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1. Biomedical articles (top 50; 2009 to 2014)
1. |||||..... 50%  Ochoa TJ, Contreras CA: Enteropathogenic escherichia coli infection in children. Curr Opin Infect Dis; 2011 Oct;24(5):478-83
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Enteropathogenic escherichia coli infection in children.
  • PURPOSE OF REVIEW: Enteropathogenic Escherichia coli (EPEC) is an important diarrheal pathogen of young children.
  • As the diagnosis of EPEC is now based mainly on molecular criteria, there has been an important change in its prevalence.
  • The purpose of this study is to review the current epidemiology of EPEC infection and the new insights into its physiopathology.
  • However, the exact mechanisms of diarrhea in EPEC infection are not completely understood.
  • [MeSH-major] Enteropathogenic Escherichia coli. Escherichia coli Infections / epidemiology

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  • (PMID = 21857511.001).
  • [ISSN] 1473-6527
  • [Journal-full-title] Current opinion in infectious diseases
  • [ISO-abbreviation] Curr. Opin. Infect. Dis.
  • [Language] eng
  • [Grant] United States / FIC NIH HHS / TW / 1K01TW007405; United States / FIC NIH HHS / TW / K01 TW007405; United States / FIC NIH HHS / TW / K01 TW007405-04
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Review
  • [Publication-country] United States
  • [Other-IDs] NLM/ NIHMS348474; NLM/ PMC3277943
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2. |||||..... 48%  Swennes AG, Buckley EM, Parry NM, Madden CM, García A, Morgan PB, Astrofsky KM, Fox JG: Enzootic enteropathogenic Escherichia coli infection in laboratory rabbits. J Clin Microbiol; 2012 Jul;50(7):2353-8
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  • [Title] Enzootic enteropathogenic Escherichia coli infection in laboratory rabbits.
  • Enteropathogenic Escherichia coli (EPEC) is the most important cause of persistent diarrhea in children, particularly in developing countries.
  • Animals serve as pathogenic E. coli reservoirs, and compelling evidence for cross-species EPEC transmission exists.
  • In this report, enzootic EPEC infection associated with up to 10.5% diarrhea-associated morbidity in a large laboratory Dutch Belted rabbit colony was investigated.
  • Fecal culture of 20 rabbits yielded 48 E. coli isolates, 83% of which were eae positive.
  • This report emphasizes the zoonotic potential of animal EPEC strains and the need for virulence determinant-based screening of E. coli isolates from diarrheic animals.
  • [MeSH-major] Animals, Laboratory / microbiology. Diarrhea / veterinary. Disease Outbreaks. Enteropathogenic Escherichia coli / isolation & purification. Escherichia coli Infections / veterinary. Rabbits / microbiology

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  • (PMID = 22573597.001).
  • [ISSN] 1098-660X
  • [Journal-full-title] Journal of clinical microbiology
  • [ISO-abbreviation] J. Clin. Microbiol.
  • [Language] eng
  • [Databank-accession-numbers] GENBANK/ JQ700206
  • [Grant] United States / NCRR NIH HHS / RR / T32 RR070036
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Anti-Bacterial Agents; 0 / DNA, Bacterial; 0 / Fluoroquinolones; 93106-60-6 / enrofloxacin
  • [Other-IDs] NLM/ PMC3405579
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3. ||||...... 44%  Schüller S, Lucas M, Kaper JB, Girón JA, Phillips AD: The ex vivo response of human intestinal mucosa to enteropathogenic Escherichia coli infection. Cell Microbiol; 2009 Mar;11(3):521-30
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  • [Title] The ex vivo response of human intestinal mucosa to enteropathogenic Escherichia coli infection.
  • In vitro organ culture (IVOC) represents a gold standard model to study enteropathogenic E. coli (EPEC) infection of human intestinal mucosa.
  • A polarized IVOC system (pIVOC) was developed in order to overcome such limitations: apical EPEC infection produced negligible bacterial leakage via biopsy edges, resulted in enhanced colonization compared with standard IVOC, and showed evidence of bacterial detachment, as in natural rabbit EPEC infections.
  • Examination of mucosal innate immune responses in pIVOC showed both interleukin (IL)-8 mRNA and protein levels were significantly increased after apical EPEC infection.
  • Immunofluorescence staining of EPEC-infected small intestinal biopsies revealed apical and basolateral distribution of Toll-like receptor (TLR) 5 on epithelium, suggesting that EPEC can trigger mucosal IL-8 responses by apical flagellin/TLR5 interaction ex vivo and does not require access to the basolateral membrane as postulated in cell culture models.
  • [MeSH-major] Enteropathogenic Escherichia coli / growth & development. Intestinal Mucosa / microbiology
  • [MeSH-minor] Escherichia coli Proteins / genetics. Escherichia coli Proteins / immunology. Gene Deletion. Gene Expression Profiling. Humans. Interleukin-8 / biosynthesis. Organ Culture Techniques / methods. Toll-Like Receptor 5 / biosynthesis

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  • (PMID = 19134113.001).
  • [ISSN] 1462-5822
  • [Journal-full-title] Cellular microbiology
  • [ISO-abbreviation] Cell. Microbiol.
  • [Language] eng
  • [Grant] United States / NIDDK NIH HHS / DK / R01 DK058957-07; United States / NIDDK NIH HHS / DK / R01 DK058957-09; United States / NIDDK NIH HHS / DK / R01DK58957; United States / NIAID NIH HHS / AI / R37 AI021657-22; United States / NIAID NIH HHS / AI / R37 AI021657-23; United States / NIAID NIH HHS / AI / R37AI21657
  • [Publication-type] In Vitro; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Escherichia coli Proteins; 0 / FliC protein, E coli; 0 / Interleukin-8; 0 / TLR5 protein, human; 0 / Toll-Like Receptor 5
  • [Other-IDs] NLM/ NIHMS105491; NLM/ PMC2676445
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4. ||||||||.. 77%  Cooper IF, Siadaty MS: 'Functional Concepts' associated with 'Enteritis Escherichia Coli Enteropathogenic': Top Publications. BioMedLib Review; FunctionalConcept;EnteritisEscherichiaColi:706380104. ISSN: 2331-5717. 2014/7/26
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  • [Title] 'Functional Concepts' associated with 'Enteritis Escherichia Coli Enteropathogenic': Top Publications.
  • There are articles published each month which present 'Functional Concept' for 'enteritis escherichia coli enteropathogenic'.
  • Wu Y et al: Enteropathogenic Escherichia coli infection triggers host phospholipid metabolism perturbations.
  • Cantarelli VV et al: Cortactin is necessary for F-actin accumulation in pedestal structures induced by enteropathogenic Escherichia coli infection.
  • Swennes AG et al: Enzootic enteropathogenic Escherichia coli infection in laboratory rabbits.
  • Schüller S et al: The ex vivo response of human intestinal mucosa to enteropathogenic Escherichia coli infection.
  • Buret A et al: Effects of orally administered epidermal growth factor on enteropathogenic Escherichia coli infection in rabbits.
  • Fagundes-Neto U et al: The gut at war: the consequences of enteropathogenic Escherichia coli infection as a factor of diarrhea and malnutrition.
  • Bockemühl J et al: [Enteritis due to Escherichia coli O142 K86 H34 in a ward of premature infants. With a discussion on the problem of pathogenicity of "enteropathogenic serogroups of E. coli" (author's transl)].
  • Petrović M et al: [Enteritis due to enteropathogenic Escherichia coli].
  • Neter E: Enteritis due to enteropathogenic Escherichia coli.

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
  • (UID = 706380104.001).
  • [ISSN] 2331-5717
  • [Journal-full-title] BioMedLib Review
  • [Language] eng
  • [Publication-type] Review
  • [Publication-country] UNITED STATES
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5. ||||...... 43%  Hemrajani C, Berger CN, Robinson KS, Marchès O, Mousnier A, Frankel G: NleH effectors interact with Bax inhibitor-1 to block apoptosis during enteropathogenic Escherichia coli infection. Proc Natl Acad Sci U S A; 2010 Feb 16;107(7):3129-34
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] NleH effectors interact with Bax inhibitor-1 to block apoptosis during enteropathogenic Escherichia coli infection.
  • The human pathogens enteropathogenic (EPEC) and enterohemorrhagic Escherichia coli and the related mouse pathogen Citrobacter rodentium subvert a variety of host cell signaling pathways via their plethora of type III secreted effectors, including triggering of an early apoptotic response.
  • During EPEC infection, NleH effectors inhibit elevation of cytosolic Ca(2+) concentrations, nuclear condensation, caspase-3 activation, and membrane blebbing and promote cell survival.
  • [MeSH-major] Apoptosis / drug effects. Enterobacteriaceae Infections / metabolism. Escherichia coli Proteins / metabolism. Membrane Proteins / metabolism. Virulence Factors / metabolism
  • [MeSH-minor] Animals. Blotting, Western. Calcium / metabolism. Caspase 3 / metabolism. Cell Nucleus / drug effects. Citrobacter rodentium. Cloning, Molecular. Cytosol / metabolism. Escherichia coli. Female. Mice. Microscopy, Electron, Scanning. Microscopy, Fluorescence. Specific Pathogen-Free Organisms. Two-Hybrid System Techniques

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  • (PMID = 20133763.001).
  • [ISSN] 1091-6490
  • [Journal-full-title] Proceedings of the National Academy of Sciences of the United States of America
  • [ISO-abbreviation] Proc. Natl. Acad. Sci. U.S.A.
  • [Language] eng
  • [Grant] United Kingdom / Medical Research Council / / ; United Kingdom / Wellcome Trust / /
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Escherichia coli Proteins; 0 / Membrane Proteins; 0 / Tmbim6 protein, mouse; 0 / Virulence Factors; EC 3.4.22.- / Caspase 3; SY7Q814VUP / Calcium
  • [Other-IDs] NLM/ PMC2840288
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6. ||||...... 41%  Gareau MG, Ho NK, Brenner D, Sousa AJ, Lebourhis L, Mak TW, Girardin SE, Philpott DJ, Sherman PM: Enterohaemorrhagic, but not enteropathogenic, Escherichia coli infection of epithelial cells disrupts signalling responses to tumour necrosis factor-alpha. Microbiology; 2011 Oct;157(Pt 10):2963-73
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  • [Title] Enterohaemorrhagic, but not enteropathogenic, Escherichia coli infection of epithelial cells disrupts signalling responses to tumour necrosis factor-alpha.
  • Enterohaemorrhagic Escherichia coli (EHEC), serotype O157 : H7 is a non-invasive, pathogenic bacterium that employs a type III secretion system (T3SS) to inject effector proteins into infected cells.
  • Using complementary techniques, stimulation with TNFα caused activation of NF-κB, as determined by luciferase reporter assay (increase in gene expression), Western blotting (phosphorylation of IκBα), immunofluorescence (p65 nuclear translocation) and immunoassay (CXCL-8 secretion), and each was blocked by EHEC O157 : H7 infection.
  • In contrast, subversion of host cell signalling was not observed following exposure to either enteropathogenic E. coli, strain E2348/69 (O127 : H6) or the laboratory E. coli strain HB101.
  • [MeSH-major] Cell Line / microbiology. Enterohemorrhagic Escherichia coli / physiology. Epithelial Cells / metabolism. Escherichia coli Infections / metabolism. Escherichia coli O157 / physiology. Signal Transduction. Tumor Necrosis Factor-alpha / metabolism

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  • (PMID = 21798984.001).
  • [ISSN] 1465-2080
  • [Journal-full-title] Microbiology (Reading, England)
  • [ISO-abbreviation] Microbiology (Reading, Engl.)
  • [Language] eng
  • [Grant] Canada / Canadian Institutes of Health Research / / MOP-89894
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / NF-kappa B; 0 / Tumor Necrosis Factor-alpha
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7. ||||...... 44%  Gouveia EM, Silva IS, Nakazato G, Araujo FR, Chang MR: Experimental infection with enteropathogenic Escherichia coli identified by PCR using enteric-coated capsules in boxer pups. Acta Cir Bras; 2011 Apr;26(2):144-8
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  • [Title] Experimental infection with enteropathogenic Escherichia coli identified by PCR using enteric-coated capsules in boxer pups.
  • PURPOSE: To verify the possibility of an experimental infection with enteropathogenic Escherichia coli and to confirm by PCR that the symptoms manifested after infection were due to the virulence factors of the studied bacteria.
  • Gelatinous capsules coated with enteric-coating solution were used for the inoculation of strains. E. coli isolation from feces was performed for all tested animals, and the extracted DNA was subjected to Polymerase Chain Reaction (PCR).
  • CONCLUSION: The efficiency of PCR for the studied strains indicates that this technique can be recommended for the diagnosis of enteropathogenic Escherichia coli as a differential from other pathogens causing diarrhea.
  • It may also be used in the future to verify whether other virulence factors (bfpA gene and EAF plasmid) persist after infection and to assess the pathogenicity of these bacteria.
  • [MeSH-major] Enteropathogenic Escherichia coli / genetics. Escherichia coli Infections / microbiology. Polymerase Chain Reaction

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  • (PMID = 21445479.001).
  • [ISSN] 1678-2674
  • [Journal-full-title] Acta cirúrgica brasileira / Sociedade Brasileira para Desenvolvimento Pesquisa em Cirurgia
  • [ISO-abbreviation] Acta Cir Bras
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Brazil
  • [Chemical-registry-number] 0 / DNA, Bacterial; 0 / Virulence Factors
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8. |......... 11%  Cooper IF, Siadaty MS: 'Diseases or Syndromes' associated with 'Enzootic': Top Publications. BioMedLib Review; DiseaseOrSyndrome;Enzootic:705310219. ISSN: 2331-5717. 2014/3/25
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  • Kenney JL et al: Genetic and anatomic determinants of enzootic Venezuelan equine encephalitis virus infection of Culex (Melanoconion) taeniopus.
  • Swennes AG et al: Enzootic enteropathogenic Escherichia coli infection in laboratory rabbits.
  • Lőrincz M et al: Sporadic re-emergence of enzootic porcine transmissible gastroenteritis in Hungary.

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
  • (UID = 705310219.001).
  • [ISSN] 2331-5717
  • [Journal-full-title] BioMedLib Review
  • [Language] eng
  • [Publication-type] Review
  • [Publication-country] UNITED STATES
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9. |||....... 32%  Yoda K, He F, Kawase M, Miyazawa K, Hiramatsu M: Oral administration of Lactobacillus gasseri TMC0356 stimulates peritoneal macrophages and attenuates general symptoms caused by enteropathogenic Escherichia coli infection. J Microbiol Immunol Infect; 2014 Apr;47(2):81-6
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  • [Title] Oral administration of Lactobacillus gasseri TMC0356 stimulates peritoneal macrophages and attenuates general symptoms caused by enteropathogenic Escherichia coli infection.
  • BACKGROUND: Enteropathogenic Escherichia coli (EPEC) is an important cause of diarrhea in human.
  • This study was conducted to investigate the ability of orally administrated probiotic lactobacilli to protect hosts from EPEC infection via enhancement of immune responses.
  • CONCLUSION: These results suggest that some of selected probiotic lactobacilli may, at least partly, protect hosts from EPEC infection by the enhancement of innate immunity of host and attenuate symptoms caused by the infection.

  • NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .
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  • [Copyright] Copyright © 2012. Published by Elsevier B.V.
  • (PMID = 23079484.001).
  • [ISSN] 1995-9133
  • [Journal-full-title] Journal of microbiology, immunology, and infection = Wei mian yu gan ran za zhi
  • [ISO-abbreviation] J Microbiol Immunol Infect
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Keywords] NOTNLM ; Enteropathogenic E. coli / Lactobacillus / Macrophage / Phagocytosis
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10. ||........ 25%  Vulcano AB, Tino-De-Franco M, Amaral JA, Ribeiro OG, Cabrera WH, Bordenalli MA, Carbonare CB, Álvares EP, Carbonare SB: Oral infection with enteropathogenic Escherichia coli triggers immune response and intestinal histological alterations in mice selected for their minimal acute inflammatory responses. Microbiol Immunol; 2014 Jun;58(6):352-9
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Oral infection with enteropathogenic Escherichia coli triggers immune response and intestinal histological alterations in mice selected for their minimal acute inflammatory responses.
  • Enteropathogenic Escherichia coli (EPEC), a leading cause of infant diarrhea, is an important public health problem in Brazil and other developing countries.
  • In this study, the effects of oral infection with EPEC on mice selected for their minimal acute inflammatory response (AIR min) were evaluated.
  • However, electron microscopy did not show bacteria attached to the intestinal epithelia to form attaching and effacing lesions, characteristic of EPEC in humans.
  • The bacteria were detected in Peyer's patches and intestinal contents up to 5 hr post-infection.
  • In summary, it was found that EPEC triggers immune responses and intestinal histological alterations but does not produce evidence of diarrheal disease in mice infected by the oral route.
  • This study of EPEC experimental infection provides a better understanding of the effects of antibodies on bacterial infections and may provide a suitable model for the design and testing of immunobiological products for active or passive immunization.

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  • [Copyright] © 2014 The Societies and Wiley Publishing Asia Pty Ltd.
  • (PMID = 24750489.001).
  • [ISSN] 1348-0421
  • [Journal-full-title] Microbiology and immunology
  • [ISO-abbreviation] Microbiol. Immunol.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Australia
  • [Keywords] NOTNLM ; anti-Enteropathogenic Escherichia coli / antibodies / enteropathogenic Escherichia coli / experimental infection / selected mouse lines
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11. |||....... 33%  Calderon Toledo C, Arvidsson I, Karpman D: Cross-reactive protection against enterohemorrhagic Escherichia coli infection by enteropathogenic E. coli in a mouse model. Infect Immun; 2011 Jun;79(6):2224-33
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Cross-reactive protection against enterohemorrhagic Escherichia coli infection by enteropathogenic E. coli in a mouse model.
  • Enteropathogenic Escherichia coli (EPEC) and enterohemorrhagic E. coli (EHEC) are related attaching and effacing (A/E) pathogens.
  • We used a mouse model to inoculate BALB/c mice intragastrically, first with EPEC and then with EHEC (E. coli O157:H7).
  • Four control groups received either a nonpathogenic E. coli (NPEC) strain followed by EHEC (NPEC/EHEC), phosphate-buffered saline (PBS) followed by EHEC (PBS/EHEC), EPEC/PBS, or PBS/PBS.
  • EPEC colonized the intestine after challenge, and mice developed serum antibodies to intimin and E. coli secreted protein B (encoded in the LEE).
  • Prechallenge with an EPEC strain had a protective effect after EHEC infection, as only a few mice developed mild symptoms, from which they recovered.
  • These mice had an increase in body weight similar to that in control animals, and tissue morphology exhibited mild intestinal changes and normal renal histology.
  • All mice that were not prechallenged with the EPEC strain developed mild to severe symptoms after EHEC infection, with weight loss as well as intestinal and renal histopathological changes.
  • These data suggest that EPEC may protect against EHEC infection in this mouse model.
  • [MeSH-major] Antibodies, Bacterial / immunology. Cross Protection / immunology. Enterohemorrhagic Escherichia coli / immunology. Enteropathogenic Escherichia coli / immunology. Escherichia coli Infections / immunology

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  • (PMID = 21402761.001).
  • [ISSN] 1098-5522
  • [Journal-full-title] Infection and immunity
  • [ISO-abbreviation] Infect. Immun.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Antibodies, Bacterial
  • [Other-IDs] NLM/ PMC3125830
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12. |||....... 32%  Girard F, Crepin VF, Frankel G: Modelling of infection by enteropathogenic Escherichia coli strains in lineages 2 and 4 ex vivo and in vivo by using Citrobacter rodentium expressing TccP. Infect Immun; 2009 Apr;77(4):1304-14
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  • [Title] Modelling of infection by enteropathogenic Escherichia coli strains in lineages 2 and 4 ex vivo and in vivo by using Citrobacter rodentium expressing TccP.
  • Enteropathogenic Escherichia coli (EPEC) strains colonize the human gut mucosa via attaching-and-effacing (A/E) lesion formation, while in vitro they employ diverse strategies to trigger actin polymerization.
  • Since mice are resistant to EPEC infection, in vivo studies are frequently done using the murine model pathogen Citrobacter rodentium, which shares with EPEC-1 strains the ability to induce A/E lesions and trigger strong actin polymerization via the Tir:Nck pathway.
  • Using a mouse intestinal in vitro organ culture model and oral gavage into C57BL/6 mice, we have shown that TccP can cooperate with Tir of C. rodentium.
  • [MeSH-major] Citrobacter rodentium / pathogenicity. Disease Models, Animal. Enteropathogenic Escherichia coli / pathogenicity. Escherichia coli Infections. Escherichia coli Proteins / metabolism

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  • (PMID = 19188355.001).
  • [ISSN] 1098-5522
  • [Journal-full-title] Infection and immunity
  • [ISO-abbreviation] Infect. Immun.
  • [Language] eng
  • [Grant] United Kingdom / Biotechnology and Biological Sciences Research Council / / ; United Kingdom / Wellcome Trust / /
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Actins; 0 / Escherichia coli Proteins; 0 / tccP protein, E coli
  • [Other-IDs] NLM/ PMC2663131
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13. |||....... 29%  Kobayashi T, Ogawa M, Sanada T, Mimuro H, Kim M, Ashida H, Akakura R, Yoshida M, Kawalec M, Reichhart JM, Mizushima T, Sasakawa C: The Shigella OspC3 effector inhibits caspase-4, antagonizes inflammatory cell death, and promotes epithelial infection. Cell Host Microbe; 2013 May 15;13(5):570-83
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  • [Title] The Shigella OspC3 effector inhibits caspase-4, antagonizes inflammatory cell death, and promotes epithelial infection.
  • Caspase-mediated inflammatory cell death acts as an intrinsic defense mechanism against infection.
  • In a screen for Shigella flexneri effectors that regulate cell death during infection, we discovered that Shigella infection induced acute inflammatory, caspase-4-dependent epithelial cell death, which is counteracted by the bacterial OspC3 effector.
  • Infection of guinea pigs with a Shigella ospC3-deficient mutant resulted in enhanced inflammatory cell death and associated symptoms, correlating with decreased bacterial burdens.
  • Salmonella Typhimurium and enteropathogenic Escherichia coli infection also induced caspase-4-dependent epithelial death.
  • These findings highlight the importance of caspase-4-dependent innate immune responses and demonstrate that Shigella delivers a caspase-4-specific inhibitor to delay epithelial cell death and promote infection.
  • [MeSH-minor] Animals. Cell Line. DNA, Bacterial / chemistry. DNA, Bacterial / genetics. Disease Models, Animal. Dysentery, Bacillary / immunology. Dysentery, Bacillary / microbiology. Dysentery, Bacillary / pathology. Escherichia coli / immunology. Escherichia coli / pathogenicity. Gene Knockout Techniques. Guinea Pigs. Humans. Molecular Sequence Data. Protein Binding. Protein Interaction Mapping. Salmonella typhimurium / immunology. Salmonella typhimurium / pathogenicity. Sequence Analysis, DNA. Virulence Factors / genetics. Virulence Factors / metabolism

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  • [Copyright] Copyright © 2013 Elsevier Inc. All rights reserved.
  • (PMID = 23684308.001).
  • [ISSN] 1934-6069
  • [Journal-full-title] Cell host & microbe
  • [ISO-abbreviation] Cell Host Microbe
  • [Language] eng
  • [Databank-accession-numbers] GENBANK/ AB819724/ AB819725/ AB819726
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Bacterial Proteins; 0 / DNA, Bacterial; 0 / Enzyme Inhibitors; 0 / Virulence Factors; EC 3.4.22.- / CASP4 protein, human; EC 3.4.22.- / Caspases, Initiator
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14. |||....... 29%  Shui JW, Larange A, Kim G, Vela JL, Zahner S, Cheroutre H, Kronenberg M: HVEM signalling at mucosal barriers provides host defence against pathogenic bacteria. Nature; 2012 Aug 9;488(7410):222-5
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  • During intestinal Citrobacter rodentium infection, a mouse model for enteropathogenic Escherichia coli infection, Hvem−/− mice showed decreased Stat3 activation, impaired responses in the colon, higher bacterial burdens and increased mortality.
  • Likewise, in pulmonary Streptococcus pneumoniae infection, HVEM is also required for host defence.
  • [MeSH-minor] Animals. Antigens, CD / immunology. Antigens, CD / metabolism. Bacterial Load. Cell Line. Disease Models, Animal. Enterobacteriaceae Infections / immunology. Enterobacteriaceae Infections / microbiology. Enteropathogenic Escherichia coli. Epithelial Cells / immunology. Epithelial Cells / metabolism. Escherichia coli Infections. GPI-Linked Proteins / immunology. GPI-Linked Proteins / metabolism. Intestines / immunology. Intestines / microbiology. Ligands. Lung / immunology. Lung / microbiology. Lymphocytes / immunology. Lymphocytes / metabolism. Mice. Mice, Inbred C57BL. Pneumococcal Infections / immunology. Pneumococcal Infections / microbiology. Protein-Serine-Threonine Kinases / metabolism. Receptors, Immunologic / immunology. Receptors, Immunologic / metabolism. STAT3 Transcription Factor / metabolism. Streptococcus pneumoniae / immunology. Survival Rate

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  • (PMID = 22801499.001).
  • [ISSN] 1476-4687
  • [Journal-full-title] Nature
  • [ISO-abbreviation] Nature
  • [Language] eng
  • [Grant] United States / NIAID NIH HHS / AI / F32 AI083029; United States / NIDDK NIH HHS / DK / F32 DK082249; United States / NIAID NIH HHS / AI / F32-AI083029; United States / NIDDK NIH HHS / DK / F32-DK082249; United States / NIDDK NIH HHS / DK / P01 DK046763; United States / NIDDK NIH HHS / DK / P01 DK46763; United States / NIAID NIH HHS / AI / R01 AI050265; United States / NIAID NIH HHS / AI / R01 AI061516; United States / NIAID NIH HHS / AI / R01 AI064584; United States / NIAID NIH HHS / AI / R01-AI061516
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Antigens, CD; 0 / Cd160 protein, mouse; 0 / GPI-Linked Proteins; 0 / Ligands; 0 / Receptors, Immunologic; 0 / Receptors, Tumor Necrosis Factor, Member 14; 0 / STAT3 Transcription Factor; 0 / Stat3 protein, mouse; 0 / Tnfrsf14 protein, mouse; EC 2.7.11.1 / Protein-Serine-Threonine Kinases; EC 2.7.11.25 / NF-kappa B kinase
  • [Other-IDs] NLM/ NIHMS406607; NLM/ PMC3477500
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15. |||....... 29%  Esmaili A, Nazir SF, Borthakur A, Yu D, Turner JR, Saksena S, Singla A, Hecht GA, Alrefai WA, Gill RK: Enteropathogenic Escherichia coli infection inhibits intestinal serotonin transporter function and expression. Gastroenterology; 2009 Dec;137(6):2074-83
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  • [Title] Enteropathogenic Escherichia coli infection inhibits intestinal serotonin transporter function and expression.
  • Whether alteration in SERT activity contributes to the pathophysiology of diarrhea induced by the food-borne pathogen enteropathogenic Escherichia coli (EPEC) is not known.
  • The present studies examined the effects of EPEC infection on SERT activity and expression in intestinal epithelial cells and elucidated the underlying mechanisms.
  • METHODS: Caco-2 cells as a model of human intestinal epithelia and EPEC-infected C57BL/6J mouse model of infection were utilized.
  • RESULTS: Infection of Caco-2 cells with EPEC for 30-120 minutes decreased apical SERT activity (P < .001) in a type 3 secretion system dependent manner and via involvement of protein tyrosine phosphatases.
  • EPEC infection decreased V(max) of the transporter; whereas cell surface biotinylation studies revealed no alteration in the cellular or plasma membrane content of SERT in Caco-2 cells.
  • EPEC infection of mice (24 hours) reduced SERT immunostaining with a corresponding decrease in SERT messenger RNA levels, 5-HT uptake, and mucosal 5-HT content in the small intestine.
  • [MeSH-major] Enteropathogenic Escherichia coli / pathogenicity. Escherichia coli Infections / metabolism. Intestinal Mucosa / metabolism. Intestine, Small / metabolism. Serotonin / metabolism. Serotonin Plasma Membrane Transport Proteins / metabolism

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  • (PMID = 19747920.001).
  • [ISSN] 1528-0012
  • [Journal-full-title] Gastroenterology
  • [ISO-abbreviation] Gastroenterology
  • [Language] eng
  • [Grant] United States / NIDDK NIH HHS / DK / DK 061931; United States / NIDDK NIH HHS / DK / DK-074459; United States / NIDDK NIH HHS / DK / DK-09930; United States / NIDDK NIH HHS / DK / DK068271; United States / NIDDK NIH HHS / DK / K01 DK074458; United States / NIDDK NIH HHS / DK / P01 DK-067887; United States / NIDDK NIH HHS / DK / P01 DK067887-03; United States / NIDDK NIH HHS / DK / R01 DK050694-04; United States / NIDDK NIH HHS / DK / R01 DK061931-07; United States / NIDDK NIH HHS / DK / R01 DK071596
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / RNA, Messenger; 0 / SLC6A4 protein, human; 0 / Serotonin Plasma Membrane Transport Proteins; 0 / Slc6a4 protein, mouse; 333DO1RDJY / Serotonin; EC 3.1.3.48 / Protein Tyrosine Phosphatases
  • [Other-IDs] NLM/ NIHMS145181; NLM/ PMC3727418
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16. |||....... 28%  Dean P, Kenny B: Cell-surface nucleolin is sequestered into EPEC microcolonies and may play a role during infection. Microbiology; 2011 Jun;157(Pt 6):1761-7
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  • [Title] Cell-surface nucleolin is sequestered into EPEC microcolonies and may play a role during infection.
  • Nucleolin is a prominent nucleolar protein that is mobilized into the cytoplasm during infection by enteropathogenic Escherichia coli (EPEC).
  • Nucleolin also exists at low levels at the cell surface of eukaryotic cells and here we show that upon infection of an intestinal cell model, EPEC recruits and subsequently sequesters cell-surface EGFP-nucleolin into extracellularly located bacterial microcolonies.
  • Incubation of host intestinal cells with different ligands that specifically bind nucleolin impaired the ability of EPEC to disrupt epithelial barrier function but did not inhibit bacterial attachment or other effector-driven processes such as pedestal formation or microvilli effacement.
  • Taken together, this work suggests that EPEC exploits two spatially distinct pools of nucleolin during the infection process.
  • [MeSH-major] Enteropathogenic Escherichia coli / growth & development. Enteropathogenic Escherichia coli / pathogenicity. Intestine, Small / microbiology. Phosphoproteins / metabolism. RNA-Binding Proteins / metabolism

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  • (PMID = 21436219.001).
  • [ISSN] 1465-2080
  • [Journal-full-title] Microbiology (Reading, England)
  • [ISO-abbreviation] Microbiology (Reading, Engl.)
  • [Language] eng
  • [Grant] United Kingdom / Wellcome Trust / /
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Phosphoproteins; 0 / RNA-Binding Proteins; 0 / enhanced green fluorescent protein; 0 / nucleolin; 147336-22-9 / Green Fluorescent Proteins
  • [Other-IDs] NLM/ PMC3167913
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17. |||....... 28%  Liu Z, Ma Y, Moyer MP, Zhang P, Shi C, Qin H: Involvement of the mannose receptor and p38 mitogen-activated protein kinase signaling pathway of the microdomain of the integral membrane protein after enteropathogenic Escherichia coli infection. Infect Immun; 2012 Apr;80(4):1343-50
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  • [Title] Involvement of the mannose receptor and p38 mitogen-activated protein kinase signaling pathway of the microdomain of the integral membrane protein after enteropathogenic Escherichia coli infection.
  • The microdomain of the integral membrane protein (MIMP) has been shown to adhere to mucin and to antagonize the adhesion of enteropathogenic Escherichia coli (EPEC) to epithelial cells; however, the mechanism has not been fully elucidated.
  • The results indicated that MIMP adhered to NCM460 cells by binding to the mannose receptor and inhibited the phosphorylation of p38 MAPK stimulated after EPEC infection via inhibition of the Toll-like receptor 5 pathway.
  • These findings indicated that MIMPs relieve the injury of NCM460 cells after enteropathogenic E. coli infection through the mannose receptor and inhibition of the p38 MAPK signaling pathway, both of which may therefore be potential therapeutic targets for intestinal diseases, such as inflammatory bowel disease.
  • [MeSH-major] Enteropathogenic Escherichia coli / pathogenicity. Escherichia coli Infections / metabolism. Escherichia coli Proteins / metabolism. Lectins, C-Type / metabolism. MAP Kinase Signaling System. Mannose-Binding Lectins / metabolism. Membrane Proteins / metabolism. Receptors, Cell Surface / metabolism. p38 Mitogen-Activated Protein Kinases / metabolism

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  • (PMID = 22290149.001).
  • [ISSN] 1098-5522
  • [Journal-full-title] Infection and immunity
  • [ISO-abbreviation] Infect. Immun.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Escherichia coli Proteins; 0 / Lectins, C-Type; 0 / Mannose-Binding Lectins; 0 / Membrane Glycoproteins; 0 / Membrane Proteins; 0 / Receptors, Cell Surface; 0 / Toll-Like Receptor 5; 0 / mannose receptor; EC 2.7.11.24 / p38 Mitogen-Activated Protein Kinases
  • [Other-IDs] NLM/ PMC3318401
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18. |||....... 28%  Bardiau M, Szalo M, Mainil JG: Initial adherence of EPEC, EHEC and VTEC to host cells. Vet Res; 2010 Sep-Oct;41(5):57
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  • Initial adherence to host cells is the first step of the infection of enteropathogenic Escherichia coli (EPEC), enterohaemorrhagic Escherichia coli (EHEC) and verotoxigenic Escherichia coli (VTEC) strains.
  • The importance of this step in the infection resides in the fact that (1) adherence is the first contact between bacteria and intestinal cells without which the other steps cannot occur and (2) adherence is the basis of host specificity for a lot of pathogens.
  • [MeSH-major] Bacterial Adhesion / physiology. Enterohemorrhagic Escherichia coli / physiology. Enteropathogenic Escherichia coli / physiology. Intestines / cytology. Shiga-Toxigenic Escherichia coli / physiology

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  • NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .
  • NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .
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  • [Copyright] © INRA, EDP Sciences, 2010.
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  • (PMID = 20423697.001).
  • [ISSN] 0928-4249
  • [Journal-full-title] Veterinary research
  • [ISO-abbreviation] Vet. Res.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't; Review
  • [Publication-country] France
  • [Other-IDs] NLM/ PMC2881418
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19. |||....... 28%  Harmidy K, Tufenkji N, Gruenheid S: Perturbation of host cell cytoskeleton by cranberry proanthocyanidins and their effect on enteric infections. PLoS One; 2011;6(11):e27267
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  • In this study, the effect of cranberry proanthocyanidins (CPACs) on the infection of epithelial cells by two enteric bacterial pathogens, enteropathogenic Escherichia coli (EPEC) and Salmonella Typhimurium was investigated.
  • Immunofluorescence data showed that actin pedestal formation, required for infection by enteropathogenic Escherichia coli (EPEC), was disrupted in the presence of CPACs.
  • [MeSH-major] Actins / metabolism. Cytoskeleton / drug effects. Enteropathogenic Escherichia coli / drug effects. Epithelial Cells / drug effects. Proanthocyanidins / pharmacology. Salmonella typhimurium / drug effects. Vaccinium macrocarpon / chemistry
  • [MeSH-minor] Bacterial Adhesion. Bacterial Proteins / metabolism. Cell Survival / drug effects. Escherichia coli Infections / drug therapy. Escherichia coli Infections / microbiology. HeLa Cells. Humans. Salmonella Infections / drug therapy. Salmonella Infections / microbiology. Signal Transduction

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  • (PMID = 22076143.001).
  • [ISSN] 1932-6203
  • [Journal-full-title] PloS one
  • [ISO-abbreviation] PLoS ONE
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Actins; 0 / Bacterial Proteins; 0 / Proanthocyanidins
  • [Other-IDs] NLM/ PMC3208605
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20. |||....... 28%  Crane JK, Naeher TM, Broome JE, Boedeker EC: Role of host xanthine oxidase in infection due to enteropathogenic and Shiga-toxigenic Escherichia coli. Infect Immun; 2013 Apr;81(4):1129-39
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  • [Title] Role of host xanthine oxidase in infection due to enteropathogenic and Shiga-toxigenic Escherichia coli.
  • Here, we present evidence that XO is released from and active in intestinal tissues and fluids in response to infection with enteropathogenic Escherichia coli (EPEC) and Shiga-toxigenic E. coli (STEC), also known as enterohemorrhagic E. coli (EHEC).
  • XO is released into intestinal fluids in EPEC and STEC infection in a rabbit animal model.
  • XO activity results in the generation of surprisingly high concentrations of uric acid in both cultured cell and animal models of infection.
  • Hydrogen peroxide (H(2)O(2)) generated by XO activity triggered a chloride secretory response in intestinal cell monolayers within minutes but decreased transepithelial electrical resistance at 6 to 22 h.
  • H(2)O(2) generated by XO activity was effective at killing laboratory strains of E. coli, commensal microbiotas, and anaerobes, but wild-type EPEC and STEC strains were 100 to 1,000 times more resistant to killing or growth inhibition by this pathway.
  • In vivo, exogenous XO plus the substrate hypoxanthine did not protect and instead worsened the outcome of STEC infection in the rabbit ligated intestinal loop model of infection.
  • XO released during EPEC and STEC infection may serve as a virulence-inducing signal to the pathogen and not solely as a protective host defense.
  • [MeSH-major] Enteropathogenic Escherichia coli / pathogenicity. Escherichia coli Infections / pathology. Host-Pathogen Interactions. Shiga-Toxigenic Escherichia coli / pathogenicity. Xanthine Oxidase / metabolism

  • MedlinePlus Health Information. consumer health - E. Coli Infections.
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  • [CommentIn] Gut Microbes. 2013 Sep-Oct;4(5):388-91 [23811846.001]
  • (PMID = 23340314.001).
  • [ISSN] 1098-5522
  • [Journal-full-title] Infection and immunity
  • [ISO-abbreviation] Infect. Immun.
  • [Language] eng
  • [Grant] United States / NIAID NIH HHS / AI / R01 AI 081528; United States / NIAID NIH HHS / AI / R21 AI 102212; United States / NIAID NIH HHS / AI / R21 AI102212
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] United States
  • [Chemical-registry-number] 268B43MJ25 / Uric Acid; BBX060AN9V / Hydrogen Peroxide; EC 1.17.3.2 / Xanthine Oxidase
  • [Other-IDs] NLM/ PMC3639607
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21. |||....... 28%  Choi HJ, Kim J, Do KH, Park SH, Moon Y: Prolonged NF-κB activation by a macrophage inhibitory cytokine 1-linked signal in enteropathogenic Escherichia coli-infected epithelial cells. Infect Immun; 2013 Jun;81(6):1860-9
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Prolonged NF-κB activation by a macrophage inhibitory cytokine 1-linked signal in enteropathogenic Escherichia coli-infected epithelial cells.
  • Intestinal epithelial activation of nuclear factor kappa B (NF-κB) exerts both detrimental and beneficial functions in response to various luminal insults, including ones associated with mucosa-associated pathogens.
  • Gastrointestinal infection with enteropathogenic Escherichia coli (EPEC) causes severe injuries in epithelial integrity and leads to watery diarrhea.
  • The present study was conducted to investigate the prolonged epithelial responses to persistent EPEC infection via NF-κB activation.
  • EPEC infection led to sustained activation of NF-κB signal in mouse intestinal epithelial cells in vivo and in vitro, which was positively associated with a type III secretion system, whereas early NF-κB is regulated.
  • Moreover, prolonged NF-κB activation was found to be a part of macrophage inhibitory cytokine 1 (MIC-1)-mediated signaling activation, a novel link between NF-κB signaling and infection-associated epithelial stress.
  • EPEC infection induced gene expression of MIC-1, a member of the transforming growth factor β (TGF-β) superfamily, which then activated TGF-β-activated kinase 1 and consequently led to NF-κB activation.
  • In summary, the results of the present study suggest that MIC-1 serves as a mediator of prolonged NF-κB activation, which is critical in maintaining gut epithelial integrity in response to infection-induced injuries.
  • [MeSH-major] Enteropathogenic Escherichia coli / physiology. Growth Differentiation Factor 15 / metabolism. NF-kappa B / metabolism. Signal Transduction
  • [MeSH-minor] Animals. Cell Line. Cyclin D1 / genetics. Cyclin D1 / metabolism. Epithelial Cells / metabolism. Epithelial Cells / microbiology. Gene Expression Regulation, Bacterial. Humans. Interleukin-8 / genetics. Interleukin-8 / metabolism. Intestinal Mucosa / cytology. Intracellular Signaling Peptides and Proteins / genetics. Intracellular Signaling Peptides and Proteins / metabolism. Mice. Mice, Inbred C57BL. Phosphorylation. Transcription Factor RelA / genetics. Transcription Factor RelA / metabolism. Transforming Growth Factor beta / genetics. Transforming Growth Factor beta / metabolism

  • NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .
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  • (PMID = 23403560.001).
  • [ISSN] 1098-5522
  • [Journal-full-title] Infection and immunity
  • [ISO-abbreviation] Infect. Immun.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Growth Differentiation Factor 15; 0 / Interleukin-8; 0 / Intracellular Signaling Peptides and Proteins; 0 / NF-kappa B; 0 / TAB3 protein, human; 0 / Transcription Factor RelA; 0 / Transforming Growth Factor beta; 136601-57-5 / Cyclin D1
  • [Other-IDs] NLM/ PMC3676000
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22. |......... 11%  Cooper IF, Siadaty MS: 'Enzymes' associated with 'Enteropathogenic': Top Publications. BioMedLib Review; Enzyme;Enteropathogenic:705377072. ISSN: 2331-5717. 2014/12/4
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  • [Title] 'Enzymes' associated with 'Enteropathogenic': Top Publications.
  • Background: There are articles published each month which present 'Enzyme' for 'enteropathogenic'.
  • Hawrani A et al: Effect of protein kinase A-mediated phosphorylation on the structure and association properties of the enteropathogenic Escherichia coli Tir virulence protein.
  • Swimm A et al: Enteropathogenic Escherichia coli use redundant tyrosine kinases to form actin pedestals.
  • Crane JK et al: Externalization of host cell protein kinase C during enteropathogenic Escherichia coli infection.
  • Bhatt S et al: CsrA and TnaB coregulate tryptophanase activity to promote exotoxin-induced killing of Caenorhabditis elegans by enteropathogenic Escherichia coli.
  • Czerucka D et al: Implication of mitogen-activated protein kinases in T84 cell responses to enteropathogenic Escherichia coli infection.
  • de Grado M et al: Enteropathogenic Escherichia coli infection induces expression of the early growth response factor by activating mitogen-activated protein kinase cascades in epithelial cells.
  • Tomson FL et al: Differing roles of protein kinase C-zeta in disruption of tight junction barrier by enteropathogenic and enterohemorrhagic Escherichia coli.
  • Shin S et al: An activator of glutamate decarboxylase genes regulates the expression of enteropathogenic Escherichia coli virulence genes through control of the plasmid-encoded regulator, Per.
  • Egea L et al: Role of secreted glyceraldehyde-3-phosphate dehydrogenase in the infection mechanism of enterohemorrhagic and enteropathogenic Escherichia coli: interaction of the extracellular enzyme with human plasminogen and fibrinogen.
  • Bommarius B et al: Enteropathogenic Escherichia coli Tir is an SH2/3 ligand that recruits and activates tyrosine kinases required for pedestal formation.

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
  • (UID = 705377072.001).
  • [ISSN] 2331-5717
  • [Journal-full-title] BioMedLib Review
  • [Language] eng
  • [Publication-type] Review
  • [Publication-country] UNITED STATES
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23. |......... 11%  Cooper IF, Siadaty MS: 'Bacteriums' associated with 'Enzootic': Top Publications. BioMedLib Review; Bacterium;Enzootic:705587863. ISSN: 2331-5717. 2014/2/3
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  • [Title] 'Bacteriums' associated with 'Enzootic': Top Publications.
  • Background: There are articles published each month which present 'Bacterium' for 'enzootic'.
  • Finding such articles is important for researchers, clinicians, and patients.
  • However these articles are spread across thousands of journals, and there are many types of 'Bacterium'.
  • This makes searching and locating the relevant publications a challenge.
  • We have used BioMedLib's semantic search technology to address the issue, and gathered all the pertinent publications in this review article.
  • Methods: We categorized the publications we found into two groups.
  • We used the strength of textual-association to separate the groups.
  • In group one there are publications with the strongest evidence of association. We focused finding the most relevant publications pertinent to our goal, rather than combining them into a conclusion section. Such textual synthesis will be the focus of our next project.
  • Results: Group one includes 33 publications, and group two 950 publications.
  • Here are the top 10.
  • Bearden SW et al: Attenuated enzootic (pestoides) isolates of Yersinia pestis express active aspartase.
  • Bearden SW et al: Recent findings regarding maintenance of enzootic variants of Yersinia pestis in sylvatic reservoirs and their significance in the evolution of epidemic plague.
  • Goethert HK et al: Enzootic transmission of Anaplasma bovis in Nantucket cottontail rabbits.
  • Ouyang Z et al: Activation of the RpoN-RpoS regulatory pathway during the enzootic life cycle of Borrelia burgdorferi.
  • Oliver JH Jr et al: An enzootic transmission cycle of Lyme borreliosis spirochetes in the southeastern United States.
  • Goethert HK et al: Enzootic transmission of the agent of human granulocytic ehrlichiosis among cottontail rabbits.
  • Clark KL et al: Host associations of ticks parasitizing rodents at Borrelia burgdorferi enzootic sites in South Carolina.
  • Foley J et al: An Ixodes minor and Borrelia carolinensis enzootic cycle involving a critically endangered Mojave Desert rodent.
  • Wheelhouse N et al: Evidence of Chlamydophila abortus vaccine strain 1B as a possible cause of ovine enzootic abortion.
  • Ewers C et al: [Mannheimia haemolytica and the pathogenesis of enzootic bronchopneumonia].

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
  • (UID = 705587863.001).
  • [ISSN] 2331-5717
  • [Journal-full-title] BioMedLib Review
  • [Language] eng
  • [Publication-type] Review
  • [Publication-country] UNITED STATES
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24. |......... 11%  Cooper IF, Siadaty MS: 'Phenomena or Processes' associated with 'E Coli Enteropathogenic': Top Publications. BioMedLib Review; PhenomenonOrProcess;EColiEnteropathogenic:706318592. ISSN: 2331-5717. 2014/8/25
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  • [Title] 'Phenomena or Processes' associated with 'E Coli Enteropathogenic': Top Publications.
  • There are articles published each month which present 'Phenomenon or Process' for 'e coli enteropathogenic'.
  • Bhat A et al: DNA looping-dependent autorepression of LEE1 P1 promoters by Ler in enteropathogenic Escherichia coli (EPEC).
  • Carvalho HM et al: Antibody against the carboxyl terminus of intimin alpha reduces enteropathogenic Escherichia coli adherence to tissue culture cells and subsequent induction of actin polymerization.
  • Gareau MG et al: Enterohaemorrhagic, but not enteropathogenic, Escherichia coli infection of epithelial cells disrupts signalling responses to tumour necrosis factor-alpha.
  • Jarvis KG et al: Enteropathogenic Escherichia coli contains a putative type III secretion system necessary for the export of proteins involved in attaching and effacing lesion formation.
  • Lee DW et al: Enteropathogenic Escherichia coli Outbreak and its Incubation Period: Is it Short or Long?.
  • Nieto-Pelegrin E et al: Crk adaptors negatively regulate actin polymerization in pedestals formed by enteropathogenic Escherichia coli (EPEC) by binding to Tir effector.
  • Kenny B et al: Co-ordinate regulation of distinct host cell signalling pathways by multifunctional enteropathogenic Escherichia coli effector molecules.
  • Senerwa D et al: Antimicrobial resistance of enteropathogenic Escherichia coli strains from a nosocomial outbreak in Kenya.
  • Makino S et al: Molecular epidemiological study of a mass outbreak caused by enteropathogenic Escherichia coli O157:H45.

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
  • (UID = 706318592.001).
  • [ISSN] 2331-5717
  • [Journal-full-title] BioMedLib Review
  • [Language] eng
  • [Publication-type] Review
  • [Publication-country] UNITED STATES
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25. |......... 11%  Cooper IF, Siadaty MS: 'Manufactured Objects' associated with 'Cortactin Chemical Ingredient': Top Publications. BioMedLib Review; ManufacturedObject;CortactinChemicalIngredient:706172480. ISSN: 2331-5717. 2014/1/23
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  • Cantarelli VV et al: Cortactin is necessary for F-actin accumulation in pedestal structures induced by enteropathogenic Escherichia coli infection.
  • Cantarelli VV et al: Cortactin is essential for F-actin assembly in enteropathogenic Escherichia coli (EPEC)- and enterohaemorrhagic E. coli (EHEC)-induced pedestals and the alpha-helical region is involved in the localization of cortactin to bacterial attachment sites.

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
  • (UID = 706172480.001).
  • [ISSN] 2331-5717
  • [Journal-full-title] BioMedLib Review
  • [Language] eng
  • [Publication-type] Review
  • [Publication-country] UNITED STATES
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26. |......... 11%  Cooper IF, Siadaty MS: 'Bacteriums' associated with 'Cortactin Chemical Ingredient': Top Publications. BioMedLib Review; Bacterium;CortactinChemicalIngredient:706168280. ISSN: 2331-5717. 2014/2/23
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  • Cantarelli VV et al: Cortactin is necessary for F-actin accumulation in pedestal structures induced by enteropathogenic Escherichia coli infection.
  • Mousnier A et al: Cortactin recruitment by enterohemorrhagic Escherichia coli O157:H7 during infection in vitro and ex vivo.
  • Cantarelli VV et al: Cortactin is essential for F-actin assembly in enteropathogenic Escherichia coli (EPEC)- and enterohaemorrhagic E. coli (EHEC)-induced pedestals and the alpha-helical region is involved in the localization of cortactin to bacterial attachment sites.
  • Varon C et al: Helicobacter pullorum cytolethal distending toxin targets vinculin and cortactin and triggers formation of lamellipodia in intestinal epithelial cells.
  • Cantarelli VV et al: Tyrosine phosphorylation controls cortactin binding to two enterohaemorrhagic Escherichia coli effectors: Tir and EspFu/TccP.

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
  • (UID = 706168280.001).
  • [ISSN] 2331-5717
  • [Journal-full-title] BioMedLib Review
  • [Language] eng
  • [Publication-type] Review
  • [Publication-country] UNITED STATES
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27. |......... 11%  Cooper IF, Siadaty MS: 'Tissues' associated with 'Enteropathogenic': Top Publications. BioMedLib Review; Tissue;Enteropathogenic:706160411. ISSN: 2331-5717. 2014/4/23
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  • [Title] 'Tissues' associated with 'Enteropathogenic': Top Publications.
  • There are articles published each month which present 'Tissue' for 'enteropathogenic'.
  • Girard F et al: Interactions of typical and atypical enteropathogenic Escherichia coli strains with the calf intestinal mucosa ex vivo.
  • Gauthier A et al: Mechanical fractionation reveals structural requirements for enteropathogenic Escherichia coli Tir insertion into host membranes.
  • Schüller S et al: The ex vivo response of human intestinal mucosa to enteropathogenic Escherichia coli infection.
  • Magalhães CA et al: The ability of haemolysins expressed by atypical enteropathogenic Escherichia coli to bind to extracellular matrix components.
  • Skurnik M et al: YadA mediates specific binding of enteropathogenic Yersinia enterocolitica to human intestinal submucosa.
  • Hecht G: Microbes and microbial toxins: paradigms for microbial-mucosal interactions. VII. Enteropathogenic Escherichia coli: physiological alterations from an extracellular position.
  • Wachter C et al: Insertion of EspD into epithelial target cell membranes by infecting enteropathogenic Escherichia coli.
  • Celli J et al: Enteropathogenic Escherichia coli (EPEC) attachment to epithelial cells: exploiting the host cell cytoskeleton from the outside.
  • Drago-Serrano ME et al: Antibody response in mice immunized by mucosal routes with formalin-inactivated enteropathogenic Escherichia coli (EPEC) strains.
  • Frankel G et al: Royal Society of Tropical Medicine and Hygiene meeting at Manson House, London, 14 December 1995. Enteropathogenic Escherichia coli--mucosal infection models.

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
  • (UID = 706160411.001).
  • [ISSN] 2331-5717
  • [Journal-full-title] BioMedLib Review
  • [Language] eng
  • [Publication-type] Review
  • [Publication-country] UNITED STATES
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28. |......... 11%  Cooper IF, Siadaty MS: 'Phenomena or Processes' associated with 'Enteropathogenic': Top Publications. BioMedLib Review; PhenomenonOrProcess;Enteropathogenic:706153309. ISSN: 2331-5717. 2014/9/23
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  • [Title] 'Phenomena or Processes' associated with 'Enteropathogenic': Top Publications.
  • There are articles published each month which present 'Phenomenon or Process' for 'enteropathogenic'.
  • Gareau MG et al: Enterohaemorrhagic, but not enteropathogenic, Escherichia coli infection of epithelial cells disrupts signalling responses to tumour necrosis factor-alpha.
  • Lee DW et al: Enteropathogenic Escherichia coli Outbreak and its Incubation Period: Is it Short or Long?.
  • Bhat A et al: DNA looping-dependent autorepression of LEE1 P1 promoters by Ler in enteropathogenic Escherichia coli (EPEC).
  • Nieto-Pelegrin E et al: Crk adaptors negatively regulate actin polymerization in pedestals formed by enteropathogenic Escherichia coli (EPEC) by binding to Tir effector.
  • Rodríguez-Escudero I et al: Enteropathogenic Escherichia coli type III effectors alter cytoskeletal function and signalling in Saccharomyces cerevisiae.
  • Barlow RS et al: A novel serotype of enteropathogenic Escherichia coli (EPEC) as a major pathogen in an outbreak of infantile diarrhoea.
  • Kresse AU et al: Modulation of host cell signalling by enteropathogenic and Shiga toxin-producing Escherichia coli.
  • Senerwa D et al: Antimicrobial resistance of enteropathogenic Escherichia coli strains from a nosocomial outbreak in Kenya.
  • Yatsuyanagi J et al: [Enteropathogenic Escherichia coli strains harboring enteroaggregative Escherichia coli (EAggEC) heat-stable enterotoxin-1 gene isolated from a food-borne like outbreak].
  • McNamara BP et al: A novel proline-rich protein, EspF, is secreted from enteropathogenic Escherichia coli via the type III export pathway.

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29. |......... 11%  Cooper IF, Siadaty MS: 'Mental Processes' associated with 'Enteropathogenic': Top Publications. BioMedLib Review; MentalProcess;Enteropathogenic:706148479. ISSN: 2331-5717. 2014/12/23
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  • [Title] 'Mental Processes' associated with 'Enteropathogenic': Top Publications.
  • There are articles published each month which present 'Mental Process' for 'enteropathogenic'.
  • Anyanful A et al: Conditioning protects C. elegans from lethal effects of enteropathogenic E. coli by activating genes that regulate lifespan and innate immunity.
  • Thomassin JL et al: Enterohemorrhagic and enteropathogenic Escherichia coli evolved different strategies to resist antimicrobial peptides.
  • Yona-Nadler C et al: Integration host factor (IHF) mediates repression of flagella in enteropathogenic and enterohaemorrhagic Escherichia coli.
  • Ochoa TJ et al: New insights into the epidemiology of enteropathogenic Escherichia coli infection.
  • Batchelor M et al: Structural basis for recognition of the translocated intimin receptor (Tir) by intimin from enteropathogenic Escherichia coli.
  • Parissi-Crivelli A et al: Recognition of enteropathogenic Escherichia coli virulence determinants by human colostrum and serum antibodies.
  • Ibenyassine K et al: Use of repetitive DNA sequences to determine the persistence of enteropathogenic Escherichia coli in vegetables and in soil grown in fields treated with contaminated irrigation water.
  • Zhao B et al: Acid stress response in enteropathogenic gammaproteobacteria: an aptitude for survival.
  • de Almeida CM et al: Egg yolk anti-BfpA antibodies as a tool for recognizing and identifying enteropathogenic Escherichia coli.
  • Anuradha R et al: Bowman-Birk inhibitor-like protein is secreted by sprouted pea seeds in response to induced colonization by enteropathogenic Escherichia coli.

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30. |......... 11%  Cooper IF, Siadaty MS: 'Findings' associated with 'Amplaxin': Top Publications. BioMedLib Review; Finding;Amplaxin:706075170. ISSN: 2331-5717. 2014/3/22
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  • Cantarelli VV et al: Cortactin is necessary for F-actin accumulation in pedestal structures induced by enteropathogenic Escherichia coli infection.

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31. |......... 11%  Cooper IF, Siadaty MS: 'Manufactured Objects' associated with 'Amplaxin': Top Publications. BioMedLib Review; ManufacturedObject;Amplaxin:705986060. ISSN: 2331-5717. 2014/11/18
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  • Cantarelli VV et al: Cortactin is necessary for F-actin accumulation in pedestal structures induced by enteropathogenic Escherichia coli infection.
  • Cantarelli VV et al: Cortactin is essential for F-actin assembly in enteropathogenic Escherichia coli (EPEC)- and enterohaemorrhagic E. coli (EHEC)-induced pedestals and the alpha-helical region is involved in the localization of cortactin to bacterial attachment sites.

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32. |......... 11%  Cooper IF, Siadaty MS: 'Bacteriums' associated with 'Amplaxin': Top Publications. BioMedLib Review; Bacterium;Amplaxin:705984253. ISSN: 2331-5717. 2014/3/18
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  • Cantarelli VV et al: Cortactin is necessary for F-actin accumulation in pedestal structures induced by enteropathogenic Escherichia coli infection.
  • Mousnier A et al: Cortactin recruitment by enterohemorrhagic Escherichia coli O157:H7 during infection in vitro and ex vivo.
  • Cantarelli VV et al: Cortactin is essential for F-actin assembly in enteropathogenic Escherichia coli (EPEC)- and enterohaemorrhagic E. coli (EHEC)-induced pedestals and the alpha-helical region is involved in the localization of cortactin to bacterial attachment sites.
  • Varon C et al: Helicobacter pullorum cytolethal distending toxin targets vinculin and cortactin and triggers formation of lamellipodia in intestinal epithelial cells.
  • Cantarelli VV et al: Tyrosine phosphorylation controls cortactin binding to two enterohaemorrhagic Escherichia coli effectors: Tir and EspFu/TccP.

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33. |......... 11%  Cooper IF, Siadaty MS: 'Therapeutic or Preventive Procedures' associated with 'Specimen Type Macrophages': Top Publications. BioMedLib Review; TherapeuticOrPreventive;SpecimenTypeMacrophages:705846385. ISSN: 2331-5717. 2014/11/24
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  • da Fonseca AC et al: Microglia and macrophages in malignant gliomas: recent discoveries and implications for promising therapies.
  • Yoda K et al: Oral administration of Lactobacillus gasseri TMC0356 stimulates peritoneal macrophages and attenuates general symptoms caused by enteropathogenic Escherichia coli infection.

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34. |......... 11%  Cooper IF, Siadaty MS: 'Therapeutic or Preventive Procedures' associated with 'Specimen Source Codes Macrophages': Top Publications. BioMedLib Review; TherapeuticOrPreventive;SpecimenSourceCodes:705845091. ISSN: 2331-5717. 2014/1/23
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  • da Fonseca AC et al: Microglia and macrophages in malignant gliomas: recent discoveries and implications for promising therapies.
  • Yoda K et al: Oral administration of Lactobacillus gasseri TMC0356 stimulates peritoneal macrophages and attenuates general symptoms caused by enteropathogenic Escherichia coli infection.

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  • [Language] eng
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  • [Publication-country] UNITED STATES
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35. |......... 11%  Cooper IF, Siadaty MS: 'Mammals' associated with 'Enzootic': Top Publications. BioMedLib Review; Mammal;Enzootic:705626259. ISSN: 2331-5717. 2014/1/2
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  • Sze CW et al: Borrelia burgdorferi needs chemotaxis to establish infection in mammals and to accomplish its enzootic cycle.

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36. |......... 11%  Cooper IF, Siadaty MS: 'Bacteriums' associated with 'Pedestals': Top Publications. BioMedLib Review; Bacterium;Pedestals:705615437. ISSN: 2331-5717. 2014/4/24
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  • Bulgin R et al: The T3SS effector EspT defines a new category of invasive enteropathogenic E. coli (EPEC) which form intracellular actin pedestals.
  • Unsworth KE et al: Dynamin is required for F-actin assembly and pedestal formation by enteropathogenic Escherichia coli (EPEC).
  • Vingadassalom D et al: Enterohemorrhagic E. coli requires N-WASP for efficient type III translocation but not for EspFU-mediated actin pedestal formation.
  • Zobiack N et al: Cell-surface attachment of pedestal-forming enteropathogenic E. coli induces a clustering of raft components and a recruitment of annexin 2.
  • Cantarelli VV et al: Cortactin is necessary for F-actin accumulation in pedestal structures induced by enteropathogenic Escherichia coli infection.
  • Goosney DL et al: Recruitment of cytoskeletal and signaling proteins to enteropathogenic and enterohemorrhagic Escherichia coli pedestals.
  • Allen-Vercoe E et al: Amino acid residues within enterohemorrhagic Escherichia coli O157:H7 Tir involved in phosphorylation, alpha-actinin recruitment, and Nck-independent pedestal formation.
  • Campellone KG et al: Tails of two Tirs: actin pedestal formation by enteropathogenic E. coli and enterohemorrhagic E. coli O157:H7.
  • Gruenheid S et al: Enteropathogenic E. coli Tir binds Nck to initiate actin pedestal formation in host cells.
  • Simovitch M et al: EspM inhibits pedestal formation by enterohaemorrhagic Escherichia coli and enteropathogenic E. coli and disrupts the architecture of a polarized epithelial monolayer.

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37. |......... 11%  Cooper IF, Siadaty MS: 'Mammals' associated with 'Infection Maternal': Top Publications. BioMedLib Review; Mammal;InfectionMaternal:705576052. ISSN: 2331-5717. 2014/5/27
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  • [Title] 'Mammals' associated with 'Infection Maternal': Top Publications.
  • Background: There are articles published each month which present 'Mammal' for 'infection maternal'.
  • Cooper IF et al: 'Mammals' associated with 'Infection Maternal': Top Publications.
  • Short SJ et al: Maternal influenza infection during pregnancy impacts postnatal brain development in the rhesus monkey.
  • Chen J et al: Vaccination with hemagglutinin or neuraminidase DNA protects BALB/c mice against influenza virus infection in presence of maternal antibody.
  • Tirabassi RS et al: Infection with viruses from several families triggers autoimmune diabetes in LEW*1WR1 rats: prevention of diabetes by maternal immunization.
  • Elbers AR et al: Schmallenberg virus antibody persistence in adult cattle after natural infection and decay of maternal antibodies in calves.
  • Dando SJ et al: Maternal administration of erythromycin fails to eradicate intrauterine ureaplasma infection in an ovine model.
  • Cabañas-Cortés MA et al: Toxoplasma gondii: effect of maternal infection in the development of lymphoid organs of BALB/c neonates.
  • Intapan PM et al: Opisthorchis viverrini: influence of maternal infection in hamsters on offspring infected with homologous parasite and their IgG antibody response.
  • Liverman CS et al: Altered expression of pro-inflammatory and developmental genes in the fetal brain in a mouse model of maternal infection.
  • Eguchi K et al: Short communication: epidemiological evidence that simian T-lymphotropic virus type 1 in Macaca fuscata has an alternative transmission route to maternal infection.

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38. |......... 11%  Cooper IF, Siadaty MS: 'Bacteriums' associated with 'Domestic Rabbit': Top Publications. BioMedLib Review; Bacterium;DomesticRabbit:705524431. ISSN: 2331-5717. 2014/7/3
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  • Swennes AG et al: Enteropathogenic Escherichia coli prevalence in laboratory rabbits.
  • Swennes AG et al: Enzootic enteropathogenic Escherichia coli infection in laboratory rabbits.
  • Sánchez del Rey V et al: Genetic analysis of Streptococcus suis isolates from wild rabbits.

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39. |......... 11%  Cooper IF, Siadaty MS: 'Bacteriums' associated with 'Intestinal Mucosa': Top Publications. BioMedLib Review; Bacterium;IntestinalMucosa:705512747. ISSN: 2331-5717. 2014/4/23
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  • [Title] 'Bacteriums' associated with 'Intestinal Mucosa': Top Publications.
  • Background: There are articles published each month which present 'bacterium' for 'intestinal mucosa'.
  • Oliveira AG et al: Helicobacter species in the intestinal mucosa of patients with ulcerative colitis.
  • Schüller S et al: The ex vivo response of human intestinal mucosa to enteropathogenic Escherichia coli infection.
  • Cui HH et al: Effects of probiotic on intestinal mucosa of patients with ulcerative colitis.
  • de Champs C et al: Persistence of colonization of intestinal mucosa by a probiotic strain, Lactobacillus casei subsp. rhamnosus Lcr35, after oral consumption.
  • Girard F et al: Interactions of typical and atypical enteropathogenic Escherichia coli strains with the calf intestinal mucosa ex vivo.
  • Troost FJ et al: Identification of the transcriptional response of human intestinal mucosa to Lactobacillus plantarum WCFS1 in vivo.
  • Ouwehand AC et al: Disease-dependent adhesion of lactic acid bacteria to the human intestinal mucosa.
  • Nemcová R et al: Effects of the administration of lactobacilli, maltodextrins and fructooligosaccharides upon the adhesion of E. coli O8:K88 to the intestinal mucosa and organic acid levels in the gut contents of piglets.
  • Gustavo Hermes R et al: Casein glycomacropeptide in the diet may reduce Escherichia coli attachment to the intestinal mucosa and increase the intestinal lactobacilli of early weaned piglets after an enterotoxigenic E. coli K88 challenge.
  • Uchida H et al: Lactobacilli binding human A-antigen expressed in intestinal mucosa.

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40. |......... 11%  Cooper IF, Siadaty MS: 'Enzymes' associated with 'Externalization': Top Publications. BioMedLib Review; Enzyme;Externalization:705446308. ISSN: 2331-5717. 2014/11/29
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  • Crane JK et al: Externalization of host cell protein kinase C during enteropathogenic Escherichia coli infection.

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41. |......... 11%  Cooper IF, Siadaty MS: 'Mental Processes' associated with 'E Coli Enteropathogenic': Top Publications. BioMedLib Review; MentalProcess;EColiEnteropathogenic:706349837. ISSN: 2331-5717. 2014/10/25
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  • [Title] 'Mental Processes' associated with 'E Coli Enteropathogenic': Top Publications.
  • There are articles published each month which present 'Mental Process' for 'e coli enteropathogenic'.
  • Anyanful A et al: Conditioning protects C. elegans from lethal effects of enteropathogenic E. coli by activating genes that regulate lifespan and innate immunity.
  • Thomassin JL et al: Enterohemorrhagic and enteropathogenic Escherichia coli evolved different strategies to resist antimicrobial peptides.
  • Dean P et al: Insights into the pathogenesis of enteropathogenic E. coli using an improved intestinal enterocyte model.
  • Parissi-Crivelli A et al: Recognition of enteropathogenic Escherichia coli virulence determinants by human colostrum and serum antibodies.
  • Crane JK et al: Externalization of host cell protein kinase C during enteropathogenic Escherichia coli infection.
  • Sekiya K et al: Supermolecular structure of the enteropathogenic Escherichia coli type III secretion system and its direct interaction with the EspA-sheath-like structure.
  • de Almeida CM et al: Egg yolk anti-BfpA antibodies as a tool for recognizing and identifying enteropathogenic Escherichia coli.
  • Anuradha R et al: Bowman-Birk inhibitor-like protein is secreted by sprouted pea seeds in response to induced colonization by enteropathogenic Escherichia coli.
  • Porter ME et al: Direct and indirect transcriptional activation of virulence genes by an AraC-like protein, PerA from enteropathogenic Escherichia coli.
  • Ibenyassine K et al: Use of repetitive DNA sequences to determine the persistence of enteropathogenic Escherichia coli in vegetables and in soil grown in fields treated with contaminated irrigation water.

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42. |......... 11%  Cooper IF, Siadaty MS: 'Phenomena or Processes' associated with 'Necrosis Tumour': Top Publications. BioMedLib Review; PhenomenonOrProcess;NecrosisTumour:706123670. ISSN: 2331-5717. 2014/10/23
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  • Gareau MG et al: Enterohaemorrhagic, but not enteropathogenic, Escherichia coli infection of epithelial cells disrupts signalling responses to tumour necrosis factor-alpha.
  • Belisle SE et al: Genomic profiling of tumor necrosis factor alpha (TNF-alpha) receptor and interleukin-1 receptor knockout mice reveals a link between TNF-alpha signaling and increased severity of 1918 pandemic influenza virus infection.

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  • [Publication-country] UNITED STATES
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43. |......... 11%  Cooper IF, Siadaty MS: 'Amino Acids, Peptides, or Proteins' associated with 'Externalization': Top Publications. BioMedLib Review; AminoAcidPeptide;Externalization:705854443. ISSN: 2331-5717. 2014/4/30
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  • Crane JK et al: Externalization of host cell protein kinase C during enteropathogenic Escherichia coli infection.

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  • [Language] eng
  • [Publication-type] Review
  • [Publication-country] UNITED STATES
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44. |......... 11%  Cooper IF, Siadaty MS: 'Bacteriums' associated with 'Infection Maternal': Top Publications. BioMedLib Review; Bacterium;InfectionMaternal:705551558. ISSN: 2331-5717. 2014/10/3
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  • [Title] 'Bacteriums' associated with 'Infection Maternal': Top Publications.
  • Background: There are articles published each month which present 'Bacterium' for 'infection maternal'.
  • Arias-Camison JM: Late onset group B streptococcal infection from maternal expressed breast milk in a very low birth weight infant.
  • Blond MH et al: [Neonatal bacterial infection by maternal-fetal contamination: for a change in approach? 1. Detection of Streptococcus agalactiae infection: methods and evaluation of results].
  • Ramirez K et al: Mucosal priming of newborn mice with S. Typhi Ty21a expressing anthrax protective antigen (PA) followed by parenteral PA-boost induces B and T cell-mediated immunity that protects against infection bypassing maternal antibodies.
  • Aydın B et al: Prematurity due to maternal brucella infection and review of the literature.
  • Cutland CL et al: Maternal HIV infection and vertical transmission of pathogenic bacteria.
  • Dando SJ et al: Maternal administration of erythromycin fails to eradicate intrauterine ureaplasma infection in an ovine model.
  • Chiu CH et al: Protection of neonatal mice from lethal enterovirus 71 infection by maternal immunization with attenuated Salmonella enterica serovar Typhimurium expressing VP1 of enterovirus 71.
  • Hentgen V et al: [Maternal antibiotic use and Gram negative bacteria infection in neonates].
  • Kageyama D et al: Spiroplasma infection causes either early or late male killing in Drosophila, depending on maternal host age.
  • Avila LA et al: Effects of probiotics and maternal vaccination on Salmonella enteritidis infection in broiler chicks.

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45. |......... 11%  Cooper IF, Siadaty MS: 'Qualitative Concepts' associated with 'Enteritis Escherichia Coli Enterohemorrhagic': Top Publications. BioMedLib Review; QualitativeConcept;EnteritisEscherichiaColi:706382120. ISSN: 2331-5717. 2014/6/26
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  • [Title] 'Qualitative Concepts' associated with 'Enteritis Escherichia Coli Enterohemorrhagic': Top Publications.
  • There are articles published each month which present 'Qualitative Concept' for 'enteritis escherichia coli enterohemorrhagic'.
  • Ceponis PJ et al: Epithelial cell signaling responses to enterohemorrhagic Escherichia coli infection.
  • Calderon Toledo C et al: Cross-reactive protection against enterohemorrhagic Escherichia coli infection by enteropathogenic E. coli in a mouse model.
  • Lukyanenko V et al: Enterohemorrhagic Escherichia coli infection stimulates Shiga toxin 1 macropinocytosis and transcytosis across intestinal epithelial cells.
  • Iida T et al: Measurement of fecal lactoferrin for rapid diagnosis of enterohemorrhagic Escherichia coli infection.
  • Cerna-Cortes JF et al: Consumption of street-vended beverage a potential exposure risk for non-O157 enterohemorrhagic Escherichia coli infection: the importance of testing for virulence loci.
  • Kudoh Y: [Current status of gastrointestinal infection in Japan--with special reference to enterohaemorrhagic Escherichia coli infection].
  • Tsutsumi R et al: Homologous and heterologous antibody responses to lipopolysaccharide after enterohemorrhagic Escherichia coli infection.
  • Tatsumi N et al: [Etiological bacterial level in enterohemorrhagic Escherichia coli infection feces and chromogenic culture medium CHROMagar STEC usefulness].
  • Shiomi M: [Indicators for early diagnosis of enterohemorrhagic Escherichia coli infection and methods for final diagnosis].

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46. |......... 11%  Cooper IF, Siadaty MS: 'Pathologic Functions' associated with 'E Coli Enteropathogenic': Top Publications. BioMedLib Review; PathologicFunction;EColiEnteropathogenic:706350635. ISSN: 2331-5717. 2014/2/25
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  • [Title] 'Pathologic Functions' associated with 'E Coli Enteropathogenic': Top Publications.
  • There are articles published each month which present 'Pathologic Function' for 'e coli enteropathogenic'.
  • Hill SM et al: Enteropathogenic Escherichia coli and life threatening chronic diarrhoea.
  • Ruchaud-Sparagano MH et al: Enteropathogenic Escherichia coli (EPEC) inactivate innate immune responses prior to compromising epithelial barrier function.
  • Khan MA et al: Flagellin-dependent and -independent inflammatory responses following infection by enteropathogenic Escherichia coli and Citrobacter rodentium.
  • Mansfield KG et al: Enteropathogenic Escherichia coli and ulcerative colitis in cotton-top tamarins (Saguinus oedipus).
  • De Rycke J et al: Enteropathogenic Escherichia coli O103 from rabbit elicits actin stress fibers and focal adhesions in HeLa epithelial cells, cytopathic effects that are linked to an analog of the locus of enterocyte effacement.
  • Savkovic SD et al: PKC zeta participates in activation of inflammatory response induced by enteropathogenic E. coli.
  • Joh K et al: Antigenicities of enteropathogenic Escherichia coli, lysozyme, and alpha-1-antichymotrypsin on macrophages of genitourinary malacoplakia.
  • Abdallah Hajj Hussein I et al: Enteropathogenic e.coli sustains iodoacetamide-induced ulcerative colitis-like colitis in rats: modulation of IL-1β, IL-6, TNF-α, COX-2, and apoptosisi.
  • Shifrin Y et al: Enteropathogenic Escherichia coli induces modification of the focal adhesions of infected host cells.

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  • (UID = 706350635.001).
  • [ISSN] 2331-5717
  • [Journal-full-title] BioMedLib Review
  • [Language] eng
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  • [Publication-country] UNITED STATES
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47. |......... 11%  Cooper IF, Siadaty MS: 'Qualitative Concepts' associated with 'Infection Mouth': Top Publications. BioMedLib Review; QualitativeConcept;InfectionMouth:706293883. ISSN: 2331-5717. 2014/1/25
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  • [Title] 'Qualitative Concepts' associated with 'Infection Mouth': Top Publications.
  • There are articles published each month which present 'Qualitative Concept' for 'infection mouth'.
  • Jensen KD et al: Toxoplasma gondii rhoptry 16 kinase promotes host resistance to oral infection and intestinal inflammation only in the context of the dense granule protein GRA15.
  • Asoh T et al: Natural defense by saliva and mucosa against oral infection by Leptospira.
  • Gonzalez MI et al: Striated muscle involvement in experimental oral infection by herpes simplex virus type 1.
  • Costanzo KS et al: Effect of oral infection of La Crosse virus on survival and fecundity of native Ochlerotatus triseriatus and invasive Stegomyia albopicta.
  • Vulcano AB et al: Oral infection with enteropathogenic Escherichia coli triggers immune response and intestinal histological alterations in mice selected for their minimal acute inflammatory responses.

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
  • (UID = 706293883.001).
  • [ISSN] 2331-5717
  • [Journal-full-title] BioMedLib Review
  • [Language] eng
  • [Publication-type] Review
  • [Publication-country] UNITED STATES
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48. |......... 11%  Cooper IF, Siadaty MS: 'Temporal Concepts' associated with 'Citrobacter Nos': Top Publications. BioMedLib Review; TemporalConcept;CitrobacterNos:706236488. ISSN: 2331-5717. 2014/12/24
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  • Khan MA et al: Flagellin-dependent and -independent inflammatory responses following infection by enteropathogenic Escherichia coli and Citrobacter rodentium.
  • Raczynski AR et al: Enteric infection with Citrobacter rodentium induces coagulative liver necrosis and hepatic inflammation prior to peak infection and colonic disease.
  • Chen CC et al: Concurrent infection with an intestinal helminth parasite impairs host resistance to enteric Citrobacter rodentium and enhances Citrobacter-induced colitis in mice.
  • Kokrdová Z et al: [Fatal course of neonatal citrobacter infection and its legal evaluation].

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
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  • [Language] eng
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49. |......... 11%  Cooper IF, Siadaty MS: 'Organism Attributes' associated with 'Citrobacter Nos': Top Publications. BioMedLib Review; OrganismAttribute;CitrobacterNos:706168008. ISSN: 2331-5717. 2014/4/23
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  • Lebeis SL et al: Interleukin-1 receptor signaling protects mice from lethal intestinal damage caused by the attaching and effacing pathogen Citrobacter rodentium.
  • Khan MA et al: Flagellin-dependent and -independent inflammatory responses following infection by enteropathogenic Escherichia coli and Citrobacter rodentium.
  • Nyagode BA et al: Altered inflammatory responses to Citrobacter rodentium infection, but not bacterial lipopolysaccharide, in mice lacking the Cyp4a10 or Cyp4a14 genes.
  • Li L et al: Cytokine IL-6 is required in Citrobacter rodentium infection-induced intestinal Th17 responses and promotes IL-22 expression in inflammatory bowel disease.

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  • (UID = 706168008.001).
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  • [Language] eng
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50. |......... 11%  Cooper IF, Siadaty MS: 'Viruses' associated with 'Depressed Symptom': Top Publications. BioMedLib Review; Virus;DepressedSymptom:706302345. ISSN: 2331-5717. 2014/3/25
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  • Bruchas RR et al: The St. Louis African American health-heart study: methodology for the study of cardiovascular disease and depression in young-old African Americans.
  • Guy JS et al: High mortality and growth depression experimentally produced in young turkeys by dual infection with enteropathogenic Escherichia coli and turkey coronavirus.
  • Arslan N et al: Depression and anxiety in chronic hepatitis B: effect of hepatitis B virus infection on psychological state in childhood.

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  • [Copyright] Copyright 2014 Siadaty and Cooper; licensee BioMedLib LLC.
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  • [Language] eng
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2. Definitions


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