse frontotemporal atrophy with multiple gliotic areas in frontal lobes.

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  • (PMID = 24251132.001).
  • [ISSN] 2230-8210
  • [Journal-full-title] Indian journal of endocrinology and metabolism
  • [ISO-abbreviation] Indian J Endocrinol Metab
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] India
  • [Other-IDs] NLM/ PMC3830278
  • [Keywords] NOTNLM ; Craniosynostosis / Say Meyer syndrome / frontotemporal atrophy / multiple gliotic areas / short stature
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5. |||||..... 47%  Kim JA, Roy RR, Kim SJ, Zhong H, Haddad F, Baldwin KM, Edgerton VR: Electromechanical modulation of catabolic and anabolic pathways in chronically inactive, but neurally intact, muscles. Muscle Nerve; 2010 Sep;42(3):410-21
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  • SI-Stim1 and SI-Stim2 paradigms attenuated plantaris atrophy by 20% and 38%, respectively, whereas only SI-Stim2 blunted soleus atrophy (24%) relative to SI-C.
  • No relationships between SI or electromechanical stimulation and expression of several atrophy markers were observed.
  • These data suggest that regulatory mechanisms for maintaining muscle mass previously shown in acute states of atrophy differ substantially from those observed in chronic states.
  • [MeSH-minor] Animals. Blotting, Western. Body Weight / physiology. DNA / genetics. Electric Stimulation. Electrophysiology. Female. Isometric Contraction / physiology. Muscle Denervation. Muscle Proteins / biosynthesis. Muscular Atrophy / metabolism. Muscular Atrophy / physiopathology. Organ Size / physiology. Physical Stimulation. RNA / biosynthesis. RNA / isolation & purification. Rats. Rats, Sprague-Dawley. Reverse Transcriptase Polymerase Chain Reaction. Urinary Bladder / innervation. Urinary Bladder / physiology

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  • [Cites] Biochim Biophys Acta. 2000 Jun 2;1497(1):77-88 [10838161.001]
  • [Cites] Muscle Nerve. 2009 Jul;40(1):69-78 [19533653.001]
  • [Cites] Nat Cell Biol. 2001 Nov;3(11):1014-9 [11715023.001]
  • [Cites] Muscle Nerve. 2002 Aug;26(2):238-51 [12210389.001]
  • [Cites] Am J Physiol Cell Physiol. 2002 Oct;283(4):C1182-95 [12225982.001]
  • [Cites] J Clin Endocrinol Metab. 2002 Dec;87(12):5553-8 [12466352.001]
  • [Cites] Biochem J. 2003 Jun 1;372(Pt 2):555-66 [12611592.001]
  • [Cites] J Appl Physiol (1985). 2003 Aug;95(2):781-90 [12716870.001]
  • [Cites] J Appl Physiol (1985). 2003 Aug;95(2):791-802 [12716877.001]
  • [Cites] Am J Physiol Regul Integr Comp Physiol. 2003 Nov;285(5):R1086-90 [12881204.001]
  • [Cites] Cell. 2004 Apr 30;117(3):399-412 [15109499.001]
  • [Cites] Biochem J. 2004 Jun 15;380(Pt 3):795-804 [15030312.001]
  • [Cites] Muscle Nerve. 2004 Jul;30(1):77-86 [15221882.001]
  • [Cites] Am J Physiol Endocrinol Metab. 2004 Oct;287(4):E591-601 [15100091.001]
  • [Cites] Biochim Biophys Acta. 1971 Aug 6;245(1):259-62 [4332100.001]
  • [Cites] Med Biol Eng Comput. 1991 Nov;29(6):NS13-9 [1813741.001]
  • [Cites] Lab Anim Sci. 1992 Aug;42(4):335-43 [1434492.001]
  • [Cites] J Appl Physiol (1985). 1993 May;74(5):2509-16 [8335584.001]
  • [Cites] J Appl Physiol (1985). 1996 Dec;81(6):2509-16 [9018499.001]
  • [Cites] Nature. 1997 May 1;387(6628):83-90 [9139826.001]
  • [Cites] Muscle Nerve. 1998 Mar;21(3):375-89 [9486867.001]
  • [Cites] Eur J Biochem. 1998 Aug 15;256(1):45-50 [9746344.001]
  • [Cites] J Appl Physiol (1985). 1998 Dec;85(6):2237-48 [9843548.001]
  • [Cites] Mol Cell Biol. 1999 Jun;19(6):4525-34 [10330191.001]
  • [Cites] J Anat. 1999 Apr;194 ( Pt 3):323-34 [10386770.001]
  • [Cites] Am J Physiol. 1999 Aug;277(2 Pt 2):R601-6 [10444569.001]
  • [Cites] J Biol Chem. 2005 Jan 28;280(4):2737-44 [15550386.001]
  • [Cites] Am J Physiol Regul Integr Comp Physiol. 2005 Jun;288(6):R1423-31 [15886351.001]
  • [Cites] J Investig Med. 2005 Apr;53(3):135-42 [15921033.001]
  • [Cites] J Am Soc Nephrol. 2006 Jul;17(7):1807-19 [16738015.001]
  • [Cites] J Physiol. 2006 Nov 1;576(Pt 3):923-33 [16916907.001]
  • [Cites] Med Sci Sports Exerc. 2006 Nov;38(11):1950-7 [17095929.001]
  • [Cites] FASEB J. 2007 Jan;21(1):140-55 [17116744.001]
  • [Cites] J Appl Physiol (1985). 2007 Jul;103(1):195-205 [17431083.001]
  • [Cites] J Appl Physiol (1985). 2007 Nov;103(5):1914; author reply 1915 [17965248.001]
  • [Cites] J Appl Physiol (1985). 2008 Jan;104(1):27-33 [17885021.001]
  • [Cites] Muscle Nerve. 2008 Apr;37(4):505-13 [18236467.001]
  • [Cites] J Appl Physiol (1985). 2008 Oct;105(4):1246-54 [18653749.001]
  • [Cites] J Appl Physiol (1985). 2009 Jan;106(1):178-86 [18988763.001]
  • [Cites] Nat Cell Biol. 2001 Nov;3(11):1009-13 [11715022.001]
  • (PMID = 20658566.001).
  • [ISSN] 1097-4598
  • [Journal-full-title] Muscle & nerve
  • [ISO-abbreviation] Muscle Nerve
  • [Language] eng
  • [Grant] United States / NINDS NIH HHS / NS / NS16333; United States / NINDS NIH HHS / NS / P01 NS016333-269001; United States / NIBIB NIH HHS / EB / R01 EB007615; United States / NCRR NIH HHS / RR / RR02-056
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Muscle Proteins; 63231-63-0 / RNA; 9007-49-2 / DNA
  • [Other-IDs] NLM/ NIHMS237322; NLM/ PMC2950751
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6. |||||..... 47%  Woo GH, Jho YS, Bak EJ: Canine distemper virus infection in fennec fox (Vulpes zerda). J Vet Med Sci; 2010 Aug;72(8):1075-9
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  • Histopathologically, there were lymphohistiocytic meningoencephalitis with malacia, mild interstitial pneumonia, lymphoid depletion of lymphoid tissues and organs, and intestinal villous atrophy with intralesional coccidia.
  • There were many intracytoplasmic and/or intranuclear inclusion bodies in the epithelial cells of the medullary velum, lungs, liver, kidneys, trachea, pancreas, stomach, gall bladder, urinary bladder, and ureters, and in macrophages of malacia foci and lymphocytes and macrophages of lymphoid organs.
  • [MeSH-major] Distemper / diagnosis. Distemper Virus, Canine / pathogenicity
  • [MeSH-minor] Animals. Atrophy. DNA Primers. Dehydration / pathology. Dehydration / veterinary. Dehydration / virology. Dogs. Emaciation / pathology. Emaciation / veterinary. Emaciation / virology. Eye Diseases / pathology. Eye Diseases / veterinary. Eye Diseases / virology. Female. Foxes. Genome, Viral. Lymphoid Tissue / pathology. Lymphoid Tissue / virology. Male. Reverse Transcriptase Polymerase Chain Reaction. Sudan

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  • (PMID = 20299771.001).
  • [ISSN] 0916-7250
  • [Journal-full-title] The Journal of veterinary medical science / the Japanese Society of Veterinary Science
  • [ISO-abbreviation] J. Vet. Med. Sci.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Japan
  • [Chemical-registry-number] 0 / DNA Primers
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7. |||||..... 47%  Tan A, Unluoglu S, Bayol U, Emil Sayhan S, Altinel D: Ureteritis cystica presenting with atrophic kidney: report of a case. ScientificWorldJournal; 2010;10:1535-8
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  • Ureteritis cystica is a rare proliferative condition that is found predominantly in the bladder, renal pelvis, and upper ureter.
  • In conclusion, ureteritis cystica is a benign and indolent lesion that needs to be kept in mind among the causes of renal atrophy.
  • [MeSH-major] Cysts / diagnosis. Kidney / pathology. Ureteral Diseases / diagnosis
  • [MeSH-minor] Aged. Atrophy / complications. Atrophy / diagnosis. Chronic Disease. Diagnosis, Differential. Female. Humans. Pyelonephritis / complications. Pyelonephritis / diagnosis

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  • (PMID = 20694450.001).
  • [ISSN] 1537-744X
  • [Journal-full-title] TheScientificWorldJournal
  • [ISO-abbreviation] ScientificWorldJournal
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] England
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8. |||||||||. 40429%  Cooper IF, Siadaty MS: 'Pathologic Functions' associated with 'Bladder Atrophy': Top Publications. BioMedLib Review; PathologicFunction;BladderAtrophy:706817524. ISSN: 2331-5717. 2014/8/1
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  • [Title] 'Pathologic Functions' associated with 'Bladder Atrophy': Top Publications.
  • [Transliterated title]
  • Background: There are articles published each month which present 'Pathologic Function' for 'bladder atrophy'.
  • Finding such articles is important for researchers, clinicians, and patients.
  • However these articles are spread across thousands of journals, and there are many types of 'Pathologic Function'.
  • This makes searching and locating the relevant publications a challenge.
  • We have used BioMedLib's semantic search technology to address the issue, and gathered all the pertinent publications in this review article.
  • Methods: We categorized the publications we found into two groups.
  • We used the strength of textual-association to separate the groups.
  • In group one there are publications with the strongest evidence of association. We focused finding the most relevant publications pertinent to our goal, rather than combining them into a conclusion section. Such textual synthesis will be the focus of our next project.
  • Results: Group one includes 15 publications, and group two 647 publications.
  • Here are the top 9.
  • Koizumi S et al: [Adenocarcinoma occurring 37 years after augmentation ileocystoplasty for tuberculous bladder atrophy: report of a case].
  • Nakata W et al: [A rare case of transitional cell carcinoma of the ileum segment arising 43 years after ureteroileocystoplasty due to tuberculous bladder atrophy].
  • Okoshi M et al: [Cystectasia for bladder atrophy in patients with a single kidney].
  • CIBERT J et al: [Entero-cystoplasty in post-tuberculous bladder atrophy].
  • Murayama K et al: [Clinical application of thyrotropin-releasing hormone to neurogenic bladder caused by olivo-ponto-cerebellar atrophy].
  • Boudes M et al: Bladder dysfunction in a transgenic mouse model of multiple system atrophy.
  • Peterson T et al: Simultaneous arterial and urinary bladder pressure recordings in multiple system atrophy and in spinal disorders with detrusor hyperreflexia.
  • Cremers CW et al: Juvenile diabetes mellitus, optic atrophy, hearing loss, diabetes insipidus, atonia of the urinary tract and bladder, and other abnormalities (Wolfram syndrome). A review of 88 cases from the literature with personal observations on 3 new patients.
  • Chaudhuri KR: Management of bladder dysfunction in patients with multiple-system atrophy.

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
  • (UID = 706817524.001).
  • [ISSN] 2331-5717
  • [Journal-full-title] BioMedLib Review
  • [Language] eng
  • [Publication-type] Review
  • [Publication-country] UNITED STATES
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9. |||||||||. 1786%  Watanabe T, Masago T, Miyagawa I: Apoptotic action of botulinum toxin on detrusor muscle in rats. Urol Int; 2010;84(3):341-6
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  • METHODS: The bladder wall and prostate in adult male Sprague-Dawley rats were injected with BTX-A (10 U at each site).
  • RESULTS: Although bladder weight was significantly increased in the BTX-A group compared to controls, no significant changes in prostate weight were identified.
  • CONCLUSIONS: The lack of significant changes in cellular dynamics of the detrusor muscle in the current study suggests that BTX-A injection into detrusor muscle for overactive bladder may not induce bladder atrophy.

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  • [Copyright] Copyright 2010 S. Karger AG, Basel.
  • (PMID = 20389167.001).
  • [ISSN] 1423-0399
  • [Journal-full-title] Urologia internationalis
  • [ISO-abbreviation] Urol. Int.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Switzerland
  • [Chemical-registry-number] 0 / Neuromuscular Agents; EC 3.4.24.69 / Botulinum Toxins, Type A
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10. |||||||||. 1781%  Pagano D, Cintorino D, Li Petri S, di Francesco F, Ricotta C, Argento J, Echeverri GJ, Bertani T, Riva S, Gridelli BG, Spada M: Laparoscopic approach for an intra-abdominal kidney allograft nephrectomy after pediatric transplantation: a case report. Transplant Proc; 2012 Sep;44(7):2036-7
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  • A 15-year-old girl underwent transplantation with a cadaveric donor kidney due to congenital pyelonephritis, vesicoureteral reflux, and secondary bladder atrophy.
  • After recurrent urinary tract infections, the patient was reintroduced to hemodialysis in 2010.

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  • [Copyright] Copyright © 2012 Elsevier Inc. All rights reserved.
  • (PMID = 22974901.001).
  • [ISSN] 1873-2623
  • [Journal-full-title] Transplantation proceedings
  • [ISO-abbreviation] Transplant. Proc.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
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11. |||||..... 53%  Boudes M, Uvin P, Pinto S, Voets T, Fowler CJ, Wenning GK, De Ridder D, Stefanova N: Bladder dysfunction in a transgenic mouse model of multiple system atrophy. Mov Disord; 2013 Mar;28(3):347-55
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  • [Title] Bladder dysfunction in a transgenic mouse model of multiple system atrophy.
  • Multiple system atrophy (MSA) is an adult-onset neurodegenerative disorder presenting with motor impairment and autonomic dysfunction.
  • To date, bladder function and structure have never been investigated in MSA models.
  • We aimed to test bladder function in a transgenic MSA mouse featuring oligodendroglial α-synucleinopathy and define its applicability as a preclinical model to study urological failure in MSA.
  • Bladder morphology and neuropathology of the lumbosacral intermediolateral column and the pontine micturition center (PMC) were analyzed in young and aged mice.
  • Urodynamic analysis revealed a less efficient and unstable bladder in MSA mice with increased voiding contraction amplitude, higher frequency of nonvoiding contractions, and increased postvoid residual volume.
  • MSA mice bladder walls showed early detrusor hypertrophy and age-related urothelium hypertrophy.
  • [MeSH-major] Multiple System Atrophy / complications. Urinary Bladder Diseases / etiology
  • [MeSH-minor] Acetylcholine / pharmacology. Age Factors. Animals. Brain / pathology. Disease Models, Animal. Disease Progression. Diuresis / drug effects. Diuresis / physiology. Female. Gene Expression Regulation / genetics. Humans. Image Cytometry. In Vitro Techniques. Male. Mice. Mice, Inbred C57BL. Mice, Transgenic. Myelin Proteolipid Protein / genetics. Urinary Bladder / drug effects. Urinary Bladder / metabolism. Urinary Bladder / physiopathology. Uterine Contraction / drug effects. Uterine Contraction / genetics. alpha-Synuclein / genetics

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  • [Copyright] Copyright © 2013 Movement Disorder Society.
  • (PMID = 23426727.001).
  • [ISSN] 1531-8257
  • [Journal-full-title] Movement disorders : official journal of the Movement Disorder Society
  • [ISO-abbreviation] Mov. Disord.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Myelin Proteolipid Protein; 0 / Plp1 protein, mouse; 0 / alpha-Synuclein; N9YNS0M02X / Acetylcholine
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12. |||||..... 53%  Hillard T: The postmenopausal bladder. Menopause Int; 2010 Jun;16(2):74-80
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  • [Title] The postmenopausal bladder.
  • The bladder and its surrounding structures are rich in estrogen receptors and there are demonstrable physiological and anatomical changes that occur around and immediately after the menopause.
  • The prevalence of many bladder symptoms, such as frequency, urgency and incontinence, does seem to increase around the menopause, but there is continuing debate over whether these effects are due to ageing, menopause or a combination of the two.
  • The impact of the menopause varies with individual symptoms; stress urinary incontinence being associated more with estrogen deficiency than urge incontinence which seems to be more age-related.
  • Based on these effects, it could be anticipated that estrogen replacement would have a positive effect on bladder symptoms.
  • It seems that topical estrogens do have some positive effects, particularly on symptoms of urgency, frequency and urge incontinence, the prevention of urogenital atrophy and in the prevention of recurrent urinary tract infections.
  • The possible explanations for this apparent contradiction are explored, but based on current evidence, it would appear preferable to use vaginal estrogens rather than systemic as part of the management of menopause-related bladder problems.
  • [MeSH-major] Estrogen Replacement Therapy. Postmenopause / physiology. Urinary Bladder / physiopathology. Urinary Incontinence / etiology. Urinary Incontinence / physiopathology
  • [MeSH-minor] Administration, Intravaginal. Age Factors. Female. Humans. Receptors, Estrogen / physiology. Receptors, Progesterone / physiology. Urinary Tract Infections / prevention & control

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  • (PMID = 20729499.001).
  • [ISSN] 1754-0461
  • [Journal-full-title] Menopause international
  • [ISO-abbreviation] Menopause Int
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Receptors, Estrogen; 0 / Receptors, Progesterone
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13. |||||..... 53%  Domoney C: Treatment of vaginal atrophy. Womens Health (Lond Engl); 2014 Mar;10(2):191-200
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  • [Title] Treatment of vaginal atrophy.
  • Vaginal or vulvovaginal atrophy is a widespread but poorly recognized condition of peri- and post-menopausal women.
  • It causes urogenital symptoms of dryness, reduced lubrication, itching, burning, irritable bladder symptoms and painful intercourse.
  • However, when embedded, bladder and sexual changes have occurred and these may be more difficult to remedy.
  • [MeSH-minor] Administration, Cutaneous. Administration, Intravaginal. Administration, Oral. Atrophy / drug therapy. Dehydroepiandrosterone / therapeutic use. Female. Humans. Hyaluronic Acid / therapeutic use. Oxytocin / therapeutic use. Testosterone / therapeutic use

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  • (PMID = 24601810.001).
  • [ISSN] 1745-5065
  • [Journal-full-title] Women's health (London, England)
  • [ISO-abbreviation] Womens Health (Lond Engl)
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Androgens; 0 / Lubricants; 0 / Oxytocics; 0 / Selective Estrogen Receptor Modulators; 3XMK78S47O / Testosterone; 459AG36T1B / Dehydroepiandrosterone; 50-56-6 / Oxytocin; 9004-61-9 / Hyaluronic Acid
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14. |||||..... 53%  Figueroa JJ, Singer W, Parsaik A, Benarroch EE, Ahlskog JE, Fealey RD, Parisi JE, Sandroni P, Mandrekar J, Iodice V, Low PA, Bower JH: Multiple system atrophy: prognostic indicators of survival. Mov Disord; 2014 Aug;29(9):1151-7
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  • [Title] Multiple system atrophy: prognostic indicators of survival.
  • Neurological and autonomic presentation in multiple system atrophy (MSA) may predict early mortality.
  • Survival was shorter in patients with early laboratory evidence of generalized (composite autonomic severity score ≥ 6) autonomic failure (7.0 [3.9-9.8] vs. 9.8 [4.6-13.8] years; P = 0.036), and early requirement of bladder catheterization (7.3 [3.1-10.2] vs. 13.7 [8.5-14.9] years; P = 0.003) compared with those without these clinical features.
  • On Cox proportional analysis, prognostic indicators of shorter survival were older age at onset (hazard ratio [95% confidence interval], 1.04 [1.01-1.08]; P = 0.03), early requirement of bladder catheterization (7.9 [1.88-38.63]; P = 0.004), and early generalized (composite autonomic severity score ≥ 6) autonomic failure (2.8 [1.01-9.26]; P = 0.047).
  • Gender, phenotype, and early development of gait instability, aid-requiring ambulation, orthostatic symptoms, neurogenic bladder, or significant anhidrosis (thermoregulatory sweat test ≥ 40%) were not indicators of shorter survival.

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  • [Copyright] © 2014 International Parkinson and Movement Disorder Society.
  • (PMID = 24909319.001).
  • [ISSN] 1531-8257
  • [Journal-full-title] Movement disorders : official journal of the Movement Disorder Society
  • [ISO-abbreviation] Mov. Disord.
  • [Language] eng
  • [Grant] United States / NINDS NIH HHS / NS / K23 NS075141; United States / NINDS NIH HHS / NS / K23NS075141; United States / NINDS NIH HHS / NS / NS 32352; United States / NINDS NIH HHS / NS / NS 44233; United States / NINDS NIH HHS / NS / P01 NS044233; United States / NINDS NIH HHS / NS / P50 NS032352; United States / NICHD NIH HHS / HD / T32 HD007447; United States / NICHD NIH HHS / HD / T32 HD07447; United States / NINDS NIH HHS / NS / U54 NS065736; United States / NINDS NIH HHS / NS / U54 NS065736; United States / NCATS NIH HHS / TR / UL1 TR000135; United States / NCATS NIH HHS / TR / UL1 TR000135
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Other-IDs] NLM/ NIHMS597796 [Available on 08/01/15]; NLM/ PMC4139446 [Available on 08/01/15]
  • [Keywords] NOTNLM ; MSA / autonomic failure / mortality / survival
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15. |||||..... 52%  Bloch F, Pichon B, Bonnet AM, Pichon J, Vidailhet M, Roze E, Perrigot M: Urodynamic analysis in multiple system atrophy: characterisation of detrusor-sphincter dyssynergia. J Neurol; 2010 Dec;257(12):1986-91
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  • [Title] Urodynamic analysis in multiple system atrophy: characterisation of detrusor-sphincter dyssynergia.
  • In multiple system atrophy (MSA), parkinsonism and a cerebellar syndrome are associated with autonomic dysfunction.
  • Both bladder neck dysfunction and external sphincter denervation have been implicated in detrusor-sphincter dyssynergia.
  • Pressure assessment at several levels of the urethra is needed to unravel the mechanisms of bladder-urethra dysfunction.
  • Here, we evaluated the use of multiple sensor pressure transducers to assess bladder-sphincter function in 52 patients with MSA in comparison to patients with Parkinson's disease (PD) who were matched for age and severity in the "off" condition.
  • Urinary dysfunction appeared significantly earlier in MSA (<2 years) than in PD (>5 years).
  • Detrusor-urethral dyssynergia in MSA patients was always better characterized by multiple sensor pressure transducer measurement of bladder and urethral pressure than by a single global measurement.
  • This new approach may prove useful for differential diagnosis of parkinsonian syndromes, and especially MSA.
  • [MeSH-major] Multiple System Atrophy / physiopathology. Urinary Bladder / physiopathology. Urinary Bladder, Neurogenic / diagnosis. Urinary Bladder, Neurogenic / physiopathology. Urination / physiology


16. |||||..... 52%  Köllensperger M, Geser F, Ndayisaba JP, Boesch S, Seppi K, Ostergaard K, Dupont E, Cardozo A, Tolosa E, Abele M, Klockgether T, Yekhlef F, Tison F, Daniels C, Deuschl G, Coelho M, Sampaio C, Bozi M, Quinn N, Schrag A, Mathias CJ, Fowler C, Nilsson CF, Widner H, Schimke N, Oertel W, Del Sorbo F, Albanese A, Pellecchia MT, Barone P, Djaldetti R, Colosimo C, Meco G, Gonzalez-Mandly A, Berciano J, Gurevich T, Giladi N, Galitzky M, Rascol O, Kamm C, Gasser T, Siebert U, Poewe W, Wenning GK, EMSA-SG: Presentation, diagnosis, and management of multiple system atrophy in Europe: final analysis of the European multiple system atrophy registry. Mov Disord; 2010 Nov 15;25(15):2604-12
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Presentation, diagnosis, and management of multiple system atrophy in Europe: final analysis of the European multiple system atrophy registry.
  • Multiple system atrophy (MSA) is a Parkinson's Disease (PD)-like α-synucleinopathy clinically characterized by dysautonomia, parkinsonism, cerebellar ataxia, and pyramidal signs in any combination.
  • In 19 European MSA Study Group centres all consecutive patients with a clinical diagnosis of MSA were recruited from 2001 to 2005.
  • Symptomatic dysautonomia was present in almost all patients, and urinary dysfunction (83%) more common than symptomatic orthostatic hypotension (75%).
  • Less than a third of patients with documented orthostatic hypotension or neurogenic bladder disturbance were receiving treatment.
  • [MeSH-major] Multiple System Atrophy / diagnosis. Multiple System Atrophy / therapy. Registries
  • [MeSH-minor] Age of Onset. Antiparkinson Agents / therapeutic use. Cerebellar Ataxia / diagnosis. Cerebellar Ataxia / physiopathology. Europe. Female. Humans. Hypotension, Orthostatic / diagnosis. Hypotension, Orthostatic / physiopathology. Levodopa / therapeutic use. Male. Middle Aged. Parkinsonian Disorders / diagnosis. Parkinsonian Disorders / drug therapy. Parkinsonian Disorders / physiopathology. Shy-Drager Syndrome / diagnosis. Shy-Drager Syndrome / physiopathology

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  • [Copyright] © 2010 Movement Disorder Society.
  • (PMID = 20922810.001).
  • [ISSN] 1531-8257
  • [Journal-full-title] Movement disorders : official journal of the Movement Disorder Society
  • [ISO-abbreviation] Mov. Disord.
  • [Language] eng
  • [Publication-type] Journal Article; Multicenter Study; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Antiparkinson Agents; 46627O600J / Levodopa
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17. |||||..... 52%  Harik LR, O'Toole KM: Nonneoplastic lesions of the prostate and bladder. Arch Pathol Lab Med; 2012 Jul;136(7):721-34
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Nonneoplastic lesions of the prostate and bladder.
  • CONTEXT: Specimens from the prostate and bladder are commonly encountered by the general surgical pathologist.
  • Emphasis is usually placed on neoplasms of the bladder and prostate, particularly if malignant, owing to their therapeutic consequences.
  • A good command of benign lesions occurring in the bladder and prostate, and knowledge of their preneoplastic potential will help pathologists confidently diagnose malignancy versus its benign mimickers and guide the urologists in choosing the appropriate therapy and follow-up for the patient.
  • CONCLUSIONS: A wide variety of benign entities are present in the prostate and bladder.
  • Benign lesions in the prostate can be age related, such as prostatic atrophy and benign prostatic hyperplasia; transition zone associated, such as basal cell hyperplasia, adenosis, and sclerosing adenosis; or prostatic urethra associated.
  • Benign lesions of the bladder encompass a wide variety of reactive changes that can occur in the urothelium, as well as hyperplastic lesions or reactive proliferations that could be misdiagnosed as malignant.
  • The bladder responds to chronic irritation through several reactive/metaplastic lesions such as cystitis cystica/glandularis, keratinizing squamous metaplasia, or nephrogenic metaplasia.
  • [MeSH-major] Prostate / pathology. Prostatitis / pathology. Urinary Bladder / pathology. Urinary Bladder Diseases / pathology
  • [MeSH-minor] Atrophy. Humans. Male

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  • (PMID = 22742546.001).
  • [ISSN] 1543-2165
  • [Journal-full-title] Archives of pathology & laboratory medicine
  • [ISO-abbreviation] Arch. Pathol. Lab. Med.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] United States
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18. |||||||||. 859%  Cooper IF, Siadaty MS: 'Pathologic Functions' associated with 'Anastomosis Of Bladder With Isolated Segment Of Ileum': Top Publications. BioMedLib Review; PathologicFunction;AnastomosisOfBladder:706882262. ISSN: 2331-5717. 2014/1/2
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  • [Title] 'Pathologic Functions' associated with 'Anastomosis Of Bladder With Isolated Segment Of Ileum': Top Publications.
  • [Transliterated title]
  • Background: There are articles published each month which present 'Pathologic Function' for 'anastomosis of bladder with isolated segment of ileum'.
  • Finding such articles is important for researchers, clinicians, and patients.
  • However these articles are spread across thousands of journals, and there are many types of 'Pathologic Function'.
  • This makes searching and locating the relevant publications a challenge.
  • We have used BioMedLib's semantic search technology to address the issue, and gathered all the pertinent publications in this review article.
  • Methods: We categorized the publications we found into two groups.
  • We used the strength of textual-association to separate the groups.
  • In group one there are publications with the strongest evidence of association. We focused finding the most relevant publications pertinent to our goal, rather than combining them into a conclusion section. Such textual synthesis will be the focus of our next project.
  • Results: Group one includes 23 publications, and group two 350 publications.
  • Here are the top 10.
  • Armah HB et al: Tubular adenoma with high-grade dysplasia in the ileal segment 34 years after augmentation ileocystoplasty: report of a first case.
  • Kishino T et al: [Assessment of surgical invasiveness of augmentation ileocystoplasty applying the systemic inflammatory response syndrome score in patients with spina bifida].
  • Desgrandchamps F et al: Spontaneous rupture of orthotopic detubularized ileal bladder replacement: report of 5 cases.
  • Mitsui T et al: Preoperative renal scar as a risk factor of postoperative metabolic acidosis following ileocystoplasty in patients with neurogenic bladder.
  • Kawamura S et al: [A case of replacement ileocystoplasty for contracted bladder].
  • Beiko DT et al: Cerebrospinal fluid pseudocyst: a postoperative complication of augmentation ileocystoplasty in myelodysplastic children.
  • Koizumi S et al: [Adenocarcinoma occurring 37 years after augmentation ileocystoplasty for tuberculous bladder atrophy: report of a case].
  • Razmaria AA et al: Does robot-assisted laparoscopic ileocystoplasty (RALI) reduce peritoneal adhesions compared with open surgery?.
  • Robles MW et al: Adenocarcinoma and dysplasia in an ileal neobladder after ileocystoplasty for interstitial cystitis.
  • HABA T et al: [Ileocystoplasty for tuberculous contracted bladder].

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
  • (UID = 706882262.001).
  • [ISSN] 2331-5717
  • [Journal-full-title] BioMedLib Review
  • [Language] eng
  • [Publication-type] Review
  • [Publication-country] UNITED STATES
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19. |||||..... 52%  Calandra-Buonaura G, Guaraldi P, Sambati L, Lopane G, Cecere A, Barletta G, Provini F, Contin M, Martinelli P, Cortelli P: Multiple system atrophy with prolonged survival: is late onset of dysautonomia the clue? Neurol Sci; 2013 Oct;34(10):1875-8
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Multiple system atrophy with prolonged survival: is late onset of dysautonomia the clue?
  • Multiple system atrophy (MSA) is a neurodegenerative disease characterised by cardiovascular autonomic failure and/or urinary dysfunctions, associated with parkinsonism, cerebellar and/or corticospinal signs, usually leading to death after an average of 7 years.
  • The time of involvement of the urogenital system was more variable (from 0 to 14 years after disease onset) and manifested with symptoms of storage disorders (urinary urgency, frequency and incontinence) and erectile dysfunction in men.
  • Conversely complains suggestive of urinary voiding dysfunction (incomplete bladder emptying and urinary retention) were not recorded and patients required catheterization only late in the disease course.
  • In conclusion, our study showed that late onset of both cardiovascular autonomic failure and urinary voiding disorders may be positive prognostic factors in MSA irrespective of the MSA subtype.
  • [MeSH-major] Multiple System Atrophy / complications. Multiple System Atrophy / mortality. Primary Dysautonomias / complications


20. |||||..... 52%  Kokubun S, Ozawa H, Aizawa T, Ly NM, Tanaka Y: Spine-shortening osteotomy for patients with tethered cord syndrome caused by lipomyelomeningocele. J Neurosurg Spine; 2011 Jul;15(1):21-7
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • RESULTS: Preoperatively, all patients displayed severe neurological deficits such as motor disturbance, muscle atrophy, and bladder dysfunction.

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  • [CommentIn] J Neurosurg Spine. 2011 Jul;15(1):19-20; author reply p 20 [21495811.001]
  • (PMID = 21495816.001).
  • [ISSN] 1547-5646
  • [Journal-full-title] Journal of neurosurgery. Spine
  • [ISO-abbreviation] J Neurosurg Spine
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] Lipomyelomeningocele
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21. |||||..... 51%  Hameed O, Humphrey PA: Pseudoneoplastic mimics of prostate and bladder carcinomas. Arch Pathol Lab Med; 2010 Mar;134(3):427-43
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Pseudoneoplastic mimics of prostate and bladder carcinomas.
  • CONTEXT: The differential diagnoses of prostatic carcinoma and bladder epithelial neoplasms include several histologic mimics that should be known to avoid misdiagnosis.
  • OBJECTIVE: To discuss pseudoneoplastic lesions of the prostate and bladder that could potentially be confused with prostatic carcinoma and bladder epithelial neoplasms, respectively, with specific focus on their distinguishing histopathologic features.
  • CONCLUSIONS: Pseudoneoplastic lesions in the prostate include those of prostatic epithelial origin, the most common being atrophy, adenosis (atypical adenomatous hyperplasia), basal cell hyperplasia, and crowded benign glands, as well as those of nonprostatic origin, such as seminal vesicle epithelium.
  • Such lesions often mimic lower-grade prostatic adenocarcinoma, whereas others, such as clear cell cribriform hyperplasia and granulomatous prostatitis, for example, are in the differential diagnosis of Gleason adenocarcinoma, Gleason grade 4 or 5.
  • Pseudoneoplastic lesions of the urinary bladder include lesions that could potentially be confused with urothelial carcinoma in situ, such as reactive urothelial atypia, and others, such as polypoid/papillary cystitis, where papillary urothelial neoplasms are the main differential diagnostic concern.
  • Diagnostic awareness of the salient histomorphologic and relevant immunohistochemical features of these prostatic and urinary bladder pseudoneoplasms is critical to avoid rendering false-positive diagnoses of malignancy.
  • [MeSH-major] Granuloma, Plasma Cell / diagnosis. Prostatic Diseases / diagnosis. Urinary Bladder Diseases / diagnosis
  • [MeSH-minor] Diagnosis, Differential. Humans. Male. Prostatic Neoplasms / classification. Prostatic Neoplasms / diagnosis. Urinary Bladder Neoplasms / classification. Urinary Bladder Neoplasms / diagnosis

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  • (PMID = 20196670.001).
  • [ISSN] 1543-2165
  • [Journal-full-title] Archives of pathology & laboratory medicine
  • [ISO-abbreviation] Arch. Pathol. Lab. Med.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] United States
  • [Number-of-references] 83
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22. |||||..... 51%  Deguchi K, Ikeda K, Goto R, Tsukaguchi M, Urai Y, Kurokohchi K, Touge T, Mori N, Masaki T: The close relationship between life-threatening breathing disorders and urine storage dysfunction in multiple system atrophy. J Neurol; 2010 Aug;257(8):1287-92
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] The close relationship between life-threatening breathing disorders and urine storage dysfunction in multiple system atrophy.
  • Survival of multiple system atrophy (MSA) depends on whether a variety of sleep-related breathing problems as well as autonomic failure (AF) occur.
  • In 15 patients with MSA, we performed autonomic function tests composed of postural challenges and administered a questionnaire on bladder condition, as well as polysomnography and laryngoscopy during wakefulness and under anesthesia.
  • Polysomnographic variables such as the apnea-hypopnea index (AHI) and oxygen saturation (SpO(2)) and the findings of laryngoscopy were compared with the degree of cardiac and urinary autonomic dysfunction.
  • [MeSH-major] Multiple System Atrophy / complications. Respiratory Insufficiency / diagnosis. Shy-Drager Syndrome / diagnosis. Shy-Drager Syndrome / physiopathology. Urinary Bladder, Neurogenic / physiopathology


23. |||||..... 51%  Dixit P, Shek KL, Dietz HP: How common is pelvic floor muscle atrophy after vaginal childbirth? Ultrasound Obstet Gynecol; 2014 Jan;43(1):83-8
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] How common is pelvic floor muscle atrophy after vaginal childbirth?
  • OBJECTIVE: To determine if there is evidence of levator ani atrophy in primiparous women.
  • Peripartum changes in bladder neck elevation and reduction of anteroposterior hiatal diameter on pelvic floor muscle contraction (PFMC) and changes in muscle thickness were analyzed.
  • There was a significant reduction in bladder neck elevation (P = 0.001) and change in anteroposterior hiatal diameter (P = 0.03) on PFMC when comparing antenatal and postnatal results, the latter being significantly associated with delivery mode (P = 0.013).
  • CONCLUSIONS: There is a reduction in sonographic measures of pelvic floor function after childbirth, but muscle atrophy is unlikely to be a significant factor.
  • [MeSH-major] Delivery, Obstetric / adverse effects. Muscular Atrophy / epidemiology. Pelvic Floor / physiopathology. Pelvic Floor Disorders / epidemiology. Pregnancy Complications / epidemiology

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  • [Copyright] Copyright © 2013 ISUOG. Published by John Wiley & Sons Ltd.
  • (PMID = 23784670.001).
  • [ISSN] 1469-0705
  • [Journal-full-title] Ultrasound in obstetrics & gynecology : the official journal of the International Society of Ultrasound in Obstetrics and Gynecology
  • [ISO-abbreviation] Ultrasound Obstet Gynecol
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Keywords] NOTNLM ; childbirth / levator ani / muscle atrophy / pelvic floor muscle / ultrasound
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24. |||||..... 51%  Hironishi M, Ishimoto S, Sawanishi T, Miwa H, Kawachi I, Kondo T: [Neuromyelitis optica following thymectomy with severe spinal cord atrophy after frequent relapses for 30 years]. Brain Nerve; 2012 Aug;64(8):951-5
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [Neuromyelitis optica following thymectomy with severe spinal cord atrophy after frequent relapses for 30 years].
  • The initial symptoms of NMO, including headache, high fever, retrobulbar optic neuritis, and neurogenic bladder, appeared at the age of 30.
  • Spinal MRI after frequent NMO attacks for 30 years revealed an extended spinal cord atrophy involving the lower cervical region and the entire thoracic region.
  • [MeSH-minor] Atrophy / pathology. Female. Humans. Magnetic Resonance Imaging. Middle Aged. Myasthenia Gravis / pathology. Prognosis. Recurrence

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  • (PMID = 22868887.001).
  • [ISSN] 1881-6096
  • [Journal-full-title] Brain and nerve = Shinkei kenkyū no shinpo
  • [ISO-abbreviation] Brain Nerve
  • [Language] jpn
  • [Publication-type] Case Reports; English Abstract
  • [Publication-country] Japan
  • [Chemical-registry-number] 0 / Aquaporin 4
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25. |||||..... 51%  Kohli J, Brandt N: Multiple-system atrophy and medications: how to minimize the risk of falling. Consult Pharm; 2011 Mar;26(3):190-6
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Multiple-system atrophy and medications: how to minimize the risk of falling.
  • Following a chart review and interview with the resident, the consultant pharmacist found that her past medical history consists of coronary artery disease, atrial fibrillation, congestive heart failure, hypertension, dyslipidemia, osteoporosis, gastroesophageal reflux disease, glaucoma, mild dementia, overactive bladder, and Parkinson's disease (PD).
  • During the review, the consultant pharmacist found a recent neurology note that concluded the resident may have multiple-system atrophy (MSA) and her therapy for PD may not be beneficial.
  • [MeSH-major] Accidental Falls / prevention & control. Autonomic Nervous System Diseases / etiology. Hypotension, Orthostatic / etiology. Multiple System Atrophy / complications


26. |||||..... 51%  Portman DJ, Gass ML, Vulvovaginal Atrophy Terminology Consensus Conference Panel: Genitourinary Syndrome of Menopause: New Terminology for Vulvovaginal Atrophy from the International Society for the Study of Women's Sexual Health and The North American Menopause Society. J Sex Med; 2014 Dec;11(12):2865-72
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Genitourinary Syndrome of Menopause: New Terminology for Vulvovaginal Atrophy from the International Society for the Study of Women's Sexual Health and The North American Menopause Society.
  • INTRODUCTION: The terminology for the genitourinary tract symptoms related to menopause was vulvovaginal atrophy, which does not accurately describe the symptoms nor is a term that resonates well with patients.
  • RESULTS: Members of the consensus conference agreed that the term genitourinary syndrome of menopause (GSM) is a medically more accurate, all-encompassing, and publicly acceptable term than vulvovaginal atrophy.
  • GSM is defined as a collection of symptoms and signs associated with a decrease in estrogen and other sex steroids involving changes to the labia majora/minora, clitoris, vestibule/introitus, vagina, urethra, and bladder.
  • The syndrome may include but is not limited to genital symptoms of dryness, burning, and irritation; sexual symptoms of lack of lubrication, discomfort or pain, and impaired function; and urinary symptoms of urgency, dysuria, and recurrent urinary tract infections.
  • Women may present with some or all of the signs and symptoms, which must be bothersome and should not be better accounted for by another diagnosis.
  • DJ Portman, MLS Gass, on behalf of the Vulvovaginal Atrophy Terminology Consensus Conference Panel.
  • Genitourinary syndrome of menopause: New terminology for vulvovaginal atrophy from the International Society for the Study of Women's Sexual Health and The North American Menopause Society.

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  • [Copyright] © 2014 International Society for Sexual Medicine, The North American Menopause Society, International Menopause Society, and European Menopause and Andropause Society.
  • (PMID = 25155380.001).
  • [ISSN] 1743-6109
  • [Journal-full-title] The journal of sexual medicine
  • [ISO-abbreviation] J Sex Med
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Keywords] NOTNLM ; Atrophic Vaginitis / Genitourinary Syndrome of Menopause / Menopause / Urinary Urgency / Vulvovaginal Atrophy / Women's Sexual Health
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27. |||||..... 51%  Sakakibara R, Tateno F, Kishi M, Tsuyuzaki Y, Uchiyama T, Yamamoto T: Pathophysiology of bladder dysfunction in Parkinson's disease. Neurobiol Dis; 2012 Jun;46(3):565-71
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Pathophysiology of bladder dysfunction in Parkinson's disease.
  • Bladder dysfunction (urinary urgency/frequency) is a common non-motor disorder in Parkinson's disease (PD).
  • In contrast to motor disorders, bladder dysfunction is sometimes non-responsive to levodopa treatment.
  • The brain pathology causing the bladder dysfunction (appearance of overactivity) involves an altered dopamine basal ganglia-frontal circuit, which normally suppresses the micturition reflex.
  • The pathophysiology of the bladder dysfunction in PD differs from that in multiple system atrophy; therefore, it might aid in differential diagnosis.
  • Anticholinergic agents are used to treat bladder dysfunction in PD, although these drugs should be used with caution particularly in elderly patients who have cognitive decline.
  • [MeSH-major] Parkinson Disease / complications. Urinary Bladder Diseases / etiology
  • [MeSH-minor] Basal Ganglia / physiopathology. Cholinergic Antagonists / therapeutic use. Dopamine / physiology. Dopamine Agents / therapeutic use. Electromyography. Humans. Male. Transurethral Resection of Prostate / adverse effects. Urinary Bladder / physiopathology. Urination / physiology. Urination Disorders / etiology. Urination Disorders / physiopathology. Urodynamics

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  • [Copyright] Copyright © 2011 Elsevier Inc. All rights reserved.
  • (PMID = 22015601.001).
  • [ISSN] 1095-953X
  • [Journal-full-title] Neurobiology of disease
  • [ISO-abbreviation] Neurobiol. Dis.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Cholinergic Antagonists; 0 / Dopamine Agents; VTD58H1Z2X / Dopamine
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28. |||||..... 51%  Portman DJ, Gass ML, Vulvovaginal Atrophy Terminology Consensus Conference Panel: Genitourinary syndrome of menopause: New terminology for vulvovaginal atrophy from the International Society for the Study of Women's Sexual Health and The North American Menopause Society. Maturitas; 2014 Nov;79(3):349-54
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Genitourinary syndrome of menopause: New terminology for vulvovaginal atrophy from the International Society for the Study of Women's Sexual Health and The North American Menopause Society.
  • RESULTS AND CONCLUSION: Members of the consensus conference agreed that the term genitourinary syndrome of menopause (GSM) is a medically more accurate, all-encompassing, and publicly acceptable term than vulvovaginal atrophy.
  • GSM is defined as a collection of symptoms and signs associated with a decrease in estrogen and other sex steroids involving changes to the labia majora/minora, clitoris, vestibule/introitus, vagina, urethra and bladder.
  • The syndrome may include but is not limited to genital symptoms of dryness, burning, and irritation; sexual symptoms of lack of lubrication, discomfort or pain, and impaired function; and urinary symptoms of urgency, dysuria and recurrent urinary tract infections.
  • Women may present with some or all of the signs and symptoms, which must be bothersome and should not be better accounted for by another diagnosis.

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  • [Copyright] Copyright © 2014 D.J. Portman. Published by Elsevier Ireland Ltd.. All rights reserved.
  • (PMID = 25179577.001).
  • [ISSN] 1873-4111
  • [Journal-full-title] Maturitas
  • [ISO-abbreviation] Maturitas
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Ireland
  • [Keywords] NOTNLM ; Atrophic vaginitis / Genitourinary syndrome of menopause / Menopause / Urinary urgency / Vulvovaginal atrophy / Women's sexual health
  •  go-up   go-down


29. |||||..... 51%  Sakakibara R, Tateno F, Nagao T, Yamamoto T, Uchiyama T, Yamanishi T, Yano M, Kishi M, Tsuyusaki Y, Aiba Y: Bladder function of patients with Parkinson's disease. Int J Urol; 2014 Jul;21(7):638-46
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Bladder function of patients with Parkinson's disease.
  • Bladder function of patients with Parkinson's disease alters significantly: the majority of patients have overactive bladder (urinary urgency/frequency) with little or no post-void residuals.
  • This seems to be the result of an altered brain-bladder relationship, as in Parkinson's disease, the frontal-basal ganglia D1 dopaminergic circuit that normally suppresses the micturition reflex is altered.
  • The pathophysiology of the bladder dysfunction in Parkinson's disease differs from that in multiple system atrophy; therefore, it might also aid in differential diagnosis.
  • The effects of levodopa, the major drug to treat motor dysfunction, on the bladder in Parkinson's disease vary significantly; therefore, add-on therapy is often required.
  • Newer modalities include deep brain stimulation that improves the bladder in Parkinson's disease; and botulinum toxin is promising, particularly in difficult cases.

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  • [Copyright] © 2014 The Japanese Urological Association.
  • (PMID = 24571321.001).
  • [ISSN] 1442-2042
  • [Journal-full-title] International journal of urology : official journal of the Japanese Urological Association
  • [ISO-abbreviation] Int. J. Urol.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Australia
  • [Keywords] NOTNLM ; Parkinson's disease / bladder function / brain / detrusor overactivity
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30. |||||..... 51%  Tsunoyama K, Sakakibara R, Yamaguchi C, Uchiyama T, Yamamoto T, Yamanishi T, Takahashi O, Sugiyama M, Kishi M, Ogawa E: Pathogenesis of reduced or increased bladder sensation. Neurourol Urodyn; 2011 Mar;30(3):339-43
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  • [Title] Pathogenesis of reduced or increased bladder sensation.
  • OBJECTIVE: Pathogenesis of reduced or increased bladder sensation is not well known.
  • Hence, we systematically investigated the frequency of reduced or increased bladder sensation in neurologic/mental diseases.
  • Data registries included a diagnosis, a lower urinary tract symptom questionnaire, a urodynamic study, and neurological examinations.
  • Reduced bladder sensation is defined as bladder volume at the first sensation >300 ml.
  • Increased bladder sensation is defined as bladder volume at the first sensation <100 ml.
  • RESULTS: Neuropathies are the most common cause of reduced bladder sensation (33.3-43.8% in diabetic neuropathy, etc.).
  • Less common is brain diseases (9.6% in multiple system atrophy, etc.).
  • In contrast, myelopathies are the most common cause of increased bladder sensation without DO (25.0-40.0% in spinal forms of systemic lupus erythematosus, Sjogren's syndrome, etc.).
  • Less common is brain/mental diseases (20.0% in psychogenic bladder dysfunction, 8.1% in Parkinson's disease, etc.).
  • CONCLUSION: The present study revealed that neuropathies are the most common cause of reduced bladder sensation in neurologic/mental diseases.
  • Increased bladder sensation without DO occurs mainly in peripheral and central sensory pathway lesions, as well as in basal ganglia lesions and psychogenic bladder dysfunction.
  • Reduced and increased bladder sensation should be a major treatment target for maximizing patients' quality of life.
  • [MeSH-major] Hypesthesia / etiology. Sensation. Urinary Bladder / innervation. Urinary Bladder Diseases / etiology

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  • [Copyright] Copyright © 2011 Wiley-Liss, Inc.
  • (PMID = 21305587.001).
  • [ISSN] 1520-6777
  • [Journal-full-title] Neurourology and urodynamics
  • [ISO-abbreviation] Neurourol. Urodyn.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
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31. |||||..... 51%  Portman DJ, Gass ML, Vulvovaginal Atrophy Terminology Consensus Conference Panel: Genitourinary syndrome of menopause: new terminology for vulvovaginal atrophy from the International Society for the Study of Women's Sexual Health and The North American Menopause Society. Climacteric; 2014 Oct;17(5):557-63
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  • [Title] Genitourinary syndrome of menopause: new terminology for vulvovaginal atrophy from the International Society for the Study of Women's Sexual Health and The North American Menopause Society.
  • RESULTS AND CONCLUSION: Members of the consensus conference agreed that the term genitourinary syndrome of menopause (GSM) is a medically more accurate, all-encompassing, and publicly acceptable term than vulvovaginal atrophy.
  • GSM is defined as a collection of symptoms and signs associated with a decrease in estrogen and other sex steroids involving changes to the labia majora/minora, clitoris, vestibule/introitus, vagina, urethra and bladder.
  • The syndrome may include but is not limited to genital symptoms of dryness, burning, and irritation; sexual symptoms of lack of lubrication, discomfort or pain, and impaired function; and urinary symptoms of urgency, dysuria and recurrent urinary tract infections.
  • Women may present with some or all of the signs and symptoms, which must be bothersome and should not be better accounted for by another diagnosis.

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  • (PMID = 25153131.001).
  • [ISSN] 1473-0804
  • [Journal-full-title] Climacteric : the journal of the International Menopause Society
  • [ISO-abbreviation] Climacteric
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Keywords] NOTNLM ; ATROPHIC VAGINITIS / GENITOURINARY SYNDROME OF MENOPAUSE / MENOPAUSE / URINARY URGENCY / VULVOVAGINAL ATROPHY / WOMEN’S SEXUAL HEALTH
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32. |||||..... 51%  VanderBrink BA, Stock JA, Hanna MK: Aesthetic aspects of reconstructive clitoroplasty in females with bladder exstrophy-epispadias complex. J Plast Reconstr Aesthet Surg; 2010 Dec;63(12):2141-5
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  • [Title] Aesthetic aspects of reconstructive clitoroplasty in females with bladder exstrophy-epispadias complex.
  • BACKGROUND: Genital anomalies in females born with classic bladder exstrophy-epispadias complex (EEC) include a bifid clitoris and anterior displacement of vagina.
  • METHODS: The study group had 26 female patients; three patients with isolated epispadias and 23 with bladder exstrophy.
  • Three patients had partial clitoral atrophy and in one patient the gap between clitoral bodies was widely separated and incapable of being joined.
  • [MeSH-major] Bladder Exstrophy / surgery. Epispadias / surgery. Genitalia, Female / surgery. Gynecologic Surgical Procedures / methods. Reconstructive Surgical Procedures / methods
  • [MeSH-minor] Atrophy. Child. Child, Preschool. Esthetics. Female. Humans. Infant. Infant, Newborn. Patient Satisfaction. Reoperation. Treatment Outcome

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  • [Copyright] Copyright © 2010 British Association of Plastic, Reconstructive and Aesthetic Surgeons. Published by Elsevier Ltd. All rights reserved.
  • (PMID = 20303329.001).
  • [ISSN] 1878-0539
  • [Journal-full-title] Journal of plastic, reconstructive & aesthetic surgery : JPRAS
  • [ISO-abbreviation] J Plast Reconstr Aesthet Surg
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Netherlands
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33. |||||..... 51%  Quinta A, Carvalho AP, Oliveira M, Oliveira C, Ribeiro Santos A: [Bladder adenocarcinoma 41 years after augmentation enterocystoplasty for tuberculosis]. Arch Esp Urol; 2013 Mar;66(2):231-3
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  • [Title] [Bladder adenocarcinoma 41 years after augmentation enterocystoplasty for tuberculosis].
  • OBJECTIVE: To report a case of adeno-carcinoma arising in an augmented bladder 41 years after the procedure.
  • METHODS: After troublesome evaluation and fistula closure with urinary diversion by percutaneous nephrostomy, the patient underwent palliative chemotherapy and radiotherapy.
  • [MeSH-major] Adenocarcinoma / pathology. Tuberculosis, Urogenital / surgery. Urinary Bladder / surgery. Urinary Bladder Neoplasms / pathology
  • [MeSH-minor] Aged. Atrophy. Bone and Bones / radionuclide imaging. Fatal Outcome. Humans. Kidney / pathology. Kidney / ultrasonography. Kidney Function Tests. Magnetic Resonance Imaging. Male. Palliative Care. Tomography, X-Ray Computed. Urologic Surgical Procedures

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  • (PMID = 23589601.001).
  • [ISSN] 1576-8260
  • [Journal-full-title] Archivos españoles de urología
  • [ISO-abbreviation] Arch. Esp. Urol.
  • [Language] eng; spa
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Spain
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34. |||||..... 50%  Portman DJ, Gass ML, Vulvovaginal Atrophy Terminology Consensus Conference Panel: Genitourinary syndrome of menopause: new terminology for vulvovaginal atrophy from the International Society for the Study of Women's Sexual Health and the North American Menopause Society. Menopause; 2014 Oct;21(10):1063-8
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Genitourinary syndrome of menopause: new terminology for vulvovaginal atrophy from the International Society for the Study of Women's Sexual Health and the North American Menopause Society.
  • RESULTS AND CONCLUSIONS: Members of the consensus conference agreed that the term genitourinary syndrome of menopause (GSM) is a medically more accurate, all-encompassing, and publicly acceptable term than vulvovaginal atrophy.
  • GSM is defined as a collection of symptoms and signs associated with a decrease in estrogen and other sex steroids involving changes to the labia majora/minora, clitoris, vestibule/introitus, vagina, urethra and bladder.
  • The syndrome may include but is not limited to genital symptoms of dryness, burning, and irritation; sexual symptoms of lack of lubrication, discomfort or pain, and impaired function; and urinary symptoms of urgency, dysuria and recurrent urinary tract infections.
  • Women may present with some or all of the signs and symptoms, which must be bothersome and should not be better accounted for by another diagnosis.

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  • (PMID = 25160739.001).
  • [ISSN] 1530-0374
  • [Journal-full-title] Menopause (New York, N.Y.)
  • [ISO-abbreviation] Menopause
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Investigator] Portman D; Gass M; Kingsberg S; Archer D; Bachmann G; Burrows L; Freedman M; Goldstein A; Goldstein I; Heller D; Iglesia C; Kagan R; Spadt SK; Krychman M; Nachtigall L; Nappi R; Pinkerton J; Shifren J; Simon J; Stuenkel C
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35. |||||..... 50%  Lin YC, Chiu KH, Shiea J, Huang HW, Mok HK: Seasonal changes in atrophy-associated proteins of the sonic muscle in the big-snout croaker, Johnius macrorhynus (Pisces, Sciaenidae), identified by using a proteomic approach. Fish Physiol Biochem; 2011 Dec;37(4):977-91
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  • [Title] Seasonal changes in atrophy-associated proteins of the sonic muscle in the big-snout croaker, Johnius macrorhynus (Pisces, Sciaenidae), identified by using a proteomic approach.
  • In most sciaenids, males possess sonic muscles and produce sound through the contraction of these muscles and amplification of the swim bladder.
  • The protein profiles of the sonic muscle, red muscle, and white muscle in the Johnius macrorhynus were shown by two-dimensional electrophoresis (2-DE) and were compared to reveal differential protein expressions.
  • Seasonal hypertrophy and atrophy of the sonic muscles related to the reproductive cycle were verified in male big-snout croaker.
  • [MeSH-minor] Animals. Atrophy. Electrophoresis, Gel, Two-Dimensional. Hypertrophy. Male. Muscle Contraction. Proteomics. Seasons

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  • (PMID = 21553060.001).
  • [ISSN] 1573-5168
  • [Journal-full-title] Fish physiology and biochemistry
  • [ISO-abbreviation] Fish Physiol. Biochem.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / Muscle Proteins
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36. |||||..... 50%  Gupta A, Garg RK, Singh MK, Verma R, Malhotra HS, Sankhwar SN, Jain A, Singh R, Parihar A: Bladder dysfunction and urodynamic study in tuberculous meningitis. J Neurol Sci; 2013 Apr 15;327(1-2):46-54
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  • [Title] Bladder dysfunction and urodynamic study in tuberculous meningitis.
  • Bladder dysfunction in tuberculous meningitis is often considered secondary to tuberculous radiculomyelopathy.
  • We, in this study, evaluated the incidence and pattern of bladder dysfunction in tuberculous meningitis.
  • RESULTS: Out of 51 patients, urinary symptoms were present in one-third of the patients.
  • Other urodynamic abnormalities were neurogenic detrusor overactivity in 10, detrusor sphincter dyssynergia in 6, normal detrusor activity in 19, reduced bladder sensation in 12, raised cystometric capacity in 9, and larger volumes of post-void residual urine in 12 patients.
  • MRI showed spinal meningeal enhancement in 37, lumbosacral arachnoiditis in 25, myelitis in 12 patients, CSF loculations in 6, and cord atrophy in 5 patients.
  • Spinal arachnoiditis and urinary symptoms showed significant association with urodynamic abnormalities.
  • CONCLUSION: Bladder dysfunctions, in tuberculous meningitis, are frequently encountered.
  • [MeSH-major] Tuberculosis, Meningeal / diagnosis. Tuberculosis, Meningeal / epidemiology. Urinary Bladder / physiopathology. Urinary Bladder Diseases / diagnosis. Urinary Bladder Diseases / epidemiology

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  • [Copyright] Copyright © 2013 Elsevier B.V. All rights reserved.
  • (PMID = 23472924.001).
  • [ISSN] 1878-5883
  • [Journal-full-title] Journal of the neurological sciences
  • [ISO-abbreviation] J. Neurol. Sci.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Netherlands
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37. |||||..... 50%  Ohannessian A, Kaboré FA, Agostini A, Lenne Aurier K, Witjas T, Azulay JP, Karsenty G: [Transcutaneous tibial nerve stimulation in the overactive bladder syndrome in patients with Parkinson's syndromes]. Prog Urol; 2013 Sep;23(11):936-9
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  • [Title] [Transcutaneous tibial nerve stimulation in the overactive bladder syndrome in patients with Parkinson's syndromes].
  • OBJECTIVES: To evaluate the efficacy of chronic transcutaneous tibial nerve stimulation (TNS) on overactive bladder syndrome in female patients with Parkinson's disease (PD) and multiple system atrophy (MSA).
  • PATIENTS AND METHODS: A prospective monocentric study enrolled six female patients with PD or MSA suffering from overactive bladder syndrome for a six-week study period.
  • The secondary outcomes measurements were symptom and quality of life scores, bladder diary and urodynamics.
  • CONCLUSION: Although urodynamics and symptoms scores did not show significant difference, an efficacy of TNS on overactive bladder in PD and MSA is possible.
  • [MeSH-major] Parkinson Disease / complications. Tibial Nerve. Transcutaneous Electric Nerve Stimulation. Urinary Bladder, Overactive / etiology. Urinary Bladder, Overactive / therapy

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  • [Copyright] Copyright © 2013 Elsevier Masson SAS. All rights reserved.
  • (PMID = 24010924.001).
  • [ISSN] 1166-7087
  • [Journal-full-title] Progrès en urologie : journal de l'Association française d'urologie et de la Société française d'urologie
  • [ISO-abbreviation] Prog. Urol.
  • [Language] fre
  • [Publication-type] Comparative Study; English Abstract; Journal Article
  • [Publication-country] France
  • [Keywords] NOTNLM ; Atrophie multisystématisée / Hyperactivité vésicale / Incontinence / Incontinence urinaire / Maladie de Parkinson / Multiple system atrophy / Neurogenic bladder / Neurostimulation tibiale / Overactive bladder / Parkinson disease / Transcutaneous tibial nerve stimulation / Vessie neurologique
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38. |||||..... 50%  Zhang Y, Sheng J, Hou C, Lin H: [Microstructural study on detrusor muscle after bladder functional reconstruction for atonic bladder caused by medullary cone injury in rats]. Zhongguo Xiu Fu Chong Jian Wai Ke Za Zhi; 2013 Jul;27(7):836-42
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  • [Title] [Microstructural study on detrusor muscle after bladder functional reconstruction for atonic bladder caused by medullary cone injury in rats].
  • OBJECTIVE: To study the microstructural change of detrusor muscle and neuromuscular junction (NMJ) after bladder functional reconstruction for atonic bladder caused by medullary cone injury and to discuss the feasibility of bladder functional reconstruction for improving the detrusor muscle degeneration.
  • No treatment was given in normal group; the medullary cone injury was established by sharp transection of spinal cord at L4,5 levels in control group; and the anastomosis of bilateral L5 ventral root (VR)-S2 VR and L5 dorsal root (DR)-S2 DR was performed for bladder functional reconstruction after modeling of medullary cone injury in experimental group.
  • HE staining and Masson trichrome staining indicated that the muscle fibers arranged in disorder with gradually aggravated atrophy and gradually increased connective tissue in control group, while the shape of the detrusor muscle recovered with no increased connective tissue at 4, 5, and 6 months after operation in experimental group; there was significant difference in cross-sectional area of detrusor muscle and percentage of connective tissue between normal group and experimental group, and between normal group and control group at each time point (P < 0.05).
  • CONCLUSION: The reconstruction of bladder function with L5 nerve roots above the paraplegic plane can effectively inhibit the degeneration of detrusor muscle and improve its microstructural changes after medullary cone injury.
  • [MeSH-major] Muscle, Smooth / pathology. Neuromuscular Junction / pathology. Spinal Cord Injuries / complications. Spinal Nerve Roots / surgery. Urinary Bladder, Neurogenic / surgery
  • [MeSH-minor] Animals. Disease Models, Animal. Feasibility Studies. Male. Muscle Fibers, Skeletal / pathology. Muscle Fibers, Skeletal / ultrastructure. Random Allocation. Rats. Rats, Sprague-Dawley. Reconstructive Surgical Procedures / methods. Urinary Bladder / innervation. Urinary Bladder / physiopathology. Urinary Bladder / surgery. Urination

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  • (PMID = 24063173.001).
  • [ISSN] 1002-1892
  • [Journal-full-title] Zhongguo xiu fu chong jian wai ke za zhi = Zhongguo xiufu chongjian waike zazhi = Chinese journal of reparative and reconstructive surgery
  • [ISO-abbreviation] Zhongguo Xiu Fu Chong Jian Wai Ke Za Zhi
  • [Language] chi
  • [Publication-type] English Abstract; Journal Article
  • [Publication-country] China
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39. |||||..... 50%  Bodareva NV, Anichkov NM, Komiakov BK: [Adaptive changes in enteric explants instead of the urinary bladder]. Arkh Patol; 2011 Jan-Feb;73(1):27-9
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  • [Title] [Adaptive changes in enteric explants instead of the urinary bladder].
  • One hundred and twenty-two biopsy specimens of enteric explants (orthotopic urinary reservoirs (OUR)) placed instead of the neoplastic urinary bladder were studied in 94 patients.
  • (1) progressive atrophy and a reduction in enteric villae; brush-border enterocyte replacement by goblet cells secreting syalomucins with the appearance of the lining devoid of the brush border and secretion;.
  • [MeSH-major] Intestine, Small. Urinary Bladder / pathology. Urinary Bladder / surgery. Urinary Bladder Neoplasms / pathology. Urinary Bladder Neoplasms / surgery. Urologic Surgical Procedures

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  • (PMID = 21506337.001).
  • [ISSN] 0004-1955
  • [Journal-full-title] Arkhiv patologii
  • [ISO-abbreviation] Arkh. Patol.
  • [Language] rus
  • [Publication-type] English Abstract; Journal Article
  • [Publication-country] Russia (Federation)
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40. |||||..... 50%  Tanidir Y, Ercan F, Tarcan T: Exogenous testosterone and estrogen affect bladder tissue contractility and histomorphology differently in rat ovariectomy model. J Sex Med; 2011 Jun;8(6):1626-37
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  • [Title] Exogenous testosterone and estrogen affect bladder tissue contractility and histomorphology differently in rat ovariectomy model.
  • INTRODUCTION: Changes in sex hormone levels may play a role in the etiology of lower urinary tract dysfunction of aging women where the possible role of testosterone is overlooked.
  • AIM: To determine the effect of testosterone with/without estrogen replacement on histological and functional deterioration in ovariectomized rat bladder tissue.
  • Nine weeks after ovariectomy, bladder strips were harvested for isometric tension and histopathological studies.
  • MAIN OUTCOME MEASURES: To assess the effect of testosterone/estradiol on ovariectomized rat bladder tissue function and histomorphology.
  • RESULTS: OVX and VA groups showed statistically significant histological changes such as urothelial damage, inflammatory cell infiltration, increase in collagen fibers and muscular atrophy compared with the control group.
  • CONCLUSION: Either estradiol or testosterone replacement alone or in combination prevents significant alterations in bladder tissue histology following ovariectomy whereas both affect the bladder tissue contractility.
  • Thus, combination treatment appears to be the best method to restore both contractility and histomorphology of bladder tissue after ovariectomy.
  • [MeSH-major] Estradiol / pharmacology. Isometric Contraction / drug effects. Ovariectomy. Testosterone / analogs & derivatives. Urinary Bladder / anatomy & histology. Urinary Bladder / drug effects. Urodynamics / drug effects

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  • [Copyright] © 2011 International Society for Sexual Medicine.
  • (PMID = 21366882.001).
  • [ISSN] 1743-6109
  • [Journal-full-title] The journal of sexual medicine
  • [ISO-abbreviation] J Sex Med
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 3XMK78S47O / Testosterone; 4TI98Z838E / Estradiol; H16A5VCT9C / testosterone undecanoate
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41. |||||..... 50%  Swaminath PV, Ragothaman M, Koshy S, Sarangmath N, Adhyam M, Subbakrishna DK, Mathias CJ, Muthane UB: Urogenital symptoms in Parkinson's disease and multiple system atrophy-Parkinsonism: at onset and later. J Assoc Physicians India; 2010 Feb;58:86-90
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  • [Title] Urogenital symptoms in Parkinson's disease and multiple system atrophy-Parkinsonism: at onset and later.
  • An autonomic questionnaire was used to document urinary and genital symptoms.
  • Urinary symptoms occurred twice as frequently in MSA-P than in PD.
  • [MeSH-major] Multiple System Atrophy / complications. Parkinsonian Disorders / complications. Sexual Dysfunction, Physiological / etiology. Urinary Bladder Diseases / etiology. Urination Disorders / etiology
  • [MeSH-minor] Adult. Age of Onset. Aged. Aged, 80 and over. Diagnosis, Differential. Female. Humans. Male. Middle Aged. Prospective Studies. Urodynamics


42. |||||..... 50%  Siracusa G, Sparacino A, Lentini VL: Neurogenic bladder and disc disease: a brief review. Curr Med Res Opin; 2013 Aug;29(8):1025-31
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  • [Title] Neurogenic bladder and disc disease: a brief review.
  • OBJECTIVE: Neurogenic bladder refers to morphofunctional alterations of the bladder-sphincter complex secondary to central or peripheral neurological lesions.
  • This review provides a brief overview of associations between neurogenic bladder and disc disease, analyzing neuroanatomy, pathophysiology, clinical and urodynamic findings.
  • METHODS: The literature search was performed on PubMed, Medline and Google scholar using the following keywords: 'neurogenic bladder', 'disc herniation', 'disc prolapse', 'disc protrusion', 'cauda equina syndrome', 'treatment', 'surgery', 'urodynamic', either alone or in combination using 'AND' or 'OR'.
  • FINDINGS: The literature reviewed confirmed correlations between neurogenic bladder and disc disease.
  • Chronic nervous damage induces reduction of bladder sensitivity and detrusor atrophy.
  • An overdistension of the bladder follows, with global and circumferential thinning of the bladder wall.
  • CONCLUSIONS: Benefits for neurogenic bladder obtained through disc disease treatment should be studied in more detail, especially conservative therapies, not yet discussed in literature.
  • [MeSH-major] Intervertebral Disc / pathology. Spinal Diseases / complications. Urinary Bladder, Neurogenic / etiology

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  • (PMID = 23701600.001).
  • [ISSN] 1473-4877
  • [Journal-full-title] Current medical research and opinion
  • [ISO-abbreviation] Curr Med Res Opin
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] England
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43. |||||..... 50%  Padrão AI, Oliveira P, Vitorino R, Colaço B, Pires MJ, Márquez M, Castellanos E, Neuparth MJ, Teixeira C, Costa C, Moreira-Gonçalves D, Cabral S, Duarte JA, Santos LL, Amado F, Ferreira R: Bladder cancer-induced skeletal muscle wasting: disclosing the role of mitochondria plasticity. Int J Biochem Cell Biol; 2013 Jul;45(7):1399-409
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Bladder cancer-induced skeletal muscle wasting: disclosing the role of mitochondria plasticity.
  • The histologic signs of non-muscle-invasive bladder tumors observed in BBN animals were related to 17% loss of body weight and high serum levels of IL-1β, TNF-α, TWEAK, C-reactive protein, myostatin and lactate and high urinary MMPs activities, suggesting a catabolic phenotype underlying urothelial carcinoma.
  • [MeSH-major] Mitochondria, Muscle / metabolism. Muscle, Skeletal / pathology. Muscular Atrophy / metabolism. Urinary Bladder Neoplasms / metabolism

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  • HSDB. structure - 1-NAPHTHYLAMINE.
  • HSDB. structure - LACTIC ACID.
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  • [Copyright] Copyright © 2013 Elsevier Ltd. All rights reserved.
  • (PMID = 23608519.001).
  • [ISSN] 1878-5875
  • [Journal-full-title] The international journal of biochemistry & cell biology
  • [ISO-abbreviation] Int. J. Biochem. Cell Biol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / Apoptosis Regulatory Proteins; 0 / Boronic Acids; 0 / Interleukin-1beta; 0 / Membrane Proteins; 0 / Myostatin; 0 / N-(o-boronic acid)benzyl-1-naphthylamine; 0 / TWEAK protein, rat; 0 / Tumor Necrosis Factor-alpha; 0 / Tumor Necrosis Factors; 33X04XA5AT / Lactic Acid; 9007-41-4 / C-Reactive Protein; 9753I242R5 / 1-Naphthylamine
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44. |||||..... 50%  Jurkiewicz B, Ząbkowski T, Shevchuk D: Ureteral quintuplication with renal atrophy in an infant after the 1986 Chernobyl nuclear disaster. Urology; 2014 Jan;83(1):211-3
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Ureteral quintuplication with renal atrophy in an infant after the 1986 Chernobyl nuclear disaster.
  • Ureteral duplication is a comparatively frequent urinary tract anomaly.
  • The left ureter was divided into 5 ureters with 5 renal pelvises within approximately 3 cm of the urinary bladder, and trace parenchyma of the kidney was noted.
  • [MeSH-minor] Atrophy / complications. Child, Preschool. Female. Humans. Time Factors

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  • [Copyright] Copyright © 2014 Elsevier Inc. All rights reserved.
  • (PMID = 24001707.001).
  • [ISSN] 1527-9995
  • [Journal-full-title] Urology
  • [ISO-abbreviation] Urology
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
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45. |||||..... 50%  Radziszewski P, Baranowski W, Nowak-Markwitz E, Rechbeger T, Suzin J, Witek A, Expert Panel: [Expert panel recommendations on therapeutic and diagnostic management of urinary incontinence and overactive bladder in women]. Ginekol Pol; 2010 Oct;81(10):789-93
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [Expert panel recommendations on therapeutic and diagnostic management of urinary incontinence and overactive bladder in women].
  • The expert panel recommendations are issued in order to increase the number of patients with urinary incontinence and overactive bladder receiving appropriate care.
  • The initial management of stress urinary incontinence should include lifestyle interventions, and physiotherapy Use of pessaries is acceptable in women who are not fit or do not want surgical therapy Local estrogen therapy should only be used in women with urogenital atrophy Duloxetin is an option in the pharmacological therapy of stress incontinence, but it doesn't cure the disease.
  • The ineffectiveness of initial procedure should be indication to surgery Alphaadrenomimetic drugs are not recommended in the therapy of urinary incontinence.
  • The initial management of overactive bladder and urgency incontinence should include lifestyle interventions, however fluid restrictions (if fluid load is less than 3000 ml) are not recommended.
  • The cornerstone of overactive bladder and urgency incontinence therapy remains the treatment with anticholinergic drugs.
  • [MeSH-major] Urinary Bladder, Overactive / diagnosis. Urinary Bladder, Overactive / therapy. Urinary Incontinence / diagnosis. Urinary Incontinence / therapy. Women's Health


46. |||||..... 49%  Diamond P, Hassonah S, Alarab M, Lovatsis D, Drutz HP: The prevalence of detrusor overactivity amongst patients with symptoms of overactive bladder: a retrospective cohort study. Int Urogynecol J; 2012 Nov;23(11):1577-80
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] The prevalence of detrusor overactivity amongst patients with symptoms of overactive bladder: a retrospective cohort study.
  • INTRODUCTION AND HYPOTHESIS: Overactive bladder (OAB) is a symptom-based condition consisting of urgency, with or without incontinence, usually with frequency and nocturia.
  • Urogenital atrophy and incomplete emptying were more common.
  • [MeSH-major] Muscle, Smooth / physiopathology. Urinary Bladder, Overactive / epidemiology. Urinary Bladder, Overactive / physiopathology

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  • [CommentIn] Int Urogynecol J. 2013 May;24(5):885 [23572087.001]
  • [CommentIn] Int Urogynecol J. 2013 May;24(5):887-8 [23572086.001]
  • (PMID = 22531957.001).
  • [ISSN] 1433-3023
  • [Journal-full-title] International urogynecology journal
  • [ISO-abbreviation] Int Urogynecol J
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Cholinergic Antagonists
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47. |||||..... 49%  Ludwig L, Seraphim D, Lorenzetti F, Bertolla RP, Ortiz V, Dambros M: Inhibitory activity of α-tocopherol on apoptosis in the rat bladder wall subjected to androgen deprivation. Neurourol Urodyn; 2011 Jan;30(1):194-8
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Inhibitory activity of α-tocopherol on apoptosis in the rat bladder wall subjected to androgen deprivation.
  • Thus to add to this knowledge in this study we studied the potential inhibitory activity on oxidative stress and process apoptosis on bladder wall in male rats subjected to androgen deprivation.
  • A causal relationship between lower testosterone levels and apoptosis, as a component of castration-induced muscle atrophy, has been shown.
  • [MeSH-major] Antioxidants / pharmacology. Apoptosis / drug effects. Oxidative Stress / drug effects. Urinary Bladder / metabolism. alpha-Tocopherol / pharmacology

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  • HSDB. structure - PROSTAGLANDIN F2ALPHA.
  • HSDB. structure - TESTOSTERONE.
  • HSDB. structure - VITAMIN E.
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  • [Copyright] Copyright © 2010 Wiley-Liss, Inc.
  • (PMID = 20665547.001).
  • [ISSN] 1520-6777
  • [Journal-full-title] Neurourology and urodynamics
  • [ISO-abbreviation] Neurourol. Urodyn.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Antioxidants; 27415-26-5 / 8-epi-prostaglandin F2alpha; 3XMK78S47O / Testosterone; B7IN85G1HY / Dinoprost; EC 3.4.22.- / Caspase 3; H4N855PNZ1 / alpha-Tocopherol
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48. |||||..... 49%  Zlotta AR, Roumeguere T, Kuk C, Alkhateeb S, Rorive S, Lemy A, van der Kwast TH, Fleshner NE, Jewett MA, Finelli A, Schulman C, Lotan Y, Shariat SF, Nortier J: Select screening in a specific high-risk population of patients suggests a stage migration toward detection of non-muscle-invasive bladder cancer. Eur Urol; 2011 Jun;59(6):1026-31
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Select screening in a specific high-risk population of patients suggests a stage migration toward detection of non-muscle-invasive bladder cancer.
  • BACKGROUND: More than 25% of bladder cancer (BC) cases are still muscle-invasive at first diagnosis.
  • MEASUREMENTS: Patients were evaluated for presence of BC and tumor stage at diagnosis.
  • Among 43 patients who underwent screening cystoscopies (median follow-up: 94 mo), 22 were first diagnosed with non-muscle-invasive BC but none with muscle-invasive tumors and none died of BC.
  • [MeSH-major] Anti-Obesity Agents / adverse effects. Aristolochic Acids / adverse effects. Cystoscopy. Drugs, Chinese Herbal / adverse effects. Kidney Diseases / chemically induced. Mass Screening / methods. Urinary Bladder Neoplasms / diagnosis
  • [MeSH-minor] Atrophy. Biopsy. Early Detection of Cancer. Female. Fibrosis. Humans. Male. Neoplasm Invasiveness. Neoplasm Staging. Predictive Value of Tests. Prognosis. Risk Assessment. Risk Factors. Time Factors

  • Genetic Alliance. consumer health - Bladder cancer.
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  • [Copyright] Copyright © 2011 European Association of Urology. Published by Elsevier B.V. All rights reserved.
  • (PMID = 21458152.001).
  • [ISSN] 1873-7560
  • [Journal-full-title] European urology
  • [ISO-abbreviation] Eur. Urol.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Switzerland
  • [Chemical-registry-number] 0 / Anti-Obesity Agents; 0 / Aristolochic Acids; 0 / Drugs, Chinese Herbal
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49. |||||..... 49%  Comiter C, Phull HS: Angiotensin II type 1 (AT-1) receptor inhibition partially prevents the urodynamic and detrusor changes associated with bladder outlet obstruction: a mouse model. BJU Int; 2012 Jun;109(12):1841-6
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Angiotensin II type 1 (AT-1) receptor inhibition partially prevents the urodynamic and detrusor changes associated with bladder outlet obstruction: a mouse model.
  • Angiotensin II is the main effector peptide in the bladder local renin-angiotensin system.
  • This experiment demonstrates the role of this local renin-angiotensin system with respect to bladder outlet obstruction.
  • OBJECTIVE: • To determine if treatment with an angiotensin II type 1 (AT-1) receptor antagonist, losartan, can prevent the structural and functional changes that occur in a mouse model of bladder outlet obstruction (BOO).
  • Six mice served as unobstructed controls, and six unobstructed mice were given oral losartan (10 mg/kg/day) to determine the effect of angiotensin II inhibition on the normal bladder.
  • Bladder capacity (C), detrusor pressure during voiding (Pdet) and volume at first non-voiding contraction (NVC1) as a percentage of C were recorded after 6 weeks.
  • • Bladders were stained with haematoxylin and eosin for measurement of detrusor muscular thickness, and graded as 1 = atrophy (<100 µm thick), 2 = normal (100-200 µm thick), 3 = hypertrophy (>200 µm thick) compared with controls.
  • BOO mice manifested greater bladder weight (93.2 ± 11.7 mg vs 26.8 ± 2.40 mg, P < 0.01) and greater detrusor muscle thickness (median 3 vs 2, P= 0.02).
  • • Treatment with losartan mediated an insignificant reduction in mean bladder weight (68.1 ± 9.1 mg vs 93.2 ± 11.7 mg, P= 0.10), but a significant reduction in detrusor muscle thickness (median 2 vs 3, P= 0.02).
  • Losartan did not mediate any significant structural or functional changes in the unobstructed mouse bladder.
  • [MeSH-major] Angiotensin II Type 1 Receptor Blockers / pharmacology. Losartan / pharmacology. Muscle, Smooth / drug effects. Urinary Bladder Neck Obstruction / drug therapy. Urodynamics / drug effects

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  • [Copyright] © 2011 THE AUTHORS. BJU INTERNATIONAL © 2011 BJU INTERNATIONAL.
  • (PMID = 21939491.001).
  • [ISSN] 1464-410X
  • [Journal-full-title] BJU international
  • [ISO-abbreviation] BJU Int.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Angiotensin II Type 1 Receptor Blockers; JMS50MPO89 / Losartan
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50. |||||..... 49%  Ali-El-Dein B, Ashamallah A: Vaginal repair of pouch-vaginal fistula after orthotopic bladder substitution in women. Urology; 2013 Jan;81(1):198-202
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Vaginal repair of pouch-vaginal fistula after orthotopic bladder substitution in women.
  • OBJECTIVE: To report the diagnosis, technique, and treatment outcome of pouch-vaginal fistula (PVF) with particular stress on the vaginal repair.
  • Certain modifications were adopted after 100 patients to prevent chronic urinary retention and PVF.
  • Transabdominal repair was used in 2 patients(high, large, vaginal atrophy) and the other patients underwent a vaginal repair in nonopposing layers 3 to 6 months after cystectomy.
  • It is recommended in low, small fistula and if postmenopausal vaginal atrophy is absent.
  • [MeSH-major] Urinary Fistula / surgery. Urinary Reservoirs, Continent / adverse effects. Vagina / surgery. Vaginal Fistula / surgery

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  • [Copyright] Copyright © 2013 Elsevier Inc. All rights reserved.
  • [CommentIn] Urology. 2013 Jan;81(1):202-3; author reply 203 [23206583.001]
  • (PMID = 23206581.001).
  • [ISSN] 1527-9995
  • [Journal-full-title] Urology
  • [ISO-abbreviation] Urology
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
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